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1 peptide of the N-terminal 16 amino acids of ADP-ribosylation factor 1.
2 release of HA was unaffected by depletion of ADP-ribosylation factor 1, a small GTPase that has been
3 sing proximity biotinylation, we identify an ADP ribosylation factor 1/adaptor protein-1 (ARFRP1/AP-1
4 FGAP1, a GTPase-activating protein (GAP) for ADP-Ribosylation Factor 1, also participate in vesicle f
5 ucleotide-exchange protein (p200 or GEP) for ADP-ribosylation factors 1 and 3 (ARF1 and ARF3) that wa
6 ponsive to activation by GTP-gammaS-liganded ADP-ribosylation factor-1 (ARF-1) and can also be activa
7 n is dependent on additional factors such as ADP-ribosylation factor-1 (ARF-1) and protein kinase Cal
8 pression of a dominant-negative (DN) form of ADP ribosylation factor 1 (Arf1) (containing a mutation
9 ed with guanosine diphosphate (GDP) bound to ADP ribosylation factor 1 (ARF1) aligned in a liquid cry
10 H2 terminus of the small GTP-binding protein ADP ribosylation factor 1 (ARF1) antagonized the inhibit
11 din A or overexpression of dominant-negative ADP ribosylation factor 1 (ARF1) caused dissociation of
13 ily GTPase Arf79F, the Drosophila homolog of ADP ribosylation factor 1 (ARF1), essential for clathrin
14 of two subcomplexes: the membrane-targeting, ADP ribosylation factor 1 (Arf1):GTP-binding betagammade
15 -terminal fragment of adenosine diphosphate (ADP) ribosylation factor 1 (ARF1) is proposed to be invo
16 e AP-3 activation by adenosine di-phosphate (ADP)-ribosylation factor 1 (Arf1), a small guanosine tri
19 e assembly is regulated by the small GTPases ADP-ribosylation factor 1 (ARF1) and Sar1, respectively.
21 ns, both reactions are greatly stimulated by ADP-ribosylation factor 1 (ARF1) but not by the GDP-boun
22 se-activating protein (GAP) that deactivates ADP-ribosylation factor 1 (ARF1) during the formation of
23 behavior of the N-terminal fragment of human ADP-ribosylation factor 1 (ARF1) in a membranelike envir
24 d from cytosol onto the TGN by myristoylated ADP-ribosylation factor 1 (ARF1) in the presence of the
32 tudies defined the STAU1-binding site within ADP-ribosylation factor 1 (ARF1) mRNA as a 19-base-pair
33 e loss of the small guanosine triphosphatase ADP-ribosylation factor 1 (Arf1) or its effector, phosph
34 (ER) and the Golgi complex, the small GTPase ADP-ribosylation factor 1 (ARF1) recruits a cytosolic co
35 pecifically associated with the small GTPase ADP-ribosylation factor 1 (Arf1) to mediate uniform dist
36 the finiteness of the cyclical activation of ADP-ribosylation factor 1 (Arf1), a fundamental step in
38 rate that chemokine receptor CXCR4 activates ADP-ribosylation factor 1 (ARF1), a small GTPase importa
39 ergic receptor (alpha(2B)-AR) interacts with ADP-ribosylation factor 1 (ARF1), a small GTPase involve
40 nal domain (NTD), which binds and stabilizes ADP-ribosylation factor 1 (ARF1), a small GTPase regulat
41 anner by protein kinase c-alpha (PKC-alpha), ADP-ribosylation factor 1 (ARF1), and Rho family members
42 c manner in vitro by protein kinase C-alpha, ADP-ribosylation factor 1 (ARF1), and Rho family members
44 FGAP1, a GTPase-activating protein (GAP) for ADP-ribosylation factor 1 (ARF1), couples to either BARS
45 and cell biological evidence for the role of ADP-ribosylation factor 1 (ARF1)-GTPase and its effector
48 Here we have investigated the role of the ADP-ribosylation factor-1 (ARF1) in this process to dete
49 nd inhibits the GTPase activating protein of ADP-ribosylation factor 1 (ARFGAP1), suggesting that QS1
51 branes by expressing a constitutively active ADP-ribosylation factor 1 mutant arrests CFTR within dis
52 (GTPase-activating protein that inactivates ADP-ribosylation factor 1), reduces seizure threshold, a
53 anosine 5'-O-(3-thio triphosphate)-activated ADP-ribosylation factor-1, Rho family GTP-binding protei
54 ion was unaffected by cytosolic depletion of ADP-ribosylation factor 1, suggesting that HA sorting re