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1 cently been unveiled and is mediated through 5-HT2 receptor.
2 ors, they overlap in their agonist action at 5-HT2 receptors.
3 involved in agonist binding in the serotonin 5-HT2 receptors.
4 can be induced by intermittent activation of 5-HT2 receptors.
5 thylamine psychedelic with high affinity for 5-HT(2) receptors.
6 rily through its actions at both 5-HT(1) and 5-HT(2) receptors.
7 rs, with some evidence of a weak affinity to 5-HT(2) receptors.
8 n-selective agonistic actions on 5-HT(1) and 5-HT(2) receptors.
9 ting that this effect of 5-HT is mediated by 5-HT(2) receptors.
10 and ketanserin, indicating the mediation by 5-HT(2) receptors.
11 (APDs) is their relatively high affinity for 5-HT(2) receptors.
12 5-HT) neurotransmission, acting primarily at 5-HT(2) receptors.
13 lower affinity than the corresponding 1a at 5-HT2 receptors, 3a and 3b had significantly greater aff
17 and developmental profile of 5-HT(1A-1D) and 5-HT2 receptors, 5-HT1A receptors, and the serotonin (5-
18 or the previously characterized 5-HT(1A) and 5-HT(2) receptors, a proportion of layer V pyramidal neu
20 ynaptic, slow-onset inhibition attributed to 5-HT2 receptor activation exciting GABAergic interneuron
21 ith a role in facilitating tonic inhibition, 5-HT2 receptor activation increased the frequency of spo
22 at require the following: Gq-protein-coupled 5-HT2 receptor activation, new BDNF synthesis, and MEK/E
25 y cells, taurine and AL34662, a non-specific 5-HT(2) receptor activator, produced a similar regulatio
27 iods of excitation, the relationship between 5-HT(2) receptor activity and self-sustained firing in h
28 s identified as a peripherally acting potent 5-HT(2) receptor agonist (EC(50) = 42.7 nM, E(max) = 89%
31 application of alpha-methyl 5-HT (a 5-HT(1) /5-HT(2) receptor agonist) and citalopram (a selective 5-
32 xyphenyl)-2-aminopropane (DOI, 50 microM), a 5-HT(2) receptor agonist, into the NACC produced greater
33 n of alpha-M-5-HT (100 microg kg(-1), LA), a 5-HT(2) receptor agonist, stimulated only two of the nin
35 t cholinergic interneurone firing, while the 5-HT2 receptor agonist alpha-methyl-5-HT (30 microm) mim
43 ioral toxicity associated with non-selective 5-HT2 receptor agonists, targeting the 5-HT2C receptor m
45 a-methylserotonin, a nonselective agonist of 5-HT2 receptors, also blocked polysynaptic responses but
46 potent full agonist with selectivity for the 5-HT2 receptor and is anticipated to serve as a useful t
47 s a useful tool in exploring the role of the 5-HT2 receptor and its effector system in controlling in
48 itro at dopamine D2 and serotonin 5-HT1a and 5-HT2 receptors and in vivo for their ability to antagon
49 vary cell lines expressing each of the human 5-HT2 receptors and in vivo in animal models of obesity.
56 5-HT were sensitive to 10 microm-ketanserin (5-HT(2) receptor antagonist) and strongly inhibited by a
62 ged increase in MPC phospho-ERK, whereas the 5-HT2 receptor antagonist ketanserin (3-[2-[4-(4-fluorob
65 TXA2 on SMC proliferation was abolished by a 5-HT2 receptor antagonist, LY281067, without affecting t
73 agonists with greater affinity for D2 DA and 5-HT2 receptors, blocked the development of locomotor se
74 he rhythmic activity of olivary neurones via 5-HT2 receptors by inhibition of the T-type calcium curr
75 ory control on cholinergic interneurones via 5-HT2 receptors, by suppressing the AHPs associated with
82 ation (LTF) is characterized by a persistent 5-HT2 receptor-dependent increase in respiratory motor o
86 s, supporting the physiological relevance of 5-HT2 receptor heterodimerization in vivo Accordingly, e
87 ogether, our results show that activation of 5-HT(2) receptors in PFC pyramidal neurons inhibits GABA
89 a group, despite equivalent upregulation of 5-HT(2) receptors in the lumbar ventral horn of lesioned
90 suggesting a relationship between IL-1RI and 5-HT2 receptors in the medial hypothalamus that is consi
93 demonstrate that GLYX-13 does not influence 5-HT2 receptor induced head twitch response or impulsivi
94 ) = 1 nM for both) and moderate affinity for 5-HT(2) receptors (K(i) = 73 and 75 nM, respectively).
95 splacement of radioligand from rat and human 5-HT2 receptors, making it one of the most potent halluc
97 owever, in experiment 3 we demonstrated that 5-HT(2)-receptor mediated phosphoinositide hydrolysis wa
99 serotonergic facilitation of TRPV1 function; 5-HT2 receptor-mediated facilitation was also inhibited
100 al motoneurones as a result of activation of 5-HT2 receptors, microinjection of 5-HT2 antagonists int
101 erotonin (5-Hydroxytryptamine, 5-HT) and the 5-HT2 receptor modulate cardiovascular and autonomic fun
102 herefore, supports a pronociceptive role for 5-HT2 receptors, most likely through modulation of 5-HT2
104 AADC cells, together with an upregulation of 5-HT2 receptors, offers a partial explanation of hyperre
106 requires mast cell-derived 5-HT to activate 5-HT(2) receptors on parasympathetic cholinergic neurons
107 om intestinal EC cells activated 5-HT(3) and 5-HT(2) receptors on vagal afferent fibers to mediate lu
108 ous, activation of postsynaptic 5-HT type 2 (5-HT(2)) receptors on hypoglossal (XII) motoneurons lead
109 on: indirect inhibition mediated through the 5-HT2 receptors on GABAergic interneurons and direct inh
110 ffects may also be linked to the presence of 5-HT2 receptors on the same groups of neurons in this re
111 preparations indicate that somato-dendritic 5-HT(2) receptors regulate the intrinsic excitability of
113 E(max) = 89%) with high selectivity for the 5-HT(2) receptors relative to other serotonergic recepto
114 d that 5-HT(2A) receptor activation with the 5-HT(2) receptor selective agonist (R)-2,5-dimethoxy-4-i
115 e suggest alternative compounds for studying 5-HT(2) receptors, should obtaining DOI for research bec
117 mine and its metabolite norfenfluramine with 5-HT(2) receptor subtypes and examined the expression of
118 BT, 5-APBT, and 6-APBT were full agonists at 5-HT(2) receptor subtypes as determined by calcium mobil
120 wo directly acting agonists for serotonergic 5-HT(2) receptor subtypes, quipazine and (+/-)-1-[2, 5]-
121 e indole nucleus of tryptamines that bind to 5-HT2 receptor subtypes and possess LSD-like behavioral
122 tention that within the VMN, both 5-HT1A and 5-HT2 receptor subtypes contribute to the modulation of
124 a greater affinity for 5-hydroxytryptamine2 (5-HT2) receptors than for D2 dopamine receptors; its eff
125 ults suggest that serotonin acts at separate 5-HT2 receptors to facilitate the acquisition and expres
126 neonatal 6-OHDA lesions involves coupling of 5-HT2 receptors to the ERK1/2/MAP Kinase cascade, a path
127 6-OHDA lesions result in a novel coupling of 5-HT2 receptors to the ERK1/2/MAP Kinase pathway, a sign
128 HT release or direct stimulation of striatal 5-HT2 receptors via the 5-HT2 agonist DOI, produced robu
129 q-coupled M3-muscarinic, thromboxane-A2, and 5-HT2 receptors was desensitized in airway smooth muscle
130 ut effect at 5-HT7 but has high affinity for 5-HT2 receptors, was also effective in attenuating the p
131 ulsions despite the fact that they have more 5-HT(2) receptors, we hypothesized that these mice may e
134 cal methods to demonstrate that IL-1beta and 5-HT2 receptors were present on the same neurons within
135 and more potent and efficacious activity at 5-HT(2) receptors, which fundamentally changed the in vi
136 ry LTF of the carotid body via activation of 5-HT(2) receptors, which involves a novel signalling mec
138 Most emphasis has been placed on 5-HT1A and 5-HT2 receptors, which inhibit and facilitate the behavi
139 ide evidence that activation of 5-HT(1A) and 5-HT(2) receptors within the medial hypothalamus exert d
140 whether pharmacological treatments targeting 5-HT2 receptors would alter the acquisition and expressi