ライフサイエンスコーパス: nerve discharge

1 d A5 neurons (11 of 16; 69%) and sympathetic nerve discharge.

2 exin-1 channels to increase bladder afferent nerve discharge.

3 z rhythm of the slow wave of the sympathetic nerve discharge.

4 ially ventilated rat reduced resting phrenic nerve discharge.

5 ed heart rate slightly and inhibited phrenic nerve discharge.

6 activity but severely attenuated sympathetic nerve discharge.

7 of 5-hydroxytryptamine (5-HT) on mesenteric nerve discharge.

8 , thereby reflexively increasing sympathetic nerve discharge.

9 ) reduced the peak magnitude of the afferent nerve discharge and abolished the initial rise in intraj

10 in the medulla oblongata lowers sympathetic nerve discharge and blood pressure.

11 induced dose-dependent increases in afferent nerve discharge and intrajejunal pressure.

12 reparations to characterize bladder afferent nerve discharge and the mechanisms of urothelial ATP rel

13 n between the specific patterns of autonomic nerve discharges and cardiac arrhythmia during CHF.

14 e the frequency and coherence of sympathetic nerve discharges and mediate LH effects on sympathetic c

15 input during I greatly reduced CSy unit and nerve discharge, as evaluated by the no-inflation test.

16 educed blood pressure and lumbar sympathetic nerve discharges but also eliminated the inhibitory effe

17 ude of motor output; respiratory hypoglossal nerve discharge decreased and its frequency steadily dim

18 e was minimized (nitroprusside), sympathetic nerve discharge did not decrease but rather increased by

19 o evaluate whether it contributes to sensory nerve discharge during distention.

20 nchrony with co-active phrenic and abdominal nerve discharge during fictive vomiting.

21 th the early and later increases in afferent nerve discharge elicited by alpha,beta-methylene-ATP (30

22 Phrenic nerve discharge, end-tidal CO2, and arterial blood gases

23 dest and non-significant increase in phrenic nerve discharge evoked by L-glutamate.

24 The sensory nerve discharges evoked by distention were potentiated b

25 Sympathetic nerve discharge fell more rapidly but to the same nadir

26 on of bradykinin elicited increases in whole nerve discharge in a concentration-dependent manner.

27 nels lowered the CO(2) threshold for phrenic nerve discharge in anesthetized rats and decreased the v

28 owever, the effect of cocaine on sympathetic nerve discharge in humans is unknown.

29 tal signals, we measured respiratory-related nerve discharge in isolated brainstem preparations and a

30 uces elevated blood pressure and sympathetic nerve discharges in hypertension.

31 lamic presympathetic neurons and sympathetic nerve discharges in hypertension.

32 in the hypothalamus and augments sympathetic nerve discharges in normotensive, but not in hypertensiv

33 terial blood pressure and lumbar sympathetic nerve discharges in SHRs.

34 The patterns of nerve discharges, in particular the inspiratory or bipha

35 mained elevated for 60 min while sympathetic nerve discharge initially was unchanged and then decreas

36 .5 into the VLM increased integrated phrenic nerve discharge, inspiratory time and respiratory drive

37 Our findings indicate that bladder afferent nerve discharge is sensitive to elevated extracellular A

38 in the hypothalamus can increase sympathetic nerve discharges leading to hypertension.

39 These findings demonstrate that phrenic nerve discharge of rat pups, like that of kittens and pi

40 l rats but produced no effect on sympathetic nerve discharge owing to the destruction of bulbospinal

41 on the cardiorespiratory responses [phrenic nerve discharge (PND) and AP] caused by injecting dl-hom

42 SND, phrenic nerve discharge (PND) and putative sympathoexcitatory va

43 s neurons that burst just before the phrenic nerve discharge (PND) and rebound after inspiration (pre

44 h gabazine increased blood pressure, phrenic nerve discharge (PND) and the firing rate of ccRTN neuro

45 the RTN with muscimol eliminated the phrenic nerve discharge (PND) at rest, during hyperoxic hypercap

46 N with muscimol instantly eliminated phrenic nerve discharge (PND) but normal PND could usually be el

47 west intensity needed to inhibit the phrenic nerve discharge (PND).

48 SND in bursts synchronized with the phrenic nerve discharge (PND).

49 ted MAP, splanchnic SNA, and rate of phrenic nerve discharge (PND; P<0.05).

50 ontine noradrenergic neurons and sympathetic nerve discharge, possibly via the release of glutamate f

51 ship between stimulus intensity and cochlear nerve discharge rate (the rate-intensity function) in tw

52 are as follows: (1) increased carotid sinus nerve discharge rate to the respiratory centers of the b

53 lation of synaptic transmission and afferent nerve discharge reported here.

54 iring patterns and phase relative to phrenic nerve discharge, respiratory neurone types, including pr

55 ose-dependent increases in renal sympathetic nerve discharge (RSND), arterial blood pressure (BP), an

56 xia evokes long term facilitation of sensory nerve discharge (sLTF) of the carotid body in rodents ex

57 rogen has been shown to decrease sympathetic nerve discharge (SND) and BP.

58 etermine whether the increase in sympathetic nerve discharge (SND) caused by carotid chemoreceptor st

59 f cardiac-related postganglionic sympathetic nerve discharge (SND) in response to elevated blood pres

60 n generating the 10-Hz rhythm in sympathetic nerve discharge (SND) of baroreceptor-denervated, uretha

61 z rhythm in the inferior cardiac sympathetic nerve discharge (SND) of urethane-anesthetized cat.

62 iac-related and 10-Hz rhythms in sympathetic nerve discharge (SND) of urethane-anesthetized cats.

63 inferior cardiac postganglionic sympathetic nerve discharge (SND) relative to the systolic phase of

64 asal mucosa increased splanchnic sympathetic nerve discharge (SND), elevated arterial blood pressure

65 s in brain P(CO(2)) increase the sympathetic nerve discharge (SND).

66 9 mW) increased BP (15 mmHg) and sympathetic nerve discharge (SND; 64%).

67 ntranasal cocaine is to increase sympathetic nerve discharge to the skeletal muscle bed.

68 , we recorded blood pressure and sympathetic nerve discharge to the skeletal muscle vasculature using

69 36-KO mouse, respiratory-coupled sympathetic nerve discharge was attenuated and responses to chemorec

70 er lidocaine, blood pressure and sympathetic nerve discharge were unchanged.

71 ectral analysis of preganglionic sympathetic nerve discharges would reveal age-related differences in