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1                                              AITC and PEITC exhibited similar stability in water and
2                                              AITC directly binds to multiple cysteine residues of the
3                                              AITC induced concentration-dependent dilation of pressur
4                                              AITC induced mitochondrion-mediated apoptosis, as shown
5                                              AITC inhalation evoked atropine-sensitive bradycardia in
6                                              AITC is widely used experimentally as an inducer of acut
7                                              AITC turned out to be the best stimulus because the coug
8                                              AITC-evoked tachycardia in decerebrate SH rats was aboli
9                                              AITC-induced dilation was attenuated by disruption of th
10                                              AITC-induced dilation was insensitive to nitric oxide sy
11                                              AITC-induced smooth muscle cell hyperpolarization was bl
12                                 The aberrant AITC-evoked reflex in SH rats was not reduced by acute b
13 sodium leak channel, nonselective), ablating AITC detection by nociceptors.
14                                Capsaicin and AITC increased the numbers of warm-responding neurons an
15 caused by c-Jun N-terminal kinase (JNK), and AITC activates JNK.
16 ted based on their structural properties and AITC molecular characteristics.
17 lth promoting compounds such as sinigrin and AITC demonstrates that besides extending shelf life and
18 within 24h while the content of sinigrin and AITC increased post irradiation and thereafter remained
19 1 is a locus for cross-sensitization between AITC and acidosis in nociceptive neurons.
20              Inhibition of caspase-9 blocked AITC-induced apoptosis.
21  in G(1) phase by hydroxyurea abrogated both AITC-induced mitotic arrest and Bcl-2 phosphorylation.
22 the mechanism underlying TRPV1 activation by AITC remains unknown.
23                            Mitotic arrest by AITC was associated with increased ubiquitination and de
24 reover, we found that apoptosis induction by AITC depended entirely on mitotic arrest and was mediate
25 o partially supported activation of TRPA1 by AITC.
26  responses to the algesic markers capsaicin, AITC and alpha, beta-methylene ATP.
27                                 By contrast, AITC pretreatment restored the distribution and expressi
28 C release, a decreased fat content decreased AITC levels in the particles and increased the amount of
29  HC subjects and the increased threshold for AITC after capsaicin treatment in patients with IR demon
30                      The lower threshold for AITC based on NMPs in patients with IR compared with HC
31                                 Furthermore, AITC acts in a membrane-delimited manner and induces a s
32  as either type I (menthol-like) or type II (AITC-like), and provide a kinetic model that faithfully
33 e strong correlation between the increase in AITC threshold in patients with IR and symptom reduction
34 bited the cardiac reflexes evoked by inhaled AITC but not injected AITC.
35                          Conversely, inhaled AITC in conscious SH rats evoked complex brady-tachycard
36 dly rectifying K(+) channels, also inhibited AITC-induced dilation.
37 exes evoked by inhaled AITC but not injected AITC.
38  reflexes evoked by inhaled but not injected AITC.
39 ypertension and is not evoked by intravenous AITC.
40                    Surprisingly, intravenous AITC only evoked bradycardia in conscious SH and WKY rat
41 he broad TRPA1 agonist allyl isothiocyanate (AITC) (an active ingredient of tear gas and wasabi), cat
42  allicin (garlic), and allyl isothiocyanate (AITC) (mustard oil).
43 a solution model using allyl isothiocyanate (AITC) and also determines the bioaccessibility and bioav
44                Inhaled allyl isothiocyanate (AITC) evoked atropine-sensitive bradycardia with atrial-
45  controlled release of Allyl isothiocyanate (AITC) from mustard seed was designed.
46 s similar to equimolar allyl isothiocyanate (AITC) in the nominal absence of calcium, suggesting a di
47 unted for the enhanced allyl isothiocyanate (AITC) in the volatile oils of the irradiated vegetable.
48 ed release of volatile allyl isothiocyanate (AITC) molecules.
49                        Allyl isothiocyanate (AITC) occurs in many commonly consumed cruciferous veget
50 ilic compounds such as allyl isothiocyanate (AITC) through covalent modification or activated by nonc
51 d (13)C NMR spectra of allyl isothiocyanate (AITC) were measured, and the exchange dynamics were stud
52 luding cinnamaldehyde, allyl isothiocyanate (AITC), and 4-hydroxynonenal, increased [Ca(2+)](i) in my
53 hese plants - menthol, allyl isothiocyanate (AITC), and capsaicin, respectively - at concentrations f
54 gent chemicals such as allyl isothiocyanate (AITC), cinnamaldehyde, and allicin, produce nociceptive
55          The effect of allyl isothiocyanate (AITC), in combination with low temperature (10 degrees C
56 chloride, pH 3.5), and allyl isothiocyanate (AITC).
57 of the noxious stimuli allyl isothiocyanate (AITC).
58                        Allyl isothiocyanate (AITC; aka, mustard oil) is a powerful irritant produced
59 the chemical irritants allyl isothiocyanate (AITC; also known as mustard oil) or capsaicin.
60  tomatoes by releasing allyl-isothiocyanate (AITC) from mustard seeds at room temperature, and to dis
61  component of mustard, allyl-isothiocyanate (AITC), activates the extreme cold receptor transient rec
62 cin (TRPV1 agonist) or allyl-isothiocyanate (AITC, TRPA1 agonist) elicited responses in only 16% and
63 aging, TRPA1 agonists allyl isothiocyanates (AITC) and hydrogen peroxide were observed to induce a tr
64 stability of four ITCs, including allyl ITC (AITC), phenethyl ITC (PEITC), erucin (ERN), and sulforap
65 ing or small intestinal transit, but luminal AITC inhibited colonic transit via TRPA1.
66                                     Menthol, AITC, and capsaicin also elicited robust calcium respons
67 ith vehicle, 1 mg norepinephrine/kg, or 5 mg AITC/kg.
68 ersal eating monitor in a test meal.In mice, AITC administration induced a 32% increase in energy exp
69       An optimum concentration of 0.05muL/mL AITC was found to be effective in maintaining the microb
70 udy was to evaluate the potential of mustard AITC to induce thermogenesis (primary outcome) and alter
71 essibility for both mycotoxins and mycotoxin-AITC conjugates, with duodenal fractions representing 63
72                            Administration of AITC by gavage did not alter gastric emptying or small i
73 in the particles and increased the amount of AITC in the headspace.
74      The highest inhibitory concentration of AITC was found in Day 3 (175.18 ppb).
75 structural flexibility of every conformer of AITC, the analysis provides a general explanation for th
76 or the cross-sensitization of the effects of AITC and low pH.
77                              The efficacy of AITC in extending the shelf life of minimally processed
78 es is proposed, which describes formation of AITC and its partitioning between the compartments of th
79 27 demonstrated dose-dependent inhibition of AITC-induced flinching in rats, validating its utility a
80 cious SHs, while inhalation and injection of AITC caused similar bradypnoea in conscious SH and WKY r
81                     Intravenous injection of AITC evoked bradycardia but no tachycardia/PVCs in consc
82  tomatoes inoculated without the presence of AITC developed a moldy aroma on the third day of storage
83  that tomatoes inoculated in the presence of AITC did not develop the microorganisms until the eighth
84 nel as mediator of the algesic properties of AITC.
85 n conversion to AITC and a higher release of AITC in the headspace.
86  external stimulus to trigger the release of AITC molecules encapsulated in MOFs.
87                  In contrast, the release of AITC molecules from all these MOFs was triggered under h
88 nd mustard seeds on formation and release of AITC was investigated and the underlying mechanisms were
89                    The conformation space of AITC contains three minima, Cs-M1 and enantiomers M2 and
90 reshold for evoking changes in NMPs based on AITC was significantly lower for patients with IR compar
91 vertheless, anaesthesia had little effect on AITC-evoked respiratory reflexes.
92     The fat content has an important role on AITC release, a decreased fat content decreased AITC lev
93   Overexpression of a Bcl-2 mutant prevented AITC from inducing apoptosis.
94 that these MOFs could encapsulate and retain AITC molecules within their pores under low (30-35%) rel
95 large region of the potential energy surface AITC(gamma,epsilon,...) with 120 degrees < gamma < 180 d
96             Here we show that, surprisingly, AITC-induced activation of TRPV1 does not require intera
97                     These data indicate that AITC acts through reversible interactions with the capsa
98 tic analysis of channel gating revealed that AITC acts by destabilizing the closed channel, whereas m
99                            Here we show that AITC (allyl isothiocyanate; mustard oil) and menthol rep
100                           Here, we show that AITC arrests human bladder cancer cells in mitosis and a
101                       We further showed that AITC-induced Bcl-2 phosphorylation was caused by c-Jun N
102                        Our data suggest that AITC inhalation in SH rats evokes de novo adrenergic ref
103                             In addition, the AITC-insensitive highveld mole-rat exhibited overexpress
104 ysis, the AITC surface exchange rate and the AITC fat solubility, an overall picture of the factors i
105  the results of the sinigrin hydrolysis, the AITC surface exchange rate and the AITC fat solubility,
106 erall picture of the factors influencing the AITC release from the particles is proposed, which descr
107 ng Ag-TA-5 could meet the requirement of the AITC Test T110-2007 and was comparable to commercial adh
108 ese MOFs, indicating that development of the AITC-MOFs delivering system is technically feasible.
109 pothesis that water vapors could trigger the AITC release from these MOFs, indicating that developmen
110 cles resulted in more sinigrin conversion to AITC and a higher release of AITC in the headspace.
111  increased the threshold for the response to AITC at 4 and 12 weeks compared with placebo (P = .0406
112 sensitive to TRPV1 (capsaicin) and/or TRPA1 (AITC) agonists.
113 lability of reaction products (alpha-ZOL/ZEA-AITC) were lower than 42.13%, but significantly higher t
114 icity and estrogenic effect of alpha-ZOL/ZEA-AITC.

 
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