戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              AHO is caused by heterozygous inactivating mutations in
2  from four crosses (B73 x OH43, Mo17 x A632, AHO x A632, Latente x A632) revealed that alleles of the
3  region (Gsalpha R258W) was identified in an AHO patient.
4 d their regulatory mechanisms in control and AHO-treated tobacco leaves.
5 n different patterns of aqueous angiographic AHO improvement whose further understanding may advance
6 regions of baseline low or high angiographic AHO in each eye (n = 2 eyes with enough space to place t
7 ablished initial baseline nasal angiographic AHO patterns.
8 line, all eyes showed segmental angiographic AHO patterns.
9 tilized to query alterations to angiographic AHO patterns.
10 ed equivalent results when comparing another AHO mutation in Galphas (D173N) with a counterpart cance
11  skeletal scintigraphy shows major-long-bone AHO if treatment response is slow.
12 und in 3 (6%) of 48 cases of major-long-bone AHO.
13 ent G proteins appeared associated with both AHO and cancer.
14 that both mRNA and miRNA regulation enhances AHO-induced SAR.
15                           Qualitatively, few AHO pathways were seen at baseline that improved at foll
16                3-Acetonyl-3-hydroxyoxindole (AHO) induces systemic acquired resistance (SAR) in Nicot
17  affinity leading to the loss-of-function in AHO whereas Galphao-R243H has a mild decrease in nucleot
18 nism might explain the variable phenotype in AHO.
19         Self-reported unfavorable modifiable AHO concerning lifestyle and psychosocial health are in
20           Compared with a low-risk group (no AHO; n = 446) and with exception of neurotoxicity, this
21          In no instance was the diagnosis of AHO indicated only by MRI.
22 ing 79 (92%) of 86 with a final diagnosis of AHO.
23 us, AA demonstrated sustained improvement of AHO for the first time after effective IOP lowering from
24 understanding may advance basic knowledge of AHO and possibly enhance intraocular pressure reduction
25 which was the other preponderant location of AHO.
26 beculotomy, there was an increased number of AHO branches (p=0.016) and branch junctions (p=0.02).
27 l scintigraphy because of the possibility of AHO.
28 previously described clinical case report of AHO.
29 at the same mutation can cause either POH or AHO was observed within a single family, in which the ph
30 ion in Albright's hereditary osteodystrophy (AHO) and a recent report of two patients with AHO who ha
31       In Albright Hereditary Osteodystrophy (AHO) heterozygous loss of function mutations of GNAS can
32 esity in Albright hereditary osteodystrophy (AHO) patients, but only when the mutations occur on the
33 s of the Albright hereditary osteodystrophy (AHO) phenotype.
34        Albright's Hereditary Osteodystrophy (AHO) was the first inherited disease associated with los
35          Albright hereditary osteodystrophy (AHO), a disorder characterized by skeletal abnormalities
36          Albright hereditary osteodystrophy (AHO), an autosomal dominant disorder characterized by sh
37  suspected acute hematogenous osteomyelitis (AHO) with nondiagnostic radiographs.
38 is study, we compared aqueous humor outflow (AHO) pathway patterns using aqueous angiography before c
39 to underlie increased aqueous humor outflow (AHO) resistance, which leads to elevated ocular pressure
40 I should be strongly considered after pelvic AHO is detected, and MRI might be substituted diagnostic
41 for suspected radiographically occult pelvic AHO.
42  of the association of abscesses with pelvic AHO, however, the use of MRI should be strongly consider
43 r puncture model, we determined that reduced AHO altered the fate of SC both during development and u
44        It has remained unclear why only some AHO patients present with multihormone resistance and wh
45 n 146 (69%) of all cases and 46 (53%) of the AHO cases.
46                                    In total, AHO treatment led to 17 up- and 6 down-regulated at leas
47 tal scintigraphy is the first test used when AHO is suspected but radiographs are negative remains hi
48 present with multihormone resistance and why AHO patients demonstrate resistance to some hormones [e.
49 expressed genes (DEGs) at least 2 folds with AHO treatment.
50  so resembles the ossification observed with AHO.
51 HO) and a recent report of two patients with AHO who had atypically extensive heterotopic ossificatio