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2 progression toward autoimmune insulin deficiency in adults is probably due to a lower genetic load overall combined with
3 vative, XAC-X-BY630, revealed that the decrease in affinity is probably due to a significant increase in dissociation rat
4 re, the emergence of bone marrow failure in Fanconi anaemia is probably due to aldehyde-mediated genotoxicity restricted
5 This effect is sex- and oestrous cycle-dependent and is probably due to an oestrous cycle-dependent change in the
6 g a variety of blockers we determined that the augmentation is probably due to insertion of GABA(A)Rs into the synapse by
8 The smaller amount of the FMO protein in the mutant cell is probably due to the degradation of the apo-FMO protein at
9 fold selectivity of PAP-1 for Kv1.3 over other Kv1 channels is probably due to its preferential affinity to the C-type in
10 epancy between observed and expected nitrite concentrations is probably due to a combination of factors, including an inc
16 ry and subsequent wet heating; this non-monotonic evolution is probably due to protein structural changes, and shows that
17 d H4K12 acetylation that occurs following acrolein exposure is probably due to the formation of acrolein-histone lysine a
18 yed temporal response between RLC phosphorylation and force is probably due to mechanical effects associated with elastic
19 di-GMP to form aggregates (as compared to that of c-di-GMP) is probably due to a higher activation barrier to convert fro
20 erence in Lisch nodule burden between light and dark irides is probably due to the photoprotective effects of pigmentatio
21 retains its native globular structure, so its activity loss is probably due to loss of specific toxin-receptor residue pa
22 This discrepancy with the earlier studies using microsomes is probably due to the known accessibility of microsomal carb
23 ts high expression correlating with worse end-point outcome is probably due to the passenger event of the nearby ERBB2's
25 The increased severity of the Lama1 null mutant phenotype is probably due to the complete loss of the inner limiting me
30 association between high transcription and genome stability is probably due to a variety of factors including an enhanced
31 ictions in the literature about the R.R:Y triplex stability is probably due to lack of molecular resolution, since shifti
32 reduced in the nuclei of transfected cells, an effect that is probably due to a general defect in 3' RNA processing.
33 s markedly enhanced upon acetylation of K56, an effect that is probably due to increased conformational entropy of the al
44 ha-Smooth Muscle Actin) upon treatment with TGF-beta1; this is probably due to intrinsic variability of cytokine concentr
45 ther than restricted developmental potential, however, this is probably due to a matter of timing.
46 antly reduced the N2 O production from these pathways; this is probably due to the observed reduction in the expression o
47 The heterogeneity among colorectal tumors is probably due to differences in developmental pathways and
48 s in human and mouse kidneys as well as MDCK cysts in vitro is probably due to enhanced levels of reactive oxygen species
49 ron and manganese remain mainly between +II and +III, which is probably due to short residence time of these particles in
50 tead of isolated pathways, may overcome this problem, which is probably due to the extreme heterogeneity of human tumors.