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1                                              AHC and AC+AH has similar adjusted mortality at 1-y, but
2                                              AHC is usually a sporadic disorder and has unknown etiol
3                                              AHC treatment showed evidence of plaque stabilization as
4                                              AHC was associated with lower 3- and 5-y post-LT surviva
5                  During the study period, 34 AHC outbreaks were reported in 20 counties of 9 cities i
6                                     Although AHCs provide an important rescue option, when regular re
7                                           An AHC outbreak dataset from January 2007 to December 2016
8                                           An AHC was used in 33% of retransplantations.
9 h LT, of whom 1900 patients (9%) received an AHC.
10 es the transcriptional activity of SF-1, and AHC mutants of DAX-1 lose repression function.
11 s, the survival probabilities of AC, AH, and AHC were compared by Kaplan-Meier survival analysis.
12 different responses of patients with CHC and AHC to pegIFN-alpha therapy.
13 ients compared with healthy subjects (YH and AHC combined).
14 ations in the human DAX1 gene (also known as AHC) cause the X-linked syndrome AHC, a disorder that is
15  that bridge the phenotypic spectrum between AHC and FHM syndromes, supporting a possible common path
16 ify ICAM-1 as an essential receptor for both AHC-causing and non-AHC strains.
17 rch or the effects of remedies undertaken by AHCs.
18                Therapy of acute hepatitis C (AHC) has not yet been standardized and several issues ar
19 reatment of patients with acute hepatitis C (AHC) is more effective, with sustained virologic respons
20 tients manage to overcome acute hepatitis C (AHC) spontaneously.
21 a cohort of patients with acute hepatitis C (AHC), analyzing epidemiological, clinical and virologica
22 outcomes of patients with acute hepatitis C (AHC).
23 ients (alcoholic cirrhosis with hepatitis C [AHC]).
24 culate in humans, only variant strains cause AHC for reasons that are yet unknown.
25  the involvement of the anterior horn cells (AHC) in the early post-stroke period using histomorpholo
26 bility of impairment of anterior horn cells (AHC-s).
27                 Loss of auditory hair cells (AHCs) is a major cause of human deafness.
28 -level presence for academic health centers (AHCs), minority-serving institutions (MSIs), health prof
29 search conducted in academic health centers (AHCs).
30         Alternating hemiplegia of childhood (AHC) is a disorder that can result from pathogenic varia
31         Alternating hemiplegia of childhood (AHC) is a neurodevelopmental disorder with no disease-mo
32         Alternating hemiplegia of childhood (AHC) is a rare, severe neurodevelopmental syndrome chara
33         Alternating hemiplegia of childhood (AHC) is typically distinguished from familial hemiplegic
34 COS and alternating hemiplegia of childhood (AHC), and for whom two distinct pathogenic de novo varia
35 (FHM2), alternating hemiplegia of childhood (AHC), and rapid-onset dystonia parkinsonism (RDP) are ca
36       Agglomerative hierarchical clustering (AHC) and principal component analysis (PCA) were used.
37 ) and agglomerative hierarchical clustering (AHC) were applied to understand interrelationships betwe
38 ive) and into subgroups with the most common AHC variants (D801N, E815K, G947R, and other).
39 Ns under the Accountable Health Communities (AHC) program from June 5, 2018, to January 31, 2022.
40 me of the left amygdala-hippocampal complex (AHC) was lower in the first-episode group (4.3 cm3 [0.6]
41 eduction of the anomalous Hall conductivity (AHC) above 90 K linearly follows the disappearance of th
42 states, enhance anomalous Hall conductivity (AHC) away from the magic angle.
43 nerate a strong anomalous Hall conductivity (AHC) due to a large Berry curvature that scales with the
44              Although aortohepatic conduits (AHCs) provide an effective technique for arterialization
45 human X-linked adrenal hypoplasia congenita (AHC) and hypogonadotropic hypogonadism (HH).
46 ause X-linked, adrenal hypoplasia congenita (AHC) and hypogonadotropic hypogonadism (HH).
47  sex reversal, adrenal hypoplasia congenita (AHC) critical region on the X chromosome, gene 1] is an
48                Adrenal hypoplasia congenita (AHC) is an X-linked disorder that typically presents wit
49       X-linked adrenal hypoplasia congenita (AHC) with hypogonadotropic hypogonadism was recently sho
50 order X-linked adrenal hypoplasia congenita (AHC), which resembles the phenotype of SF-1-deficient mi
51 lt in X-linked adrenal hypoplasia congenita (AHC).
52           Acute haemorrhagic conjunctivitis (AHC) outbreaks are reported frequently in China.
53            Acute hemorrhagic conjunctivitis (AHC) is a painful, contagious eye disease, with millions
54 tically confirmed; and non-demented control (AHC) patients N = 27.
55  of non-demented apparently healthy control (AHC) cases.
56 stals, including the anomalous Hall crystal (AHC) with nonzero Chern number.
57 hree donor-acceptor azahelical coumarins (DA-AHCs), namely, H-AHC, Me-AHC, and Ph-AHC, were rationall
58                                 All three DA-AHCs are shown to display very high fluorosolvatochromic
59 rgest absolute magnitude miR-15/16-dependent AHC peak.
60 oding Ahch, the mouse homologue of DAX1 (DSS-AHC Region on Human X Chromosome, Gene1) which is the ge
61 as been made towards identifying the elusive AHC mechanoreceptor channel.
62 b induces a high SVR in chronically evolving AHC patients.
63 th the CT genotype with chronically evolving AHC.
64 one inherited mutation in a case of familial AHC.
65 ggests a potential one-time PE treatment for AHC and establishes the ability of PE to rescue a neurol
66 or azahelical coumarins (DA-AHCs), namely, H-AHC, Me-AHC, and Ph-AHC, were rationally designed and sy
67 g in the excited state promotes quenching (H-AHC), (ii) efficient crystal packing promotes high emiss
68                            Of those, 647 had AHC (HCV + alcohol), 353 had AH, and 7369 had AC.
69                                  A-type HCs (AHC) are axonless and extensively coupled via connexin (
70 d increasingly provide new insights into how AHCs are programed and maintained.
71 nce disorders are also shedding light on how AHCs are specified and, maintained and handle ion flux.
72                           Mutations in human AHC cause X-linked, adrenal hypoplasia congenita (AHC) a
73  are in the form of alkyl hydroxycinnamates (AHCs), with alkyl caffeates predominating.
74 uencing of crosslinking immunoprecipitation (AHC) combined with gene expression profiling in normal a
75 in the DAA era compared with 2009 to 2013 in AHC, but remained worse in AHC group versus AC and/or AH
76        This may underlie the SUD etiology in AHC.
77   Malignancy-related mortality was higher in AHC (15% versus 9.3% in AC) in the DAA era.
78 ; P = 0.0004) mortality rates were higher in AHC.
79  that did not respond to therapy, but not in AHC.
80 th ALD without HCV and the worse outcomes in AHC group continued in the DAA era.
81 t the low frequency dispersion of phonons in AHC, despite the presence of Berry curvature, resembles
82 FN alpha-2b alone or with ribavirin (RBV) in AHC patients.
83 etermine the types of mutations resulting in AHC and to locate single-amino-acid changes in a DAX1 st
84 greater overshoot was observed in YH than in AHC (21 +/- 8 %HbO(2) vs. 10 +/- 5 %HbO(2); p = 0.0116).
85 h 2009 to 2013 in AHC, but remained worse in AHC group versus AC and/or AH.
86 ers perceive clinical research activities in AHCs to be less healthy, of poorer quality, and facing g
87 d the challenges facing clinical research in AHCs to be urgent or extremely urgent.
88 nd quantum tuning of Berry curvature induced AHC via external tuning of magnetic order.
89 ge followed by increase in size of the large AHC-s with a reduction of their synaptic coverage.
90 dents by US Census division and county-level AHCs, MSIs, HPSAs, and rurality.
91  independently associated with self-limiting AHC (odds ratio, 5.3; 95% confidence interval, 1.1-26.5)
92 ents with the CC genotype with self-limiting AHC had a stronger (P = .02) and broader (P = .013) CMI
93        Eighteen patients had a self-limiting AHC that was associated with female sex (P = .028), olde
94                                Self-limiting AHC was independently associated with CC genotype.
95                                         Many AHCs do not have policies or mechanisms to address chall
96 lical coumarins (DA-AHCs), namely, H-AHC, Me-AHC, and Ph-AHC, were rationally designed and synthesize
97 t crystal packing promotes high emission (Me-AHC) by precluding deactivations via vibrational motions
98  were less represented in counties with more AHCs (mean [SD] RQ, 0.00 [0.06]; P < .001).
99                                Notably, most AHC cases are caused by one of seven recurrent ATP1A3 mu
100 ential receptor for both AHC-causing and non-AHC strains.
101 er spread worldwide, accounting for numerous AHC outbreaks and two pandemics.
102  has been deleted in all naturally occurring AHC deletion mutants described to date.
103 r N-CoR to SF-1, whereas naturally occurring AHC mutations of DAX-1 permit the SF-1-DAX-1 interaction
104 g an Na(+)/K(+) ATPase subunit, cause 70% of AHC cases.
105 ovo ATP1A3 mutations as the primary cause of AHC and offers insight into disease pathophysiology by e
106 24v) emerged, which is now the main cause of AHC.
107 erin23 has been identified as a component of AHC stereocilia tip links, and progress has been made to
108  be associated with a self-limited course of AHC in HIV(+) patients.
109 iller (NK) cells in modulating the course of AHC in HIV(+) patients.
110 ents with chronic and self-limited course of AHC.
111 he effective reproduction number (R(eff)) of AHC.
112 udied two kindreds with clinical features of AHC and HHG.
113 es exemplify the phenotypic heterogeneity of AHC/HHG, and suggest that DAX-1 mutations impair gonadot
114  of Ahch, we have generated a mouse model of AHC-HH that allows the function of Ahch to be examined i
115 X-1 may account in part for the phenotype of AHC.
116                      The transmissibility of AHC is high in small-scale outbreaks in China.
117             However, the transmissibility of AHC remains unclear.
118                           The mean R(unc) of AHC outbreaks was 8.28 (95% CI: 6.46-10.11).
119 s of suberin-associated waxes in the form of AHCs.
120 ntation in counties with greater presence of AHCs (mean [SD] RQ, 0.77 [0.04]; P = .007).
121 he published evidence of outcome and risk of AHCs in LT using bibliographic databases and following t
122 es (cells clustered together) than non-AD or AHC cell lines.
123             On the basis of these parameters AHC classified samples into three main groups, with acce
124 hat cause rapid-onset dystonia-parkinsonism, AHC-causing mutations in this gene caused consistent red
125 ntigenic construct expressing three peptides AHC (ApoB100, HSP60 and outer membrane protein of chlamy
126 ins (DA-AHCs), namely, H-AHC, Me-AHC, and Ph-AHC, were rationally designed and synthesized, and their
127 count for low quantum yields of emission (Ph-AHC).
128 , followed by five oral dosing with purified AHC or ovalbumin on alternate days and continued on HFD
129                                    Recently, AHC was shown to be caused by mutations in DAX-1, a prot
130                    Mammals cannot regenerate AHCs, but birds and other lower vertebrates can.
131                                    The right AHC showed a similar pattern (absolute volumes 4.5 cm3 [
132           In contrast, a comparatively small AHC is observed in noncollinear antiferromagnets.
133  at-risk individuals with particularly small AHC or thalami are most likely to develop schizophrenia,
134        Mutation analysis in classic sporadic AHC patients and in an additional five kindreds in which
135 and 3) age-matched healthy control subjects (AHC) (n = 8; mean ABI 1.1 +/- 0.1, mean age 68 +/- 9 yea
136                        Fifty-six symptomatic AHC patients were enrolled and followed prospectively.
137 so known as AHC) cause the X-linked syndrome AHC, a disorder that is associated with hypogonadotropic
138 e as crystal geometry shifts, revealing that AHC can become "soft" under certain conditions.
139                                      For the AHC in RMG, we obtain estimates of phonon speed and shea
140 009) survival was significantly lower in the AHC compared to the nonconduit group.
141              We demonstrate further that the AHC below T(N) results from the nonvanishing Berry curva
142 alth-related social needs assessed under the AHC program included housing stability, housing quality,
143                       Interestingly, a total AHC of 2,800 Omega(-1) cm(-1) is observed at ~45 K, i.e.
144 d Atp1a3 mutations in human cells and in two AHC mouse models.
145 cts Atp1a3 D801N and E815K in the CNS of two AHC mouse models, yielding up to 48% DNA correction and
146                                        While AHC is primarily a neurologic disease, some individuals
147 Among the 222 individuals included (148 with AHC and 74 control), the mean (SD) age at diagnostic ele
148                              The cohort with AHC consisted of 148 largely unrelated probands (mean [S
149 001) were higher in AC + AH as compared with AHC.
150 ations identified among our 17 families with AHC brought the total number of families with AHC to 48
151 HC brought the total number of families with AHC to 48 and the number of reported mutations to 42; 1
152                 We studied 120 patients with AHC (64 male; 37 +/- 16 years old) and 96 healthy indivi
153            Twenty-seven HIV(+) patients with AHC (self-limited course: n = 10; chronic course: n = 17
154 level of IP-10 was lower among patients with AHC and spontaneous clearance (764 [113-2470] pg/mL) tha
155 used exome sequencing of seven patients with AHC and their unaffected parents to identify de novo non
156 CS3 between liver samples from patients with AHC and those with CHC.
157  altered in liver samples from patients with AHC compared with controls.
158 notype was more frequent among patients with AHC than controls (62.5% vs 39.6%; P < .001) and among p
159 2 centers across 10 countries, patients with AHC were grouped by ATP1A3 variant status (positive vs n
160 er transplantation outcomes of patients with AHC when compared with those with alcoholic cirrhosis (A
161 and SNPs in IL28B can identify patients with AHC who are most likely to undergo spontaneous clearance
162 rdiomyocytes were derived from patients with AHC who were positive for the D801N variant of ATP1A3 (i
163 nt for the greater response of patients with AHC, compared with those with CHC, to treatment with peg
164 pression in liver samples from patients with AHC, patients with CHC, and individuals without hepatiti
165 analysis of ATP1A3 in 98 other patients with AHC, we found that ATP1A3 mutations were likely to be re
166  induced in liver samples from patients with AHC, whereas expression of IFN-alpha-stimulated genes wa
167 ked immunosorbent assays of 62 patients with AHC.
168 respond to therapy, but not in patients with AHC.
169 ion occurred in 7% (3%-16%) of patients with AHCs, compared to 2% (1%-3%) without conduit (OR 3.70; 1

 
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