ライフサイエンスコーパス: A-cell

1 A cell (325 muL) with distant side walls was formed by p

2 A cell adhesion assay was used to determine the proporti

3 A cell adhesion assay, a cell-binding assay, receptor bl

4 A cell autonomous mechanism with sustained cAMP/PKA acti

5 A cell autonomous molecular oscillator, consisting of in

6 A cell autonomous role for Klf6 in endoderm and hepatic

7 A cell becomes senescent when one or more of its telomer

8 A cell can be thought of as a highly sophisticated micro

9 A cell cluster at the anterior tip of planarian head bla

10 A cell contains numerous protein molecules.

11 A cell culture model demonstrates that cells on chemFN a

12 A cell culture model of freshly isolated corneal stromal

13 A cell culture model of freshly isolated rabbit corneal

14 A cell culture model was used to investigate the role of

15 A cell culture study evaluated the effect of MC on MMP p

16 A cell culture system consisting of mouse S17 stromal ce

17 A cell culture system has been established to detect the

18 A cell cycle analysis revealed that the cellular toxicit

19 A cell cycle regulatory module in islets predicts diabet

20 A cell death assay revealed that apoptotic-related DNA f

21 A cell death detection ELISA kit was used to evaluate HR

22 A cell delivery strategy was investigated that was hypot

23 A cell distribution experiment showed that flk-1:fas fus

24 A cell dose escalation was conducted, starting at 10(7)

25 A cell envelope-associated proteinase gene (prtH) was id

26 A cell fraction separated by density gradient centrifuga

27 A cell fraction that would today be termed "the nuclear

28 A cell fractionation experiment using cisplatin-treated

29 A cell free system was developed, and a succession of co

30 A cell fusion assay was used to assess functional activi

31 A cell fusion complementation assay in fibroblasts from

32 A cell growth inhibition assay was used to measure varia

33 A cell is a minifactory in which structures and molecule

34 A cell kinetics model is developed to describe the evolu

35 A cell line (410.4) derived from a mouse mammary adenoca

36 A cell line (AmE-711) has been established that is compo

37 A cell line (EKT2) was established from an affected kidn

38 A cell line derived from one such tumor expresses vGPCR

39 A cell line expressing the membrane-bound ADAM15 without

40 A cell line lacking functional copies of both NDUFA9 all

41 A cell line representative of human high-grade serous ov

42 A cell line selected for stable resistance to geldanamyc

43 A cell line that is adherent in serum-containing media a

44 A cell line that is established from lung metastases rel

45 A cell line was cloned from a single progenitor cell.

46 A cell line with a strong Trop-2 expression (CVX-8) was

47 A cell line, KG-1, behaves similarly.

48 A cell maintains normal centrosome numbers by coupling c

49 A cell may arrest division via checkpoint activation to

50 A cell membrane biosensor was constructed by using the c

51 A cell membrane can be considered a liquid-phase plane i

52 A cell membrane protein named ATP-binding cassette trans

53 A cell membrane-permeable form of 2',3'-cAMP and 3'-AMP

54 A cell membrane-permeable fusion peptide derived from th

55 A cell metabolic labeling experiment can be completed in

56 A cell permeant peptide derived from this sequence displ

57 A cell permeant phosphopeptide mimetic of Niban (NiPp) w

58 A cell plated on a two-dimensional substrate forms adhes

59 A cell population with progenitor-like phenotype (CD45-C

60 A cell printer that can print gels, single cells and cel

61 A cell proliferation assay revealed potent functional ac

62 A cell proliferation assay revealed that Notch signaling

63 A cell regulates the number, size, and kind of each orga

64 A cell responds to a chemotactic signal by activating ac

65 A cell slurry containing both cell types added to silico

66 A cell surface biotinylation assay and live cell confoca

67 A cell surface cytokine CD40 ligand (CD40L) enhances TF

68 A cell surface fraction from S. epidermidis 0-47 grown i

69 A cell suspension model was designed to validate the imp

70 A cell synchronization protocol was established in which

71 A cell system in which the impact on transcription can b

72 A cell therapeutic approach with a tissue-engineered bio

73 A cell tracing experiment was included to observe the pr

74 A cell type expressing this receptor is the osteoblast,

75 A cell type-centric interface allows the identification

76 A cell undergoing mitosis is presented with a potentiall

77 A cell viability assay showed potentiation of ATP-induce

78 A cell viability assay showed that alpha-tocopheryl succ

79 A cell viability assay showed that IC(50)s for human col

80 A cell viability assay was used to determine the sensiti

81 A cell viability screen identified Abeta(31-42) as the m

82 A cell viability test was conducted on MgSO(4) solutions

83 A cell voltage of 1.035V and power density of 3.154mWcm(

84 A cell wall-associated beta-glucosidase activity that re

85 A cell's ability to regulate gene transcription depends

86 A cell's ability to tolerate DNA damage is directly conn

87 A cell's decision to growth arrest, apoptose, or differe

88 A cell's decision to undergo meiosis is regulated by mul

89 A cell's epigenome arises from interactions among regula

90 A cell's perception of morphogen concentration is though

91 A cell-autonomous RelA activity amplified the yield of C

92 A cell-based assay confirmed that all 4 patients, but no

93 A cell-based assay shows that nonhomologous end joining

94 A cell-based assay was developed by fusing a green fluor

95 A cell-based bioassay was generated that detects SMN2 pr

96 A cell-based biological pacemaker is based on the differ

97 A cell-based fluorometric assay was used to measure intr

98 A cell-based functional assay suggests that S100A6 modul

99 A cell-based high-throughput screen to identify small mo

100 A cell-based method for the detection of nanomolar conce

101 A cell-based model preserving the evolving molecular pro

102 A cell-based mortality model was developed and refined.

103 A cell-based MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphe

104 A cell-based pharmacodynamic explanation was suggested b

105 A cell-based phenotypic screen for inhibitors of biofilm

106 A cell-based proteomic study validates the ability of SA

107 A cell-based readout system was created to functionally

108 A cell-based receptor trafficking assay was used to furt

109 A cell-based reglucosylation assay was used to investiga

110 A cell-based reporter assay linking LasR function with b

111 A cell-based screen of chemical libraries was carried ou

112 A cell-based screen performed in our laboratories identi

113 A cell-based standard was developed to compare the COBAS

114 A cell-based, drug-screening assay is also described, wh

115 A cell-based, functional high-throughput screen of appro

116 A cell-binding assay and fluorescence cell staining were

117 A cell-binding assay was performed to quantify the abili

118 A cell-cell fusion assay revealed that representative en

119 A cell-cell signalling system mediated by the fatty acid

120 A cell-configuration system was built utilizing V(2)O(5)

121 A cell-coupling analysis for Pinus sylvestris showed hig

122 A cell-culture system was developed that allows identifi

123 A cell-culture-based system was designed previously to m

124 A cell-cycle-dependent shuffling of multiple centromeric

125 A cell-free (axenic) culture system was developed, which

126 A cell-free assay of prion protein misfolding was used t

127 A cell-free assay showed that the R143A mutant partially

128 A cell-free assay that reproduces transport between the

129 A cell-free assay was developed for the orphan nuclear r

130 A cell-free cellulosomal GH25 lysozyme may provide a bac

131 A cell-free conversion assay was used to measure the int

132 A cell-free conversion reaction approximating physiologi

133 A cell-free ECM was prepared from cultured femoral marro

134 A cell-free in vitro translation assay containing human

135 A cell-free membrane-fusion system demonstrates that eng

136 A cell-free mRNA decay system reconstitutes TPA-induced

137 A cell-free plasma gap representing the ECG could be ima

138 A cell-free Ser(20) phosphorylation site assay was used

139 A cell-free system depleted of intersectin failed to sup

140 A cell-free system derived from Xenopus eggs has been es

141 A cell-free system that reconstructs these events has re

142 A cell-free system was used to evaluate the activation o

143 A cell-free system was used.

144 A cell-free translation-based small molecule screen was

145 A cell-free, mate-paired library provided single DNA mol

146 A cell-impermeable peptide PC inhibitor blocks the appea

147 A cell-intrinsic timer helps control when rodent oligode

148 A cell-matrix composite was constructed by applying fibr

149 A cell-membrane fraction isolated from Escherichia coli

150 A cell-penetrant peptide that disrupts this interaction

151 A cell-penetrating pepducin, X1/2pal-i3, which targets t

152 A cell-penetrating peptide (CPP)-morpholino oligonucleot

153 A cell-penetrating peptide containing this sequence comp

154 A cell-penetrating peptide-modified fusogenic liposomal

155 A cell-penetrating, anti-DNA, lupus autoantibody, 3E10,

156 A cell-permeable alpha7 helix peptide competitively inhi

157 A cell-permeable decoy peptide corresponding to the same

158 A cell-permeable ester of alpha-ketoglutarate reversed t

159 A cell-permeable NBD peptide blocked association of NEMO

160 A cell-permeable peptide aminofluorocoumarin substrate d

161 A cell-permeable peptide conjugated to the Akt-binding d

162 A cell-permeable peptide corresponding to this region di

163 A cell-permeable peptide inhibitor of GSK-3beta, and lit

164 A cell-permeable peptide inhibitor specific for the zeta

165 A cell-permeable peptide that contained a polyproline se

166 A cell-permeable peptide that selectively disrupted NF-k

167 A cell-permeable peptide that uncouples G alpha(s) from

168 A cell-permeable PKCzeta pseudosubstrate peptide inhibit

169 A cell-permeable TIRAP blocking peptide attenuated LPS-i

170 A cell-permeant Ca(2+) chelator reduced the CDDO-induced

171 A cell-permeant inhibitor based on the PKC-binding seque

172 A cell-permeant inhibitor of inositol trisphosphate rece

173 A cell-permeant peptide that blocks binding of PAK to PI

174 A cell-permeant TAT-tagged peptide encompassing the seco

175 A cell-permeant version of the peptide encompassing amin

176 A cell-permeant, bioactivatable Ins(3,4,5,6)P4 analog el

177 A cell-permeant, phosphoinositide-binding peptide attenu

178 A cell-population nucleosome map requires two observable

179 A cell-proliferation assay was used to determine the res

180 A cell-rounding phenotype associated with the transient

181 A cell-scaled microbead system was used to analyze the f

182 A cell-sparse gap, observed within layer III, was not fi

183 A cell-specific regulation of redox state thus balances

184 A cell-specific targeting peptide, RGD-4C which binds al

185 A cell-surface marker was identified by comparing the mR

186 A cell-surface protein of approximately 82 kd recognized

187 A cell-to-surface attachment assay shows that addition o

188 A cell-type-specific deletion of Tbx1 in osteochondro-pr

189 A cell-wall/membrane (CW/M) antigen from the vaccine str

190 A cells contain heavy chain DJ rearrangements.

191 rs that drive gene changes in 4 settings: 1) A cell line expressing or lacking single TF, 2) Breast t

192 Extracts of hPMS2-deficient HEC-1-A cells, which express hMLH1, hMLH3, and hPMS1, are only

193 nd cell death caused by tBHP in neuronal N(2)A cells (EC(50) in nm range).

194 ely bound to TNFR1 when overexpressed in N(2)A cells, with DENN/MADD abrogating TNFR1 binding to TRAD

195 G hepatocellular carcinoma cell lines than A/A cell lines.

196 uced NFkappaB activation is delayed in MEF(A/A) cells and in an MCF-7 cell line that is stably transf

197 d that this inhibition is prevented in MEF(A/A) cells in which the phosphorylation site, Ser-51 in th

198 Their islets show marked alpha (A)-cell hyperplasia, suggesting a possible defect in glu

199 ned the proliferation dynamics of B1, C, and A cells in vivo.

200 onstrated very few differences between F and A cells at the RNA level either under baseline condition

201 of B1 cells, C cells divide three times and A cells once, possibly twice.

202 until they encounter and attach to anterior A cells.

203 (P) compartment cells and induces Anterior (A) cells to create a developmental organizer at the AP c

204 experimental models were used: model A, B10.A cells presensitized to B6 (a anti-b cells) were inject

205 long intervals, we found a richer behavior: A cell swimming in the dark stochastically switches betw

206 tative Hh receptor expressed specifically by A cells, reduced Fu*.

207 Following delivery of E6 siRNA to C33-A cells using oligofectamine or His6 reducible polymers,

208 d late erythroblast F cells and non-F cells (A cells) from the human HUDEP2 erythroid cell line and p

209 t Madin-Darby canine kidney, HT29, and Clone A cells and from solutions of purified spectrin and alph

210 a dominant negative RhoA (N19RhoA) in clone A cells inhibited alpha6beta4-dependent membrane rufflin

211 d its enhanced detergent solubility in Clone A cells that harbor a defective alpha-catenin.

212 or hydrolysis of the carboxyl ester in clone A cells, although in primary human hepatocytes, both Cat

213 he alpha6beta4-mediated interaction of clone A cells with laminin promoted the translocation of RhoA

214 (CP-B, CP-C, CP-D), and nondysplastic BE (CP-A) cells were incubated with pharmacologic inhibitors or

215 After C cells, Doublecortin(+) A cells were the second-most common dividing cell type i

216 Ege A overexpressing and ege A(-) cells display similar phenotypes, suggesting that a

217 n of a fully functional cAR1-YFP enables ege A(-) cells to form loose aggregates, but cAR1-YFP/ege A(

218 s to form loose aggregates, but cAR1-YFP/ege A(-) cells are still unable to express GP80, suggesting

219 the present study the association of ephrin-A cell surface ligands and EphA receptor tyrosine kinase

220 ly ET(A)(-/-) --> (+/+) chimeric embryos, ET(A)(-/-) cells are excluded from the caudoventral aspects

221 cardiac outflow tract and great vessels, ET(A)(-/-) cells are excluded from the walls of the develop

222 HbF together with adult hemoglobin (HbA) (F+A+ cells).

223 sults were obtained upon correction of an FA-A cell line, with a reciprocal increase in the half-life

224 FA-A cells also showed defects in both cell-cell and cell-m

225 toration of alphaIISp levels to normal in FA-A cells corrects these deficiencies.

226 Knocking down mu-calpain by siRNA in FA-A cells restored levels of alphaIISp to normal and rever

227 entation group D) cells but not in mutant FA-A cells.

228 was able to rescue the adhesion defect of FA-A cells.

229 ion of FANCA readily restored adhesion of FA-A cells.

230 Fanconi anemia, complementation group A (FA-A) cells, there is a deficiency in these incisions.

231 Fanconi anemia, complementation group A (FA-A) cells, where alphaIISp levels are 35-40% of normal, I

232 lls (AA4.1(+)/natural killer 1.1(-) Fraction A cells) and cells capable of generating pro-B colonies

233 Wnt1-Cre-activated diphtheria toxin fragment-A cell-killing system was employed.

234 A expression in inflammatory cells and FXIII-A(+) cell numbers were significantly increased in NP.

235 , CD19+, CD3+, CD34+, CD33+, and glycophorin A+ cells and granulocytes were isolated from the periphe

236 A special target of the granzyme A cell death pathway is an endoplasmic reticulum-associa

237 on group O or B cells of infants or on group A cells of adults.

238 syndrome but not Xeroderma pigmentosum group A cells providing evidence that ssDNA regions generated

239 In Group A, cell-free polymerase assays confirm that mutations in

240 HEK-A cells were used to compare achievable cell signals for

241 ranules in the hyperplastic and hypertrophic A-cells, along with morphological evidence of high rates

242 yed-type hypersensitivity was dependent on I-A+ cells and was genetically restricted.

243 bly caused by a reduction in Slit1 ligand in A cells.

244 Notably, prolonged stimulation of R-/IR-A cells with insulin, but not with IGF-II, targeted the

245 Using R-/IR-A cells, we discovered that insulin evoked significant I

246 for the production of glucagon in the islet A-cells.

247 alpha-linked sialic acid (alpha-SA) and JAM-A cell-surface receptors.

248 replication that involves the protein kinase A cell-signaling pathway and a possible down-regulation

249 and viability of TNBC cells, but not Luminal A cells, and (3) is associated with a poor prognosis in

250 In MAT A: cells responding to alpha-factor, we found that Dnf1,

251 to Balb/C mice inoculated with syngenic meth/A cells demonstrated statistically significant dose-depe

252 lished in mechanically stimulated mouse MRTF-A(-/-) cells or upon inhibition of CREB-binding protein

253 ecific feature of MYCN-amplified cells (MYCN-A cells) that represent a particularly aggressive subcla

254 ed in motility (myosin light chain 1, myosin A), cell morphology (PhIL1), and host cell invasion (api

255 iate progenitors (C cells), and neuroblasts (A cells) in the V-SVZ and the number of times these cell

256 ing (type C) cells, chains of young neurons (A cells), and blood vessels.

257 under conditions that inhibit the growth of A cells and normal hepatocytes.

258 A 30% cell suspension of A-cells or B-cells was used for haemagglutination on the

259 PC-A cells were capable of further differentiation to periv

260 PC-A cells were not osteogenic or adipogenic under standard

261 s (Y iB), (iv) stable PR-B plus inducible PR-A cells (B iA), and (v) stable PR-A plus inducible PR-B

262 e control cells (Y iNull), (ii) inducible PR-A cells (Y iA), (iii) inducible PR-B cells (Y iB), (iv)

263 comB has a cell-autonomous role in prestalk A cell differentiation.

264 Activated CD11b+SRA+ (scavenger receptor A) cells were found in blood and cervical lymph node and

265 As expected, acc1(S/A) cells exhibit a reduction in acetate/acetyl-CoA avail

266 In ngn3(-/-) mice, glucagon secreting A-cells, somatostatin secreting D-cells, and gastrin sec

267 In contrast, all serotype A cells harvested from brain tissue bound the same O-ace

268 erall rate of accumulation of C3 on serotype A cells that was strongly correlated with increasing ant

269 In phagocytosis assays, using serotype A cells and resident murine peritoneal macrophages, rim-

270 the use of cell culture models, such as SVG-A cells (SVGAs), an immortalized, mixed population of gl

271 tributes to CAG*CTG tract instability in SVG-A cells.

272 In contrast, the A cell population expands precociously, mostly fails to

273 n that localizes to the cell membrane in the A cell and physically interacts with the Notch receptor.

274 The A cells play a crucial role in night-time (scotopic) vis

275 boundaries, organises the pattern of all the A cells.

276 e present study we aimed to characterize the A cells in human retina.

277 tentials and on the postsynaptic IPSP in the A-cell was occluded by previous paired (but not unpaired

278 acellular Ca2+ in the sensitization of toxin A/cell-surface interactions.

279 Type A cells appear to correspond to type II neurones of Namb

280 Type A cells were readily detected only in animals in which t

281 over the overall numerically dominating Type A cells, which in turn leads to an acceleration of the o

282 e number of action potentials evoked in type A cells by AF test stimulation and a concomitant increas

283 zole, seven of ten breast cancer lines (type A cells) arrested in mitosis, whereas the other three (t

284 e conductances of type B cells, but not type A cells.

285 The somata of type A cells were variable in shape (20 x 12 micrometer) whil

286 may be necessary for the production of type A cells.

287 interactions among the more stationary Type A cells, but unexpectedly accelerates again when these A

288 driven synaptically by AF stimulation (type A cells) were concentrated in the deep half of the GCL a

289 ge specific and differ in the number of UCYN-A cells involved.

290 repair-deficient xeroderma pigmentosum (XP)-A cells that stably express photoproduct-specific photol

291 XP-A cells were unable to repair these oxidative DNA base l

292 mplex formation was similar in normal and XP-A cells, but was reduced in CS-B cells.

293 PCNA relocated as compared to normal and XP-A cells.

294 ulation of active p53 in repair-deficient XP-A cells occurred despite the lack of DNA strand break in

295 icient PCNA complex formation observed in XP-A cells following oxidative damage suggests that formati

296 e repair of oxidative DNA base lesions in XP-A cells in vivo and in vitro.

297 significantly following UV irradiation in XP-A cells in which sumoylation of XPC does not occur.

298 However, unlike in XP-A cells, the absence of pol eta expression resulted in a

299 zation observed was comparable to that of XP-A cells deficient in nucleotide excision repair, a recog

300 in nucleotide excision repair-deficient (XP-A) cells but were not found in repair-proficient cells.