ライフサイエンスコーパス: ACC

1 ACC activates Ca(2+)-containing ion currents via GLUTAMA

2 ACC and vaterite remained stable for up to 4 days, possi

3 ACC is often used to induce ethylene responses.

4 ACC rates by size were 0.1%, 2.4%, and 19.5% for AIs of

5 ACC regulates these choices by providing a stable popula

6 ACC represents the complete state space of the task, wit

7 ACC risks by Hounsfield density were 0%, 0.5%, and 6.3%

8 ACC tracking of subjective value also correlated with ch

9 ACC/AHA risk alone misclassified plaque extent compared

10 ions, with peak oxygen uptake of 16.2+/-5.2 (ACC/AHA), 14.1+/-4.2 (ESC), and 12.7+/-3.1 mL.kg(-1).min

11 py eligibility was determined using the 2013 ACC/AHA and 2018 AHA/ACC guidelines criteria.

12 IDH, by 2017 ACC/AHA (systolic BP <130 mm Hg, diastolic BP >=80 mm Hg

13 Achieving and maintaining 2017 ACC/AHA BP goals could prevent a greater number of CVD e

14 er ten years, achieving and maintaining 2017 ACC/AHA guideline goals compared with current BP levels,

15 The document is a focused update of the 2017 ACC Expert Consensus Decision Pathway on the Management

16 IDH was more common when defined by the 2017 ACC/AHA BP guideline compared with the JNC7 guideline.

17 ce of IDH in the NHANES was 6.5% by the 2017 ACC/AHA definition and 1.3% by the JNC7 definition (abso

18 motensive ARIC participants, IDH by the 2017 ACC/AHA definition was not significantly associated with

19 external cohorts (eg, HRs of IDH by the 2017 ACC/AHA definition were 1.17 [95% CI, 0.87-1.56] in the

20 s recommended BP pharmacotherapy by the 2017 ACC/AHA guideline based solely on the presence of IDH.

21 or antihypertensive drug therapy in the 2017 ACC/AHA guideline, 2003 Seventh Joint National Committee

22 e of cardiovascular outcomes ranged from 75 (ACC/AHA) to 298 events per 1000 person-years (HFSA).

23 Accordingly, ACC is necessary only for updating model-based strategie

24 mmatory potential, with predictive accuracy (ACC) exceeding 90%.

25 on of 1-aminocyclopropane-1-carboxylic acid (ACC) synthases (ACSs), the rate-limiting enzymes in ethy

26 ursor 1-aminocyclopropane-1-carboxylic acid (ACC) was found to promote the emergence of nodal roots.

27 rsor, 1-aminocyclopropane-1-carboxylic acid (ACC), is a non-proteinogenic amino acid produced by ACC

28 cursor (aminocyclopropane-1-carboxylic acid, ACC) peak before veraison.

29 development of NAFLD and NASH by activating ACC.

30 In advanced ACC that is microsatellite stable, pembrolizumab provide

31 We enrolled 39 patients with advanced ACC and herein report after a median follow-up of 17.8 m

32 Patients with advanced ACC were enrolled in a phase II study to evaluate the cl

33 mations, including corpus callosum agenesis (ACC) and microcephaly.

34 termined using the 2013 ACC/AHA and 2018 AHA/ACC guidelines criteria.

35 herapies (very high risk) under the 2018 AHA/ACC guidelines, by assessing the composite of all-cause

36 and the American College of Cardiology (AHA/ACC) changed 3-hydroxy-3-methyl-glutaryl-coenzyme A redu

37 The AHA/ACC risk calculator first triages patients into 4 risk c

38 Dysregulation of the amygdala, ACC, or hippocampus (HPC) has been hypothesized to contr

39 ackknife validation, StackDPPred achieved an ACC of 89.96%, MCC of 0.799 and AUC of 94.50%.

40 is (PCA) produced three clear components: an ACC glutathione-glutamate component; an insula-visual gl

41 CLOPROPANE-1-CARBOXYLATE (ACC) SYNTHASE7, an ACC synthase homolog involved in ethylene biosynthesis,

42 nectivity and integrity between amygdala and ACC were also decreased in IA subjects.

43 s after cooking was found to be genotype and ACC dependent, which suggest that genotype and ACC induc

44 C dependent, which suggest that genotype and ACC induces differential expression of genes for the bio

45 hair cortisol and children's hippocampal and ACC structure, including across hippocampal subfields, a

46 4-parameter sigmoidal model in which PFC and ACC TSPO V(T) accounted for 84% and 92% of the variance,

47 eks of treatment was plotted against PFC and ACC TSPO V(T), showing a significant nonlinear relations

48 emission tomography scan to measure PFC and ACC TSPO V(T).

49 S2 and ACC-projecting neurons showed no changes in excitability

50 ging from solution to hydrated and anhydrous ACC, and with concentrations ranging between 1 and 15 M.

51 As ACC strength is primarily a function of the position of

52 rdiology and the American Heart Association (ACC/AHA) Pooled Cohort Risk Equation.

53 ge of Cardiology/American Heart Association (ACC/AHA), 205 met European Society of Cardiology (ESC),

54 sis, and evaluation of carboxylic acid-based ACC inhibitors with organic anion transporting polypepti

55 of subjects had large discrepancies between ACC/AHA risk and plaque burden at imaging.

56 fMRI results showed a relationship between ACC activation and androgen levels before treatment but

57 ements, we assessed the relationship between ACC-striatal rsFC and nicotine dependence severity.

58 Both ACC and BLA inhibition block this enhancement but via di

59 ea and fungi in saline soils was affected by ACC amendments; whereas community composition in non-sal

60 ken to further understand the role played by ACC in the early stages of OA.Tibio-femoral joints were

61 s a non-proteinogenic amino acid produced by ACC SYNTHASE (ACS).

62 Amorphous calcium carbonate (ACC) is an unstable mineral phase, which is progressivel

63 rospectroscopy, amorphous calcium carbonate (ACC) was observed first in the drops, followed by vateri

64 in the form of amorphous calcium carbonate (ACC).

65 activation of acetyl-coenzyme A carboxylase (ACC) and increased lipid content in human hepatocytes an

66 al data suggest that acetyl-CoA carboxylase (ACC) inhibitors have the potential to rebalance disorder

67 nsecticide targeting acetyl-CoA carboxylase (ACC) of pest insects and mites upon foliar application.

68 ing protein [SREBP], acetyl-CoA carboxylase [ACC], peroxisome proliferator-activator receptors [PPARs

69 dentified 1-AMINOCYCLOPROPANE-1-CARBOXYLATE (ACC) SYNTHASE7, an ACC synthase homolog involved in ethy

70 d risk factors for adrenocortical carcinoma (ACC) in adrenal incidentaloma (AI).

71 Adrenocortical carcinoma (ACC) is a rare and aggressive malignancy with limited th

72 Adrenocortical carcinomas (ACC) are rare and aggressive malignancies with limited t

73 iation (AHA)/American College of Cardiology (ACC) atherosclerosis cardiovascular disease (ASCVD) risk

74 rgeons (STS)/American College of Cardiology (ACC) Transcatheter Valve Therapies Registry from June 20

75 most recent American College of Cardiology (ACC)/American Heart Association (AHA) and European Socie

76 In the 2017 American College of Cardiology (ACC)/American Heart Association (AHA) guideline, the def

77 has been no American College of Cardiology (ACC)/American Heart Association guideline update specifi

78 te hippocampal volume and rostral and caudal ACC thickness and surface area (n = 37 with both child h

79 assessed using the Acoustic Change Complex (ACC) from EEG recordings.

80 fects of genotype, agro-climatic conditions (ACC), and cooking method as well as their interactions o

81 We also demonstrate that EVs contain ACC in uterine fluid using spectroscopic analysis.

82 In contrast, ACC outputs to the visual cortex facilitate sensory proc

83 S and their respective age-matched controls (ACC; ANOVA main effect of diagnosis: F(1,58) = 0.407, p

84 t a novel atom-confinement and coordination (ACC) strategy to achieve the synthesis of rare-earth sin

85 as identified the anterior cingulate cortex (ACC) among other brain regions involved in this process.

86 , ~120 ms) in mid-anterior cingulate cortex (ACC) and a later beta power suppression (13-22 Hz, ~350

87 levels in dorsal anterior cingulate cortex (ACC) and glutamate and Glx levels in left thalamus.

88 thalamus (MThal), anterior cingulate cortex (ACC) and striatum play important roles in affective-moti

89 as focused on the anterior cingulate cortex (ACC) as a key brain region in the mitigation of the comp

90 activation of the anterior cingulate cortex (ACC) corresponding to the noxious stimulus condition.

91 ase in NAc-dorsal anterior cingulate cortex (ACC) FC relative to non-responders (p < 0.001) which was

92 as implicated the anterior cingulate cortex (ACC) in attaching value to social outcomes.

93 al neurons of the anterior cingulate cortex (ACC) in mice with a mutation in Shank3, a high-confidenc

94 tex including the anterior cingulate cortex (ACC) in social cognition in adolescence.

95 lism in pregenual anterior cingulate cortex (ACC) in type 1 diabetes (T1D) without depression affects

96 The anterior cingulate cortex (ACC) is important for decision-making as it integrates m

97 rtex known as the anterior cingulate cortex (ACC) is particularly well suited for this function.

98 o pain areas like anterior cingulate cortex (ACC) might underlie maladaptive pain phenotypes.

99 al neurons in the anterior cingulate cortex (ACC) of adult male mice we found good coupling, particul

100 cortex (PFC) and anterior cingulate cortex (ACC) predicts reduction of depressive symptoms following

101 to show that the anterior cingulate cortex (ACC) predicts the state that actions will lead to, not s

102 at removal of the anterior cingulate cortex (ACC) prevents monkeys from learning what actions are pro

103 nnectivity of the anterior cingulate cortex (ACC) was evaluated with: the precuneus (P), the posterio

104 thalamus and the anterior cingulate cortex (ACC) were measured using 1[H]-magnetic resonance spectro

105 n activity in the anterior cingulate cortex (ACC), a brain region important in mood regulation.

106 neural signals in anterior cingulate cortex (ACC), a brain region thought to contribute to attentiona

107 phenomenon in the anterior cingulate cortex (ACC), a brain structure implicated in signaling cost-ben

108 FC with adjacent anterior cingulate cortex (ACC), a crucial region of emotion modulation.

109 formation, to the anterior cingulate cortex (ACC), a key area for processing pain affect, in freely b

110 ween amygdala and anterior cingulate cortex (ACC), and had increased positive FC between amygdala and

111 entrations in the anterior cingulate cortex (ACC), left insula, and visual cortex using 7T proton mag

112 ing the amygdala, anterior cingulate cortex (ACC), nucleus accumbens (NAcc), and orbitofrontal cortex

113 sual cortex (V1), anterior cingulate cortex (ACC), posterior parietal cortex (PPC), while freely movi

114 l (BP(ND)) in the anterior cingulate cortex (ACC).

115 he mouse PFC, the anterior cingulate cortex (ACC).

116 tical region, the anterior cingulate cortex (ACC, experiment 2), in macaques.

117 cortical (insula, anterior cingulate cortex [ACC]) regions even in the absence of current symptoms.

118 Across 26 current ACC/AHA guidelines (2930 recommendations; median, 121 re

119 C) and of the Antarctic Circumpolar Current (ACC) is accurately predicted.

120 The Antarctic Circumpolar Current (ACC) spills across the Falkland Plateau into the South A

121 north of the Antarctic Circumpolar Current (ACC) than in those restricted to Antarctic/sub-Antarctic

122 cation of the Antarctic Circumpolar Current (ACC).

123 However, descending ACC projections control fear generalization independent

124 with an optimal cut-off of 20 HU to diagnose ACC.

125 -unemotional traits) showed decreased dorsal ACC activation compared with typically developing youths

126 (e.g., amygdala) and DBD-only (e.g., dorsal ACC) groups.

127 patients had lower levels of GABA in dorsal ACC (p = .03), and the subgroup of patients with a schiz

128 thalamus (p = .01), but Glx levels in dorsal ACC and thalamus did not differ between groups.

129 te that decreased GABAergic levels in dorsal ACC are involved in schizophrenia and psychotic disorder

130 Glx levels in dorsal ACC were positively associated with working memory (loga

131 consistent with diverse roles of the dorsal ACC in motor evaluation and the ventral ACC in affective

132 heories propose that the MFC, and the dorsal ACC in particular, integrates information related to sub

133 However, dPMC input to the dorsal ACC was densest in deep layers, while amygdalar inputs p

134 In the dorsal ACC, outputs to dPMC and the amygdala strongly overlap i

135 in the voxel pattern response of the dorsal ACC/pre-SMA.

136 and p53/Rb signaling is sufficient to drive ACC in mouse.

137 Thus, dysfunctional ACC/HPC signaling to the BLA may be a predominant underl

138 s that were LOE A did not increase in either ACC/AHA (median, 9.0% [current] vs 11.7% [prior]) or ESC

139 d The Cancer Genome Atlas (TCGA) dataset for ACC and found that patients harboring alterations in bot

140 ear growth were independent risk factors for ACC.

141 The optimal size cut-off for ACC in AI was 4.6 cm.

142 ne optimal size and attenuation cut-offs for ACC.

143 minantly use tumor size to stratify risk for ACC.

144 gether, we demonstrate dissociable roles for ACC and BLA in transmitting confidence and learning unde

145 Here we correlate the occurrence of CPA from ACC precursor particles with nanoparticulate fabric and

146 that changes in attention are decoupled from ACC firing.

147 To address this issue, we recorded from ACC as rats performed a Pavlovian task that predicted wh

148 These AC-clay granules (ACC-G) were tested for remediation potential (PCB-bioacc

149 Furthermore, modulation of this S1->ACC projection regulates aversive responses to pain.

150 During HG, ACC glutamate increased (1.2 mmol/kg, 10% P = 0.014) whi

151 responses of the same cells and compare how ACC neurons respond during the same effortful behavior w

152 Here we demonstrate that the human ACC NCI-H295R cell line is remarkably sensitive to induc

153 Importantly, ACC outputs exert control by reducing the innate respons

154 We next looked for value coding cells in ACC during effortful behavior and reward consumption pha

155 me of the fMRI substudy (N=20) was change in ACC activity.

156 ab is a standard therapy, are more common in ACC than has been recognized.

157 MRS, we measured glutamate concentrations in ACC and occipital lobe cortex (OCC) in 13 subjects with

158 levels before treatment but no difference in ACC activation with testosterone compared with placebo.

159 lated negatively with tCr and tCho levels in ACC and mIns levels in hippocampus and thalamus, but not

160 tNAA and Glu (neuron-related metabolites) in ACC, DLPFC, hippocampus, and thalamus.

161 recording studies have shown that neurons in ACC are modulated by reward or shock when delivered to a

162 ask while we recorded from single neurons in ACC.

163 Neural responses in ACC were weaker in the choice compared with the no-choic

164 increases the response to noxious stimuli in ACC neurons.

165 d thalamus, total creatine (tCr) and tCho in ACC and hippocampus; lower levels of glutamate (Glu) wer

166 and hippocampus and total choline (tCho) in ACC.

167 ere observed for mIns in hippocampus, tCr in ACC and hippocampus, tCho in hippocampus, and total N-ac

168 hat SPT-enol acts on nematodes by inhibiting ACC.

169 increase after cooking), by the interactions ACC x cooking method (up to 2.1-fold increase) and genot

170 Patients with histologically confirmed R/M ACC of any primary site with radiographic and/or symptom

171 ng antitumor activity with lenvatinib in R/M ACC patients.

172 or metastatic adenoid cystic carcinoma (R/M ACC) is a malignant neoplasm of predominantly salivary g

173 se inhibitor lenvatinib in patients with R/M ACC.

174 For example, single neurons in macaque ACC selectively code reward delivery to the self, a part

175 rimentally against seven independent MeONPs (ACC = 86%).

176 exercise filling pressures), of whom 222 met ACC/AHA, 161 met ESC, and 41 met HFSA criteria.

177 Moreover, ACC, the direct precursor of ethylene, accentuates the e

178 Furthermore, SPT-enol inhibits nematode ACC activity, affects storage lipids and fatty acid comp

179 id biosynthesis by interfering with nematode ACC is a new nematicidal mode of action addressed by SPT

180 These findings uncover a novel ACC function and provide insights for unraveling new phy

181 The novel ACC-G material was compared to GAC of the same particle

182 HCO[Formula: see text]), for accumulation of ACC in vesicles.

183 ation by stimulating GPR55 and activation of ACC.

184 we tested the hypothesis that amendments of ACC in soils reshape the structure of soil microbiome an

185 Exogenous application of ACC led to BS symptoms, while ethephone application led

186 at these early calcifiers used attachment of ACC particles to form their biominerals.

187 nts a new tool to investigate the biology of ACC and to identify new treatment strategies.

188 f visual stimuli, suggesting contribution of ACC but not BLA to confidence computations.

189 Meticulous design of ACC-HNFs provided a versatile approach for constructing

190 Disruption of ACC, via either chemogenetic inhibition or excitation, r

191 ce was delayed and the promotional effect of ACC on nodal root emergence decreased.

192 n deficits, whereas selective enhancement of ACC activity, restoration of SHANK3 expression in the AC

193 Inhibition or excitation of ACC reduced effort toward the high-value option, but onl

194 levated ethylene responses and expression of ACC Synthase 7 (ACS7), which encodes a rate-limiting enz

195 revious reports, we found that the firing of ACC neurons was modulated by aversive stimuli delivered

196 use models that recapitulate the genetics of ACC has hampered progress in the field.

197 undertaken to evaluate the immunogenicity of ACC.

198 unraveling new physiological implications of ACC in plants.

199 performed unilateral excitotoxic lesions of ACC while recording downstream from the ipsilateral hemi

200 This autochthonous model of ACC represents a new tool to investigate the biology of

201 ion, as manifested by an increased number of ACC neurons that respond to S1 inputs and the magnified

202 ting theoretical predictions for the role of ACC in cognitive control.

203 nces within-treatment groups (specificity of ACC measurement) were significant for grass AIT (P < 0.0

204 ission electron microscopy (TEM), a study of ACC in wild type (WT) mice, and mice with genetic osteoa

205 vely redefine the molecular underpinnings of ACC progression and identify further targets for precisi

206 processes, with only the former depending on ACC.

207 ons, first-in-human clinical trials with our ACC inhibitor PF-05175157 led to robust reduction of de

208 Overall, ACC incidence in AI was 1.7%.

209 In ovules, ACC stimulates transient Ca(2+) elevation, and Ca(2+) in

210 whereas manipulations of adjacent perigenual ACC (pgACC, area 32) have no effect.

211 In COS-7 cells expressing moss PpGLR1, ACC induces the highest cytosolic Ca(2+) elevation compa

212 FC) versus appraisal (dorsally, in pregenual ACC).

213 These data indicate that the primate ACC is necessary for acquisition of prosocial preference

214 olism resulted in lower ethylene production, ACC oxidase activity, membrane permeability, <beta>-gala

215 tion was linked to the initiation of a proto-ACC, which occurred ~6 Ma earlier than the currently est

216 n attenuates attention and attention-related ACC signals in an intake-dependent manner, and that chan

217 In return, ACC is used by these microbes as a source of nitrogen.

218 = 221), and positively correlated with right ACC-seeded FC with the right precentral gyrus and the bi

219 Among all areas, the rostral ACC (A32) had the lowest relative density of connections

220 Silencing of H. schachtii ACC by RNAi induced similar phenotypes and thus mimics t

221 ta suggest that the identified trans-species ACC-striatal circuit relationship with nicotine dependen

222 suggest unique interactions within specific ACC compartments that are likely important for the tempo

223 of vesicular transport to provide stabilized ACC in chicken uterine fluid where eggshell mineralizati

224 across a cortico-striatal circuit (striatum, ACC, insula).

225 The STS-ACC TVT Registry (Society of Thoracic Surgeons-American

226 components of vesicular transport to supply ACC in a vertebrate model of biomineralization.

227 rcement task before and after they sustained ACC lesions.

228 Targeted (ACC-allergen = AIT-allergen) and untargeted (ACC-allerge

229 Here, we demonstrate that ACC exhibits ethylene-independent signaling in Arabidops

230 ver, to date, no study has demonstrated that ACC is necessary for this form of behavioral flexibility

231 This contemporary analysis demonstrates that ACC risk per size in AI is less than previously reported

232 e mutant with reduced seed set, we find that ACC signaling in ovular sporophytic tissue is involved i

233 We hypothesize that ACC is stabilized by ovalbumin and/or lysozyme or additi

234 of social behavior in mice and indicate that ACC dysfunction may be involved in social impairments in

235 These results reveal that ACC is a critical node in model-based control, with a sp

236 ional perturbation experiments revealed that ACC functioned downstream of DMFC and was directly and s

237 ging and optogenetic manipulations show that ACC outputs to the superior colliculus, a key midbrain s

238 The findings show that ACC-G has a significantly higher remediation potential t

239 flexibility, nor have any studies shown that ACC acts by modulating downstream brain regions such as

240 These results suggest that ACC can process information about outcomes (i.e., identi

241 Results suggest that ACC glutamate control mechanisms are disrupted in T1D, w

242 Collectively, our findings suggest that ACC is necessary for the mitigation of competing inputs

243 The ACC CVD in Women Committee undertook a review of the rec

244 The ACC is implicated in effort exertion and choices based o

245 The ACC strength initially increases due to changes in the w

246 The ACC/AHA guideline recommends its use in patients >20/10

247 xanthin content was highly influenced by the ACC (up to 631-fold change), chlorogenic acid was simila

248 First, the ACC is interconnected with prefrontal, parietal, and sub

249 Fourth, the ACC dynamically controls instrumental- and non-instrumen

250 estigate the functional projections from the ACC and vHPC to the amygdala and their role in governing

251 unctional signaling to the amygdala from the ACC or the HPC could underlie overgeneralized fear respo

252 o inhibit glutamatergic projections from the ACC or vHPC that terminate within the BLA at recent (1 d

253 However, the ACC was uniquely activated by quality, quantity, and the

254 egatively correlated with GABA levels in the ACC (N = 221), and positively correlated with right ACC-

255 Here, we measured GABA levels in the ACC and FC in a subsample (aged 10.5-13.4 years) of a la

256 Furthermore, L5 pyramidal neurons in the ACC did not exhibit dendritic Ca(2+) spikes as prominent

257 Critically, none of the monkeys in the ACC lesion group acquired social preferences with a new

258 signaling of reward, but not effort, in the ACC of freely-moving rats.

259 sis, we find that a subset of neurons in the ACC receives S1 inputs, and activation of the S1 axon te

260 Conditional knockout of Shank3 in the ACC was sufficient to generate excitatory synaptic dysfu

261 Only the fastest EPSPs in the ACC were reduced to a similar degree as in primary somat

262 three metabolite concentration values in the ACC were significantly reduced in this group.

263 ity, restoration of SHANK3 expression in the ACC, or systemic administration of an alpha-amino-3-hydr

264 rite to the soma in pyramidal neurons in the ACC, which was significantly better compared with S1.

265 MThal inputs to the pyramidal neurons in the ACC.

266 e neurons to the nociceptive response in the ACC.

267 (outputs) and axon terminals to (inputs) the ACC of adult rhesus monkeys (Macaca mulatta).

268 er than the currently estimated onset of the ACC at 30 Ma.

269 motor connections within subdivisions of the ACC is not well understood.

270 As such, other standing meanders of the ACC may also act as biological hotspots where trophic in

271 ctive, bilateral, excitotoxic lesions of the ACC, and the other half served as unoperated controls.

272 They underwent 7T (1)H-MRS of the ACC, DLPFC, hippocampus, and thalamus and performed a vi

273 nary history, following the direction of the ACC, which might have promoted dispersal and admixture.

274 A24a) and dorsal (A24b,c) subregions of the ACC, while amygdalar connections are more evenly distrib

275 did not result in greater activation of the ACC.

276 hrenia had significantly lower scores on the ACC glutathione-glutamate component, an effect almost en

277 Second, the ACC integrates diverse, behaviorally relevant informatio

278 different options.SIGNIFICANCE STATEMENT The ACC is implicated in effort-based decision-making.

279 We previously found that the ACC and ventral hippocampus (vHPC) regulate generalized

280 Our findings demonstrate that the ACC and vHPC regulate fear expressed in novel, nonthreat

281 vide direct evidence for the notion that the ACC has a role in the regulation of social behavior in m

282 Further, our data indicate that the ACC-BLA circuit supports generalization in a time-indepe

283 We also show that the ACC-related KIF21B variant independently perturbs axonal

284 Third, the ACC signals behaviorally relevant information flexibly,

285 Here, we assessed whether the ACC's contribution to social cognition is causal by test

286 ontrast, alterations in both pathways led to ACC with pulmonary metastases.

287 input from the mediodorsal thalamus (MD) to ACC, using sciatic nerve injury and chemotherapy-induced

288 r results ascribe motor and sensory roles to ACC projections to the superior colliculus and the visua

289 Cyp11b1 and upregulation of Ezh2, similar to ACC patients with a poor prognosis.

290 ACC-allergen = AIT-allergen) and untargeted (ACC-allergen != AIT-allergen) treatment effects were ass

291 rized by increases in FC between NAc-ventral ACC for environmental, NAc-thalamus for physical, and NA

292 rsal ACC in motor evaluation and the ventral ACC in affective and contextual memory.

293 In the ventral ACC, entorhinal and amygdalar connections strongly overl

294 f BLA shortens waiting times overall whereas ACC inhibition renders waiting times insensitive to conf

295 recordings suggest that a mechanism by which ACC mediates effortful decisions is in the discriminatio

296 A mechanism by which ACC regulates effortful decisions is in providing a stab

297 increased (1.2 mmol/kg, 10% P = 0.014) while ACC fALFF4 was unchanged (-0.007, -2%, P = 0.449) in the

298 e carotid wall volume and were compared with ACC/AHA risk quartiles.

299 s preference was reduced in the monkeys with ACC lesions.

300 and artificially-developed saline soils with ACC in different concentrations for 14 days.