1 ta- and alpha-cells; however, the function of islet TSPAN-
7 has not been determined.
2 d for females who participate in activity, however
accuracy has not been determined.
3 However, it remains controversial
and has not been determined for any of the bacterial enzymes that
4 own; however, its structure bound to the VirB1-11
apparatus has not been determined.
5 rt failure (HF), but the efficacy of noninvasive
approaches has not been determined.
6 +) neurons, chandelier cells (ChCs) and basket cells (
BCs),
has not been determined.
7 change of the S protein is initiated after receptor
binding has not been determined.
8 protein-protein interaction domains of PCDH15 in hair
cells has not been determined.
9 anges on large-scale network activity in myenteric
circuits has not been determined.
10 mit any of these viruses (that is, their vector
competence)
has not been determined.
11 zing metabolites at physiologically relevant
concentrations has not been determined.
12 e methods, the exact number of active conformations of
CueR has not been determined.
13 While the exact role of
CytM has not been determined, a mutant deficient in the thylakoid-
14 following reduced human papillomavirus (HPV) vaccine
doses has not been determined.
15 c nervous activity and hypoxia-induced vascular
dysfunction has not been determined.
16 impact of this intracellular pathogen on the bAM
epigenome has not been determined.
17 r S-acylation affects ASBT function and membrane
expression has not been determined.
18 ogous state transitions in mixtures of supercritical
fluids has not been determined, and it is not clear whether a Widom
19 in, however, has a role in normal human macrophage
function has not been determined.
20 information independent of sensory inputs and future
goals has not been determined.
21 The function of Cox16p,
however,
has not been determined.
22 enescence, the contribution of TRF1 to senescence
induction has not been determined.
23 abolic deficit can be targeted for therapeutic
intervention has not been determined.
24 It has not been determined whether the elevation of inflammatory
25 It has not been determined, however, whether the cell death-enab
26 important in HD and fl-mHTT is expressed in astrocytes,
it has not been determined whether the expression of fl-mHTT in
27 counts are increased during human tuberculosis (TB) but
it has not been determined whether Mycobacterium tuberculosis (M
28 However,
it has not been determined functionally whether promoter-enhance
29 Although CXCL13 is produced by pulmonary tissues,
it has not been determined which cells are involved.
30 the activity of the mPFC and amygdala at the synaptic
level has not been determined previously.
31 of MHC I with a chaperone that facilitates peptide
loading has not been determined.
32 steroid therapy, are common options, but optimal
management has not been determined.
33 But its Cooper pairing
mechanism has not been determined.
34 use in the pharmaceutical industry, the reaction
mechanism has not been determined.
35 However, the role of vimentin in amoeboid
migration has not been determined.
36 d template, but the exact axial disposition of the
molecule has not been determined.
37 tease, the structure of the N-terminal RNA helicase (
nsP2h)
has not been determined.
38 however, its role in pulmonary arterial hypertension (
PAH)
has not been determined.
39 me of day of chlamydia infection on chlamydial
pathogenesis has not been determined.
40 multiple myeloma; however its role in disease
pathogenesis has not been determined.
41 at baseline, but the impact of TB treatment on this
pattern has not been determined.
42 CAPS1 binding to DCVs and for its regulation of vesicle
pH has not been determined.
43 ever, the mutational landscape that may predict
progression has not been determined.
44 the Ni sites (Ni-N(2)C(2), Ni-N(3)C(1), Ni-N(4)) in Ni-
SACs has not been determined experimentally.
45 depth and cell numbers, two important factors in scRNA-
seq,
has not been determined for a complex cell population such as
46 a condition characterized by aberrant type I IFN
signaling,
has not been determined.
47 t animal models, a possible embryonic root of this
syndrome has not been determined.
48 asis of variation in the progression of primary
tauopathies has not been determined.
49 However, the effect of vector genome size on
titre has not been determined.
50 a for hydrophobic molecules, yet the mechanism of
transport has not been determined.