ライフサイエンスコーパス: methyl bromide

1 e crop production systems in a world without methyl bromide.

2 the distinctiveness of industrially produced methyl bromide.

3 ents, rats received infusions of scopolamine methyl bromide (10 microg/0.5 microl) into the nucleus a

4 osylation of 5 with (1,3-dihydroxy-2-propoxy)methyl bromide (22) followed by a ring annulation with h

5 Methyl bromide, a fungicide often used in strawberry cul

6 epithelium by inhalation of the olfactotoxin methyl bromide activates a population of multipotent glo

7 implies that about 1 percent of atmospheric methyl bromide and 5 percent of methyl iodide arise from

8 tems is preplant fumigation with mixtures of methyl bromide and chloropicrin.

9 budgets for the ozone-depleting halocarbons, methyl bromide and methyl chloride.

10 By observing emissions of methyl chloride, methyl bromide, and methyl iodide from flooded Californi

11 information system to estimate the amount of methyl bromide applied within 5 km of a woman's residenc

12 e inhibited by atropine sulfate and atropine methyl bromide but not by hexamethonium.

13 Methyl chloride (CH(3)Cl) and methyl bromide (CH(3)Br) are the primary carriers of nat

14 The atmospheric budget of methyl bromide (CH(3)Br), an ozone-depleting gas, is hig

15 luding methane (CH(4)), isoprene (C(5)H(8)), methyl bromide (CH(3)Br), dimethyl sulfide, and oxygen (

16 Atmospheric methyl bromide (CH3Br) and methyl chloride (CH3Cl), comp

17 need for technologies to capture and destroy methyl bromide (CH3Br) emissions from postharvest fumiga

18 The atmospheric residence time for methyl bromide (CH3Br) has been estimated as 0.8 +/- 0.1

19 Methyl bromide (CH3Br) is widely used as a fumigant for

20 Little is known about the effects of methyl bromide exposure during pregnancy.

21 and patches of it are rendered acellular by methyl bromide exposure.

22 tution after direct experimental injury with methyl bromide, expression of the GBC-1 antigen overlaps

23 r is utilized for the generation of volatile methyl bromide from aqueous solutions of bromide and bro

24 Methyl bromide from combined natural and anthropogenic s

25 ch rat's epithelium was subjected to 330 ppm methyl bromide gas for 6 hr.

26 We used the inhalation of methyl bromide gas to produce a near-complete destructio

27 the bulb are reconstituted after ablation by methyl bromide gas, and some measure of olfactory functi

28 of the anterior bulb in the food-restricted, methyl bromide gas-lesioned animals indicates that the m

29 Because methyl bromide has provided a reliable return on investm

30 n during the recovery of the epithelium from methyl bromide-induced injury.

31 nstitution of the OE, rats were subjected to methyl bromide-induced OE lesion and their OE and OBs ex

32 Methyl bromide is an effective pre-plant soil fumigant u

33 ion following injury by the olfactotoxic gas methyl bromide is coincident with the down-regulation of

34 ium and during epithelial regeneration after methyl bromide lesion or olfactory bulbectomy.

35 t epithelium that recover as olfactory after methyl bromide lesion with those undergoing respiratory

36 Immediately after methyl bromide lesion, but not at any time after bulbect

37 ulated after activation of the epithelium by methyl bromide lesion.

38 covery of the rat olfactory epithelium after methyl bromide lesion.

39 ommitted to making sustentacular cells after methyl bromide lesion.

40 n the olfactory epithelium from unilaterally methyl bromide-lesioned and control rats.

41 marker-labeled cells into the epithelium of methyl bromide-lesioned, unmarked host mice to dissect p

42 how nematode management practices other than methyl bromide may be incorporated.

43 as a stratospheric ozone-depleting compound, methyl bromide (MeBr) is being phased out despite being

44 Methyl bromide (MeBr) is used increasingly as a biocidal

45 1 day after exposure to the olfactotoxic gas methyl bromide (MeBr) to assess the lineage relationship

46 llent soil fumigant for killing plant pests, methyl bromide (MeBr) was phased out in 2005 in the USA,

47 All spared GBCs express Ki-67 in the methyl bromide (MeBr)-lesioned OE initially after lesion

48 This is related to a different gas product (methyl bromide, MeBr) removal mechanism, changing from c

49 may have prostate-specific biologic effects (methyl bromide (odds ratio = 1.62, 95% confidence interv

50 hed and abandoning gas fumigation containing methyl bromide resulted in the recurrence of an almost f

51 With the impending phase-out of methyl bromide, the agricultural industries that rely on

52 After epithelial lesion by exposure to methyl bromide, the remaining cells, which are mostly GB

53 elationship between residential proximity to methyl bromide use and birth outcomes.

54 Residential proximity to methyl bromide use during the second trimester was assoc

55 outcomes were also associated with moderate methyl bromide use during the second trimester.

56 s among applicators over 50 years of age and methyl bromide use were significantly associated with pr