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   1 ith the visceralizing species of Leishmania, Leishmania chagasi.                                     
     2 vironment surrounding the invading protozoan Leishmania chagasi.                                     
     3 fore, we investigated protective immunity to Leishmania chagasi, a cause of visceral leishmaniasis.  
     4  efficiently inhibited the in vivo growth of Leishmania chagasi and Leishmania amazonensis promastigo
     5 to a cloned antigen, recombinant (r) K39, of Leishmania chagasi are specific for members of the Leish
  
     7 ssue cytokine response of dogs infected with Leishmania chagasi, cytokine mRNA levels were measured i
     8 crapings from cutaneous leishmanial lesions, Leishmania chagasi in dermal scrapings of atypical cutan
     9 sness of 50 sentinel dogs exposed to natural Leishmania chagasi infection was assessed through time b
  
    11 l leishmaniasis (VL) caused by the protozoan Leishmania chagasi is ongoing outside Natal, northeast B
  
    13 te level of GP46 mRNA increases >>30-fold as Leishmania chagasi promastigotes develop in vitro from a
    14 ly, resulting in about a 30-fold increase as Leishmania chagasi promastigotes grow in vitro from loga
  
    16 ectants, we developed a method to label live Leishmania chagasi short-term with fluorescent dyes.    
  
    18 ry from the intracellular amastigote form of Leishmania chagasi, the cause of South American visceral
  
  
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