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1 nance of remission of mild to moderate UC is 5-aminosalicylic acid.
2 eatment with infliximab (0.36; P = .0005) or 5-aminosalicylic acid (0.44; P < .0001) were associated
4 one group treated with the anti-inflammatory 5-aminosalicylic acid (5-ASA) and one group untreated),
5 lial oxygenation, but the PPAR-gamma agonist 5-aminosalicylic acid (5-ASA) functionally replaced Clos
6 y used inflammatory bowel disease (IBD) drug 5-aminosalicylic acid (5-ASA) has been attributed, in pa
8 ilable for inflammatory bowel disease (IBD), 5-aminosalicylic acid (5-ASA) is still the key medicatio
9 IL-1-mediated COX-2 gene expression whereas 5-aminosalicylic acid (5-ASA) or indomethacin had no eff
10 NSAIDs) 4-aminophenylacetic acid (4-APAA) or 5-aminosalicylic acid (5-ASA) with peptides, including a
11 and its metabolites, sulfapyridine (SP) and 5-aminosalicylic acid (5-ASA), on components of angiogen
12 dy two strategies for delivering mesalazine (5-aminosalicylic acid, 5-ASA) to the colon were compared
13 ferences in the in vitro response of PPK1 to 5-aminosalicylic acid, a known PPK1 inhibitor, and obser
14 atory drugs (OR, 0.1; 95% CI: 0.03-0.5), and 5-aminosalicylic acid agents (OR, 0.4; 95% CI: 0.2-0.9),
15 f sulfasalazine (SSZ) with its two moieties, 5-aminosalicylic acid (ASA) and sulfapyridine (SP), in p
19 Treatments for ulcerative colitis include 5-aminosalicylic acid drugs, steroids, and immunosuppres
22 ntal data to predict the release of the drug 5-aminosalicylic acid from polysaccharide-based coatings
26 ssification; and use of sulfasalazine, other 5-aminosalicylic acid preparations, prednisone, cyclospo
28 rotective factors colonoscopic surveillance, 5-Aminosalicylic Acid, thiopurines, and smoking was mode
29 fective treatment for mild to moderate UC is 5-aminosalicylic acid, whereas moderate to severe UC can
30 e sensitization of Eu3+ is accomplished with 5-aminosalicylic acid, which provides energy transfer fo