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1 the active site of prolyl 4-hydroxylase and lysyl hydroxylase.
2 acting as both an arginine demethylase and a lysyl-hydroxylase.
3 he alpha-subunit of prolyl 4-hydroxylase and lysyl hydroxylases.
4 ydroxylases, such as prolyl 4-hydroxylase or lysyl hydroxylases.
5 ent dioxygenases, such as collagen prolyl or lysyl hydroxylases.
7 in B with the P-domain of calreticulin, with lysyl hydroxylase 1, and probably other proteins, such a
8 m of clinical phenotypes than the absence of lysyl-hydroxylase 1 and additionally includes myopathy,
11 on of the alternatively spliced long form of lysyl hydroxylase 2 (LH2), a collagen telopeptide LH.
12 signaling in macrophages to the induction of lysyl hydroxylase 2 (LH2), an enzyme that directs persis
14 -function studies in tumor cells showed that lysyl hydroxylase 2 (LH2), which hydroxylates telopeptid
15 se associated with inactivating mutations in lysyl hydroxylase 2 (LH2/PLOD2) or FK506 binding protein
16 T3-E1 (MC)-derived clones stably suppressing lysyl hydroxylase 3 (LH3) (short hairpin (Sh) clones) an
19 th its partner proteins, regulate sorting of lysyl hydroxylase 3 (LH3, also known as PLOD3) into newl
20 and IV, respectively, while mutations in the lysyl hydroxylase and ATP7A genes, with roles in collage
22 y 2,2'-dipyridyl, an inhibitor of prolyl and lysyl hydroxylase, and by 2 mM dithiothreitol, but unaff
23 ing hypoxia-inducible factor 1 [HIF-1]), the lysyl hydroxylase JMJD6, and the RNA hydroxylase TYW5 bu
24 sults from mutations in the genes coding for lysyl hydroxylase (LH) 2 or peptidyl-prolyl cis-trans is
25 emically by the severely diminished level of lysyl hydroxylase (LH) activity in the patient's skin fi
26 y reports the expression of functional human lysyl hydroxylase (LH), a post-translational modifying e
27 R may also act as an arginine demethylase, a lysyl hydroxylase, or an RNA-binding protein through its
29 We further demonstrate that the procollagen lysyl hydroxylase (PLOD1) promoter is regulated by Nkx2.
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