ライフサイエンスコーパス: we do not know

1 h the help of another multi-component complex dynactin, but we do not know enough about factors that may affect the assem

2 However, we do not know exactly how cholesterol is distributed and wha

3 The role of proteins often changes during evolution, but we do not know how cells adapt when a protein is asked to par

4 Yet, we do not know how distinct endothelial and haematopoietic fa

5 a defining feature of twenty-first-century US politics, but we do not know how it relates to citizens' policy opinions.

6 olecular components that mediate kinetochore binding [5-7], we do not know how kinetochores physically interact with poly

7 Yet, we do not know how many people live on deltas and their expos

8 ite our strong understanding of the genetic basis of ADPKD, we do not know how most variants impact channel function.

9 While harmful effects of male age on sperm are well known, we do not know how much seminal fluid deteriorates in compari

10 Again mechanistic insight is missing such that we do not know how muscle senses its "inactivity status" or w

11 However, we do not know how reliably these measures predict the streng

12 ilitate cell shape change and buffer mechanical stress, but we do not know how reservoir dynamics are regulated.

13 However, we do not know how such intracomplex reactions depend on the

14 However, we do not know how symbionts affect host fitness when the lat

15 But we do not know how the activity of neurons in PFC collectivel

16 However, we do not know how the spatial distribution of PM(2.5) has ev

17 In particular, we do not know how the temporal organization of cell division

18 nt roles in nitrogen and carbon cycling in fresh waters but we do not know how these two processes compete for their comm

19 However, when quantum effects are involved, we do not know how to compute channel capacities.

20 Despite intense study of AMPs on model membranes, we do not know how well the mechanism of attack translates to

21 However, we do not know if and how much of the buttressing regions of

22 However, when walking at faster speeds, we do not know if and how these reflexes are changed.

23 exercise is not routinely undertaken in most HD units, and we do not know if home-based (HB) programs are as effective a

24 At present we do not know if published reports of guideline implementati

25 Currently, we do not know if the efficiency of transmission is very low

26 What we do not know is the ultimate mechanism of action; that is,

27 However, we do not know precise mechanisms on how lipid metabolic chan

28 formation on the CCT was collected at the end of the study, we do not know the exact timing for when each patient was reg

29 linking different cortical domains have been described, but we do not know the extent to which connections also exist bet

30 However, we do not know the heritability of the development of the neu

31 Currently, we do not know the importance of global protein localization

32 g host lipid metabolism and trafficking, but in most cases, we do not know the molecular mechanisms responsible or how sp

33 For example, we do not know the optimal sample preparation methods or imag

34 in patients undergoing assessment for epilepsy surgery, but we do not know their potential for providing helpful clinical

35 ing of the forces dynamically maintaining poles is limited: we do not know where and how quickly they act or their streng

36 e bears the load of chromosome movement far from poles, but we do not know where and how-physically and molecularly-this

37 In particular, we do not know whether alien species will continue to accumul

38 Yet, we do not know whether change in reward neurocircuitry as a f

39 H during major extinctions in the geological past, and thus we do not know whether human impacts are pruning the tree of

40 Additionally, we do not know whether previously observed correlations betwe

41 The study was limited as we do not know whether the additional patients presenting to

42 Furthermore, we do not know whether the conventional deep neural networks,

43 in urban conditions differ from those in rural conditions, we do not know whether these differences provide a combined r

44 ansduction genes or the same genes at different levels, but we do not know which molecular differences were most importan

45 genes or the same genes at different expression levels, but we do not know which molecular differences were most importan

46 ive method to enhance endogenous opioid signaling; however, we do not know which specific peptidase(s) to target.

47 As we do not know which specific proteins, if any, regulate BRM,

48 However, we do not know why some patients develop more QT prolongation

49 In particular, we do not know why these infections respond poorly to antibio

50 It has been suggested that we do not know within an order of magnitude the number of all