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1  transcriptional regulation of both Ant1 and Ant2.
2 sues but muscle, in marked contrast to human ANT2.
3  of transcription, as hypothesized for human ANT2.
4                                      Loss of Ant2, a crucial protein mediating ADP/ATP exchange betwe
5 ion of the adenine nucleotide translocase 2 (ANT2), an inner mitochondrial membrane protein, which le
6 AT, and knockdown of the major ANT isoforms, ANT2 and ANT3, did not collapse DeltaPsi after respirato
7 , represented by two highly similar isoforms ANT2 and ANT3.
8 ing enzyme and the ADP/ATP carrier proteins, ANT2 and ANT3.
9                   The finding that the mouse Ant2 and Pem loci are tightly linked suggests that human
10 a gene implicated in Graves' disease and it, ANT2 and two others are confirmed by EST matches.
11 irectly interacts with mitochondria proteins ANT2 and VDAC2, and formation of the DNA-PKcs/ANT2/VDAC2
12 leotide translocase-1 and -2 genes (Ant1 and Ant2) and assigned the loci to mouse chromosomes 8 and X
13 sequenced the genomic loci of mouse Ant1 and Ant2, and compared them to the three human ANT loci.
14 aved between two extant groups, 0.ANT1 and 0.ANT2, and is distant from strains associated with the se
15 endent tyrosine phosphorylation of L1, ANT1, ANT2, and MMP14.
16 e sequence of 135 038 nt contains the entire ANT2 cDNA as well as four other candidates suggested by
17                                              Ant2 cosegregates with DXMit49 and DXMit50 and lies dist
18               Our findings thus suggest that Ant2-deficient T cells bypass the typical metabolic repr
19                    Our data demonstrate that ANT2 depletion in renal proximal tubule cells (RPTCs) le
20 his segment of the Hprt region as: Agtr2-Pem-Ant2-DXMit50-Lamp2-DXMit49.
21 and proinflammatory activation by increasing ANT2 expression and mitochondrial permeability transitio
22  we describe the genomic organization of the Ant2 gene and its regional map position on the X chromos
23 rmatogenesis where the sex chromosome-linked Ant2 gene is inactivated.
24                                    The mouse Ant2 gene, like human ANT2, has an upstream GRBOX, yet t
25 re promoters of the mouse and human ANT1 and ANT2 genes are very similar.
26                 The mouse and human ANT1 and ANT2 genes showed substantial homology starting about 30
27              The mouse Ant2 gene, like human ANT2, has an upstream GRBOX, yet this element is not ass
28 e we demonstrate that targeted disruption of Ant2 in mouse liver enhances uncoupled respiration witho
29 s analyses to gain insights into the role of ANT2 in regulating mitochondrial function, RPTC physiolo
30                             The depletion of Ant2 in RPTCs led to a fundamental rewiring of their pri
31       We propose a significant role for RPTC-Ant2 in the development of obesity-induced CKD.
32 al role of adenine nucleotide translocase 2 (ANT2) in the pathogenesis of obesity-induced CKD.
33 hermore, we show that ADP/ATP translocase 2 (ANT2) interacts with Fe-S apoproteins and MMS19 in the C
34                                        Mouse Ant2 is strongly expressed in all tissues but muscle, in
35 hrough the mitochondrial inner membrane, and Ant2 is the predominant isoform expressed in the liver.
36                   We generated RPTC-specific ANT2 knockout ( RPTC-ANT2-/- ) mice, which were then sub
37                Interestingly, liver specific Ant2 knockout mice are leaner and resistant to hepatic s
38  loci are tightly linked suggests that human ANT2 may be useful as a marker for isolating the human P
39 ell-specific ADP/ATP translocase-2 knockout (Ant2(-/-)) mice.
40        Our findings revealed that obese RPTC-ANT2-/- mice displayed preserved renal morphology and fu
41 generated RPTC-specific ANT2 knockout ( RPTC-ANT2-/- ) mice, which were then subjected to a 24-week h
42 of wild-type mice exhibited higher levels of Ant2 mRNA compared with hindlimb muscle, which may compe
43                               Interestingly, Ant2(-/-) naive T cells exhibit enhanced activation, pro
44 3000 translocation breakpoint and six genes (ANT2, NDUFA1, LAMP2, OCRL, IGSF1, and HDGF) at better th
45 enetic or pharmacologic inhibition of either ANT2 or HIF-1alpha can prevent or reverse these pathophy
46 f ANT in wild-type T cells recapitulates the Ant2(-/-) phenotype and improves adoptive T cell therapy
47 complex and that the structurally homologous ANT2 protein does not bind the complex.
48 cate that plasma membrane-localized ANT1 and ANT2 regulate L1-mediated neurite outgrowth in conjuncti
49 15kb of Ant1 sequence and 36% of the 27kb of Ant2 sequence and included SINEs, LINEs and LTR elements
50 leotide translocator (ANT) isoforms ANT1 and ANT2 that are present in the plasma membrane of mouse ce
51                    The nullification of RPTC-Ant2 triggers a cascade of cellular mechanisms, includin
52 NT2 and VDAC2, and formation of the DNA-PKcs/ANT2/VDAC2 (DAV) complex supports optimal exchange of AD
53      Of interest, in contrast to its paralog Ant2, which is encoded by the X chromosome and ubiquitou
54   In this study, we investigated the role of ANT2, which serves as the primary regulator of cellular