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1 up of a mevalonate metabolite and preventing protein prenylation.
2 D) is characterized by a severe reduction in protein prenylation.
3 in-2 (SREBP-2) cleavage, causing decrease of protein prenylation.
4 terol trafficking secondary to inhibition of protein prenylation.
5 e synthase (FPPS) in osteoclasts, preventing protein prenylation.
6 carboxyl-terminal mutation used to eliminate protein prenylation.
7 ful to the community of researchers studying protein prenylation.
8 through direct interference of small GTPases protein prenylation.
9 yl pyrophosphate synthase (FPPS) and inhibit protein prenylation.
10 ocalization was not altered by inhibitors of protein prenylation.
11 mino-terminal domain, even in the absence of protein prenylation.
12 with an isoprenyl lipid via a process called protein prenylation.
13  CAAX motif, a well-characterized signal for protein prenylation.
14 umulation appears dissociated from increased protein prenylation.
15 IFN-gamma in MVECs through interference with protein prenylation.
16  reductase] pathway synthesizes lipids for G-protein prenylation.
17 erol and isoprenoid synthesis, and increased protein prenylation.
18 , probably as a consequence of inhibition of protein prenylation.
19                                              Protein prenylation, a well-defined protein consensus mo
20 zoledronic acid monohydrate, an inhibitor of protein prenylation, act synergistically to reverse outc
21                             We conclude that protein prenylation, acting downstream of Hmgcr1b and po
22 ition of geranylgeranyl transferase 1 (GGT1) protein prenylation activity also resulted in abnormal g
23     In addition, aplexone treatment inhibits protein prenylation and blocking the activity of geranyl
24  we investigated the effect of lovastatin on protein prenylation and cell signaling.
25 sses the mevalonate pathway and reduces KRAS protein prenylation and function, which in turn inhibits
26 e advantageous for unraveling unknown native protein prenylation and further developments in profilin
27  a significant inhibitory effect of Abeta on protein prenylation and identify SREBP-2 as a target of
28 iosynthesis inhibitor, selectively inhibited protein prenylation and induced apoptosis in MESN cells,
29 peptides that should be useful in studies of protein prenylation and other structurally related biolo
30 udies establish context-dependent effects of protein prenylation and unique roles of geranylgeranylat
31 s being important for dolichol biosynthesis, protein prenylation, and modification of other proteins
32  on a subbranch of the pathway important for protein prenylation, and showed improved mitochondrial f
33 ieties on proteins, differentiation specific protein prenylation, and the ability of peptidomimetic p
34         Significant to AD, reduced levels of protein prenylation are present in the cerebral cortex o
35  with its potential utility for the study of protein prenylation, are discussed.
36 ith concurrent reversal of the inhibition of protein prenylation as shown by protein RhoA geranylgera
37  in CD43 redistribution is through decreased protein prenylation because the cholesterol-dependent li
38 effects of FPP could not be accounted for by protein prenylation, because inhibition of farnesylation
39                       The ability to measure protein prenylation before and after FTI and GGTI treatm
40 ts, we provide evidence that isoprenoids and protein prenylation, but not cholesterol, are required i
41                               The pattern of protein prenylation by epithelial and fiber cells was si
42  describe protocols to measure the degree of protein prenylation by farnesyl transferase or geranylge
43   This study demonstrates that inhibition of protein prenylation by lovastatin leads to disruption of
44              Direct identification of native protein prenylation by mass spectrometry (MS) has been c
45 dia containing 3H-mevalonolactone to examine protein prenylation by mevalonate-derived isoprenes.
46 ndicating that increased cellular demand for protein prenylation cannot explain increased statin sens
47  pyrophosphate (GGPP), which is required for protein prenylation, caused cell stress in monocytes, fo
48  cells with lovastatin, a drug that disrupts protein prenylation, changed the relative electrophoreti
49 hree post-translational processing reactions-protein prenylation, endoproteolysis, and carboxymethyla
50      Previously, we generated mutations in a protein prenylation enzyme, GGB, and showed that it is e
51 l or geranylgeranyl lipids, a process called protein prenylation, facilitates interactions of protein
52                         To determine whether protein prenylation (farnesyl/geranylgeranylation) regul
53 oves the confidence in the identification of protein prenylation from large-scale samples, which enab
54                               Aberrations in protein prenylation have been indicated in multiple dise
55 pleiotropic effects that establish roles for protein prenylation in abscisic acid (ABA) signaling and
56  synthesis of isoprenoid lipids required for protein prenylation in bone-resorbing osteoclasts.
57 of prenylated proteins and reveal a role for protein prenylation in host defense against viral infect
58  these peptides open the door for studies of protein prenylation in living cells, including enzymatic
59 beta production and elucidate the effects of protein prenylation in monocytes.
60 gulation in the synthesis of isoprenoids for protein prenylation in obesity might be a factor determi
61 on of clodronate and alendronate, effects on protein prenylation in osteoclasts and macrophages in vi
62 ese results demonstrate unique properties of protein prenylation in P. falciparum: a limited specific
63 an cells and yeast, however, the function of protein prenylation in plants is not well understood and
64 or farnesol (FOH) to investigate the role of protein prenylation in platelet-derived growth factor (P
65 sferase specificity and functional roles for protein prenylation in Rho GTPase function.
66 d showed improved mitochondrial function and protein prenylation in the presence of statins.
67  not squalene, indicating the involvement of protein prenylation in this process.
68 cant progress has been made in understanding protein prenylation in vitro, we have been interested in
69 a an arteriovenous-dependent requirement for protein prenylation in zebrafish and human endothelial c
70 nyl pyrophosphate (GGPP), which are used for protein prenylation, including the oncoproteins of the R
71                                              Protein prenylation involves the attachment of one or tw
72                                              Protein prenylation, involving the alkylation of a speci
73                                              Protein prenylation is a common post-translational modif
74                                              Protein prenylation is a post-translational modification
75                                              Protein prenylation is a post-translational modification
76                                              Protein prenylation is a posttranslational lipid modific
77                                   Inhibiting protein prenylation is an attractive means to modulate c
78                                              Protein prenylation is an essential post-translational m
79                                              Protein prenylation is an important post-translational m
80                                              Protein prenylation is believed to be catalyzed by three
81                                              Protein prenylation is carried out by a pair of cytosoli
82 mised individuals, is one pathogen for which protein prenylation is essential for survival.
83 Using conditional mutant mice, we found that protein prenylation is essential for sympathetic axon in
84 vel of residual enzyme activity, below which protein prenylation is impaired.
85                                              Protein prenylation is required for a variety of growth
86 f prenyltransferase inhibitors indicate that protein prenylation is required for malaria parasite dev
87                                     Although protein prenylation is widely studied, there are few goo
88 osis induced by statins (other inhibitors of protein prenylation) is dependent on protein synthesis.
89  inhibitor-2147 (GGTI-2147), an inhibitor of protein prenylation, markedly increased cytosolic accumu
90  in-vitro studies suggest that inhibition of protein prenylation may underlie the myotoxic effects of
91                                              Protein prenylation occurs in the protozoan that causes
92                We have previously shown that protein prenylation occurs in the Trypanosomatids Trypan
93                          It is not clear how protein prenylation of small GTPases relates to GTP hydr
94  production versus mevalonate pathway-driven protein prenylation, on the emergence of the so-called p
95  plants were treated with some inhibitors of protein prenylation or by further monoterpenes.
96 two branches of mevalonate metabolism-fueled protein prenylation pathway in thymocyte egress and immu
97 the last of the three-step posttranslational protein prenylation process for the so-called CaaX prote
98 ence on both sterol metabolite synthesis and protein prenylation processes.
99 o oAbeta(42)-treated neurons recovers normal protein prenylation, reduces cholesterol sequestration,
100                                Inhibition of protein prenylation represents a mechanism of oAbeta(42)
101 retreatment with lovastatin, an inhibitor of protein prenylation, resulted in superinduction of NOS-2
102  Exogenous farnesol, which enhances membrane protein prenylation, reversed viperin-mediated inhibitio
103 poptotic cell death induced by inhibitors of protein prenylation such as bisphosphonates.
104 tatins are caused primarily through impaired protein prenylation that results in mitochondria dysfunc
105                                              Protein prenylation, the specific prenyl modification (f
106 induced lowering of isoprenoids required for protein prenylation triggered NLRP3/caspase-1 inflammaso
107 cific inhibitors, the relative importance of protein prenylation versus terminal cholesterol synthesi
108 ailability of the intermediates required for protein prenylation was responsible for decreased gammah
109 o the unprenylated form of RaplA, effects on protein prenylation were assessed by Western blot analys
110 of senescent human fibroblasts by inhibiting protein prenylation, without affecting the senescent gro
111 doubts about whether any treatment targeting protein prenylation would be particularly effective.

 
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