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1 ons suggest that LSD's slow binding kinetics may be due to a "lid" formed by extracellular loop 2 (EL
2           The observed loss of DNA over time may be due to a bulk diffusion process in many cases, hi
3                                The phenotype may be due to a change in the expression of one or more
4 vo of cells pulsed with low doses of peptide may be due to a combination of heterogeneity in peptide
5 ggested that differences in male mate choice may be due to a couple loci with large effects.
6    A learning problem with older individuals may be due to a decrease in stability rather than a decr
7                                         This may be due to a decrease in the Ag-specific T cell popul
8 the RRM2B modifier effect on timing of onset may be due to a DNA instability mechanism.
9 type associated with the LMX1BR246Q mutation may be due to a dominant negative effect on WT1(-KTS) is
10 oms only for shorter exposure windows, which may be due to a drop in power resulting from the decreas
11 urs in conditions such as autism and anxiety may be due to a failure to appropriately modulate sensor
12 ritis can develop in some individuals, which may be due to a failure to eliminate viral RNA and Ag an
13                              These increases may be due to a faster shell formulation, enabling PLGA
14                                         This may be due to a high prevalence of horizontally oriented
15 pha-diversity was higher than soybean, which may be due to a high relative abundance of Rhizobiales.
16                              This difference may be due to a lack of detailed information on the Pi r
17                              This resilience may be due to a less extreme sleep loss than in previous
18 ttending worship services and mortality risk may be due to a number of different factors.
19 ence at older ages observed in Latin America may be due to a population-level difference in the effic
20 ex statistical features, such as bimodality, may be due to a problem of acquiring the priors rather t
21 m is unlikely attributed to NAHR, but rather may be due to a recombination-coupled DNA replicative re
22  axonal sprouting in the 125-day-old rat SON may be due to a reduction in CNTF or the CNTF receptor c
23 e decreased preload by lidocaine in CHF rats may be due to a reduction in peripheral vascular resista
24 ototype CCR9 antagonist, were not successful may be due to a relatively poor pharmacokinetic (PK) pro
25                                         This may be due to a true paucity of iron stores (absolute ir
26 xplained inconsistencies across studies that may be due to ( a) insufficient attention to measurement
27                                         This may be due to accumulated cellular damage, decreases in
28 s of the enzymes in different organisms that may be due to adaptation to the specific environments in
29                              This deficiency may be due to adiposity-related inflammation that increa
30 an adult CD8(+) T cells postinfection, which may be due to age-related differences in Lin28b expressi
31                                         This may be due to age-related dopamine (DA) decline affectin
32 er-molecular-weight compounds, although this may be due to alkane formation by thermocracking of othe
33  is stiffer than age-matched controls, which may be due to alterations in ECM cross-linking.
34     Global differences in gray matter volume may be due to alterations in the general mechanisms unde
35 ggest that LPS-induced changes in motivation may be due to alterations to mesolimbic dopamine.
36                                   The latter may be due to altered cell adhesion, as loss of Pxt or F
37 l evidence suggests that these perturbations may be due to altered epigenetic profiles and chromatin
38 elective antitumor activity displayed by OPA may be due to altered membrane PE levels in cancer cells
39 hat the agonist-independent activity of US28 may be due to an amino acid network evolved in the viral
40                            Low VTE incidence may be due to an early time to enoxaparin initiation and
41 mpared with Ad.E6E7 (DC.E6E7), a result that may be due to an effect of p16 on cyclin-dependent kinas
42 eferential clearance of male gametocytes and may be due to an effect on gametocyte fitness.
43 sults suggest that perceptual hallucinations may be due to an imprecise and biased state of sensory c
44                                         This may be due to an increase in serum hepcidin and a decrea
45  with partial Foxp3 DNA remethylation, which may be due to an increased activity of the glutaminolysi
46 yed pronounced bacterial infiltration, which may be due to an observed decrease in infiltrating immun
47            We hypothesized that such failure may be due to anergy, as CLL cells exhibit variable leve
48 he unusual rotational-translational coupling may be due to anisotropic interactions originating from
49 pha kinetics indicating increases in product may be due to APP shunting from the beta-secretase to th
50  Archaea to synthesize proteins using (2)H2O may be due to archaeal heterotrophy, whereby Archaea off
51  include the possibility that spouse effects may be due to assortative mating and the relatively smal
52 the generally low virus recovery we observed may be due to bead saturation or hindrance by existing g
53  protective association of statins on bleeds may be due to bias.
54 tions for California versus Nevada gun shows may be due to California's stricter firearm regulations.
55 the pathogenesis of Parkinson's disease (PD) may be due to cell-to-cell transmission of misfolded pre
56                                     Findings may be due to chance or explained by residual confoundin
57                       Because these findings may be due to chance, they need confirmation in larger s
58     Exploratory analyses suggested that this may be due to changes in parental functioning: Mothers i
59                       Some of this variation may be due to changes in the rate of food intake with po
60 monalities between letter and Vernier acuity may be due to common bottlenecks in early visual cortex
61 of forest ecosystems in south-central Alaska may be due to competition with tall shrubs and/or more c
62 ns and poor responses to vaccinations, which may be due to compromised B cell function.
63                                         This may be due to conditions associated with fetal growth re
64                     Left bundle branch block may be due to conduction system degeneration or a reflec
65 lts, including evidence for the association, may be due to confounding, which is rarely addressed in
66                              Most infections may be due to contact with adult men.
67 f association with diseases by CRS phenotype may be due to CRSwNP sample size limitations or reflect
68 e mechanism of which is uncertain, but which may be due to cyclic interruption and exaggeration of pu
69 rs, indicating that downward migration of Pu may be due to cycling between Pu(V) and Pu(IV).
70                  This decrease in heart rate may be due to decreased central sympathetic output as we
71 bserved in the absence of the HIGD1 proteins may be due to decreased proton pumping by complex IV (CI
72 lly, the attenuated virulence of 10-del ZIKV may be due to decreased viral RNA synthesis and increase
73         We hypothesized that this difference may be due to degradation of the sterile alpha motif and
74  sterilizing immunity induced by TB vaccines may be due to delayed activation of mucosal dendritic ce
75 race, and Hispanic ethnicity; these findings may be due to delays in diagnosis for these groups.
76 reatment in the mouse model, an effect which may be due to depletion of T-regulatory and myeloid-deri
77 nd if their initial area is >500 um(2); this may be due to deposition occurring in the outer regions
78 erved to be different between species, which may be due to differences between species in the gene ex
79                               The variations may be due to differences in abundance, recent exposure,
80 CTD on topo IIalpha and topo IIbeta function may be due to differences in CTD charge distribution and
81          We hypothesized that this variation may be due to differences in how the DNA damage response
82 c differences observed (robusta vs. Arabica) may be due to differences in ploidy level (2n = 22 vs. 2
83                           Sexual dimorphisms may be due to differences in sex hormones, sex chromosom
84 % CI, 88%-93%]); variability and imprecision may be due to differences in study designs or CTC protoc
85  randomized trials with mixed results, which may be due to differences in the study participant chara
86 hetic control of resting blood pressure (BP) may be due to differences in the transduction of sympath
87                                         This may be due to different drug release rates resulting fro
88 e substitutions in C. elegans, which in part may be due to different methodological approaches, inclu
89                                         This may be due to different regulatory mechanisms.
90        Thus, unilateral restriction of HSV-1 may be due to differential kinetics of virus arrival in
91 e that the inability of Mtb LAM to prime PMN may be due to differential localization of TLR2/1 signal
92                           These observations may be due to differing characteristics and underlying p
93       Ex vivo studies demonstrated that this may be due to diminished activation-induced cell death t
94 ative stress related chronic diseases, which may be due to direct antioxidant effects, or indirect me
95 s of tobacco products on periodontal tissues may be due to direct inhibition of normal fibroblast fun
96                            Specifically this may be due to disruption of hydrophobic interactions wit
97 sue damage in response to influenza A, which may be due to downregulated expression of a viral patter
98 wn, but further modelling revealed that this may be due to dysfunctional noradrenaline reuptake.
99                       In part, these changes may be due to dysregulation of ganglioside-mediated olig
100 al extracorporeal membrane oxygenation which may be due to early death driven by the underlying disea
101 lations in zones of active sulfate reduction may be due to either the transport of free methane gas f
102         Here we explain how this discrepancy may be due to englacial solar warming, whereby meteorite
103 t features whose association with an outcome may be due to epistasis or statistical interactions with
104 rnal and foetal morbidity and mortality, and may be due to excessive release of placental syncytiotro
105 NIV-induced extracellular matrix remodelling may be due to extensive remodelling/disassembly of the c
106 ion or stability in cones, a difference that may be due to extracellular versus intradiscal localizat
107                                 This outcome may be due to failure to attenuate the progressive subst
108 he cytotoxicity of Phe oligomers and fibrils may be due to favorable interactions between the hydroph
109  during APAs for step initiation in freezers may be due to FoG.
110  loss of PSI for step initiation in freezers may be due to FoG.
111 ng the role of AMPK in cancer, some of which may be due to functional differences between isoforms.
112                                These changes may be due to genetic predisposition, environmental pert
113 is increased death following BDNF withdrawal may be due to glutamate toxicity, as the N-methyl-d-aspa
114 iments suggest that some river-bed armouring may be due to granular segregation from below-rather tha
115 ntation with flow vector at any shear, which may be due to greater vorticity and thus instability in
116 ed with SSL risk and part of the association may be due to HCA intake.
117       The inconsistency in reported outcomes may be due to heterogeneity in study design, supplement
118  net nitrification in some highly acid soils may be due to high rates of NO3(-)-N immobilization caus
119 ficacy of available antidepressant therapies may be due to how they affect the underlying brain netwo
120 location as a driver of poor prognosis which may be due to IMiD resistance.
121 n awareness that characterizes schizophrenia may be due to impaired communication between these areas
122 ack of clinical translation of these systems may be due to, in part, to a lack of understanding of mi
123 dence of prostate cancer than ESR1 KO, which may be due to increased abundance of P. acnes.
124                              This difference may be due to increased degradation of FcgammaRIIa, whic
125 phosphomimetic T112D Lsr2 suggests that this may be due to increased dynamics of the DNA-binding doma
126                                  This effect may be due to increased near visual activity or reduced
127 detectably different seizure characteristics may be due to increased penetration of PTZ related to co
128 lar syphilis increased from 2014 to 2015 and may be due to increased recognition of ocular manifestat
129 environmental variables in the later decades may be due to increasing human influence on moose densit
130 est that the pathogenesis of this form of AI may be due to ineffective ligand binding of ITGB6 result
131 ooxygenase-1, whereas its anticancer effects may be due to inhibition of cyclooxygenase-2 and other p
132 he effect of the VEx regardless of treatment may be due to interactions of attention allocation and l
133        Instead, GPER1 actions in the synapse may be due to interactions with synaptic scaffolding pro
134 dance between amyloid biomarker measurements may be due to interindividual differences in total amylo
135 ects of the glutamatergic modulator ketamine may be due to its ability to restore synaptic plasticity
136  that oxytocin's effects on social behaviors may be due to its anxiolytic effects.
137      Air pollution's association with asthma may be due to its augmentation of allergenic effects, bu
138   The higher responses after rHA vaccination may be due to its higher HA content.
139 igration, adhesion and tube formation; which may be due to its inhibition on STAT3/HIF-1alpha/VEGF si
140 t the unexplained variance in ADHD decisions may be due to less value tracking.
141  becomes ambiguous because negative outcomes may be due to limited information or bad strategy.
142 o received their own fecal microbiota, which may be due to limited numbers.
143                                         This may be due to low expectations because people lack knowl
144                            Low total 25(OH)D may be due to low vitamin D-binding proteins (DBPs) or f
145 metabolized NMP in larvae relative to adults may be due to lower cytochrome P450 enzyme activity in f
146 n placental expression of 20alpha-HSD, which may be due to lower prolactin levels observed in these w
147 els and other indicators of average glycemia may be due to many factors but can be measured as the gl
148 ween dietary and urinary sodium or potassium may be due to measurement error in one or both estimates
149                      Up to 50% to 75% of IAP may be due to microlithiasis, which is undetectable by c
150 pancies between the gamma-probe and SPECT/CT may be due to missed SNs during surgery, but with respec
151 he brains of MAV-1-infected susceptible mice may be due to MMP activity produced by endothelial cells
152 hus, the stronger immune response of females may be due to more activated innate immune pathways prio
153 onicotinoid exposure on foraging performance may be due to motivational and/or sensory impairments.
154                     High-altitude adaptation may be due to multiple genes that act in concert with on
155                                       Ageing may be due to mutation accumulation across the lifespan,
156 The mechanism underlying this is unclear but may be due to N-methyl-D-aspartate receptor (NMDAR) hypo
157 e substitutions at positions S195D and M217T may be due to natural fluctuation of the RVA.
158 o a highly consistent, stereotyped procedure may be due to neuroanatomical variation in the patients.
159      Blue-yellow defects are most common and may be due to neurotoxicity or retinal damage.
160                        These inconsistencies may be due to NOAA satellite orbit changes and MODIS sen
161 s are not associated with HLA mismatches and may be due to non-immunologic causes (hypertension, obes
162 ever, ARB effectiveness varies widely, which may be due to non-synonymous single nucleotide polymorph
163 ests that the effect reported for the latter may be due to off-target cytotoxic effects.
164                                         This may be due to ongoing bacterial infection, translocated
165                                      Illness may be due to ongoing fungal growth that produces acute
166 ch is larger in younger than older subjects, may be due to optic nerve tethering in adduction.
167 res or disease-modifying therapies, and this may be due to our inability to detect the disease before
168 reported protumorigenic activities of sema3C may be due to p65-Sema3C produced by tumor cells.
169 ceptions and guideline assessment of control may be due to partially controlled asthma being associat
170                                         This may be due to passive seroprotection afforded by the ubi
171 pairment among those with versus without TBI may be due to poor sensitivity of the cognitive battery
172  iDC steps delivered after sustained current may be due to post-adaptation changes in afferent sensit
173 associated with Bosnia and Kosovo deployment may be due to predeployment psychological and behavioral
174 of the extinction rate from the 1960s onward may be due to prior loss of the most sensitive species a
175 monstrated that the oncogenic effect of SLPI may be due to protection of growth factor progranulin fr
176 ty among heavily stripped jellyfish galaxies may be due to ram pressure causing gas to flow towards t
177 ggesting that bidirectional pairing of genes may be due to random event.
178  improved biomarker potential of these genes may be due to reduced functional complexity.
179 enlafaxine perturbs brain development, which may be due to reduced serotonin, leading to altered larv
180 at the beneficial effect of cimetidine in PD may be due to reduction of IL-6 immunolabeling in the in
181 ssociation studies of prostate cancer (PrCa) may be due to regulatory effects of genetic variants on
182 nt tuberculosis is a major health burden and may be due to relapse with the original strain or reinfe
183 he stronger association in older individuals may be due to relatively longer duration of calcium supp
184                            This invigoration may be due to release of dopamine before movement onset,
185 ry different hypothesis: purifying selection may be due to removing harmful mutations from the popula
186 reased odds of CRC, however this association may be due to residual confounding by BMI or another con
187 Prior reports of an increased risk of stroke may be due to residual confounding related to chronic co
188 isk of diabetes associated with hysterectomy may be due to residual confounding, such as the reasons
189 d intraocular pressure control in such eyes, may be due to resolution of edema of the RNFL.
190 ation shared between closely related species may be due to retention of ancestral polymorphisms becau
191 r changes after ocriplasmin injection, which may be due to retinal traction release.
192 e; however, at least part of the association may be due to reverse causation.
193 environmental associations for atopic eczema may be due to reverse causation.
194  obesity and lower serum 25-hydroxyvitamin D may be due to reversed causation with increased adiposit
195      Association of ribosomes with the 3'UTR may be due to ribosome migration through the stop codon
196                                         This may be due to salary subsidies provided by the UHLT and
197 ases may be inherited and up to 85% of those may be due to segregation of susceptibility genes with l
198 athological changes in the paranasal sinuses may be due to selection of unsuitable CT windows.
199 nce in coloration between males and females, may be due to sexual selection for ornamentation and mat
200                                These changes may be due to shifts in the OH:HO2 ratio from SO2 oxidat
201 nsulin sensitivity in LRep1(-/-) mice, which may be due to significantly lower TG content in the live
202 that the rarity of polyploidy in gymnosperms may be due to slow diploidization in this clade.
203 enotypic influence on microbiome composition may be due to stable vertical transmission of the microb
204  high expressing PDACs have better OS, which may be due to stable vessels that supply anti-cancer imm
205                            These differences may be due to steric hindrance by the 11-keto group in C
206      This immediate extinction deficit (IED) may be due to stress-induced dysregulation of neural cir
207                                         This may be due to subjective assessment of stridor or inabil
208                                         This may be due to sulfide bridging and cation bridging, resp
209                   This increased sensitivity may be due to surface-dependent lasR induction initiatin
210 cales in amyotrophic lateral sclerosis (ALS) may be due to symptom heterogeneity, particularly coexis
211          We hypothesized that these findings may be due to systematic bias and examined the independe
212 ding artery, produce variable results, which may be due to technical difficulties associated with the
213 dges, a substantial portion of the spreading may be due to tectonic stress building up to a critical
214 es challenged with lipopolysaccharide, which may be due to the absence of amine groups.
215                                 This failure may be due to the absence of truly representative experi
216 -type genes in plants than in animals, which may be due to the abundance of intron retention cases in
217               A large part of this variation may be due to the ages of individuals who are tested and
218 suggest that the efficacy at low frequencies may be due to the amplitude of motion being of the order
219 g that the reduced MMP20 and KLK4 expression may be due to the attenuated epithelial BMP/Smad4 signal
220                              This phenomenon may be due to the coexpression of dominant and subdomina
221 r results suggest that this atypical pattern may be due to the combination of sex-specific life histo
222 ulation of multiple proteins in this pathway may be due to the combined effects of schizophrenia risk
223                            Such difficulties may be due to the competition generated in memory by irr
224                                         This may be due to the complex, branched and highly sulfated
225 in participants in the childhood abuse group may be due to the constant need to monitor their own act
226 rption and emission changes upon HSA binding may be due to the dehydration of the dye and/or stabiliz
227                   This remarkable resilience may be due to the depth-dependent mechanical properties,
228 eported differences in behavioral strategies may be due to the different types of tasks used in diffe
229  metabolite accumulation between two species may be due to the differential activation of CHS promote
230                                These results may be due to the direct effects of temperature, or medi
231 lored whether differences in flight capacity may be due to the effect of infection on glycogen and li
232                                         This may be due to the effect of viscous drag from surroundin
233       The difference between the two methods may be due to the effects of differences in milk homogen
234              It has been suggested that this may be due to the effects of pathology outside the brain
235 ons of space or a priori expectations (which may be due to the experimental setup, instructions, or d
236                                         This may be due to the fact that many studies investigated mo
237                                         This may be due to the fact that parboiling releases the oil.
238                                         This may be due to the fact that this subfamily contains an u
239                                         This may be due to the greater ability of lipoplexes relative
240                     We hypothesize that this may be due to the growth rates and kinetics of the virus
241  and that the heterogeneity of the responses may be due to the heterogeneity of the astrocyte-to-astr
242                                 This finding may be due to the high rate of disease relapse or the in
243            We assume here that this decrease may be due to the increase in adsorption of gaseous pest
244 wer MDA levels than non-meat proteins, which may be due to the increase in Grx1 and Trx1 expression a
245  targets during early HIV-1 infection, which may be due to the increased activation of these cells.
246                              This difference may be due to the indirect nature of the associations an
247 the pathophysiological contribution of IL-17 may be due to the induction of chemokines from the epith
248 iated with decreased population sizes, which may be due to the inflexible nature of their strict diet
249 reening for obesity has been reported, which may be due to the influence of quantitative trait loci b
250                                         This may be due to the large variation in cancer types or met
251            The failure of these technologies may be due to the limitations of the signals measured.
252                                         This may be due to the loss of other neurotransmitters, inclu
253 levels and decreased glycogen storage, which may be due to the manifestation of hepatic steatosis obs
254 essed the proliferation of LoVo cells, which may be due to the modulation on cell energy metabolism a
255 reduction in nitrate reductase activity that may be due to the overaccumulation of PMP in pdx3.
256                                         This may be due to the overuse of terminology that stresses r
257 e that the reduction in alternative splicing may be due to the partitioning of ancestral splice forms
258 ding of the cognate peptide to MHC-II, which may be due to the presence of a noncognate molecule.
259 d vulnerability to arrhythmia in AF patients may be due to the proarrythmic influence of alternans at
260                                         This may be due to the protective effects of the flavonoids e
261                                These results may be due to the reorganisation of skull structure in b
262                             This association may be due to the rich content of choline, especially ph
263  time in particular contexts, this evolution may be due to the selection of these attributes for incr
264                This negative impact on sleep may be due to the short-wavelength-enriched light emitte
265                                         This may be due to the small number of studies that were able
266 nces in USL composition were observed, which may be due to the small sample size and high variability
267                                         This may be due to the strong appeal of being able to visuali
268  Lee', ABA and JA decreased in winter, which may be due to the strong requirement of zeaxanthin for r
269                          These discrepancies may be due to the subtlety of brain alterations caused b
270 a(2 - q)[1 - beta(1 - q)tau](1/(1-q)), which may be due to the superstatistical nature of the occurre
271                       The observed increases may be due to the synergistic effect of new traits intro
272 se and another one at lower temperature that may be due to the transition from the HO to the LMAFM ph
273 by the energy release during friction, which may be due to the transition of electrons between the su
274 e suggest that the preference for navigation may be due to the triggering of automatic object finding
275 sence of prestin protein in DeltaIDR KI OHCs may be due to the unexpected splicing of the hybrid 17-1
276 oxicity of soluble metal-based nanomaterials may be due to the uptake of metals in both dissolved and
277                           Low matching rates may be due to the use of limited gene panels, restrictiv
278 n-level impacts between the 2 delivery areas may be due to the varied rotavirus vaccine coverage and
279            Some have noted that this decline may be due to the ways in which digital platforms direct
280                                         This may be due to the wide range of aroma qualities associat
281                                         This may be due to the wide variety of disease definitions.
282                                         This may be due to their interfacial properties as demonstrat
283 global MMP inhibitors have low efficacy that may be due to their lack of selectivity.
284 of plants at different concentrations, which may be due to their physico-chemical properties.
285                                         This may be due to transfer of high-avidity self-specific T c
286  recurrence of atrial tachyarrhythmia (ERAT) may be due to transient proarrhythmic factors.
287                            Treatment failure may be due to transplant complications or relapse.
288                                Although this may be due to under-reporting of bleaching events, it ma
289 ses in extracellular matrix transcripts that may be due to underlying profibrogenic changes in this g
290 Ps and reveal that apparent toxicity results may be due to unintended free wrapping agents such as po
291 ear what share of bariatric episode spending may be due to unnecessary variation and thus modifiable
292 uring the A(H3N2)-dominated 2017-2018 season may be due to vaccine virus adaptation to growth in eggs
293                                         This may be due to variability in dopaminergic damage between
294                                In part, this may be due to variable cortical neuron orientation relat
295         These differences in firing patterns may be due to variations in behavioral conditions or cou
296  characterized by large heterogeneity, which may be due to variations in childhood socioeconomic cond
297                                         This may be due to variations in serum 25-hydroxyvitamin D (2
298  indicating that the attentional blink to T2 may be due to very early T1-driven attentional capture.
299  suggest that the sequelae of LASV infection may be due to virus persistence resulting in systemic va
300  RUNX1 and suggests that sensitivity to THZ1 may be due to vulnerability conferred by the RUNX1 super

 
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