ライフサイエンスコーパス: lack the ability to

1 might be because the mature mammalian retina lacks the ability to accept and incorporate stem cells o

2 on using molecular dynamics simulations that lacked the ability to account for charge transfer and di

3 importantly, every theory (and every model) lacks the ability to account for some key autoimmune dis

4 Deltazrc1Deltacot1 lacks the ability to accumulate Zn in the vacuole and ha

5 Yet, we lack the ability to accurately link genes with their dis

6 Unlike CXCL12, these truncates either lack the ability to act as a chemoattractant for CD34(+)

7 which, although often described as "smart," lack the ability to act autonomously.

8 beta-Catenin-deficient mice, which lack the ability to activate canonical Wnt signaling, ex

9 ike NeuroD, the c-Myc transactivation domain lacked the ability to activate insulin gene expression.

10 were enhanced in muscle and adipose of mice lacking the ability to activate the signal transducer an

11 Importantly, this mutant lacks the ability to activate p53-dependent apoptotic ge

12 ther, a compound that binds to PPARgamma but lacks the ability to activate transcription, also inhibi

13 lymeric IgR knockout (pIgR(-/-)) mice, which lack the ability to actively secrete IgA into the mucosa

14 nt transport, such as cystic fibrosis, might lack the ability to adapt to the infection and present w

15 fmt) mutant strains of L. monocytogenes that lacked the ability to add formyl groups to nascent polyp

16 health-related impacts of climate change but lacking the ability to address these impacts.

17 The glnD null mutant completely lacked the ability to adenylylate GlnK.

18 cause these methods have a limited scale and lack the ability to afford particles of varied shapes.

19 pocket and surrounding hydrophobic ridge, it lacks the ability to agglutinate guinea pig erythrocytes

20 in K14E6(WT)), the mice expressing E6(I128T) lacked the ability to alter the radiation-induced block

21 a mixed population of viruses containing or lacking the ability to antagonize BST-2.

22 ct into JH-deleted embryonic stem cells that lack the ability to assemble HC variable region genes an

23 f mu1, unable to release mu1N or form pores, lack the ability to associate with membranes.

24 eintroduction of an N-terminal FMRP fragment lacking the ability to associate with polyribosomes norm

25 variants with interface point substitutions lack the ability to be fully activated by Vps75, and one

26 LIP-1 was attenuated in VILIP-1 mutants that lacked the ability to be myristoylated or to bind calciu

27 al calcium binding domains as well as HA-CaM lacking the ability to be phosphorylated at residues Tyr

28 ns 2 and 3, and this poly(A)-binding protein lacks the ability to be regulated by oxidation or reduct

29 logous domain termed Zbeta, which appears to lack the ability to bind left-handed nucleic acids.

30 ce, the three follistatin domains inherently lack the ability to bind or neutralize activin.

31 18BPd isoforms lack a complete Ig domain and lack the ability to bind or neutralize IL-18.

32 tion kinetics via binding to Na(V)1.x site 3 lack the ability to bind phospholipids, whereas site 4 t

33 fied using genomic approaches alone, as they lack the ability to bind specific DNA sequences.

34 the propagation of targeted Ad vectors that lack the ability to bind the native fiber receptor.

35 ll-size antibodies, these antibody fragments lack the ability to bind the neonatal Fc receptor (FcRn)

36 ifying viral envelope proteins, so that they lack the ability to bind to their cognate receptor but s

37 Expression of Ng mutants that lack the ability to bind to, or dissociate from, calmodu

38 Mutants of Bro1 that lack the ability to bind Ub were dramatically impaired i

39 GPIHBP1-W109S lacked the ability to bind LPL but had a reduced propens

40 A p21 mutant that lacked the ability to bind proliferating cell nuclear an

41 Recently we have shown that mutants of Kv4.2 lacking the ability to bind an intersubunit Zn(2+) betwe

42 7 with wild type CaM or CaM1,2,3,4, a mutant lacking the ability to bind Ca2+, rescued surface expres

43 ransgenes encoding mutant versions of CBP60a lacking the ability to bind calmodulin failed to complem

44 s adjuvanticity, we examined how a PA mutant lacking the ability to bind EF (PA-U7) or another mutant

45 ting moiety of P947 with a scrambled peptide lacking the ability to bind MMPs.

46 e expression of beta(1)Pix SH3m(W43K), which lacks the ability to bind Pak, and beta(1)PixDHm(L238R/L

47 ation knock-in mouse (EPCR(R84A/R84A)) which lacks the ability to bind PC/APC.

48 However, GABA(B(1e)) lacks the ability to bind the radiolabeled antagonist [(

49 The p53 mutant that lacks the ability to bind to mortalin remains at centros

50 w here that GFI136N, in contrast to GFI136S, lacks the ability to bind to the Gfi1 target gene that e

51 Mutant NKCC1 that lacks the ability to bind to this kinase showed K+ trans

52 However, they lack the ability to bond along specific directions as at

53 ine receptor mutation resulted in a receptor lacking the ability to catalyze the binding of guanosine

54 ave diminished CAAX proteolytic activity and lack the ability to cleave the amino terminus of the a-f

55 of a palmitoylation-deficient PSD-95 mutant lacking the ability to cluster and to interact with NMDA

56 tive medicine, but standard assays generally lack the ability to combine different cell stimulations

57 y lipoprotein receptor-deficient mice, which lack the ability to control lipid levels in the blood, i

58 at maintain tumor antigen responsiveness but lack the ability to control melanoma outgrowth.

59 Synthetic polymer approaches generally lack the ability to control the primary sequence, with s

60 se conventional approaches, although useful, lack the ability to control the spatial and temporal asp

61 growth inhibition was not observed in cells lacking the ability to correctly upregulate this protein

62 Finally, we also showed that HSV strains lacking the ability to counter autophagy and the PKR-dri

63 A splice variant of MyD88 (MyD88(S)), which lacks the ability to couple IRAK-1 to NF-kappaB, has bee

64 Current tools lack the ability to create highly customizable and publi

65 e when dealing with thousands of samples and lacks the ability to create scores that could be used as

66 hat, unlike typical circulating HIV strains, lack the ability to degrade IRF3.

67 Filaments of nhx1 nhx2 did not elongate and lacked the ability to dehisce and release pollen, result

68 er, individual solid-state plasmonic devices lack the ability to deliver multiple functionalities.

69 Mannose receptor knockout (MR(-/-)) mice lack the ability to detect trimannose.

70 struction methodologies for cDNA microarrays lack the ability to determine array integrity prior to h

71 niform sampling of the genome and inherently lack the ability to differentiate highly similar paralog

72 Gfi-null progenitors accumulate because they lack the ability to differentiate.

73 inding of NADP(+) but resulted in the enzyme lacking the ability to differentiate between the oxidize

74 ndocytosis motif in K5 resulted in a protein lacking the ability to direct an increased rate of MHC-I

75 transcription or translation, they generally lack the ability to directly sense cellular signals.

76 In addition, many studies lack the ability to distinguish between lifetime and rec

77 Specifically, conventional approaches lack the ability to distinguish differences across cells

78 ic cleavage, while a guide-specific approach lacked the ability to distinguish between the wild-type

79 nal cellulosic aerogel processing approaches lack the ability to easily fabricate complete aerogel st

80 The mammalian CNS lacks the ability to effectively compensate for injury b

81 re that primary fibroblasts from CS patients lack the ability to efficiently repair these particular

82 These mutants uniformly lacked the ability to either grow photoautotrophically o

83 own until recently - largely because we have lacked the ability to elucidate and then probe the under

84 vealed that Rce1-deficient embryos and cells lacked the ability to endoproteolytically process Ras pr

85 are isosteric to natural DNA bases but which lack the ability to engage in Watson-Crick (W-C) hydroge

86 ucing adult level responses on occasion, but lack the ability to engage systems mediating response in

87 ic mouse-human DII-FL MAb (E28) variant that lacks the ability to engage FcgammaR and C1q also did no

88 HuR-deficient PDAC cells lacked the ability to engraft successfully in immunocomp

89 ation and the C-terminal deletion completely lacked the ability to enhance phagocytosis.

90 Further analysis showed that Tok07 PB1 alone lacked the ability to enhance the pathogenicity of the r

91 Hdm2NLS lacks the ability to enter the nucleus, whereas Hdm2NES

92 n through activation of RIG-I signaling, but lacks the ability to evade the human IFN response.

93 ons between wild-type and deficient mice and lack the ability to examine reversal/inhibition of injur

94 was virtually nonexistent because all OPMRs lacked the ability to exchange data electronically with

95 Males appear to lack the ability to exhibit an LH surge, but it is uncle

96 16-Delta strains of Saccharomyces cerevisiae lacking the ability to export all RNAs, including poly(A

97 strategy, a GPCMV mutant virus was used that lacked the ability to express an essential capsid gene (

98 l(+/+) CTLs because these remained immature, lacking the ability to express costimulatory molecules C

99 nother population of similar cells but which lacked the ability to fire phasically.

100 Lacking the ability to fly away from predators, this des

101 produce bone within the injected bone, they lack the ability to form mineralized bone nodules when e

102 l lines expressing the ANG-ALS variants also lack the ability to form stress granules.

103 CD133KD cells also lacked the ability to form CD144(+) VM-like channels in

104 on ancestor of tetrapods and ray-finned fish lacked the ability to form ectomesenchyme and its deriva

105 DM action, we prepared peptide variants that lacked the ability to form one or more of the hydrogen b

106 f 24,444 and an isoelectric point of 7.7 and lacked the ability to form the cystine loop structure ch

107 A Cys-to-serine mutant of wheat eIF4E, which lacked the ability to form the disulfide, crystallized w

108 tified that did not elicit cell rounding and lacked the ability to form typical plaques.

109 ntiospecificity was observed for 2-pyridones lacking the ability to form H-bonds.

110 3,5-benzenetricarboxyester (BTE) derivative, lacking the ability to form the H-bonding network, demon

111 , a SipW mutant protein was constructed that lacks the ability to form a solid-surface biofilm but st

112 an the others, divides at a slower rate, and lacks the ability to form an eye.

113 trical function and syntaxin 1A binding, but lacks the ability to form clusters, does not enhance gra

114 tion when 3-deazaguanine, a base analog that lacks the ability to form minor-groove hydrogen bonds wi

115 ic variant of SIAE was catalytically active, lacked the ability to function in a dominant negative ma

116 tibodies against conformational epitopes but lacked the ability to function in cell-cell fusion assay

117 This gRNA lacks the ability to function in trans.

118 Here, we show that CD73(-/-) mice, which lack the ability to generate extracellular adenosine, ar

119 Mice that lack the ability to generate NE or epinephrine show incr

120 An ackA mutant, lacking the ability to generate ATP from acetyl phosphat

121 nstrate that stem cell transplant recipients lacking the ability to generate or signal IL-17 develop

122 Prosthetic valves currently used in children lack the ability to grow with the patient and often requ

123 ltahtx strain was mutagenized and one mutant lacking the ability to grow on methylphosphonate as the

124 otype similar to that of a crp mutant, which lacks the ability to grow anaerobically with DMSO, fumar

125 h thymine replaced by difluorotoluene, which lacks the ability to hydrogen bond.

126 We demonstrate that human Tdp1 lacks the ability to hydrolyze a phosphodiester linked 5

127 plex departures from the null hypothesis but lack the ability to identify the specific alternative hy

128 Permeabilized cells of a dltD::erm mutant lacked the ability to incorporate D-alanine into LTA.

129 Mutants also lack the ability to increase mEJP rate in response to sp

130 Consequently, they lack the ability to increase the physiological salience

131 ability to organize axis formation, but they lack the ability to induce neuroectodermal tissues, a ch

132 that lack the NF-kappaB essential modulator lack the ability to induce the IFN genes following DNA d

133 A Fab fragment of the antibody, however, lacked the ability to induce cell death.

134 macrophages prepared from the same monocytes lacked the ability to induce IL-12 or IFN-gamma response

135 icroscopy demonstrated that the tssE mutants lacked the ability to induce multinucleated giant cell f

136 t the N-terminal-truncated CXCL12/SDF-1alpha lacks the ability to induce the migration of CD34(+) cor

137 r support for our hypothesis that drugs that lack the ability to inhibit transport by all three monoa

138 Axin mutants with C-terminal truncations lacked the ability to inhibit Lef-1 reporter activity, e

139 d 3) all TRPV1 mutations insensitive to ACEA lacked the ability to inhibit MO-evoked calcium accumula

140 EC strain that has a disruption in this gene lacks the ability to inhibit lymphokine production and l

141 ion of transport to the plasma membrane, and lacks the ability to inhibit the formation of CopII coat

142 he LIM domains is sufficient to bind DAT but lacks the ability to inhibit transporter activity.

143 They lack the ability to initiate pheromone-induced G1 cell c

144 h Pol II is a complex, 12-subunit enzyme, it lacks the ability to initiate transcription and cannot c

145 Some model eukaryotic organisms lack the ability to insert selenocysteine, and prokaryot

146 structure-based BCAR1 and BCAR3 mutants that lack the ability to interact, we show that BCAR3-induced

147 of Scs2p (scs2Delta) and a mutant, OPI1FFAT, lacking the ability to interact with Scs2p were utilized

148 nulogenic truncation mutant of SgIII (1-210) lacks the ability to interact with CgA.

149 an IgG1 antibody with a constant region that lacks the ability to interact with Fcgamma receptors.

150 overexpression of a mutant of p85alpha that lacks the ability to interact with the p110alpha catalyt

151 cts with another bZIP protein TaFDL13, which lacks the ability to interact with the VRN1 promoter, su

152 The toolbox of rat genetics currently lacks the ability to introduce site-directed, heritable

153 responsive tissues, we have used female mice lacking the ability to localize ERalpha to the membrane

154 ond to patterning and hydrodynamic cues, but lack the ability to maintain their precise orientation.

155 We have studied Sl/Sl(d) mice, which lack the ability to make membrane-bound stem cell factor

156 As such, we lack the ability to make predictions about the responses

157 Mice that lacked the ability to make both CD8(+) and CD4(+) iTregs

158 protein receptor Rag 1 double-knockout mice (lacking the ability to make immunoglobulins due to loss

159 Seven M. truncatula mutants, lacking the ability to make nodules, were tested for Nod

160 the same dose of AMPH is not blunted in mice lacking the ability to make norepinephrine and epinephri

161 Whereas wild-type C. bescii lacks the ability to make ethanol, 70% of the fermentati

162 ility of a C10S/V30M TTR double mutant which lacks the ability to make mixed disulfides, but retains

163 tant of Streptococcus pyogenes Manfredo that lacks the ability to make streptococcal acid glycoprotei

164 ory activity; however, the monomeric species lacked the ability to manifest anti-proliferative activi

165 However, this approach lacks the ability to measure and evaluate important meta

166 lidated bioprocessing of lignin, and it also lacks the ability to metabolize sugars or organic acids;

167 Lysates from Icmt-/- embryos lacked the ability to methylate either recombinant K-Ras

168 Lysates from Icmt-/- cells lacked the ability to methylate farnesyl-K-Ras4B or smal

169 In addition, Icmt-/- cells lacked the ability to methylate Rab proteins.

170 d this hypothesis using Stat1-/- mice, which lack the ability to mount Th1 immune responses.

171 10,000 clones were screened for mutants that lacked the ability to move away from the edge of a colon

172 re many ways to detect kinase activity, most lack the ability to "multiplex" the analysis (i.e., to d

173 cterized bevacizumab as species specific and lacking the ability to neutralize murine (m) VEGF-A.

174 sferase, hepatocarcinoma cells were found to lack the abilities to oxidize choline to betaine and to

175 e determined that RDH10(trex) mutant protein lacks the ability to oxidize retinol to retinal, resulti

176 on reaction leading to lariat formation, but lacks the ability to participate in the second reaction.

177 rained operator, cannot be miniaturized, and lack the ability to perform automated real-time high-thr

178 Bax toxicity was reduced in strains lacking the ability to perform oxidative phosphorylation

179 -7 assemble motile, full-length flagella but lack the ability to phototax.

180 actin machinery was intact, Cdc42 null cells lacked the ability to polarize their Golgi and coordinat

181 c-based antimicrobial susceptibility testing lacks the ability to predict clinical response to antimi

182 MxiE-regulated genes yielded no mutants that lacked the ability to prevent apoptosis.

183 HCV Y16F replication, and the Y16F protease lacked the ability to prevent IRF3 activation or interfe

184 Despite advances in nanotechnology, we still lack the ability to print arbitrary colors and shapes in

185 Furthermore, most techniques lack the ability to process multiple samples simultaneou

186 beta converting enzyme (ICE)-deficient mice, lacking the ability to process mature IL-18 and IL-1beta

187 the absence of anti-Gal B cells, these mice lack the ability to produce anti-Gal.

188 33 identifies Treg cells in human blood that lack the ability to produce effector cytokines (IL-2, IF

189 Most importantly, they lack the ability to produce interferon-gamma in response

190 possess any antimicrobial activity and hence lack the ability to produce microbial resistance, but wh

191 a-hydroxylase knock-out (Dbh-/-) mice, which lack the ability to produce the SNS transmitters, norepi

192 ), and the respective isogenic mutants which lacked the ability to produce alginate, for their suscep

193 is cell line to generate a viral mutant that lacked the ability to produce B2.

194 Mice lacking the ability to produce BMP2 in their limb bones

195 Transcription of rrn operons in strains lacking the ability to produce either NusA or NusB was e

196 Mice lacking the ability to produce eosinophils should prove

197 220 or its isogenic mutant strain (Deltafmt) lacking the ability to produce formylated peptides.

198 tratracheal transfer of airway CD8 TRM cells lacking the ability to produce IFN-gamma were less effec

199 NK cells with a normal mature phenotype, but lacking the ability to produce IFN-gamma, whereas cocult

200 the presence of PCA, a P. aeruginosa mutant lacking the ability to produce the siderophores pyoverdi

201 lly equivalent to the inner cell mass, which lacks the ability to produce all extraembryonic tissues.

202 This approach, however, lacks the ability to produce large and continuously vari

203 cell line that stably expresses human TP and lacks the ability to produce TXA(2) was created by gene

204 However, these catalysts generally lack the ability to promote CO(2) reduction beyond the t

205 Moreover, a Bcr mutant that lacks the ability to promote hydrolysis of Rac-GTP bound

206 They also lacked the ability to propel latex spheres and were resi

207 However, these methods lack the ability to provide spatial information for thes

208 in large cell populations and, as a result, lack the ability to quantify cell-to-cell variations.

209 Existing approaches lack the ability to quantify the enrichment levels accur

210 imated strength of physical interactions and lack the ability to rank interactants as a function of t

211 This was because it lacked the ability to rearrange itself upon binding.

212 s algebraic reconstruction technique (SART), lack the ability to recover the unacquired project infor

213 MCT-ND4, we found that the PSMalpha peptides lacked the ability to recruit beta-arrestin and induce n

214 g and short isoforms, with the short isoform lacking the ability to recruit HDACs.

215 to hyperphosphorylated RNA polymerase II but lacks the ability to recruit SR proteins because of repl

216 l domain, whereas the TLS-ERG fusion protein lacks the ability to recruit SR proteins due to replacem

217 t the discretion of the attending physician, lack the ability to redefine the standard of care and mi

218 xicity, whereas obinutuzumab, a type II mAb, lacks the ability to redistribute into lipid rafts and i

219 P450 reconstituted system, Mn-cyt b5, which lacks the ability to reduce oxyferrous cyt P450 2B4, was

220 porary bladder tissue engineering strategies lack the ability to reform bladder smooth muscle, vascul

221 Mammals, in contrast to zebrafish, lack the ability to regenerate hair cells.

222 the developed world, partly because mammals lack the ability to regenerate heart tissue.

223 XDbf4 mutant that inhibits Wnt signaling but lacks the ability to regulate Cdc7.

224 were tightly adherent to surfaces but which lacked the ability to release cells into the medium and

225 , dilute bleach, and mechanical cleaning all lack the ability to remove cellular debris completely, w

226 ApoA-I lacked the ability to remove alpha-TOH from Tangier dise

227 Mitochondria lack the ability to repair certain helix-distorting lesi

228 ic acids, proteins, and in some cases lipids lack the ability to replicate outside their target cells

229 In general, nonlegumes were assumed to lack the ability to respond to the rhizobial lipo-chitin

230 HCs are formed before birth, and mammals lack the ability to restore the sensory deficits associa

231 d a sedimentation coefficient of 5 to 7S and lacked the ability to restore translation in extracts of

232 s a harsh microenvironment for islets and it lacks the ability to retrieve the graft.

233 nference software offerings that use ABC-SMC lack the ability to scale in parallel computing environm

234 imeras in which only either B-1 or B-2 cells lack the ability to secrete IgM show mortality rates sim

235 ted conditional Wntless (Wls) KO mice, which lacked the ability to secrete Wnts from either liver epi

236 assessed the functional capacity of T(regs) lacking the ability to secrete both IL-10 and IL-35, whi

237 that mice deficient in the IL-17 receptor or lacking the ability to secrete IL-17 are highly suscepti

238 We show that this peptide lacks the ability to seed aggregation of tau244-372 in c

239 typical bovine insulin fibrils, although it lacks the ability to seed the intact protein.

240 ogical inactivation of AC, but these methods lack the ability to selectively manipulate projection ne

241 However, multipotent progenitors lack the ability to self-renew, therefore their mitotic

242 However, NF180 lacked the ability to self-assemble, suggesting that lik

243 es possessing the same functional groups but lacking the ability to self-assemble do not catalyze sub

244 Articular cartilage (AC) lacks the ability to self-repair and cell-based approach

245 Api g 1, the Bet v 1 homolog in celery, lacks the ability to sensitize and is devoid of major T-

246 ic inhibitor but not with an HBx mutant that lacked the ability to sequester Crm1 in the cytoplasm.

247 Furthermore, knockout mice lacking the ability to signal through the type I interfe

248 In addition, mice lacking the ability to signal through TLR4 had significa

249 CXC families with nanomolar affinity, yet it lacks the ability to signal upon ligand binding.

250 The majority of existing vaccine adjuvants lack the ability to significantly stimulate the cellular

251 MM11453 lacked the ability to significantly activate RARs and re

252 a popular standard coalescent simulator, it lacks the ability to simulate sequences with recombinati

253 suggesting that some S. aureus isolates may lack the ability to site-specifically integrate SCCmec i

254 ect macrophages, it had reduced survival and lacked the ability to spread cell to cell, indicating pp

255 ABCA1-dependent cholesterol efflux, and they lacked the ability to stabilize ABCA1.

256 and thyroxine preferential binding sites and lacked the ability to stabilize TTR.

257 ommitment at a first encounter, primary IPCs lacked the ability to stimulate naive T cells.

258 3 cell lines, while the PF-deficient mutants lacked the ability to stimulate, and the complemented PF

259 binding and activation of the HGF receptor, lacks the ability to super-activate the Ras-MAP kinase p

260 ate that under low Ci conditions, the mutant lacks the ability to supply Rubisco with adequate CO(2)

261 c imaging revealed that beta1-depleted cells lacked the ability to sustain protrusions into the suben

262 igoadenylate synthetase-like proteins, which lack the ability to synthesize 2'-5' A, have been recent

263 In contrast to most animals, humans lack the ability to synthesize ascorbic acid as a result

264 provement in the development of fetuses that lack the ability to synthesize cholesterol, such as thos

265 DD mice lack the ability to synthesize dopamine (DA) specificall

266 se knockout (Dbh -/-) mice that specifically lack the ability to synthesize NE.

267 r bacteria comprising the order Chlamydiales lack the ability to synthesize nucleotides de novo and m

268 Cilia lack the ability to synthesize proteins and thus require

269 All parasitic protozoa lack the ability to synthesize purine nucleotides de nov

270 parasites, they are purine auxotrophs (i.e. lack the ability to synthesize purines de novo) and ther

271 completely devoid of selenoprotein genes and lack the ability to synthesize Sec.

272 xpression profile in a luxS-deficient strain lacking the ability to synthesize AI-2.

273 Transcriptome analysis of mutants lacking the ability to synthesize cAMP or the cAMP bindi

274 But children lacking the ability to synthesize cortisol (because of v

275 missive to HMPV infection, mutant cell lines lacking the ability to synthesize glycosaminoglycans (GA

276 Members of the Nematoda are heme auxotrophs, lacking the ability to synthesize heme; however, the hum

277 xpression were still observed in backgrounds lacking the ability to synthesize JA or the key transcri

278 depletion of available carbon, swarmer cells lacking the ability to synthesize ppGpp or polyP imprope

279 ic analysis we show that B. subtilis strains lacking the ability to synthesize pulcherriminic acid fo

280 ultisensory neurons are strikingly immature, lacking the ability to synthesize the cross-modal inform

281 With a highly reduced genome, it lacks the ability to synthesize any of its own amino aci

282 ltalro1Deltaare1Deltaare2Delta strain, which lacks the ability to synthesize both TAG and SE, is not

283 istolytica, is a nucleo-base auxotroph (i.e. lacks the ability to synthesize purines or pyrimidines d

284 mophilum is an aerobic photoheterotroph that lacks the ability to synthesize several essential nutrie

285 The results argue that people with autism lack the ability to take into consideration what others

286 only the CL20i4-EF1alpha-hgamma(c)OPT vector lacked the ability to transactivate LMO2 protein express

287 We establish that the human Fat homolog FAT4 lacks the ability to transduce Hippo signaling in Drosop

288 ein (MTP), Chinese hamster ovary (CHO) cells lack the ability to translocate apoB into the lumen of t

289 hat the newly isolated H5N2 and H5N8 viruses lacked the ability to transmit between ferrets and exhib

290 Moreover, these influenza H5Nx viruses lacked the ability to transmit between ferrets in a dire

291 xpected, the addition of AI-2 to cells which lack the ability to transport AI-2 (lsr null mutant) fai

292 The defective GPIHBP1 proteins also lacked the ability to transport LPL from the basolateral

293 Consequently, these cells lack the ability to undergo activation-induced cell deat

294 round cells under all growth conditions and lack the ability to undergo morphological differentiatio

295 Exosc3-deficient B cells lack the ability to undergo normal levels of class switc

296 tigations showed that IDO2-deficient B cells lacked the ability to upregulate the costimulatory marke

297 and GLUT3 double-knockout (DKO) mice, which lack the ability to use glucose, failed to increase acti

298 The dipeptide system mutants lacked the ability to use certain dipeptides, hexapeptid

299 C. dubliniensis has been reported to lack the ability to utilize xylose (XYL) and alpha-methy

300 es such as urease and xylose utilization and lacking the ability to utilize nitrate and nitrite.