ライフサイエンスコーパス: ADH

1 ADH (alcohol dehydrogenase) gene expression, enzyme acti

2 ADH and its cofactor nicotinamide adenine dinucleotide (

3 ADH is caused by mutations in the low-density lipoprotei

4 ADH peptides induced the production of interferon-gamma

5 ADH performed an onsite investigation at clinic A on 7 S

6 ADH performed an onsite investigation at Clinic A on Sep

7 ADH-1 enhancement of response to melphalan was associate

8 ADH-1 improved responses to regional LPAM but had variab

9 ADH-1 in combination with LPAM ILI improved antitumor re

10 ADH-1 increased vascular permeability without effecting

11 ADH-1 is a cyclic pentapeptide that disrupts N-cadherin

12 ADH-41 targets Abeta in a sequence and structure-specifi

13 ADH-41 was also effective at inhibiting the seed-catalyz

14 ADH-specific peripheral T-cell responses were assessed b

15 ADH-specific T-cell responses have not been characterize

16 tion and results in disinhibition of Class 1 ADH transcription.

17 tabolizing enzymes alcohol dehydrogenase 1B (ADH-1B; rs1229984) and alcohol dehydrogenase 1c (ADH-1C;

18 1B; rs1229984) and alcohol dehydrogenase 1c (ADH-1C; rs698).

19 odococcus ruber (ADH-A), whereas evo-1.1.200 ADH led to their counterpart (R)-enantiomers also with c

20 s, 93 were analyzed, providing data for 6458 ADHs (5911 were managed with surgical excision and 547 w

21 gical excision was performed in 66 of the 72 ADH cases and in 117 of 118 DCIS cases.

22 Similar to PBMCs in abstinents, ADH peptides induced weak T-cell proliferation and a sim

23 no-3,7-dideoxy-d-threo-hept-6-ulosonic acid (ADH) synthase, the product of the Mj0400 gene, catalyzes

24 nerates the endocrine lobe [adenohypophysis (ADH)] of the pituitary, a master gland controlling growt

25 arison of the enzyme with related aldolases, ADH synthase is classified as a new member of the class

26 ehydrogenase (either metagenomic ADH-150, an ADH from Sphingobium yanoikuyae (SyADH), or a variant of

27 Ten years after an ADH diagnosis, an estimated 5.7% (95% CI, 4.3%-10.1%) of

28 eved claudin-14 gene silencing and caused an ADH-like phenotype.

29 vating mutation in this region, V836L, in an ADH patient, we studied the remaining residues in this r

30 the absolute protein expression level of an ADH isoenzyme, ADH1C1, in human liver.

31 obility shift assays were conducted using an ADH-specific SRE site.

32 rate specificity compared with the ancestral ADH.

33 This imbalance between gene expression and ADH activity at 10 degrees C, as well as the unexpected

34 ng that ChREBP regulates EtOH metabolism and ADH activity through its direct control of sirtuin 1 exp

35 administration of the N-cadherin antagonist, ADH-1, or saline.

36 irected to alcohol dehydrogenase (ADH), anti-ADH titers being associated with disease severity and ac

37 We aimed to define anti-ADH cellular immune responses and their association with

38 Proliferative anti-ADH immune responses in alcoholic hepatitis focused on i

39 ial hypercholesterolemia 4 (FH4), defined as ADH in absence of mutations in these genes and thereafte

40 ment of highly homologous isoenzymes such as ADHs where multiple signature peptides can be examined b

41 ressed genes between DCIS-HN and 447 between ADH-HN, with >90% of the ADH-HN genes also present among

42 ced a co-ordinated regulatory action between ADH genes, especially between ADH1A and ADH1C within the

43 Only 61 genes were identified between ADH-DCIS.

44 1 (E-47 cells) were exposed to ethanol, both ADH- and CYP2E1-generated products reduced STAT1 phospho

45 and bienzymatic (anchoring sequentially both ADH and aldehyde dehydrogenase) systems were tested.

46 Employing Lactobacillus brevis ADH, it was possible to achieve the synthesis of enantio

47 Acetaldehyde generated by ADH in both liver and Schwann cells surrounding nocicept

48 ction of alpha-helicity in Abeta mediated by ADH-41.

49 Inhibition of Candida ADH enzyme using disulfiram and 4-methylpyrazole resulte

50 For the first time among characterized ADHs, the high-resolution structures of all reaction ste

51 1 to catalysis was studied by characterizing ADH with His-51 substituted with Gln (H51Q).

52 d to assess for calcifications and confirmed ADH in 101 cases with subsequent surgical excision.

53 uct or terminal duct-lobular unit containing ADH was considered a focus and counted.

54 used the mouse hepatocyte cell line (CYP2E1/ADH-transfected HepB5 cells) or primary mouse hepatocyte

55 ydrogenase (ALDH) and alcohol dehydrogenase (ADH) active sites reside at the outer surface and the in

56 because inhibition of alcohol dehydrogenase (ADH) activity blunted ChREBP EtOH-induced acetylation in

57 sic functional trait, alcohol dehydrogenase (ADH) activity in D. melanogaster, across both historical

58 ionships between some alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH) genes and alcohol

59 e catabolism in which alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH) play central role

60 cells), which express alcohol dehydrogenase (ADH) and CYP2E1.

61 the activation of the alcohol dehydrogenase (ADH) and greater anaerobic metabolism in comparison with

62 betaL crystallin and alcohol dehydrogenase (ADH) and significantly less effective than wt alphaB cry

63 e dismutase (SOD) and alcohol dehydrogenase (ADH) as protein models] showed the integrity of the Zn-b

64 (OECT) modified with alcohol dehydrogenase (ADH) as the sensor.

65 hol oxidase (AOx) and alcohol dehydrogenase (ADH) biocatalysis towards butanol-1 oxidation by incorpo

66 tigated bioanode with alcohol dehydrogenase (ADH) catalysing oxidation of glycerol and glyceraldehyde

67 enetically dissecting alcohol dehydrogenase (ADH) enzyme activity.

68 ns in hepatic Class 1 alcohol dehydrogenase (ADH) expression in ethanol-fed rats correspondent with r

69 solutely quantify the alcohol dehydrogenase (ADH) expression level in a human liver sample.

70 The alcohol dehydrogenase (ADH) family of enzymes catalyzes the reversible oxidatio

71 trameric Zn-dependent alcohol dehydrogenase (ADH) from Cupriavidus necator JMP134.

72 aimed to test whether alcohol dehydrogenase (ADH) gene variants were associated with alcohol use befo

73 in the region of the alcohol dehydrogenase (ADH) genes, affected risk for alcoholism.

74 ions of AD with seven alcohol dehydrogenase (ADH) genes.

75 l (PBMC) responses to alcohol dehydrogenase (ADH) in patients with alcohol-related cirrhosis, associa

76 s to demonstrate that alcohol dehydrogenase (ADH) is downregulated in Candida biofilms.

77 ine-51 in horse liver alcohol dehydrogenase (ADH) is part of a hydrogen-bonded system that appears to

78 Inhibition of alcohol dehydrogenase (ADH) or deletion of transient receptor potential ankyrin

79 Polymorphism in the alcohol dehydrogenase (ADH) protein of Drosophila melanogaster, like genetic va

80 yde produced from the alcohol dehydrogenase (ADH) reaction was shown to improve the linearity of NAD(

81 ll extant short-chain alcohol dehydrogenase (ADH) through retroposition, provides an opportunity to e

82 the immobilization of alcohol dehydrogenase (ADH) via Nafion entrapment, with excellent analytical ch

83 mammals catalyzed by alcohol dehydrogenase (ADH), aldehyde dehydrogenase (ALDH), flavin-containing m

84 zyme, hepatic Class I alcohol dehydrogenase (ADH), and this mechanism involves regulated CCAAT/enhanc

85 ntibodies directed to alcohol dehydrogenase (ADH), anti-ADH titers being associated with disease seve

86 nzyme family) with an alcohol dehydrogenase (ADH), applying the in situ substrate feeding product rem

87 a potent inhibitor of alcohol dehydrogenase (ADH), decreased the conversion of [3H]20-HETE to 20-COOH

88 the CYP71 clan and an alcohol dehydrogenase (ADH).

89 arboxylases (KDC) and alcohol dehydrogenase (ADH).

90 relation of maternal alcohol dehydrogenase (ADH)1B genotype (rs1229984) with these outcomes (the A a

91 zing a single enzyme, alcohol dehydrogenase (ADH)] and bienzymatic (anchoring sequentially both ADH a

92 structural changes of alcohol dehydrogenase (ADH, 4mer) under varying degrees of in-source activation

93 ependent alcohol and aldehyde dehydrogenase (ADH and AldDH) enzymes for biofuel cell applications.

94 ude any genes of the arginine dehydrogenase (ADH) pathway.

95 latinum) and enzymes (alcohol dehydrogenase, ADH; lactate dehydrogenase, LDH; xanthine oxidase, XOx;

96 haeal homotetrameric alcohol dehydrogenases (ADHs) and also to the mammalian dimeric ADHs.

97 nto retinaldehyde by alcohol dehydrogenases (ADHs) or retinol dehydrogenases (RDHs); and oxidation of

98 y of metal-dependent alcohol dehydrogenases (ADHs).

99 s encoding zebrafish alcohol dehydrogenases (ADHs).

100 s obtained here, DET using the PQQ-dependent ADH and AldDH still lacks high current density, while th

101 entiate between the methods used to diagnose ADH, which may be related to the size of the ADH focus.

102 e the risk in women with presently diagnosed ADH.

103 roups of the CWP to adipic acid dihydrazide (ADH)-derivatized protein, and (ii) binding of the carbod

104 ses (ADHs) and also to the mammalian dimeric ADHs.

105 When cells expressing either ADH (VA-13 cells) or CYP2E1 (E-47 cells) were exposed to

106 (21%), the diagnosis was upgraded to either ADH or DCIS (exact two-sided 95% CI = 11.4%, 36.4%).

107 new architecture can up-regulate endogenous ADH activity by > 20-fold in transgenic Arabidopsis.

108 on the coupled alcohol dehydrogenase enzyme(ADH).

109 Human Schwann cells express ADH/TRPA1/NOX1 and recapitulate the proalgesic functions

110 CI: 1.2, 10) more likely in mass (n = 20 for ADH and n = 20 for DCIS) than in non-mass (n = 46 for AD

111 = 20 for DCIS) than in non-mass (n = 46 for ADH and n = 97 for DCIS), compared with nonunderestimati

112 ears, met modified Simon-Broome criteria for ADH and were screened for mutations in the exons and con

113 demonstrated these sites to be essential for ADH transcription.

114 Ten variants in four ADH genes were genotyped in women from South-West Englan

115 The Lactobacillus gasseri ADH beta-glucuronidase gene, gusA, was cloned previously

116 mans have seven alcohol dehydrogenase genes (ADH) falling into five classes.

117 On the other hand, ADH-A mediated bioreduction of 4,9-dihydro-1H-carbazol-3

118 uly 2018, the Arkansas Department of Health (ADH) was notified by hospital A of 3 patients with blood

119 uly 2018, the Arkansas Department of Health (ADH) was notified by Hospital A of three patients with b

120 oncentration, the antiparallel double helix (ADH) conformation was observed to be most abundant for G

121 patient-matched histologically normal (HN), ADH, and DCIS from 12 patients with estrogen receptor po

122 of a thermophilic alcohol dehydrogenase (ht-ADH): Y25A (at the dimer interface) and V260A (at the co

123 tant thermophilic alcohol dehydrogenases (ht-ADH), presenting evidence for Arrhenius prefactor values

124 Arrhenius curves previously reported for ht-ADH are proposed to arise, at least in part, from a chan

125 dynamical transition at 30 degrees C for ht-ADH, the temperature dependence of the KIE is seen to in

126 The reciprocal mutation in ht-ADH, ht-Y25A, results in kinetic behavior similar to tha

127 esidues in the cofactor-binding pocket of ht-ADH (Leu176 and V260) have been mutated to a series of h

128 a thermolabile psychrophilic homologue of ht-ADH, ps-A25Y, leads to a more thermostable enzyme and a

129 ogenase from Bacillus stearothermophilus (ht-ADH) has been mutated at an aromatic side chain in the a

130 ation of normal Arrhenius behavior in the ht-ADH reaction occurs at elevated temperatures.

131 ced temperature alters the ability of the ht-ADH variants to sample the catalytically relevant region

132 lapping peptides representing the full human ADH protein (beta 1 subunit).

133 25 overlapping peptides, spanning the human ADH beta1 subunit, were constructed.

134 Autosomal-dominant hypercholesterolemia (ADH) is characterized by elevated low-density lipoprotei

135 ND- Autosomal dominant hypercholesterolemia (ADH), characterized by elevated plasma levels of low-den

136 a diagnosis of atypical ductal hyperplasia (ADH) (75 of 6,081 [1.2%]) were reviewed; these patients

137 erestimation of atypical ductal hyperplasia (ADH) and ductal carcinoma in situ (DCIS) at magnetic res

138 djacent foci of atypical ductal hyperplasia (ADH) in eight, and well-differentiated papillary ductal

139 Atypical ductal hyperplasia (ADH) is a known risk factor for breast cancer.

140 diagnosed pure atypical ductal hyperplasia (ADH) is an unresolved clinical issue.

141 plasia (SH) and atypical ductal hyperplasia (ADH), are candidate precursors to ductal carcinoma in si

142 breast tissue, atypical ductal hyperplasia (ADH), DCIS and invasive breast cancer.

143 tients with autosomal dominant hypocalcemia (ADH) repressed the transcription of miR-9 and miR-374 ge

144 bjects with autosomal dominant hypocalcemia (ADH), five appear at the junction of TM helices 6 and 7

145 The antigenic distance hypothesis (ADH) predicts that negative interference from prior seas

146 Products of the three class I ADH genes that share 95% sequence identity are believed

147 a classical competitive inhibitor of class I ADH, failed to inhibit ADH8A.

148 ction of optimal codons led to a decrease in ADH activity.

149 eated hepatocytes, a significant decrease in ADH protein content and/or acetylation was observed.

150 (Adh) gene led to a significant decrease in ADH protein expression.

151 ated genes and pathways were dysregulated in ADH and maintained in DCIS.

152 optimal leucine codons led to an increase in ADH activity in third-instar larvae.

153 DCIS), compared with nonunderestimation, in ADH and DCIS respectively.

154 This suggests that plasticity in ADH activity is likely influenced by many loci with smal

155 Conclusion The rates of underestimation in ADH and DCIS diagnosed at MR imaging-guided vacuum-assis

156 The EcD-based biosensor that incorporates ADH, NAD(+), Pd-NPs and Nafion showed no loss of enzyme

157 the molecular mechanism for ethanol-induced ADH expression during the UEC pulse in adult male rats f

158 Insulin inhibited ADH gene expression, and this was abolished by LY294002

159 her these results indicate that class I-like ADH is conserved in zebrafish, albeit with mixed functio

160 le alcohol dehydrogenase (either metagenomic ADH-150, an ADH from Sphingobium yanoikuyae (SyADH), or

161 times higher than the risk in women with no ADH.

162 We mapped a novel ADH locus at 4p13 and identified 4 variants in STAP1 tha

163 the other pathway leads to the activation of ADH and ACS9.

164 paucity of data about the molecular basis of ADH among ethnic groups other than those of European or

165 Here, we examined the molecular basis of ADH in a multiethnic patient cohort from lipid clinics i

166 binding of most of the substrate classes of ADH, ALDH, and CYP.

167 Combination of ADH-1 with TMZ ILI did not improve tumor response in A37

168 hen catalyzes deamination and cyclization of ADH, resulting in DHQ, which is fed into the canonical p

169 at NPS 2143 corrects the molecular defect of ADH mutations for treatment of this disease are also dis

170 included 955331 women with 1727 diagnoses of ADH, 1058 (61.3%) of which were diagnosed by core biopsy

171 ALH at CNB and in those with a diagnosis of ADH at CNB was performed (Pearson chi(2) test).

172 invasive breast cancer after a diagnosis of ADH may be lower than those previously reported.

173 Diagnosis of ADH on core needle biopsy or excisional biopsy in women

174 ted with underestimation when a diagnosis of ADH was made at MR imaging-guided biopsy.

175 A diagnosis of ADH was obtained after biopsy in 72 cases, and a diagnos

176 Ten years following a diagnosis of ADH, the cumulative risk of invasive breast cancer was 2

177 mammography with and without a diagnosis of ADH.

178 ansgenes to map the functional divergence of ADH enzyme activity in vivo, we find that amino acid sub

179 Cell-based assays to assess the effect of ADH-41 on Abeta are underway and will be presented in du

180 ranscription) reduced in vitro expression of ADH mRNA by 2-fold.

181 tly higher in cases of three or more foci of ADH (15 [28%] of 53 cases) than in cases of fewer than t

182 the direct electron exchange between heme of ADH and modified AuNPs.

183 ing proteins to mediate ethanol induction of ADH in vivo.

184 the phenotypes of cell and animal models of ADH.

185 From 1996 to 2012, the proportion of ADH diagnosed by core needle biopsy increased from 21% t

186 to ethanol vapor, the enzymatic reaction of ADH and ethanol transforms NAD(+) into NADH, which cause

187 The reversible oxidation and reduction of ADH heme proceeded at around -0.05V vs. SCE.

188 Three crystal structures of ADH synthase were determined in this work: a complex wit

189 the charge at the "unfolding" N-terminus of ADH decreased at high in-source activation energies afte

190 a-ions produced by UVPD at the N-terminus of ADH.

191 pattern of regulation is similar to that of ADH that encodes alcohol dehydrogenase, which we have re

192 No other parameters were associated with of ADH or DCIS upgrade at surgery.

193 tudied the effects of insulin and ethanol on ADH gene expression in a highly differentiated rat hepat

194 ional biopsy is supported when LCIS, ALH, or ADH is diagnosed at CNB.

195 en it was placed under anoxia; the two other ADH homologs encoded on the Chlamydomonas genome do not

196 We hypothesized that the novel pentapeptide (ADH-1), which disrupts N-cadherin adhesion, could sensit

197 Unlike BvADHbeta and other plant ADHs that are strongly inhibited by Tyr, BvADHalpha exhi

198 tion of OYE2 with a Prelog or an anti-Prelog ADH allowed the preparation of the secondary alcohols wi

199 pgrade rate of percutaneously diagnosed pure ADH.

200 tivation was not observed for charge-reduced ADH, which likely adopted compact structures that are re

201 EtOH metabolism as a consequence of reduced ADH activity.

202 lcohol dehydrogenase from Rhodococcus ruber (ADH-A), whereas evo-1.1.200 ADH led to their counterpart

203 Further, endoderm can generate a rudimentary ADH-like structure in the near absence of ectodermal con

204 Seven ADH genes exist in a segment of ~370 kb on 4q21.

205 eotide polymorphisms (SNPs) across the seven ADH genes and analyzed their association with alcoholism

206 We also found that several ADH genes and the ALDH2 gene were susceptibility loci fo

207 ] showed the integrity of the Zn-binding SOD/ADH under the OFFGEL electrophoretic conditions.

208 xpression system for Sulfolobus solfataricus ADH-10 (Alcohol Dehydrogenase isozyme 10) and its use in

209 -to-matrix adhesion molecules in human SW480-ADH colon carcinoma cells.

210 Systemic ADH-1 was associated with increased growth and activatio

211 In a preclinical animal model, systemic ADH-1 given with regional melphalan demonstrated synergi

212 e found that across all environments tested, ADH activity was largely influenced by a single QTL enco

213 ssels convert 20-HETE to 20-COOH-AA and that ADH catalyzes the reaction.

214 We conclude that ADH and DCIS share highly similar gene expression and ar

215 n D. melanogaster has also demonstrated that ADH activity is plastic in response to alcohol concentra

216 These data provide genetic evidence that ADH (but not SH) are often precursors to cancer and sugg

217 and atomic force microscopy (AFM) show that ADH-41 wholly suppresses the aggregation of Abeta at a s

218 Overall, we speculate that ADH-41 directs Abeta into off-pathway structures, and th

219 two peaks in the ADH region suggesting that ADH populations are composed of two distinct conformers.

220 The ADH and DCIS underestimation rates were 25.8% (17 of 66)

221 The ADH-ALDH pathway also governs the metabolism of retinol

222 e-nucleotide polymorphisms (SNPs) across the ADH clusters in a global sampling of 42 populations.

223 Because the ADH promoter contains two canonical sterol response elem

224 At 30 degrees C, the ADH activity (ethanol to acetaldehyde direction), increa

225 At 10 degrees C, the ADH activity increased at 20%wl and continued to rise ev

226 The limit of detection for the ADH, LDH, XOx, and GOx was equal to 0.18, 0.14, 0.0031,

227 Furthermore, the ADH-Nafion bonding for the S. cerevisiae strain was conf

228 has the highest F(st) of the 54 SNPs in the ADH cluster, and it is significantly above the mean F(st

229 -95 and other discontinuous sequences in the ADH peptide, whereas only one sequence was targeted in a

230 spectrometry data revealed two peaks in the ADH region suggesting that ADH populations are composed

231 were attributed to structural changes of the ADH 4mer.

232 Although the sequence identity of the ADH family members is relatively low (34-37%), in vitro

233 ADH, which may be related to the size of the ADH focus.

234 bium yanoikuyae (SyADH), or a variant of the ADH from Thermoanaerobacter ethanolicus (TeSADH W110A))

235 that SREBP-1 is a negative regulator of the ADH gene and may work in concert with the CCAAT/enhancer

236 lin-induced transcriptional repressor of the ADH gene.

237 genes, including the two known genes of the ADH pathway, kauB and gbuA, were found to be inducible b

238 encode enzymes for the initial steps of the ADH pathway, the potential physiological functions of th

239 tOH attenuates the antiviral function of the ADH-ALDH pathway, which suggests the possibility that Et

240 -HN and 447 between ADH-HN, with >90% of the ADH-HN genes also present among the DCIS-HN genes.

241 Functional analysis of the ADH-SREs demonstrated these sites to be essential for AD

242 mic amperometric detection of ethanol on the ADH-Nafion/NiOxNPs/GC modified electrode gives linear re

243 In this study, we defined that the ADH-ALDH pathway serves as a potent antiviral host facto

244 nd identified novel risk loci mapping to the ADH gene cluster on chromosome 4 and extending centromer

245 g protein-beta expression and binding to the ADH promoter.

246 pecificity of nuclear protein binding to the ADH-SRE site was confirmed using antibody and UV cross-l

247 o identify an endodermal contribution to the ADH.

248 In hypophysectomized rats, in which the ADH protein increased by approximately 6-fold, the nucle

249 s C already at 10%wl in a synchrony with the ADH activity, indicating a rapid response likely due to

250 y during dehydration in combination with the ADH activity.

251 fluenza vaccination were consistent with the ADH and may have contributed to findings of low VE acros

252 significantly by season, consistent with the ADH.

253 three genes are tandemly arrayed within the ADH cluster on chromosome 4 and have very high nucleotid

254 cination effects were interpreted within the ADH framework.

255 nt study, we genotyped 16 markers within the ADH gene cluster (including the ADH1A, ADH1B, ADH1C, ADH

256 We genotyped 16 markers within the ADH gene cluster and 38 unlinked ancestry-informative ma

257 Further, sequence analysis shows that these ADHs form a monophyletic group along with additional fam

258 7 cases in which there were fewer than three ADH foci and all calcifications were removed.

259 alcoholic hepatitis recognised one to three ADH peptides (SI </=5.7).

260 The in vivo binding status of SREBP-1 to ADH-SRE sites, as measured by the chromatin immunoprecip

261 ggest that additional loci may contribute to ADH, especially in understudied populations such as blac

262 vated terminal phosphate group of the CWP to ADH-derivatized protein.

263 utbreak include adding extrapulmonary NTM to ADH's reportable disease list and providing more oversig

264 Proliferation to ADH peptides (1 x 10(5) cells per well, cultured with 10

265 This suggests that Th 1 responses to ADH in ARC are induced by alcohol consumption.

266 bstantial number of lesions were upgraded to ADH and DCIS at excision.

267 A tripyridylamide, ADH-41, was identified as one of the most potent antagon

268 In turn, ADH acetylation was reduced, suggesting that ChREBP regu

269 e dehydrogenases (TyrAa /ADH) encoded by two ADH genes (BvADHalpha and BvADHbeta).

270 a plastidic arogenate dehydrogenases (TyrAa /ADH) encoded by two ADH genes (BvADHalpha and BvADHbeta)

271 N(tz)AD(+) and N(tz)ADH serve as substrates for NADase, which selectively cl

272 tored in real time, show N(tz)AD(+) and N(tz)ADH to be substrates for yeast alcohol dehydrogenase and

273 e is seen as N(tz)AD(+) is converted to N(tz)ADH, reflecting a complementary photophysical behavior t

274 remaining 60 patients (65%) had unexplained ADH.

275 tes (57%) or Hispanics (53%) had unexplained ADH.

276 s with LDLR variants, those with unexplained ADH had lower levels of LDL-cholesterol (292 +/- 47 mg/d

277 These unique ADHs have exquisite substrate specificity, unusual metal

278 Chaperone activity was determined by using ADH, insulin, and betaL-crystallin as the target protein

279 cestor with mammalian class I, II, IV, and V ADHs.

280 37, blocked activation of the CaR by various ADH mutations.

281 We then compared the in vivo ADH activities imparted by the wild-type and mutant alle

282 Surgical excision is recommended even when ADH involves fewer than three foci and all mammographic

283 most effective bioanode was fabricated when ADH was immobilized on gold nanoparticles (AuNPs) modifi

284 he upgrade rate is significantly higher when ADH involves at least three foci.

285 We aimed to define whether ADH-specific cellular immunity is present in alcoholic h

286 e based on cohorts that included women whose ADH was diagnosed before widespread use of screening mam

287 d with ADH is slightly lower for women whose ADH was diagnosed by needle core biopsy compared with ex

288 fied 4 variants in STAP1 that associate with ADH.

289 the risk of invasive cancer associated with ADH diagnosed using core needle biopsy vs excisional bio

290 The risk associated with ADH is slightly lower for women whose ADH was diagnosed

291 port here that melphalan in combination with ADH-1 significantly reduced tumor growth up to 30-fold o

292 %) of 75 (95% CI: 7.7%, 24.3%) patients with ADH, lesions were upgraded.

293 similar to the upgrade rate in patients with ADH.

294 When the bioreductions were performed with ADH-A from Rhodococcus ruber overexpressed in E. coli, n

295 95% CI, 3.0%-12.8%) compared with those with ADH diagnosed via core needle biopsy (5%; 95% CI, 2.2%-8

296 ents with LCIS or ALH and that in those with ADH was not significant (P =.88).

297 Women with ADH diagnosed on excisional biopsy had a slightly higher

298 sphate and l-aspartate semialdehyde to yield ADH.

299 The genes encoding these zebrafish ADHs have been named Adh8a and Adh8b by the nomenclature

300 Phylogenetic analysis of these zebrafish ADHs indicates that they share a common ancestor with ma