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1 ntrol, randomized, open-label study in HIV-1-infected antiretroviral therapy-naive adults (CD4+ >=50
2 nation eliminated reactivated latently HIV-1-infected cells in an ex vivo quantitative viral outgrowt
3 smaller reduction in the proportion of HIV-1-infected cells within LNs per year on therapy that was s
4 entions that do not rely on daily ART, HIV-1-infected children face lifelong medications to control i
6 In three independent experiments in HIV-1-infected humanized mice and one pivotal experiment in si
7 bneutralizing plasma concentrations in HIV-1-infected humanized mice but elicited CD4-binding site mu
8 1-18 alone fully suppressed viremia in HIV-1-infected humanized mice without selecting for resistant
9 proteome arrays from chronic untreated HIV-1-infected individuals using the classificatory random for
10 dependent t-tests were used to compare HIV-1-infected individuals within or outside an identified clu
11 ured polarized endocervical cells with HIV-1-infected lymphocyte-derived cells, we discovered endocer
14 llers (EC) represent a small subset of HIV-1-infected people that spontaneously control viral replica
15 ced pathogenic levels of VEGF-A within HSV-1-infected corneas, and CD4(+) cell depletion promoted rei
16 expression was transiently elevated in HSV-1-infected neurons, as well as in the presence of antivira
19 were observed in the colonic mucosa of 11G5-infected Apc(Min/+)/Atg16l1(DeltaIEC) mice vs 11G5-infec
22 onfirmed coronavirus disease 2019 (COVID-19)-infected patients and specificity using 1,204 samples su
26 outcomes of 17 previously healthy SARS-CoV-2-infected children and adolescents with an inflammatory p
28 Recent research suggests that SARS-CoV-2-infected individuals can be highly infectious while asym
32 ence of antibody sequences across SARS-CoV-2-infected patients, highlighting stereotyped naive respon
34 did not receive treatment, and 67% of HIV-2-infected mothers and 77% of their infants received ineff
37 ll functions in vivo Importantly, H5N1 (2:6)-infected mice displayed decreased numbers of tissue-resi
38 st T cell responses in the lungs, H5N1 (2:6)-infected mice showed inefficient and delayed viral clear
39 xpression by cytotoxic T cells in H5N1 (2:6)-infected mice, suggesting that delayed viral clearance o
43 ood meal is not known; however, an arbovirus-infected mosquito can inoculate extravascular host tissu
46 CD4(+) T-cells among influenza A/H3N2 and B-infected patients (p = 0.006 and 0.004 respectively).
49 yces dermatitidis and Histoplasma capsulatum-infected canine and feline lungs and airway epithelial c
52 CDRH2 that was isolated from the chronically-infected individual from whom the bent CDRH3 bNAbs were
53 ) and that natural killer (NK) cells in CL13-infected mice are reduced in numbers and have an immatur
55 n but lower capacity to respond to human CMV-infected cells; 2) term pregnancy dNK are not skewed tow
65 ased acquisition of Th cells in Coccidioides-infected lungs compared with vaccinated wild-type mice,
66 raze toxic endophyte (Epichloe coenophialum)-infected tall fescue (E+) and are hallmark signs of fesc
67 tive donor samples, but in 299 HIV confirmed-infected donor samples, 46 (15.4%; 95% confidence interv
69 oid assay in colonic crypts isolated from CR-infected mice revealed elevated levels of LRP5/6 and FZD
71 ly with brain homogenate from individual CWD-infected elk of various genotypes (LL132, LM132, or MM13
74 nocytes isolated in an acute phase from DENV-infected pediatric patients correlates with severe disea
76 he wild-type virus and found that the DUBmut-infected mice had a statistically significant reduction
79 ather rewires intracellular signaling in EBV-infected B cells that optimizes cell survival and prolif
82 ctor memory activated phenotype, whereas EBV-infected B cells expressed plasma cell differentiation m
83 aining lymph node of ectromelia virus (ECTV)-infected mice or at becoming effectors but proliferated
84 ment downregulated CCR7 on Y. enterocolitica-infected bone marrow-derived DCs and purified MLN DCs, w
86 t of Tir clustering on the viability of EPEC-infected intestinal epithelial cells (IECs) is unknown.
88 ophylaxis and bites from 12-15 P. falciparum-infected mosquitoes (CVac-chloroquine arm) at 3 monthly
89 participants underwent Plasmodium falciparum-infected mosquito challenge (controlled human malaria in
91 T cell control of B cell lymphoma in gammaHV-infected mice overlap with those necessary for control o
92 xpression of Wnt3a and Dvl3 in P. gingivalis-infected gingival tissues, and increases disease severit
94 e-resident memory T cells compared with H1N1-infected mice; however, despite the decreased number of
96 uld be an effective strategy to identify HBV-infected pregnant women at risk of perinatal transmissio
97 countries hinders the identification of HBV-infected pregnant women at risk of perinatal transmissio
99 Indeed, survival of autophagy-inhibited HCMV-infected monocytes was rescued when MLKL and RIPK3 were
102 virin) that enrolled chronic genotype 1a HCV-infected persons coinfected with suppressed HIV: 5 of 6
103 STAT-1 and STAT-4 phosphorylation in 29 HCV-infected LTx-recipients and 17 HCV-infected patients dur
105 strated that ATM expression is higher in HCV-infected hepatocytes and chronic HCV-infected liver biop
106 loping hepatocellular carcinoma (HCC) in HCV-infected patients who achieve sustained virological resp
107 2 (Day 7 in 4 of 5 participants), mean %HCV-infected cells = 1.0% (95% CI, 0.2%-1.7%) (P < .05 for c
109 ession of miR-181c promoted apoptosis of HCV-infected hepatocytes and can be inhibited by overexpress
110 ntaining CDRH3 is specific to particular HCV-infected individuals, we solved a crystal structure of t
111 amplifying partial viral genomes from 6 HeV-infected tissue samples from Syrian hamsters and 4 tissu
113 rinatally human immunodeficiency virus (HIV)-infected adolescents (PHIV+) compared to age-, sex-, eth
114 tudies in human immunodeficiency virus (HIV)-infected individuals suggest excess weight gain with int
115 luding 57 human immunodeficiency virus (HIV)-infected men with KS, multicentric Castleman disease, or
117 ll incidence rate of 0.74 cases per 1000 HIV-infected person-years (95% confidence interval, 0.43-1.0
119 replication, we studied blood pDCs in 29 HIV-infected participants who initiated antiretroviral thera
120 were performed on stool samples from 405 HIV-infected and 111 uninfected participants of the Copenhag
122 nowledge regarding mechanisms that allow HIV-infected cells to persist in individuals during combinat
123 anti-CD3 stimulation (P = .19) although HIV-infected participants had significantly higher CD8+ T-ce
125 to infinity) but declined rapidly among HIV-infected participants (half-life; 39 years; 24-108 years
127 ears old (age-ineligible for PCV13), and HIV-infected adults (18 to 40 years old) on antiretroviral t
129 the JCI, Chaillon and coworkers assessed HIV-infected cells from various anatomic compartments obtain
131 gen receptor (CAR) T cell to target both HIV-infected CD4(+) T cells and the FDC reservoir in vitro A
133 natomic CMV compartmentalization in five HIV-infected mothers and identify the possibility of congeni
134 Kidney transplant (KT) outcomes for HIV-infected (HIV+) persons are excellent, yet acute rejecti
135 at fractionated SP enriched for EVs from HIV-infected men induces potent transcriptional responses in
138 ession profiles, and immune responses in HIV-infected adult solid organ transplant (SOT) recipients o
139 ospinal fluid (CSF) by flow cytometry in HIV-infected adults with cryptococcal (n = 31) and noncrypto
142 cal, cOgnitive and VIsual performance in hiv-infected Children cohort showed significant cognitive im
143 Our results suggest that timing of TI in HIV-infected children has a long-term and measurable impact
146 (TAF) improves renal tubular markers in HIV-infected individuals but the impact on estimated glomeru
147 telomere erosion, and cell apoptosis in HIV-infected individuals on antiretroviral therapy (ART).
148 for bacterial respiratory infections in HIV-infected inpatients, but its value is limited as quantit
150 lymorphisms that were overrepresented in HIV-infected patients in Japan sharing the same HLA genotype
156 on is the main barrier to cure, and most HIV-infected cells reside in the gut, where distinct but unk
162 he abundance of ABCA1 in brain tissue of HIV-infected human subjects diagnosed with HAND was lower, a
164 leading to profound immunodeficiency of HIV-infected patients is still only partially understood.
165 igen-specific CD4 T cells in a cohort of HIV-infected persons starting antiretroviral treatment (ART)
167 humoral response to KSHV in a cohort of HIV-infected Zambian mothers without KS and identify potenti
169 we analyze the TCR repertoire of single HIV-infected cells harboring translation-competent proviruse
171 Median survival was 82 days among the HIV-infected HCC patients compared to 181 days among those w
172 antiretroviral therapy, children born to HIV-infected (HI) mothers are at a higher risk of early-life
175 tiretroviral therapy (ART) in vertically HIV-infected children limits the size of the virus reservoir
178 mprovements over time for both, but with HIV-infected patients having greater improvements (P-trends
181 recipitation experiments with lysates of HSV-infected neurons showed that UL16 and three other tegume
182 nd explain, at least in part, why every HSV1-infected person is not equally likely to develop HSV1-as
184 Pneumococcal ability to grow in the IAV-infected LRT depends on the nutrient-rich milieu with in
190 The NE gene expression observed in KSHV-infected cells was recapitulated in uninfected endotheli
191 and underlying mechanisms of HIV Tat in KSHV-infected endothelial cells undergoing viral lytic reacti
193 BEC and LEC, respectively) to show that KSHV-infected BECs progressively lose viral genome as they pr
195 ed infection rates in surviving S411A Kunjin-infected Culex quinquefasciatus mosquitoes were observed
198 FN-gamma binds to its receptor on Leishmania-infected macrophages, resulting in their activation, pro
199 ized the interactions of lymphocytes and LVS-infected macrophages using confocal microscopy and chara
203 ms, including circulating rings from malaria-infected patients and artemisinin-induced quiescent para
205 e individuals and one from a previously MARV-infected individual were selected for testing as HCDR3 l
214 azole are two top-scoring metabolites in Mtb-infected KCs and that acetylcholine is the top-scoring i
215 dings and show that in vivo treatment of Mtb-infected C57BL/6 mice with doramapimod, a p38 MAP-kinase
216 ysosomal system is a defining feature of Mtb-infected macrophages and suggest that this altered lysos
219 ian hamsters and 4 tissue samples from a NiV-infected African green monkey with viral loads as low as
221 nvestigate changes in blood chemistry in NiV-infected Syrian hamsters that survived or succumbed to d
222 tive control material, the evaluation of non-infected cells expressing coronavirus (SARS, MERS) spike
223 ells of the human body to eliminate pathogen-infected or tumorigenic cells (i.e., target cells).
230 thod for separating all stages of Plasmodium-infected erythrocytes through lysis and removal of uninf
233 harvested from 1-, 2-, 3-, and 6-month post-infected hamsters compared to uninfected liver tissues.
234 ile brain homogenates from symptomatic prion-infected mice are highly toxic to cultured neurons, exce
235 ions in gastric epithelial cells of H pylori-infected INS-GAS mice was assessed by whole-exome sequen
238 e quaking-induced conversion assays with RML-infected cell lysates, we observed a strong signal over
239 e (n = 4446), and full-term (n = 33 417) RSV-infected infants were matched to 424, 791, 8875, and 66
240 on 3-wk- (juvenile) and 8-wk-old (adult) RSV-infected C57BL/6 mice to investigate age-related differe
242 ecovered more apoptotic neutrophils from RSV-infected cultures (>40%) than from mock-infected culture
244 5 in the bronchoalveolar lavage fluid of RSV-infected mice, without increasing viral replication in t
245 Airway sensitization of naive mice with RSV-infected BMDCs exacerbate a live challenge with RSV infe
248 epletion was performed in five SHIV(SF162P3)-infected RMs treated with ART for 12 months and with pla
250 When infused into viremic simian HIV (SHIV)-infected rhesus macaques, there was a 21% difference in
251 n simian-human immunodeficiency virus (SHIV)-infected rhesus macaques, an earlier and sharper decline
254 al coordinates analysis (PCoA) clustered SIV-infected untreated animals away from healthy and treated
255 ith sustained viral control after ATI in SIV-infected RMs.IMPORTANCE While effective, antiretroviral
257 ication, both in vitro and in vivo in SIVmac-infected macaques, by upregulating antioxidant pathways
260 nt therapy (RRT) (secondary outcome) in STEC-infected children without HUS at initial presentation.
263 ulation by fitting compartmental susceptible-infected-susceptible (SIS) transmission models simultane
266 lity data to estimate a modified susceptible-infected-recovered (SIR) model in the United States.
267 sing a computer-generated list, T. trichiura-infected adolescents were randomly assigned to 7 treatme
270 tes to mount responses to cancer and virally-infected cells, dendritic cells must capture antigens pr
272 tance capable of destroying tumors and virus-infected cells through cytotoxicity and rapid cytokine p
277 (ART)-treated simian immunodeficiency virus-infected rhesus macaques (RMs) undergoing CD8alpha deple
278 a methodology for analyzing influenza virus-infected lung in vivo by two-photon imaging microscopy.
281 a three-dimensional culture AD model, virus-infected APP/PS1 mice and the brains of patients with AD
282 nal capacity to accelerate the loss of virus-infected cells through Fc gamma receptor (FcgammaR)-medi
283 ecially true for scheduled necropsy of virus-infected ferrets, a standard component in evaluation of
284 Therefore, EBV BGLF2 might protect virus-infected cells from the type I interferon response in ce
285 n lipopolysaccharide-treated or Sendai virus-infected U937 or THP-1 cells, the mNLS variant reduced T
286 (ifnar1(-/-)ifngr1(-/-)) bitten by the virus-infected AaVA-1-deficient mosquitoes present a lower vir
288 udy 2, n = 24) were inoculated with P. vivax-infected red blood cells to initiate infection, and were
290 were detected, yet only supernatant from VZV-infected cells induced amylin aggregation and, to a less
291 induce a sustained antiviral response in WHV-infected woodchucks; the identification of a baseline in
292 lions of competitive, sterile male Wolbachia-infected mosquitoes, and use of these males in a large-s
298 hypothesized that vaginal deposition of ZIKV-infected baboon semen would lead to maternal infection a
300 ructural and functional MRI showed that ZIKV-infected infant rhesus macaques had persistent enlargeme