ライフサイエンスコーパス: ABA

1 ABA agonists selective for individual ABA receptors migh

2 ABA application to wheat grown under near-field conditio

3 ABA content was very low in nced2569 seeds, similar to t

4 ABA increases expression of important glycogen synthase,

5 ABA is derived from the cleavage of 9-cis-isomers of vio

6 ABA rapidly increases FRET efficiency in N. benthamiana

7 ABA was also tested in dopamine transporter knockdown mi

8 ABA-independent SnRK2s act at the posttranscriptional le

9 ABA-triggered SnRK2 activation, transcription factor pho

10 d (JA) concentrations, and activate Ca(2+) , ABA and JA signaling transductions in plants.

11 4-ABA functionalization induces separation of the RGO laye

12 of negatively charged 4-aminobenzoic acid (4-ABA) onto a graphene functionalized carbon paper electro

13 due to the electrostatic repulsion between 4-ABA-grafted graphene layers.

14 Electrochemically grafted 4-ABA not only leads to a favorable orientation of BOD as

15 ne oxide to RGO and covalent attachment of 4-ABA are achieved by applying alternating cathodic and an

16 ) models, while SIRT1 activation accelerates ABA phenotypes.

17 duce the accumulation of leaf abscisic acid (ABA) and jasmonic acid (JA) concentrations, and activate

18 ore, the endogenous levels of abscisic acid (ABA) and proline were also reduced in stress-treated hss

19 effects of the application of abscisic acid (ABA) and sucrose on the postharvest ripening were also e

20 lation (A), vein embolism and abscisic acid (ABA) concentration during dehydration were quantified, a

21 rp-imposed dormancy, diaspore abscisic acid (ABA) concentration, and phenotypic plasticity of dimorph

22 evel water potential and leaf abscisic acid (ABA) concentration.

23 nse to dehydration stress and abscisic acid (ABA) in Arabidopsis thaliana.

24 induced by water deficit and abscisic acid (ABA) in the root elongation zone.

25 The stress hormone abscisic acid (ABA) initiates a signaling cascade, which leads to incre

26 Abscisic acid (ABA) is a drought stress signaling molecule, and simple

27 Abscisic acid (ABA) is a key phytohormone that mediates environmental s

28 Abscisic acid (ABA) levels increase significantly in plants under stres

29 to develop opabactin (OP), an abscisic acid (ABA) mimic with up to an approximately sevenfold increas

30 The plant hormone abscisic acid (ABA) plays a central role in the regulation of stomatal

31 o-receptors, the phytohormone abscisic acid (ABA) prevents premature germination and seedling growth

32 The phytohormone abscisic acid (ABA) promotes stomatal closure and inhibits light-induce

33 hich positively regulates the abscisic acid (ABA) response in Arabidopsis.

34 rity of the genes involved in abscisic acid (ABA) response pathway, containing the Abscisic Acid Resp

35 ABI3 also contributes to the abscisic acid (ABA) response.

36 vivo interactions with NO and abscisic acid (ABA) responses.

37 Plasma membrane-associated abscisic acid (ABA) signal transduction is an integral part of ABA sign

38 a complex osmotic-stress and abscisic acid (ABA) signal transduction network.

39 immune signaling antagonizes abscisic acid (ABA) signal transduction require further investigation.

40 s a critical regulator in the abscisic acid (ABA) signaling and drought response pathways in Arabidop

41 The turnover of abscisic acid (ABA) signaling core components modulates the plant's res

42 ponents in osmotic stress and abscisic acid (ABA) signaling pathways; however, the upstream component

43 hosphatases known to regulate abscisic acid (ABA) signaling.

44 c stress responses, including abscisic acid (ABA) signaling.

45 mplicates a gene important in abscisic acid (ABA) signalling in seeds.

46 een AtNBR1 overexpression and abscisic acid (ABA) signalling was suggested by an interaction network

47 activated the promoter of the abscisic acid (ABA) synthesis key gene PdNCED3, resulting in a signific

48 on the action of the hormone abscisic acid (ABA) that acts through a receptor-signal transduction pa

49 s trigger the accumulation of abscisic acid (ABA), a key plant stress-signaling hormone that activate

50 Abscisic acid (ABA), a plant hormone synthesized from carotenoids, func

51 hat the established ethylene, abscisic acid (ABA), and GA growth regulatory module for underwater elo

52 l closure as the phytohormone abscisic acid (ABA), but underlying mechanisms remain elusive.

53 analyses of the phytohormone abscisic acid (ABA), Ca(2+), protons (H(+)), chloride (anions), the glu

54 ulation of the stress hormone abscisic acid (ABA), reprogramming of gene expression, and altering of

55 a) SnRK2 family comprises the abscisic acid (ABA)-activated protein kinases SnRK2.2, SnRK2.3, SnRK2.6

56 enetic diversification of the abscisic acid (ABA)-dependent seed maturation programs by addressing qu

57 response to CO(2) , light and abscisic acid (ABA).

58 eedling growth in response to abscisic acid (ABA).

59 s, plants produce the hormone abscisic acid (ABA).

60 ses that are under control by abscisic acid (ABA).

61 ts of polyethylene glycol and abscisic acid (ABA).

62 Active buoyancy adjustment (ABA) is a behavioural response to environmental stressor

63 Although ABA signaling inhibits PP2C activity through ABA-recepto

64 the most prominent element, representing an ABA response element and a potential CAMTA-binding site.

65 sted that the expression levels of auxin and ABA signaling genes in the STTM-directed double mutant w

66 AREB3), BASIC LEUCINE ZIPPER67 (bZIP67), and ABA INSENSITIVE3 (ABI3) with those regulated by LEC1.

67 antly in plants under stress conditions, and ABA is thought to serve as a key stress-response regulat

68 ), and are more tolerant to dehydration; and ABA strongly upregulates class 1 Glbs.

69 ible pRAB18::GFP in the presence of DFPM and ABA.

70 ired in the induction of gene expression and ABA accumulation in response to osmotic stress.

71 demonstrate an interaction between Glbs and ABA on several grounds: Glb1 and Glb2 scavenge NO produc

72 We conclude that light and ABA signal competitively in order for algae to position

73 cing of genes involved in osmotic-stress and ABA responses, light signaling, and mRNA splicing, inclu

74 s displayed dehydration stress tolerance and ABA hypersensitivity in terms of stomatal closure.

75 of the RAFs in osmotic stress tolerance and ABA responses as well as in growth and development.

76 ed stomatal opening remained unaffected, and ABA responses showed slowing in some experiments.

77 n nxd1 increases both 9-cis-violaxanthin and ABA accumulation.

78 duces high amounts of 9-cis-violaxanthin and ABA, aba4 nxd1 exhibits reduced levels in both leaves an

79 Utilizing the activity-based anorexia (ABA) model and touchscreen operant learning paradigms, w

80 were tested in the activity-based anorexia (ABA) model, with an extended period of food restriction

81 of AN behaviors in activity-based anorexia (ABA) models, while SIRT1 activation accelerates ABA phen

82 to their distinctive molecular architecture, ABA triblock copolymers will undergo specific self-assem

83 es (RAFs) in early osmotic stress as well as ABA signaling in Arabidopsis thaliana.

84 The ability of DFPM to attenuate ABA signaling was reduced in rda mutants (resistant to D

85 n, and the physiological importance of basal ABA signaling in stomatal regulation by CO(2) and, as hy

86 that stomatal CO(2) signaling requires basal ABA and SnRK2 signaling, but not SnRK2 activation.

87 de novel insights into the interplay between ABA and the light signaling component in the modulation

88 ioxygenase (NCED) and inactivation of ABA by ABA 8'-hydroxylase (CYP707A) are key regulatory metaboli

89 , and hampers the inhibition of ABI1/ABI2 by ABA-bound PYR1/PYL4, thereby terminating ABA signaling.

90 in kinase2s (SnRK2s), which are activated by ABA-triggered inhibition of type-2C protein-phosphatases

91 In lamellae formed by ABA amphiphiles, the fraction of B blocks "bridging" adj

92 ners, which are transcriptionally induced by ABA but suppress pathogen-induced expression of ABA-depe

93 ABT is induced by ABA in a PYR1/PYL/RCAR-PP2C-dependent manner.

94 are directly transcriptionally regulated by ABA-RESPONSIVE ELEMENT BINDING PROTEIN3 (AREB3), BASIC L

95 nd that this interaction is not regulated by ABA.

96 gested to have stomata that are regulated by ABA.

97 RD26 protein is stabilized by ABA and dehydration in a BIN2-dependent manner.

98 ess was dependent on the signal triggered by ABA and differed from in vivo ripening, resulting in fru

99 th greater stomatal sensitivity triggered by ABA production and leading to greater WUE(i) provides dr

100 l a negative feedback mechanism triggered by ABA that acts via ALIX to control the accumulation of sp

101 essed in multiple tissues and upregulated by ABA and drought treatments.

102 In conclusion, ABA levels, being critical for Imprime-mediated immune a

103 ative method for fabricating and controlling ABA triblock copolymer hierarchical structures using sol

104 Recent work has established a core ABA signaling pathway in which A-type PP2C protein phosp

105 The core ABA signalling components are snf1-related protein kinas

106 In the dark, ABA induced a negative geotropic movement of the algae t

107 electively and efficiently yields degradable ABA-block polymers, incorporating 6-23 wt % CO(2).

108 ical processes, including plant development, ABA-mediated signalling pathway, ubiquitin-dependent pro

109 During drought, endogenous ABA did not play a role in stomatal closure, and exogeno

110 y associated with an increase in endospermic ABA levels.

111 wever, TMB inhibits germination by enhancing ABA levels and reducing the activity of hydrolytic enzym

112 eraison, we observed high ABA-glucose ester (ABA-GE) and low indole-3-acetate aspartate (IAA-Asp) and

113 entified previously, but only aba4 exhibited ABA-deficient phenotypes.

114 ay a role in stomatal closure, and exogenous ABA applied to live, intact leaves did not induce stomat

115 showed a bell-shaped dependency on exogenous ABA, and their regulation by WD was attenuated in genoty

116 levels in darkness in response to exogenous ABA treatment by binding directly to the G-box motifs in

117 abidopsis (Arabidopsis thaliana) RING Finger ABA-Related1 (RFA1) and RFA4 E3 ubiquitin ligases, membe

118 endent directional taxis response to a fixed ABA source, moving horizontally towards the source in th

119 O produced in stomatal guard cells following ABA supply; plants overexpressing Glb1 show higher const

120 , these parameters were comparable following ABA treatment.

121 SnRK2/OST1 site, which is indispensable for ABA-induced reactivation of PP2C-dephosphorylated SnRK2

122 en characterized as the natural receptor for ABA.

123 onstrate that this M3K clade is required for ABA- and osmotic-stress-activation of SnRK2 kinases, ena

124 olic flexibility in soleus muscle cells from ABA-treated mice with DIO.

125 rt a new series of vitamin E-functionalized 'ABA' triblock copolymers with carbamate block junction,

126 Further, ABA synergized with insulin to dramatically increase the

127 Furthermore, ABA promoted indoylacetic acid transport in root tips, w

128 mprime administration for IPD changes (e.g., ABA, circulating immune complexes, complement activation

129 During post-veraison, we observed high ABA-glucose ester (ABA-GE) and low indole-3-acetate aspa

130 Tighter stomatal control mediated by higher ABA accumulation during dehydration in these species res

131 Responsive to Dehydration (RD29A) and Highly ABA-Induced 2 (HAI2), and are more tolerant to dehydrati

132 The Highly ABA-Induced 1 (HAI1) protein phosphatase is a central co

133 This raises the key question of how ABA signaling arose in the earliest land plants.

134 east two-hybrid assays were used to identify ABA signaling components that interact with COP1, and bi

135 at its retention therein strongly implicates ABA in the regulation of their relevant tropisms.

136 by WD was attenuated in genotypes altered in ABA biosynthesis and response.

137 to elucidate the regulatory role of COP1 in ABA signaling.

138 DRT111 knock-out mutants are defective in ABA-induced stomatal closure and are hypersensitive to A

139 PA in response to salt show an enrichment in ABA signaling, and in the response to stresses such as s

140 (ABI5), which encodes an activator of FLC in ABA-regulated flowering signaling.

141 Although much of the focus in ABA triblock copolymer self-assembly has been on equilib

142 known factors critical for NCED3 function in ABA synthesis (expression, chloroplast import, and thyla

143 aliana E3 ubiquitin ligase COP1 functions in ABA-mediated stomatal closure.

144 tochondrial ATP production were increased in ABA-treated human myotubes.

145 through activation of key genes involved in ABA and sucrose metabolic pathways.

146 evel of OST1, a crucial SnRK2-type kinase in ABA signaling.

147 mPFC-AcbSh pathway prevented weight loss in ABA and improved flexibility during early reversal learn

148 Parallel attenuation of weight loss in ABA and improvement of cognitive flexibility following s

149 Loss of SINE1 or SINE2 results in ABA hyposensitivity and impaired stomatal dynamics but d

150 uttling protein and plays a positive role in ABA responses by interacting with and maintaining the st

151 ABA agonists selective for individual ABA receptors might offer an approach to phenocopy the w

152 Z signaling module channels pathogen-induced ABA signaling toward cell wall defense while simultaneou

153 es a rate-limiting enzyme for stress-induced ABA synthesis.

154 glycemic control without increasing insulin, ABA extract modulates the metabolic activity of muscle.

155 tion, and stomatal movements, and integrates ABA- and light-regulated pathways to control seed germin

156 e observed that in Rhododendron 'Elsie Lee', ABA and JA decreased in winter, which may be due to the

157 In high light ABA enhanced HCO(3)(-) uptake, while under low light upt

158 ng that CDK8 could link the SnRK2.6-mediated ABA signaling to RNA polymerase II to promote immediate

159 wledge of the molecular mechanisms mediating ABA-induced stomatal closure in the past decade.

160 nsights into the roles of CDK8 in modulating ABA signaling and drought responses.

161 resilient and vulnerable phenotypes in mouse ABA significantly advances the utility of the model for

162 ong bis(anthraoxa)quinodimethanes with nine (ABA) and ten (ANA) consecutively fused six-membered ring

163 tivation of internal nodes in the absence of ABA elicited stomatal closure in wet bench experiments,

164 by affecting stress-induced accumulation of ABA and proline.

165 anced sensitivity to ABA and accumulation of ABA receptors compared with the wild type.

166 l localization and increased accumulation of ABA receptors.

167 sensitive loci by monitoring the activity of ABA-inducible pRAB18::GFP in the presence of DFPM and AB

168 th 22 interactions requiring the addition of ABA.

169 The application of ABA or proline could alleviate stress-induced oxidative

170 ight play role in maintaining the balance of ABA signalling by controlling their level and/or activit

171 locally well-segregated yet the B blocks of ABA amphiphiles are significantly less stretched than in

172 lization and promotes nuclear degradation of ABA receptors.

173 further supports the clinical development of ABA in the treatment of pre-diabetes, type 2 diabetes an

174 ggest that SINE1 and SINE2 act downstream of ABA but upstream of Ca(2+) and F-actin.

175 2 in skeletal muscle abrogated the effect of ABA extract in the DIO model and increased fasting blood

176 scriptomic analysis suggested enhancement of ABA responses, ABA levels were unchanged in the OX shoot

177 hat ICE1 binds to and inhibits expression of ABA INSENSITIVE 3.

178 but suppress pathogen-induced expression of ABA-dependent genes.

179 erize the efficacy of a fig fruit extract of ABA in promoting glycemic control.

180 ST1 levels are also depleted within hours of ABA signal onset.

181 The identification of ABA receptors and the ABA signaling core consisting of P

182 To evaluate the genome-wide impact of ABA deficiency in developing seeds at the maturation sta

183 Implications of ABA for future marine management and policy are discusse

184 enoid dioxygenase (NCED) and inactivation of ABA by ABA 8'-hydroxylase (CYP707A) are key regulatory m

185 rda mutants (resistant to DFPM inhibition of ABA signaling).

186 -mediated immune signaling and inhibition of ABA signaling.

187 DA2 functions in DFPM-mediated inhibition of ABA-mediated reporter expression.

188 dvances in understanding the interactions of ABA and other stomatal signaling pathways are reviewed h

189 ttuce seed germination by reducing levels of ABA and enhancing the activity of hydrolytic enzymes, wh

190 ects were accompanied by increased levels of ABA and its derivatives (phaseic and dehydrophasic acids

191 ther, our results reveal a core mechanism of ABA signaling termination that is critical for seed germ

192 in illuminating the regulatory mechanisms of ABA signal transduction, and the physiological importanc

193 ) signal transduction is an integral part of ABA signaling.

194 producing both cis-xanthophyll precursors of ABA.

195 tably, we found that even in the presence of ABA, OST1 levels are also depleted within hours of ABA s

196 ion and seedling greening in the presence of ABA, whereas knockout of ABT has the opposite effect.

197 etermined PP2CA stability in the presence of ABA.

198 e suggest that SIRT1 promotes progression of ABA, in part through its interaction with NRF1, leading

199 of AtNBR1 with three regulatory proteins of ABA pathway (ABI3, ABI4 and ABI5) was observed in planta

200 -type RSL1/RFA family, are key regulators of ABA receptor stability in root and leaf tissues, targeti

201 mixture was more than doubled as a result of ABA-induced monomerization, which leads to halt of quenc

202 ts provide further insights into the role of ABA and sucrose in the regulation of postharvest ripenin

203 Arabidopsis thaliana) the regulatory role of ABA in mechanisms that determine root hydraulic architec

204 eeds, also highlighted an inhibitory role of ABA on remobilization of reserves, reactive oxygen speci

205 n by IBI1, as well as the regulatory role of ABA therein, remain unknown.

206 ogy enrichments revealed a large spectrum of ABA activation targets involved in reserve storage and d

207 bidopsidis is associated with suppression of ABA-inducible abiotic stress genes.

208 ard and well-controlled one-pot synthesis of ABA triblocks, namely poly(ether-b-ester-b-ether), and A

209 f RAP2.6 and RAP2.6-mediated upregulation of ABA-responsive genes, indicating that CDK8 could link th

210 view recent studies investigating the use of ABA signaling mechanisms for the manipulation of stomata

211 T), a WD40 protein, efficiently switches off ABA signaling and is critical for seed germination and s

212 induction of ripening, which is dependent on ABA and its derivatives.

213 nalyzed the effect of ABA4 overexpression on ABA and carotenoid accumulation in wild-type and mutant

214 4 weeks; some mice were given antibiotics or ABA-PEG20k-Pi20 (Pi-PEG), which inhibits collagenase pro

215 sion of root development by water deficit or ABA.

216 the radicles and was not enhanced by NaCl or ABA.

217 s interact with ABA receptors to orchestrate ABA signaling in darkness by controlling ABI5 expression

218 egulated independently and upstream of other ABA-induced effects such as rapid growth and flowering.

219 d onto electrografted p-aminobenzoic acid (p-ABA).

220 mplate molecule and p-aminobenzeneboronic (p-ABA) acid as a functional monomer.

221 GA) on the amino groups of the 3D-Au-PAMAM-p-ABA-SPCE, where tau protein was sandwiched with a second

222 part of a novel signaling pathway promoting ABA-mediated stomatal closure by regulating the stabilit

223 Endogenous levels of PYR1 and PYL4 ABA receptors were higher in the rfa1 rfa4 double mutant

224 hat CO(2) signaling is mediated by PYL4/PYL5 ABA-receptors could not be supported here in two indepen

225 germinative growth through the PYR1/PYL/RCAR ABA receptors and PP2C co-receptors, the phytohormone ab

226 BA signaling core consisting of PYR/PYL/RCAR ABA receptors, PP2C protein phosphatases and SnRK2 prote

227 TART domain proteins are the 14 PYR/PYL/RCAR ABA receptors, while the other members of the superfamil

228 M3K triple knock-out plants show reduced ABA sensitivity and strongly impaired rapid osmotic-stre

229 at AtPTPN is required for HSFA6a to regulate ABA and drought responses.

230 ht signaling factor HY5 negatively regulates ABA-mediated inhibition of post-germination seedling gro

231 ysis suggested enhancement of ABA responses, ABA levels were unchanged in the OX shoots.

232 activation of SnRK2 kinases, enabling robust ABA and osmotic stress signal transduction.

233 resulting in a significant increase in root ABA content in poplars subjected to water deficit.

234 aphene (TLG) stacks in either a semimetallic ABA or a semiconducting ABC configuration with a gate-tu

235 A repeating triplet-sequence ABA- of non-overlapping brief tones, A and B, is a value

236 At the end of infusion, free serum ABA levels decreased, circulating immune complex levels

237 Yeast two-hybrid assays involving seven ABA receptor proteins (PtrRCAR) against 12 PtrHAB protei

238 ively regulates the transcription of several ABA-responsive genes, probably through promoting the rec

239 gas exchange analyses of quintuple/sextuple ABA receptor mutants show that stomatal CO(2) signaling

240 onversely, DRT111 overexpressing plants show ABA-hyposensitive seed germination.

241 tability in root and leaf tissues, targeting ABA receptors for degradation in different subcellular l

242 by ABA-bound PYR1/PYL4, thereby terminating ABA signaling.

243 nxd1) and Arabidopsis (Arabidopsis thaliana) ABA-deficient4 (aba4), were identified previously, but o

244 Here, we report that ABA Signaling Terminator (ABT), a WD40 protein, efficien

245 s (Arabidopsis thaliana) roots revealed that ABA treatment and uptake did not trigger rapid cytosolic

246 The ABA signaling pathway is highly complex and relies on a

247 The ABA-induced stomatal closure phenotype is, in part, attr

248 The identification of ABA receptors and the ABA signaling core consisting of PYR/PYL/RCAR ABA recept

249 K2.3, SnRK2.6, SnRK2.7, and SnRK2.8, and the ABA-independent subclass 1 protein kinases SnRK2.1, SnRK

250 As the ABA concentration increased from <1 muM to 1 mM, the int

251 e spectral region are adequate to assess the ABA concentration of a solution.

252 ate that PTPN-mediated crosstalk between the ABA signaling and AsA biosynthesis pathways positively c

253 and resilient phenotypes emerged in both the ABA and food-restricted mice without wheels.

254 e also showed that CDK8 is essential for the ABA-induced expression of RAP2.6 and RAP2.6-mediated upr

255 How the ABA and AsA signaling pathways interact in stress respon

256 However, little is known about how the ABA-mediated inhibition of seed germination and seedling

257 ng enzymes responsible for most steps of the ABA biosynthesis pathway have been identified, enzymatic

258 It also highlights the utility of the ABA model for understanding the biological bases of cogn

259 ar locations, in which the complexity of the ABA receptor family is mirrored in the partner RBR-type

260 1 show higher constitutive expression of the ABA responsive genes Responsive to ABA (RAB18), Responsi

261 ere, we show that the desensitization of the ABA signal is achieved by the regulation of OST1 (SnRK2.

262 t AtNBR1 is essential for fine-tuning of the ABA signalling pathway.

263 y, we show that PIFs positively regulate the ABA signaling pathway during the seedling stage specific

264 and overexpression of ABI5 could rescue the ABA-insensitive phenotypes of pifq mutants in the dark.

265 the complex rheostat dynamics underlying the ABA-induced stress response and desensitization.

266 When the ABA signal terminates, protein phosphatases ABI1/2 promo

267 d that PIFs can physically interact with the ABA receptors PYL8 and PYL9, and that this interaction i

268 This ABA-enriched extract, at 0.125 ug ABA/kg body weight, im

269 ABA signaling inhibits PP2C activity through ABA-receptor complex, it remains unknown if other mechan

270 Arabidopsis thaliana) ALIX directly binds to ABA receptors in late endosomes, promoting their degrada

271 Moreover, in contrast to ABA, the stomatal closing stimuli, elevated CO(2) and Me

272 d stomatal closure and are hypersensitive to ABA during seed germination.

273 tion mutants of AtPTPN were hyposensitive to ABA but hypersensitive to drought stresses, whereas plan

274 The cop1 mutants are hyposensitive to ABA-triggered stomatal closure under light and dark cond

275 ld increase in receptor affinity relative to ABA and up to 10-fold greater activity in vivo.

276 tics, particularly for closure, responded to ABA in all three taxa.

277 romote immediate transcriptional response to ABA and drought signals.

278 components modulates the plant's response to ABA and is regulated by ubiquitination.

279 Stomatal kinetic responses to ABA have not been considered.

280 on of the ABA responsive genes Responsive to ABA (RAB18), Responsive to Dehydration (RD29A) and Highl

281 Stomatal kinetics were responsive to ABA in fern.

282 Here, we identified SAB1 (Sensitive to ABA 1) as a novel negative regulator of ABI5 that simult

283 double mutant showed enhanced sensitivity to ABA and accumulation of ABA receptors compared with the

284 indicate that DRT111 controls sensitivity to ABA during seed development, germination, and stomatal m

285 , cdk8 mutants showed reduced sensitivity to ABA, impaired stomatal apertures and hypersensitivity to

286 -state g(s) did not respond significantly to ABA in fern or cedar but responded strongly in soybean.

287 overexpressor lines were more susceptible to ABA and other abiotic stresses.

288 atal conductance (g(s) ) was unresponsive to ABA in some studies, supporting this model.

289 This ABA-enriched extract, at 0.125 ug ABA/kg body weight, improves glucose tolerance, insulin

290 lar to the severe mutant aba2; unexpectedly, ABA Glc ester was detected in aba2 seeds, suggesting the

291 also activated by a rapid, largely unknown, ABA-independent osmotic-stress signalling pathway.

292 in solutions without ABA and monomerize upon ABA binding.

293 ts reveal a sophisticated mechanism by which ABA receptors are targeted by ubiquitin at different sub

294 we used a nced2569 quadruple mutant in which ABA deficiency is mostly restricted to seeds, thus limit

295 st hubs of this network were associated with ABA signalling.

296 lude that Glbs modulate NO and interact with ABA in crucial physiological processes such as the plant

297 members of the RSL1/RFA family interact with ABA receptors at plasma membrane, cytosol, and nucleus,

298 our data demonstrate that PIFs interact with ABA receptors to orchestrate ABA signaling in darkness b

299 rsensitivity were rescued by treatments with ABA, a hormone known to inhibit protein synthesis.

300 YL3 protein form dimers in solutions without ABA and monomerize upon ABA binding.