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1 an ABH-glycan microarray, we quantified ABO-A antigen-subtype (A-subtype)-specific IgM and IgG in 53
2 firmed that alpha1,2-fucose-containing H and A antigens of the HBGA family were recognized by CNV.
4 s produced a vigorous memory response to B10.A antigens, suggesting immune activation was not inhibit
7 ucing terminus of the blood type-determining A-antigen and as the initial point of attachment to the
10 develop alloantibodies to human glycophorin A antigen, we found reduced in vitro and in vivo Treg-su
12 (HMP) strategies, we demonstrate blood group A antigen loss of approximately 80% in as little as 2 h
13 y, we identified oligosaccharide blood group A antigen tetraose 6 (BGA6) as a biologically relevant G
18 plautii to enzymatically convert blood group A antigens from the renal vasculature of human kidneys t
20 uman alloreactive CD8(+) T cells against HLA A antigens were MHC restricted and demonstrated a public
22 including transfusion, small variability in A antigen levels, and rare ABO*A2.06 and ABO*A2.16 seque
23 validated with both SARS-CoV-2 and influenza A antigens from clinical nasopharyngeal swab samples (PC
24 ome coronavirus 2 (SARS-CoV-2) and influenza A antigens in nasopharyngeal swab samples at diagnostica
25 (+) or CD8(+) T cells to conserved influenza A antigens in peripheral blood did not affect nasopharyn
27 eg, A(2)) subtype donors, which express less A antigen, can be safely transplanted into group B recip
32 e immunodominant T cell-defined MART-1/Melan-A antigen and downregulation of the TAP-1 gene in a recu
37 chemical studies demonstrated high levels of A antigen on various glycoproteins (GPs) from high-expre
38 elets contained higher than normal levels of A antigen were subdivided into 2 groups, designated Type
41 A panel of 17 Salmonella Typhi and Paratyphi A antigens was purified and used to determine antigen-sp
42 Histo-blood group A transferase produces A antigens and transfers GalNAc to the acceptor substrat
43 and VA387 (GII-4), which recognize the same A antigen but differ in that NV is unable to bind to the
44 tibodies specific for A-II glycans, the sole A-antigen subtype in vascular endothelium, from other an
48 r of cells expressing alpha-gustducin or the A antigen in regenerated taste buds; in the CTX animals
49 he porcine and bovine mucins, suggesting the A antigen as a possible factor for cross-species transmi
51 ral studies indicate that 5-FdU binds in the A'-antigen-binding pocket of MR1, positioning the deoxyr
52 bserved a new pattern of binding to the type A antigen which is distinct from that of the P[II] RVs.
54 ase 109 (GH109) that is active on blood type A-antigen, along with a new subfamily of glycoside hydro
56 erotype 8 vector that expresses an anti-VEGF-A antigen-binding fragment, which provides potential for