戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              AITD and PA were associated bidirectionally (P <= 8 x 10
2                                              AITD was causally associated with impaired erythropoiesi
3                                              AITD was causally associated with increased erythrocyte
4                                              AITDs and rheumatologic disorders have significant commo
5 n some people might actually protect against AITD in others, depending on which additional risk varia
6 he FCRL3 risk variant was protective against AITD.
7         Multipoint analysis, designating all AITD sib pairs as affected, showed a peak NPL score of 3
8 e was at least 1 member who had both SLE and AITD (Graves' disease or Hashimoto thyroiditis).
9 bors a susceptibility gene shared by SLE and AITD.
10                              Because T1D and AITD are individually strongly associated with different
11                                      T1D and AITD frequently occur together in the same individual, a
12  confer joint susceptibility to both T1D and AITD in the same individual (APS3v).
13 between specific rheumatologic disorders and AITDs and manifestations of AITDs that mimic rheumatolog
14 usion, the bidirectional association between AITD and PA suggests a shared heritability for these two
15  interact with one another to cause clinical AITD.
16 ave (1) a genetic susceptibility to clinical AITD, along with (2) a separate predisposition to develo
17                  Autoimmune thyroid disease (AITD) and pernicious anemia (PA) often coexist, but the
18 ased the risk of autoimmune thyroid disease (AITD) in the RA patients, whereas the FCRL3 risk variant
19                  Autoimmune thyroid disease (AITD) is a common autoimmune disease.
20 ften detected in autoimmune thyroid disease (AITD), but the mechanisms underlying lymphocyte entry an
21                  Autoimmune thyroid disease (AITD), including Graves' disease (GD) and Hashimoto's th
22 pondylitis (AS), autoimmune thyroid disease (AITD), multiple sclerosis (MS) and breast cancer (BC).
23 abetes (T1D) and autoimmune thyroid disease (AITD).
24  a high risk for autoimmune thyroid disease (AITD).
25 ngly linked with autoimmune thyroid disease (AITD).
26 rs affected with autoimmune thyroid disease (AITD).
27                 Autoimmune thyroid diseases (AITD) affect 2-5% of the population, while thyroid cance
28                 Autoimmune thyroid diseases (AITD) arise from complex interactions between genetic, e
29             The autoimmune thyroid diseases (AITD), Graves' disease and chronic lymphocytic thyroidit
30 erring risk for autoimmune thyroid diseases (AITD).
31                 Autoimmune thyroid diseases (AITDs) are highly prevalent, affecting 1% to 5% of the p
32             The autoimmune thyroid diseases (AITDs) include two related disorders, Graves disease (GD
33             The autoimmune thyroid diseases (AITDs), comprising Graves disease (GD) and Hashimoto thy
34 architecture of Autoimmune Thyroid Disorder (AITD).
35                Autoimmune thyroid disorders (AITDs) are the most common organ-specific autoimmune dis
36 to express HLA-DR molecules harboring either AITD susceptibility or resistance DR pocket sequences.
37 ncluded that Tg is a susceptibility gene for AITD, both in humans in and in mice.
38 tic variant in CTLA4 that increases risk for AITD in some people might actually protect against AITD
39                                Treatment for AITD is still restricted to management rather than preve
40 ee homozygous carriers of rs781745126-T have AITD, of whom one also has two other T-cell mediated dis
41 wo Tg SNPs conferred susceptibility to human AITD.
42 ld potentially have a role in treating human AITD.
43 pecific epitopes recognized by antibodies in AITD and has confirmed the increased affinity of stimula
44 g, and whose expression has been detected in AITD, is involved in the migration of lymphocytes to the
45 e thyroid, suggest that CCL21 is involved in AITD pathogenesis, and establish TGCCL21 transgenic mice
46 els support a role for the gut microbiota in AITD, which has been confirmed in some reports from huma
47 s the previously identified 6p and 14q loci (AITD-1 and GD-1, respectively), but the Xq (GD-3) and 13
48 ve established thyroglobulin (TG) as a major AITD susceptibility gene.
49                                    The major AITDs include Graves disease (GD) and Hashimoto's thyroi
50  data set of 53 multiplex, multigenerational AITD families (323 individuals), using highly polymorphi
51              We found evidence for seven new AITD risk loci (P < 1.12 x 10(-6); a permutation test de
52 on of the previously reported association of AITD with TSHR and FCRL3.
53 develop the autoantibodies characteristic of AITD, and they also have (3) a predisposition to develop
54 ther investigate the genetic determinants of AITD, we conducted an association study using a custom-m
55 t signatures contribute to the initiation of AITD.
56 LA-DR3, which is key to the pathoetiology of AITD.
57  interactions underlying the pathogenesis of AITD is essential to uncover new therapeutic targets.
58 ibility genes for and the pathophysiology of AITD.
59 ls with interferon alpha, a known trigger of AITD, increased TG promoter activity only when it intera
60 as clinical and laboratory manifestations of AITDs are reviewed.
61 ic disorders and AITDs and manifestations of AITDs that mimic rheumatologic disorders.
62 mic means were employed to identify putative AITD-susceptible HLA-DR3 binders.
63 trongly and specifically to both recombinant AITD-susceptible HLA-DR3 protein and HLA-DR3 molecules e
64 et 4 was critical for the development of T1D+AITD; all disease-associated amino acids were linked to
65 ignature confers joint susceptibility to T1D+AITD in the same individual by causing significant struc
66 e identified had a marked preference for the AITD-susceptibility DR signatures and not to those signa
67                                          The AITDs are multifactorial and develop in genetically susc
68 1 diabetes (T1D) and autoimmune thyroiditis (AITD).
69    Seven loci showed evidence for linkage to AITD.
70 ntifying genetic variants that predispose to AITD and thyroid cancer, but the increasing prevalence o
71  The susceptibility genes that predispose to AITD can be subdivided into those that affect the immune
72 de polymorphism (SNP) variant predisposes to AITD.
73 ze carcinogens, all of which are relevant to AITD and thyroid cancer.
74 FNalpha) and genetic (TG) factors to trigger AITD.
75 atures and not to those signatures that were AITD-protective.
76 ong statistical support for a model in which AITD is the result of "hits" along three distinct geneti
77 roups was more significantly associated with AITD (P < 0.001).
78 on 33 SNP were significantly associated with AITD (P < 0.01).
79 an SNP (-1623A-->G) that was associated with AITD in the Caucasian population (p = 0.006).
80 nce variants at 225 loci are associated with AITD.
81 nded data set of 102 multiplex families with AITD (540 individuals), through use of 400 microsatellit
82 by which environmental factors interact with AITD susceptibility genes.
83 x loci that showed evidence for linkage with AITD in a data set of 56 multiplex families.
84  whole-genome linkage study of patients with AITD, in order to identify their susceptibility genes.