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1 binding sites upstream and downstream of the AT-rich region.
2 separation is not precisely localized in the AT-rich region.
3 eletion of both the Fis binding site and the AT-rich region.
4 e core and then 400 to 600 bp beyond into an AT-rich region.
5 aA box sequences followed by unwinding of an AT-rich region.
6 GC-rich region prevented TBP binding to the AT-rich region.
7 ymines situated at the 5' and 3' ends of the AT-rich region.
8 lete repair of breaks in microhomology-poor, AT-rich regions.
9 method, on a test dataset and on relatively AT-rich regions.
10 e of cisplatination at sites upstream of the AT-rich regions.
11 increase in base substitution error rates in AT-rich regions.
12 a Fis binding site, a DnaA binding site, an AT-rich region, an inverted repeat and a 10 bp site betw
13 uct and dinucleotide adduct sites within the AT-rich region and owing to the influence of cisplatinat
17 ycin binds the minor groove of duplex DNA at AT-rich regions and has been a valuable probe of protein
18 th potential single-stranded character, like AT-rich regions and sequences that can form cruciform st
20 stone chromatin proteins able to bind DNA in AT-rich regions and to interact with various transcripti
21 narrowed the 1p21.2 breakpoint to a 1990 bp AT-rich region, and junction fragments were amplified by
22 for overall AT content, for distribution of AT-rich regions, and for the abundance of several MAR-re
25 preferential insertions into intergenic and AT-rich regions as well as intronic regions of longer ge
26 e of the sterile antisense transcripts to an AT-rich region, as is typical for GIARDIA: It is unclear
27 ells, with the most prominent changes in the AT-rich region associated with the dA/dT(23) repeat and
32 sponse to the stimulants LPS and LTA and the AT-rich region between nt -2571 and -2338 in the Saa3 pr
33 a localized increase in accessibility at the AT-rich regions bound by homeo-proteins and perhaps at a
34 tion start site, and depend on a TATA box or AT-rich region but not the downstream promoter element (
36 ng the improved reliability with which large AT-rich regions can be efficiently manipulated in linear
37 unit, while pentanucleotides 2 and 4 and the AT-rich region constitute a second, relatively weak, ass
39 cription activity revealed that the extended AT-rich region does not affect the kinetics of abortive
41 ze and contain three functional elements: an AT-rich region flanked by binding sites for Cbf1 and CBF
42 enetic assay in yeast, we found that a short AT-rich region (Flex1) within FRA16D increases chromosom
43 ch extension (ext-a1) of the core ORF and an AT-rich region following the core extension (dsORF-a1).
44 statin, the sequence specificity lies in the AT-rich region for hybrids and is similar to that of DNA
46 nd can be functionally substituted for by an AT-rich region from the human lamin B2 IR that differs i
48 with a highly conserved thymine (-14) and an AT-rich region in the middle between the hallmark RpoN-r
51 nalysis of Fugu sequences homologous to very AT-rich regions in the human genome may, therefore, be a
52 in vitro and in vivo; (iii) mutation of the AT-rich regions inhibits HMGA1a binding in vitro; and (i
53 t made up of pentanucleotide 2 and 4 and the AT-rich region is initiated by assembly of a hexamer on
54 est that most A. gambiae MITEs are in highly AT-rich regions, many of which are closely associated wi
55 of dnaA to the end of yaaA that includes the AT-rich region melted during the initiation stage of DNA
60 o be essential for DNA strand opening at the AT-rich region of the replication origin of the Escheric
62 s in gel mobility shift assays with TrcR, an AT-rich region of the trcR promoter was shown to be esse
67 ferentiate among bulk DNA and three discrete AT-rich regions of genomic DNA examined by quantitative
69 s MvaT and MvaU are thought to bind the same AT-rich regions of the chromosome and function coordinat
73 ressing HMGA1a; (ii) HMGA1a protein binds to AT-rich regions of the KL promoter DNA both in vitro and
75 also no evidence of preferential cleavage in AT-rich regions or other target DNA sites implicated in
77 tion previously observed for deletion of the AT-rich region results from deletion of both the Fis bin
78 ence of a second repeat bound to an adjacent AT-rich region results in intramolecular cooperativity i
79 es adjacent to cruciform structures abutting AT-rich regions, similar to the CRISPR leader sequence.
81 rotein one (AP-1) and RIP60 in vitro, and an AT-rich region that contains a dA/dT(23) dinucleotide re
83 rom DNase I digestion an approximately 73-bp AT-rich region that includes the entire mntA promoter.
84 onucleotide repeats in two sets, flanking an AT-rich region that may be the site of initial melting.
85 terated initiator protein binding sites), an AT-rich region that melts upon initiator-iteron interact
87 I promoters are characterized by an extended AT-rich region upstream of -17, which is often interrupt
89 Eighteen putative DnaA boxes with several AT-rich regions were identified in the dnaA-dnaE interge
90 l proteins that act cooperatively to melt an AT-rich region where the replicative helicase is loaded
91 on at many double-stranded sites and also at AT-rich regions where single-stranded DNA is exposed dur
92 performance of sequencing and PCR primers in AT-rich regions, where short primers yield poor sequenci
93 hree times higher in GC-rich regions than in AT-rich regions, while the opposite is true for LINE1.
94 site is mapped to a sequence embedded in an AT-rich region with four scattered GC base pairs (bps) (
95 ound that ERVs integrate in late-replicating AT-rich regions with abundant microsatellites, mirror re
96 fied TEF protein can specifically bind to an AT-rich region within the core of the telokin promoter.