ライフサイエンスコーパス: AA

1 AA also increased 5-hydroxymethylcytosine (5hmC) levels

2 AA amyloidosis is associated with higher risk of mortali

3 AA coils were not used in attenuation correction process

4 AA content reached the maximum value at 10 min, correspo

5 AA following PCI were infrequent but were associated wit

6 AA had more prolonged length of hospital stays (11.1 +/-

7 AA included (1) >=3 s electrical pause or asystole; (2)

8 AA measured by adipose tissue compartment volumes-abdomi

9 AA subjects (n = 270) were initially studied between 199

10 AA were 25% less likely (95% confidence interval, 0.60-0

11 AA-bound crystal structures of PKM2 displayed distinctiv

12 AA/AG genotype carriers were 5.7 and 2.8 more likely to

13 AAs comprised 36.2% of the UAB cohort compared with only

14 AAs have worse outcomes compared to whites while facing

15 AAs present with more advanced disease and eventually ha

16 h AL (AHR = 6.2, 95% CI 4.6-8.5; p < 0.001), AA (AHR = 2.3, 95% CI 1.4-3.7; p = 0.001), or AM (AHR =

17 fidence interval [CI], 1.18, 2.60, P = .01), AA identity (aOR, 1.67; 95% CI, 1.02, 2.72, P = .04), pe

18 (a) elevated placental HIF-1alpha by AT(1) -AA or LIGHT upregulates CD73 and ADORA2B expression and

19 pe 1 receptor agonistic autoantibody (AT(1) -AA)-induced mouse model of PE.

20 he genetic shifts of seed composition for 18 AAs in 13 soybean genotypes released between 1980 and 20

21 2-AA-labeled glycans have increased mass spectrometric sen

22 2-Aminobenzoic acid (2-AA) is widely used as a labeling reagent to derivatize r

23 Coupling 2-AA to glycans results in significant increases of glycan

24 Moreover, we observed robust increases in 2-AA-LPC, 2-AA-LPE, and their downstream 12-LOX oxidation

25 calcium-stimulated production of 2-AA-LPC, 2-AA-LPE, and 12-HETE-lysophospholipids in mouse platelets

26 we observed robust increases in 2-AA-LPC, 2-AA-LPE, and their downstream 12-LOX oxidation products,

27 of 2-arachidonoyl-lysophosphatidylcholine (2-AA-LPC) and 2-arachidonoyl-lysophosphatidylethanolamine

28 arachidonoyl-lysophosphatidylethanolamine (2-AA-LPE).

29 Here, we report use of 2-AA for labeling N-glycans on a MALDI target through nonr

30 eased the calcium-stimulated production of 2-AA-LPC, 2-AA-LPE, and 12-HETE-lysophospholipids in mouse

31 This 2-AA on-MALDI-target glycan derivatization eliminates tedi

32 beled ((14)C-AA) and (13)C-labeled ((13)C(3)-AA) materials.

33 ectal tumor and nontumor samples (30 CRC; 32 AAs) from patients in Spain.

34 ncestry (EA) with SVs in comparison with 408 AA and 262 controls, respectively.

35 A nested case-control study of 56 AA subjects was conducted.

36 years, 58% female) included 45,645 EA, 7,597 AA, and 2,493 HE individuals.

37 ounts of DHA in infant formula up to ~0.64%, AA contents should at least equal the DHA contents.

38 with the FAAH C385A polymorphism (20/30 AC + AA).

39 for the FAAH C385A polymorphism (36/79 AC + AA).

40 Methanol carbonylation to acetic acid (AA) is a large-scale commodity chemical production proce

41 uba wax (CW)-based oleogel with adipic acid (AA) was prepared and its potential for application in th

42 Adipic acid (AA), 6-aminocaproic acid (6-ACA) and 1,6-hexamethylenedi

43 They were composed by adipic acid (AA), diethylene glycol (DEG) and isophthalic acid (IPA)

44 osition of beer wort in terms of amino acid (AA) content affects the final product quality, once it i

45 ttle data are available on their amino acid (AA) ileal digestibility.

46 support T cell growth even under amino acid (AA) replete conditions.

47 ed DQ haplotypes, converted into amino acid (AA) residues, and assessed for their associations with T

48 reduction of its precursor arachidonic acid (AA) in free form.

49 luding linoleic acid (LA), arachidonic acid (AA), docosahexaenoic acid (DHA), or eicosapentaenoic aci

50 aturated fatty acid (PUFA) arachidonic acid (AA), which entails risk for developing IBD.

51 ed to also include omega-6 arachidonic acid (AA; 20:4n-6).

52 + citric acid (CA), pectin + ascorbic acid (AA) and pectin alone after osmotic treatment and were dr

53 al phenolic content (TPC) and ascorbic acid (AA) as well as colour changes during IMCD processing.

54 ntioxidant activity (TAA) and ascorbic acid (AA) decreased during cold storage, all these changes bei

55 HCSVs are prepared by loading ascorbic acid (AA) in the core and poly(lactic-co-glycolic acid) shell

56 (ADD) for direct detection of ascorbic acid (AA) via FRET quenched.

57 measured by the detection of ascorbic acid (AA), dopamine (DA), uric acid (UA), and serotonin (5-HT)

58 rape-seed) in comparison with ascorbic acid (AA), sulfur dioxide (SO(2)) and bentonite.

59 hile supplemented with limiting amino acids (AA) results in decreased growth.

60 Nitrogen can be generated from amino acids (AAs) and utilized for biosynthetic processes through tra

61 N) isotope values of individual amino acids (AAs) in endogenous stores (e.g. muscle) and in egg yolk

62 tion of delta(15)N values of 14 amino acids (AAs), accounting for ca. 75% of plant protein and collag

63 of essential and non-essential amino acids (AAs).

64 Acrylamide (AA) formation during coffee roasting happens rapidly, re

65 ation and partial degradation of acrylamide (AA), asparagine and low molecular weight sugars were eva

66 ) and to assess the evolution of acrylamide (AA), trigonelline, nicotinic acid and caffeoylquinic aci

67 lics together with the antioxidant activity (AA) in almonds.

68 relationship with the antioxidant activity (AA), resveratrol, and naringin in the fruit from SLP.

69 ase (1.8-4.8 times) in antioxidant activity (AA).

70 Da) showed the highest antioxidant activity (AA); therefore, selected and loaded into nanoliposomes.

71 wed by those with baseline advanced adenoma (AA) (21.9%; 95% CI 15.7-28.1).

72 vity for the detection of advanced adenomas (AAs).

73 l glycemia on offspring abdominal adiposity (AA) in infancy and early childhood.

74 .4) after AL, 14.1% (95% CI 10.8-18.3) after AA, 7.4% (95% CI 6.7-8.1) after AM, and 4.5% (95% CI 3.9

75 linear molecule, N-alanyl-aminoacetone (Ala-AA), also bound and activated VqmA.

76 d-binding analyses revealed that DPO and Ala-AA occupy the same binding site.

77 improving knowledge among African American (AA) kidney transplant candidates compared to other races

78 state cancer (PCa) is that African American (AA) patients have a higher mortality rate than European

79 is two-fold higher in the African-American (AA) compared to non-Hispanic white.

80 TL mapping study on 1,032 African Americans (AA) and 801 European Americans (EA) in the GENOA cohort.

81 e ADHD cases included 116 African Americans (AA) and 89 of European Ancestry (EA) with SVs in compari

82 African Americans (AA) have lower rates of kidney transplantation (KT) comp

83 entations and outcomes in African Americans (AAs) compared to white patients.

84 es), 651 (4.2%) with artesunate-amodiaquine (AA, 6 studies), 7,340 (47.8%) with artesunate-mefloquine

85 ve symptoms" (EDS: CES-D(total) >= 16) among AA (HR = 1.28, 95% CI: 1.04-1.56, P = 0.017).

86 and promising to impact IRD knowledge among AA and non-AA kidney transplant candidates.

87 AAs, we demonstrated similar outcomes among AA and white recipients and better outcomes than the US

88 gnificantly lower graft survival rates among AA than white recipients (kidney 5 years: 76.5% vs 82.3%

89 1) overall and from 5 to 7 (P < 0.001) among AAs.

90 and delta(15) N, and isotopic offsets among AAs (e.g. Delta(15) N(Pro-Phe) ), to explore the allocat

91 We conducted an online simulation among AAs (N = 585) about interest in ApoL1 testing and its co

92 (N = 14, 47%), serum amyloid A amyloidosis (AA) (N = 11, 37%), localized AL (N = 3, 10%), wild-type

93 In an AA model, IL-6 deficiency led to increased lung inflamma

94 cohorts, their validity in African ancestry (AA) and Hispanic ethnicity (HE) individuals is unclear.

95 Individuals of African ancestry (AA) experience the disease more severely and with an inc

96 cestry (EA, n= 69,819) and African ancestry (AA, n = 15,603) individuals.

97 tuce, and predicted the FC (R(2) = 0.96) and AA (R(2) = 0.92) levels very well.

98 o examine the balanced groups of CC, AC, and AA individuals (n = 25/group).

99 ermolecular hydrogen bonding between ADD and AA, which was confirmed by Density Functional Theory cal

100 miRNAs significantly deregulated in CRC and AA tissues, respectively, 7 and 5 of these miRNAs were a

101 cid (IPA) and their structure was AA-DEG and AA-DEG-IPA-DEG.

102 ould evaluate the optimal intakes of DHA and AA in infants at different ages based on relevant outcom

103 the state of scientific research on DHA and AA, and to discuss the questions arising from the new Eu

104 tly enhanced the physical stability, EE, and AA retention of nanoliposomes after powder reconstitutio

105 (1.13-1.43) for incident CHD in EA, HE, and AA individuals, respectively.

106 y related to DeltaCES-D(total) among men and AA.

107 ths (0-95), ATTR amyloidosis: 17 months, and AA, 15 months (0-77).

108 Endogenous N and AA flows did not differ (P > 0.05) for diets C and HC, w

109 Total nitrogen and AA contents were quantified in effluents.

110 ry of dietary AAs on endogenous nitrogen and AA flows at the end of the ileum was studied.

111 idance for approaching patients with PNH and AA who have CH.

112 s could underestimate endogenous protein and AA losses by ~6% (the proportion of recycled 15N divided

113 nd carriers of any A at NLRP1 rs12150220 and AA for SRL rs13334970 in APD KRAS wild-type patients (HR

114 zer did not significantly affect protein and AAs concentrations.

115 of nitrogen (N) fertilization on protein and AAs trends.

116 Vitiligo and alopecia areata (AA) are common autoimmune conditions characterized by wh

117 s, the phytochemicals of Astragalus armatus (AA), A. caprinus (AC), and A. gombiformis (AG) have not

118 MRI coil technology for the anterior array (AA) surface coil in both a phantom and in vivo study.

119 differences in post LVAD atrial arrhythmias (AA) (Adjusted OR = 0.45 [0.18-1.06], p = 0.31) and ventr

120 ed in vivo in an adjuvant-induced arthritis (AA) rat model in order to identify the ideal prodrug des

121 STEM), two stacking orders are determined as AA stacking with negligible displacement across layers,

122 amilial Mediterranean fever (FMF)-associated AA amyloidosis.

123 al transplant recipients with FMF-associated AA amyloidosis (group 1) and propensity score-matched tr

124 Allergic asthma (AA) is characterized as a Th2-biased airway inflammation

125 (SE) and lower number of average awakenings (AA) (P =0.007 0.023 and 0.011, respectively) in the acup

126 ohort, graft survival did not differ between AA and white recipients; in contrast, the US cohort expe

127 and response to standard medications between AA and EA populations is partially conferred by genomic

128 rous ions through the redox reaction between AA and IONCs.

129 ) The genes differentially expressed between AAs' and EAs' tumors of the same grade category are rela

130 ) grants were awarded to fewer than 12 Black/AA women surgeons.

131 U.S. medical schools and residencies, Black/AA women have historically been underrepresented in acad

132 ool surgical faculty, 123 (0.79%) were Black/AA women surgeons with only 11 (0.54%) being tenured fac

133 epartment Chairs of surgery, none were Black/AA women.

134 allele with the ATA(+) subset of SSc in both AA and EA patients demonstrated a transancestry effect.

135 ccurate expression prediction models in both AA and EA, facilitating powerful transcriptome-wide asso

136 tro antimicrobial tests, followed closely by AA/PA-MEs.

137 cohort of SPK transplants overrepresented by AAs, we demonstrated similar outcomes among AA and white

138 was studied using (14)C-radiolabeled ((14)C-AA) and (13)C-labeled ((13)C(3)-AA) materials.

139 inine/proline metabolism, and branched-chain AA (BCAA) metabolism at baseline.

140 supplementing VLP diets with branched-chain AA (BCAA) would reverse the negative effects of these di

141 f four dynamically equilibrated constituents AA', AB', BA', and BB' is orthogonally triggered by two

142 four equilibrated nucleic acid constituents, AA', AB', BA', and BB', and the accompanying "dormant" s

143 e used for in vitro Treg expansion cultures, AA Tregs were able to expand.

144 ipherally NBN-doped PAHs to form H-bonded DD.AA- and ADDA.DAAD-type complexes with suitable complemen

145 strength is around 27 +/- 1 M(-1) for the DD.AA complexes in C(6)D(6), whereas it rises to 1820 +/- 1

146 he effect of the form of delivery of dietary AAs on endogenous nitrogen and AA flows at the end of th

147 verextension on different sequences of DNA ((AA)12, (AT)12, (CC)12 and (CG)12) with supercoiling dens

148 behaviors from random-sequence genomic DNA, AA-TT condenses in all alkaline earth metal ions.

149 ionale for testing combinations of high-dose AA and anti-PD1 agents in patients with aggressive B cel

150 The identification of three HLA-DQ AAs (alpha44, beta57, beta135) conferring T1D risk shoul

151 h 100% AT content, a poly(A)-poly(T) duplex (AA-TT) and a poly(AT)-poly(TA) duplex (AT-TA).

152 n the regulation of lung inflammation during AA and the control of S. pneumoniae bacterial disease.

153 range from 52.2 (AG) to 54.7 g 100 g(-1) DW (AA).

154 Ileal endogenous AA flows were determined by isotope dilution.

155 In contrast, almost all non-essential AAs differed in delta(13) C values between muscle and yo

156 We found that over half of essential AAs had delta(13) C values that did not differ between m

157 odel, the function and stability of expanded AA Tregs were tested.

158 A custom algorithm detected extrafoveal AA (EAA) excluding the central 1-mm circle in projection

159 owth factors, platelet-derived growth factor-AA, vascular endothelial growth factor, and others) were

160 eveloped and implemented a new algorithm for AA able to recognize microspheres and to analyze the att

161 re highest for GG homozygotes and lowest for AA homozygotes.

162 vely refined a culturally targeted video for AAs with input from multiple stakeholders.

163 An additional meal (A) containing a free AA mixture (306 mmol N) simulating the AA composition of

164 were observed with eicosanoids derived from AA by LOX enzymes.

165 The 1:1 alpha/Sulfono-gamma-AA peptides could be an alternative strategy to antagoni

166 s suggested that these helical sulfono-gamma-AA peptides could mimic the critical side chains of p53

167 was noted that these 1:1 alpha/Sulfono-gamma-AA peptides were completely resistant to proteolytic deg

168 elical heterogeneous 1:1 alpha/Sulfono-gamma-AA peptides were shown to bind tightly to MDM2 and MDMX,

169 now facilitates deeper understanding of GPCR AA function at the molecular level.

170 Many GPCR AAs modulate receptor signaling, but molecular details o

171 Most GPCR AAs are allosteric modulators and exhibit a broad range

172 Understanding GPCR AAs is not only important for defining how these unusual

173 production of more palatable kefir with high AA.

174 Recent studies have demonstrated how AAs regulate mTORC1 activity through Rags.

175 oped the "Angiogenesis Analyzer" for ImageJ (AA), a tool allowing analysis of ETFA-derived images, ac

176 ociated with progression to T2D, implicating AA and phospholipid metabolism.

177 g of the metabolic routing of this important AA.

178 In AA, 24-h BP monitoring provides limited added value as a

179 nd characteristics of somatic alterations in AA and PNH and will explore their prognostic significanc

180 othesized to be the site of immune attack in AA.

181 = 0.77) but were significantly attenuated in AA individuals (p(interaction)= 2.9 x 10(-3)).

182 arly ubiquitous clonal hematopoiesis (CH) in AA patients.

183 ed knowledge about these vascular changes in AA subjects.

184 ) and tortuosity at severity stages of DR in AA subjects.

185 We identified a total of 354,931 eSNPs in AA and 371,309 eSNPs in EA, with 112,316 eSNPs overlappe

186 fatal cardiovascular/renal disease events in AA subjects.

187 cells to regenerate anagen hair follicles in AA and intraepidermal melanocytes in vitiligo.

188 Larger studies are needed in AA to confirm these findings.

189 ction in a specific subpopulation of Treg in AA, which predicts response to immunosuppression.

190 tion that prostate tumors are more lethal in AAs than in EAs is reasonable regarding AAs' higher PHG,

191 is demonstrated in EAs (p < 0.01) but not in AAs; (2) PHG and its racial disparity are differentiated

192 ighlight strong support for ApoL1 testing in AAs and the need to examine such opinions among PLDs and

193 The digestible indispensable AA (IAA) score was 1.03 (histidine) for WPI and close to

194 with MPs isolated from healthy individuals (AA).

195 About 50% of initial AA went into the water extract, either unchanged or tran

196 Interestingly, AA were younger, with a mean age of 63 +/- 13.4 compared

197 the concentrations of the 4 fatty acids (LA, AA, DHA, and EPA) were measured in maternal plasma.

198 2 vs no coil) and -2.4% with the lightweight AA coil (P = .004 vs no coil).

199 bias of -4.3% and -4.0% with the lightweight AA coil.

200 In a humanized mouse model, AA homozygous mice were similarly protected against stre

201 he regulation of lung inflammation in murine AA caused by Aspergillus fumigatus as well as its conseq

202 ation between maternal glycemia and neonatal AA was observed across the whole range of maternal mid-g

203 tailed these two algorithms and used the new AA version to compare both methods (i.e. ETFA and FBA) i

204 articipants were recruited (according to NIA-AA guidelines, 14 healthy controls, 14 mild Alzheimer's

205 ing to impact IRD knowledge among AA and non-AA kidney transplant candidates.

206 in patients with AA (6.5% versus 0.3% in non-AA [P<0.001]).

207 Approximately 15% to 20% of AA patients and 2% to 6% of PNH patients go on to develo

208 The antimicrobial activity of AA/PA-MEs used as a washing solution on cucumber and str

209 As a result, the addition of AA in CW-based oleogels caused to form new intramolecula

210 also needed for the factorial calculation of AA requirements.

211 omically cheap platform for the detection of AA in food samples.

212 cellent selectivity towards the detection of AA in presence of possible biological active interferenc

213 190 uM was achieved towards the detection of AA with a LOD of 10 nM.

214 In this research, the fate of AA during roasting followed by drip brewed-like extracti

215 Additionally, the increase of AA concentration higher than 3% of oleogel formulation s

216 robe was drastically quenched in presence of AA, and shown excellent selectivity towards the detectio

217 Recurrence of AA amyloidosis was diagnosed in 21 patients (group 1a),

218 MF triggers the release of AA, resulting in the increase of ferrous ions through th

219 laccase activity, was comparable to that of AA or SO(2) at the typical doses employed in oenology fo

220 he biocatalytic conversion/transformation of AA to 6-ACA and HMD by carboxylic acid reductases (CARs)

221 For the transformation of AA to HMD (via 6-ACA), the wild type CAR was combined wi

222 f the samples were not changed by the use of AA/PA-MEs at 0.05 or 0.1% TVO.

223 increasing profile in the concentrations of AAs (6.5-55 ug mL(-1)), which can be explained by the pr

224 Over half of AAs that have labile nitrogen atoms (i.e. 'trophic' AA)

225 n between gestational glycemia and offspring AA measured by MRI in the neonatal period and during the

226 ime, and little is known about its impact on AAs.

227 n the ascorbic acid depletion (denoted as OP(AA)) and hydroxyl radical formation (denoted as OP(*OH))

228 In addition, we found OP(AA) and OP(*OH) are well-correlated while the proportion

229 orrelated while the proportion of OP(*OH)/OP(AA) by Cu is noticeably lower than that by Fe, indicatin

230 ous suppression or enhancement effects on OP(AA) and OP(*OH) by TMs.

231 ts show that carboxylic acids enhance the OP(AA) and OP(*OH) by TMs while imidazoles suppress them.

232 vation and confirmed by B3LYP/6-31+G(d)/OPLS-AA calculations with averaging over many explicit solvat

233 Although optimal AA intake amounts remain to be defined, we strongly reco

234 Others were AA3 (n = 4), or AA status missing (n = 1).

235 Coatings containing CA or AA promoted beta-carotene retention in dried apricot pre

236 d to fecal tests for the detection of CRC or AAs.

237 fy patients with advanced neoplasms (CRCs or AAs) based on their miRNA profiles, using findings from

238 We added OT, AA, SO(2) and bentonite to botrytized must obtained by i

239 eluting with baseline resolution from other AAs.

240 The other AAs concentrations showed linear rates of decrease over

241 t that metabolic dysregulation, particularly AA dysmetabolism, is present years prior to diabetes ons

242 UOD and pectin + AA coated samples showed lower L* and b*, and higher a*

243 will then discuss the available data on post-AA and post-PNH progression to secondary MDS/AML and pro

244 In the final product, AA content was close to 300 ug kg(-1).

245 e phosphorylation of RAPTOR in a new Raptor (AA) mouse model, in which AMPK phospho-serine sites Ser7

246 (cascades) of reduction/amination reactions (AA -> 6-ACA -> HMD).

247 l in AAs than in EAs is reasonable regarding AAs' higher PHG, while high grade alone could not imply

248 Pathway analysis revealed up-regulated AA metabolism, arginine/proline metabolism, and branched

249 rvival between the patients with FMF-related AA amyloidosis and glomerulonephropathies (GN) to define

250 sk of early CVD in patients with FMF-related AA amyloidosis.

251 ity were evaluated (rs1045642 and rs1128503 -AA genotype associated with lowest P-gp activity).

252 The rs2521634-AA (close to NPY gene) presented increased risk for seve

253 The rs2521634-AA was associated with higher percentage of interproxima

254 skin (vitiligo) and bald spots on the scalp (AA), which significantly impact patients' lives by damag

255 trade-off between yield improvement and seed AAs concentrations and will enable future genetic yield

256 he delta(15) N offset between trophic-source AAs (Delta(15) N(trophic-source) ) may provide an index

257 PUFAs and specifically AA trigger a cytokine response of IECs which is restrict

258 Successively, AA content decreased very quickly and at 14 min (203-205

259 experiments and simulations demonstrate that AA-TT condensation is driven by non-specific ion-DNA int

260 in to be defined, we strongly recommend that AA should be provided along with DHA.

261 tumor immune microenvironment revealed that AA strikingly increased intratumoral infiltration of CD8

262 Current dogma suggests that AAs have worse survival following SPK than white recipie

263 ased frequency and severity of SSc among the AA population.

264 enolic content in skin (~9.8%), PAs, and the AA.

265 Serine was omitted from the AA mixture to allow direct determination of its ileal en

266 a species revealed that chromosomes from the AA, BB or CC genomes generated specific and intense sign

267 In the AA population, the African ancestry-predominant HLA-DRB1

268 the challenge posed by low frequency of the AA genotype (appr.

269 ture-sensitive (ts) residue signature of the AA/60 MDV into a 2009 pandemic A/California/04/09 H1N1 v

270 free AA mixture (306 mmol N) simulating the AA composition of casein was included.

271 nd in situ XAS measurements suggest that the AA formation mechanism involves methanol activation on R

272 The results confirmed that the AA levels and antioxidant activity reached a maximum in

273 ed for providing additional resources to the AA communities.

274 05) compared to AG heterozygotes whereas the AA homozygotes trended towards a protective effect again

275 ferences in the side-chain polarity of these AAs trigger distinct allosteric responses in PKM2.

276 lk or albumen, suggesting that most of these AAs were directly routed from muscle into eggs.

277 free, gas phase carbonylation of methanol to AA.

278 Time from end of procedure to AA was 5.5 (0.5, 24.5) hours.

279 heir delta(15)N(Arg) values to be related to AA biosynthesis.

280 Whereas the host response to AA protected against S. pneumoniae lung disease, the IL-

281 bited stable (for 60 h) > 93% selectivity to AA with single pass conversion >60%.

282 onal animated video culturally responsive to AAs and conduct feasibility testing.

283 n SiO(2) resulted in >96% selectivity toward AA production at volumetric reaction rates comparable to

284 e value error was -5.9% with the traditional AA coil (P = .002 vs no coil) and -2.4% with the lightwe

285 bias of -9.0% and -8.6% with the traditional AA coil and a mean PET quantification bias of -4.3% and

286 t have labile nitrogen atoms (i.e. 'trophic' AA) had higher delta(15) N values in yolk and albumen th

287 wed that addition of propylene glycol to TVO/AA or PA:T80/water MEs gave dilutable systems with parti

288 The results introduce dilutable TVO:AA/PA-MEs for incorporation of TVO in aqueous systems fo

289 e (Leu) is a critical mTORC1 regulator under AA-starved conditions, how Leu regulates autophagy is po

290 sults provide a quantitative basis for using AA delta(13) C and delta(15) N, and isotopic offsets amo

291 ophthalic acid (IPA) and their structure was AA-DEG and AA-DEG-IPA-DEG.

292 ented with no insurance, and 34 of them were AA.

293 Today, the mechanisms by which AA is reduced during roasting remain unclear.

294 moral epigenome revealed increased 5hmC with AA treatment, consistent with in vitro findings.

295 Compared with AA MPs, SS MPs increased EC ICAM-1 messenger RNA and pro

296 ions, and broader peak widths, compared with AA stacking, indicating stronger interlayer interaction.

297 measuring aortic strain at DDA compared with AA, variability was acceptable at both anatomic location

298 ty was significantly higher in patients with AA (6.5% versus 0.3% in non-AA [P<0.001]).

299 Patients with AA were older, presented with acute congestive heart fai

300 moglobin in feces can identify patients with AAs or CRC more accurately than fecal hemoglobin concent

301 data on ileal digestibility of WPI and zein AAs in healthy humans and, in contrast to WPI, zein is p