ライフサイエンスコーパス: AT

1 AT also increased after exposure to MIH (Day 1: 1.13 +/-

2 AT and AI have a direct correlation with rotator cuff te

3 AT apoM deserves further investigation as a potential bi

4 AT inflammation is histologically indicated by the forma

5 AT is home to a large variety of immune cells, which are

6 AT maps showed rho (AT)=0.66 (0.53-0.73) and E (AT)=24 (

7 AT pO2 was negatively associated with AT gene expression

8 AT pO2 was positively associated with the expression of

9 AT(2)R-eGFP(+) neurons in the amygdala were predominantl

10 AT(N) biomarkers improved the prediction of memory perfo

11 AT-related metabolism in the dorsal amygdala, including

12 AT-rich interaction domain-containing protein 1A (ARID1A

13 AT-rich interactive domain 5A (ARID5A/Arid5a) is a known

14 enhances the angiotensin II receptor type 1 (AT(1) R) axis associated with oxidative stress.

15 nd between two repeating sequences with 100% AT content, a poly(A)-poly(T) duplex (AA-TT) and a poly(

16 sion on different sequences of DNA ((AA)12, (AT)12, (CC)12 and (CG)12) with supercoiling densities at

17 e structures of Env trimers on aldrithiol-2 (AT-2)-inactivated virions in ligand-free, antibody-bound

18 S (p)-Me-PTE in the sequence contexts of 5'-AT-3', 5'-CT-3', or 5'-GT-3', DNA replication was highly

19 We found 555 AT-related transcripts, 14 of which were confirmed with

20 B1) (30.7%), tumor protein 53 (TP53; 18.7%), AT-rich interaction domain 1A (ARID1A) (13%), albumin (1

21 days 4 and 5) or a suspension of 2.5 x 10(7) AT-MSCs (MSC; days 4 and 5).

22 e abnormal (+) or normal (-), resulting in 8 AT(N) profiles.

23 all three sequence contexts contained 85-90% AT and 5-10% TG.

24 rotein genes in Balanophora consist of >=90% AT, with several between 95% and 98% AT, resulting in th

25 f >=90% AT, with several between 95% and 98% AT, resulting in the most biased codon usage in any geno

26 hes reveal key catalytic features of FAS ACP-AT interactions.

27 te ACP, AcpP, to interrogate type II FAS ACP-AT interactions.

28 the X-ray crystal structure of a type II ACP-AT complex.

29 Formation of productive ACP-AT interactions is required for catalysis and specificit

30 and PKS are initiated by an acyltransferase (AT), which loads monomer units onto acyl carrier protein

31 focusing on engineering the acyltransferase (AT) domains of polyketide synthases (PKSs) responsible f

32 n the modulation of T cell responses against AT-3-induced breast tumors.

33 n of repeated doses of 2.5 x 10(7) allogenic AT-MSCs did not induce clinical or immunological respons

34 long kinetochore-bound regions that span an AT-rich core and are depleted of the canonical histone H

35 Here, we show that an AT-hook motif-containing nuclear localized (AHL) protein

36 activation, early and persistently in AT and AT MDCs of wild-type mice fed a high-fat diet (HFD).

37 tabolic conditions score, APOE genotype, and AT(N) biomarker profiles.

38 jK, the repressive histone mark H3K9me3, and AT-rich flanking sequence.

39 17-HDHA and AT-RvD1 used as adjuvants resulted in elevated serum and

40 We also explored if LG and AT are correlated with apnea severity and indices of upp

41 BM-MSCs and AT-MSCs were characterized based on their expression of

42 served in molecular cues between BM-MSCs and AT-MSCs, both cells had the ability to induce bone tissu

43 obesity-induced MDC M1-like polarization and AT inflammation and contributes to insulin resistance an

44 pro-resolution activity of AT-resolvins and AT-lipoxins may explain some of aspirin's broad anticanc

45 ment of mice with AT-RvDs (e.g., AT-RvD1 and AT-RvD3) or AT-LXA(4) inhibited primary tumor growth by

46 ns as the major difference between AA-TT and AT-TA, which originates from the continuous and close st

47 on of the ACE-2-angiotensin(1-7)-angiotensin AT(2) receptor and the ACE-2-angiotensin(1-7)-Mas recept

48 nverting enzyme-1-angiotensin II-angiotensin AT(1) receptor pathway contributes to the pathophysiolog

49 t of arrestin recruitment to the angiotensin AT(1) and vasopressin V(2) receptors.

50 Antithrombin (AT) replacement has been suggested to prevent venous thr

51 ease the risk, a deficiency in antithrombin (AT), one of the most important natural inhibitors of blo

52 IRs are AT-rich sequences flanked by more GC-rich regions and lo

53 nation with translational NHP models such as AT, promise to provide insights into the brain systems u

54 SP or who developed thrombosis before L-ASP, AT supplementation did not have a significant impact on

55 DNA substrates, showing enhanced activity at AT-rich regions and at elevated temperatures.

56 on at many double-stranded sites and also at AT-rich regions where single-stranded DNA is exposed dur

57 ively distinct, and H2A.Z.2 incorporation at AT-rich enhancers and expression of their associated gen

58 ptor type 1 receptor agonistic autoantibody (AT(1) -AA)-induced mouse model of PE.

59 unction with an inverse relationship between AT pO2 and plasma BCAA concentrations.

60 We found that Lsr2 binds AT-rich sequences throughout the chromosome, and broadly

61 Despite these new bottlenecks, AT engineering provided the first full-length polyketide

62 nd increased numbers of ST2+ T regs in brown AT, but not VAT.

63 strating the prominent contribution of brown AT (BAT) thermogenesis.

64 ed that (a) elevated placental HIF-1alpha by AT(1) -AA or LIGHT upregulates CD73 and ADORA2B expressi

65 the placentas of PE mouse models induced by AT(1) -AA or LIGHT, a TNF superfamily cytokine (TNFSF14)

66 nstrate a primary renal proximal tubule cell AT(2)R natriuretic defect in SHR that may contribute to

67 gnificantly from those of the archetypal cis-AT type systems represent a new paradigm in natural prod

68 12.8%; P = 0.07) and surgical complications (AT: 12.8% vs ST: 13.6%; P = 0.66) were equivalent.

69 Under baseline conditions, AT cells showed concurrent increased levels of proinflam

70 evealed the vast diversity of HCS-containing AT-less type I PKSs.

71 g 17-HDHA and aspirin-triggered-resolvin D1 (AT-RvD1) as adjuvants.

72 g plasma PAI-1 concentrations and decreasing AT BCAA catabolism and thereby increasing plasma BCAA co

73 MHC-II-dependent AT-II cells were isolated from IDLA-induced Treg expansi

74 our approach can be applied for determining ATs against common lepidopteran and aphid pests in many

75 that Pol I occupancy increases as downstream AT content increases and decreases as downstream GC cont

76 plex (AA-TT) and a poly(AT)-poly(TA) duplex (AT-TA).

77 maps showed rho (AT)=0.66 (0.53-0.73) and E (AT)=24 (21-32) ms, RT maps showed rho (RT)=0.55 (0.41-0.

78 dict different cognitive consequences of EC, AT, and PM tau.

79 VTE developed despite extensive AT supplementation, which suggests the need for addition

80 Sixty percent of mRNAs from its extremely AT-rich (81%) genome harbor long polyadenosine (polyA) r

81 Here, we use the Escherichia coli FAS AT, FabD, and its cognate ACP, AcpP, to interrogate type

82 administration of two doses of bovine fetal AT-MSCs in healthy cows did not induce changes in clinic

83 ith a 2.5 x 10(7)-suspension of bovine fetal AT-MSCs on experimental days 1 and 10.

84 To our knowledge, we conducted the first AT/RT-specific cooperative group trial, ACNS0333, to exa

85 te constant (0.336 s(-1) and 0.590 s(-1) for AT and VC respectively), and other electrochemical param

86 arge transfer coefficient (0.52 and 0.44 for AT and VC respectively), standard heterogeneous electron

87 mple, gammadelta T cells are fundamental for AT innervation and thermogenesis, and macrophages are re

88 , we found that IDLA cells mainly arise from AT-II cells, which are the major source of MHC-II.

89 es can also bind to receptors different from AT(1)R, in particular, angiotensin receptor type II (AT(

90 important lipids that differentiated HT from AT.

91 erent MSCs (PA MSCs) versus CD271+ MSCs from AT.

92 by inhibiting intragenic transcription from AT-rich target regions, preventing them from sequesterin

93 ctivity and form co-polymers, only one, FtsZ(AT) , is required for cell division.

94 eover, treatment of mice with AT-RvDs (e.g., AT-RvD1 and AT-RvD3) or AT-LXA(4) inhibited primary tumo

95 horizontally acquired genes also have a high AT-content.

96 ocytes and stromal cells isolated from human AT, have led to the identification of apolipoprotein M (

97 Transcriptomic studies on human AT, and a combination of analyses of transcriptome and p

98 inhibitors and angiotensin receptor type I (AT(1)R) antagonists.

99 We identified AT-related transcripts using RNA-sequencing data from do

100 and the impact of intrarenal angiotensin II AT(1) receptor blockade using candesartan and mineraloco

101 Furthermore, blockade of the angiotensin II AT(1) receptor restored cortical partial pressure of oxy

102 ults suggest that blockade of angiotensin II AT(1) receptors has a major impact upon oxygen delivery

103 in particular, angiotensin receptor type II (AT(2)R), resulting in biological and physiological effec

104 MHC-II on alveolar type-II (AT-II) cells is associated with immune tolerance in an i

105 therapies inhibiting the detrimental Ang II/AT(1) receptor axis.

106 ptor or postreceptor defect causing impaired AT(2)R signaling and renal sodium (Na(+)) retention by u

107 tisense transcriptome in cells with impaired AT regulators (Rho, H-NS and RNaseIII) and searched for

108 FMD: + 3.2% (95%CI 1.7, 4.6) (p < 0.001) in AT; + 4.0% (95%CI 2.1, 5.7) (p < 0.001) in RT; and +6.8%

109 mmHg in SBP (95%CI -10.1, 0.0; p = 0.003) in AT; -4.0 mmHg in SBP (95%CI -7.8, -0.5; p = 0.027) in RT

110 fy proresolving mediators that are active in AT and AR and propose SPMs, including 15-epi-LXA(4) or M

111 rk appears to drive higher tau deposition in AT than in the posterior-medial (PM) memory network.

112 olecular bases for individual differences in AT in the dorsal amygdala, which take advantage of novel

113 that is linked to individual differences in AT, which includes the dorsal amygdala.

114 jor retinol carrier in serum, is elevated in AT and has proinflammatory effects which are mediated pa

115 Overall features of Env trimer embedded in AT-2-treated virions appear well-represented by current

116 esity by modulating transcript expression in AT.

117 reas selective reactivation of ALMS1 gene in AT of an ALMS conditional knockdown mouse model (Alms (f

118 ly, we found TNF-alpha upregulated MHC-II in AT-II in vitro.

119 im of this study was to characterize MGCs in AT and unravel the function of these cells.

120 sufficient to increase the number of MGCs in AT, whereas other factors may be more important for endo

121 oint to the importance of neuroplasticity in AT.

122 ciently induce C-to-G edits, particularly in AT-rich sequence contexts in human cells.

123 cating activation, early and persistently in AT and AT MDCs of wild-type mice fed a high-fat diet (HF

124 e FabD structure that may play a key role in AT activity and substrate selectivity.

125 text, we confirmed their significant role in AT regulation.

126 In Wistar Kyoto, but not SHR, C-21 induced AT(2)R translocation to apical plasma membranes of renal

127 xamined the role of Stat1 in obesity-induced AT MDC polarization and inflammation and insulin resista

128 and eosinophils in perigonadal and inguinal AT, and enhanced inguinal AT browning, with increased en

129 nadal and inguinal AT, and enhanced inguinal AT browning, with increased energy expenditure.

130 The PTGDS inhibitor AT-56 caused intense pain behaviors in male mice but was

131 anti-tuberculosis drug induced liver injury (AT-DILI).

132 Integrating AT DNAm profiles with transcript profile data measured i

133 /86 +/- 6 mmHg) were randomly allocated into AT (n = 14, 40 min of cycling, 50-75% heart rate reserve

134 ng in 47 young rhesus monkeys to investigate AT's molecular underpinnings by focusing on neurons from

135 mitochondrial genome of S. microneptunus is AT-rich and includes 13 protein-coding genes (PCGs), 2 r

136 178 to 197 of the LEDGF that encompasses its AT-hook DNA-binding elements.

137 ing neurons, and identify a new role for its AT-hooks domains as critical determinants of its kinetic

138 ously identified and novel loci for many key AT transcripts influencing insulin resistance and obesit

139 with M2-like macrophages predominant in lean AT and M1-like macrophages in obese AT.

140 growth from a mouse breast cancer cell line (AT-3, Gpr81-negative) implanted in mammary fat pad of wi

141 n of the central amygdala (CeM), with little AT(2)R-eGFP expression in the basolateral amygdala or la

142 n sensitivity, effects associated with lower AT inflammation and increased numbers of ST2+ T regs in

143 The presence of HCSs in many AT-less type I PKSs suggests their co-evolutionary relat

144 ate RAPs as key determinants of Rho-mediated AT regulation in E. coli.

145 sphorylation, but not apical plasma membrane AT(2)R recruitment.

146 ls (BM-MSCs) or adipose tissue-derived MSCs (AT-MSCs) on the regeneration of rat calvarial defects.

147 Since HMGA1 proteins also carry multiple "AT-hook" DNA-binding motifs, suramin is expected to inhi

148 ward nuclear factor of activated T-cells (NF-AT) in T-lymphocytes.

149 ctural features, but contains a noncanonical AT-hook-like motif through which it strongly binds to DN

150 RNA sequencing identified numerous AT-related CeL transcripts, and we used immunofluorescen

151 Most importantly, MGCs in obese AT had a higher capacity to phagocytize oversized partic

152 he consequences of ATM accumulation in obese AT, lending further insight into obesity-related comorbi

153 in lean AT and M1-like macrophages in obese AT.

154 Thus, the pro-resolution activity of AT-resolvins and AT-lipoxins may explain some of aspirin

155 pendent pathway or-especially in the case of AT-rich DNA sequences-be transcribed and initiate MAVS-d

156 Thus, tumor cells of AT-II cellular origin contribute to Treg expansion in an

157 versed MHC-II upregulation on tumor cells of AT-II cellular origin in IDLA.

158 high expression of MHC-II in tumor cells of AT-II cellular origin, which was correlated with increas

159 ed in inflamed lung tissue and IDLA cells of AT-II cellular origin.

160 In particular, the characteristics of AT(2)R and tools to modulate its functions will be discu

161 dipocyte-driven IR may assist development of AT-targeted therapeutic strategies for diabetes.

162 ster memory decline, and that the effects of AT and PM FTP, but not EC, were driven by Abeta+ individ

163 We determined the effects of AT, RT, and CT on endothelial function and systolic (SBP

164 ng HMGA2 from binding to the minor groove of AT-rich DNA sequences.

165 h as adipocytes, as shown by live imaging of AT, 45-um bead uptake ex vivo, and higher lipid content

166 ere, we evaluated the potential influence of AT oxygenation on AT biology and insulin sensitivity in

167 vely) and renal interstitial fluid levels of AT(2)R downstream signaling molecule cGMP (cyclic guanos

168 NAC should be considered in management of AT-DILI.

169 Imaging measures of AT(N) (amyloid and tau positron emission tomography [PET

170 n = 14, 2 x 12 repetitions of RT + 20 min of AT).

171 AT) characterized by an increasing number of AT macrophages (ATMs) and linked to type 2 diabetes.

172 ium-potassium-atpase) and phosphorylation of AT(2)R-cGMP downstream signaling molecules Src (Src fami

173 utatively regulated by HAI1 was S313/S314 of AT-Hook-Like10 (AHL10), a DNA-binding protein of unclear

174 high-fat diet in mice, accompanying signs of AT inflammation and then representing ~3% of ATMs in mic

175 Exploration of the neural substrates of AT in NHPs has revealed a distributed neural circuit tha

176 Although the exact values of ATs are likely to have regional limits, our approach can

177 ency inhibited the upregulation of MHC-II on AT-II cells in inflamed lung tissue.

178 -mediated inflammation upregulates MHC-II on AT-II cells to trigger Treg expansion in inflammation-dr

179 the potential influence of AT oxygenation on AT biology and insulin sensitivity in people.METHODSWe e

180 an important role in MHC-II upregulation on AT-II cells.

181 asures competitively in the same model, only AT FTP significantly predicted memory decline.

182 dentified at the time of surgical operation (AT: 21.7% vs ST: 12.8%; P = 0.07) and surgical complicat

183 erapy based on local injection of BM-MSCs or AT-MSCs is effective in delivering cells that induced a

184 ministration had no effect on natriuresis or AT(2)R recruitment in SHR.

185 with AT-RvDs (e.g., AT-RvD1 and AT-RvD3) or AT-LXA(4) inhibited primary tumor growth by enhancing ma

186 O3 as an important determinant of peripheral AT storage capacity.

187 roxide generation, measured in permeabilised AT fibers ex vivo and intra vital confocal microscopy of

188 a poly(A)-poly(T) duplex (AA-TT) and a poly(AT)-poly(TA) duplex (AT-TA).

189 lete repair of breaks in microhomology-poor, AT-rich regions.

190 d with 22 patients excluded due to premature AT termination, noninducibility or left atrial appendage

191 ic treatment, the additional effect of preop-AT on valve culture results per 2-day interval was minor

192 C risk reduction in 2-day intervals of preop-AT ranged from 0.64 (95% CI: 0.61 to 0.68) at day 7 to 0

193 e [PVC] vs. negative valve culture) on preop-AT.

194 by pre-operative antibiotic treatment (preop-AT).

195 The mammalian high mobility group protein AT-hook 2 (HMGA2) is a multi-functional DNA-binding prot

196 Term infants received AT (20/28; 71%) or AC (8/28; 29%) with median durations

197 Preterm infants received AT (32/44; 73%) or ATM (12/44; 27%) with median duration

198 Most patients received AT supplementation (87%).

199 pressions of angiotensin II type 1 receptor (AT(1) R), NADPH oxidase (NOX) subunits, D(5) R, and NaCl

200 the angiotensin II (AngII) type 2 receptor (AT(2)R) to ameliorate renal fibrogenesis in vitro and in

201 nzyme 2, the type 2 angiotensin II receptor (AT(2)R), the proto-oncogene Mas receptor and the Mas-rel

202 ese studies may provide approaches to reduce AT inflammation and insulin resistance in obesity and di

203 hese results support the notion that reduced AT oxygenation in individuals with obesity contributes t

204 We have shown a relative AT failure in a monogenic obese cohort, a finding suppor

205 nce, we show for the first time the relative AT failure in human obese cohorts to be a major determin

206 AT maps showed rho (AT)=0.66 (0.53-0.73) and E (AT)=24 (21-32) ms, RT maps s

207 central amygdala injections of the selective AT(2)R agonist compound 21 prior to tests for cued or co

208 (Na(+)) retention by utilizing the selective AT(2)R agonist compound-21 (C-21).

209 ); liver and whole-body insulin sensitivity; AT expression of genes and pathways involved in inflamma

210 The sequence, itself a short AT-tract, resides 5 base pairs upstream of otherwise cry

211 that acts at multiple promoters and silences AT-rich genes (such as those in the AggR regulon).

212 or T. thermophila, an organism with similar AT-content.

213 rp1 encodes a reportedly Drosophila-specific AT-hook, bZIP protein responsible for many of the effect

214 on spectrophotometry method (Mo coated-T-SQT-AT-FAAS) was developed for the determination of cadmium

215 y changes based upon systemic energy status, AT macrophages (ATMs) must adapt phenotypically and meta

216 y in people.METHODSWe evaluated subcutaneous AT oxygen partial pressure (pO2); liver and whole-body i

217 sis and an in-depth analysis of subcutaneous AT histology.

218 SHR rats were studied after 24-hour systemic AT(1)R (Ang II type-1 receptor) blockade.

219 dren exhibiting extreme anxious temperament (AT) are at an increased risk for developing anxiety and

220 hibition, which we term anxious temperament (AT), reveals that it is trait-like.

221 en genotypes exposed to ambient temperature (AT) or HT during flowering.

222 anterolateral EC and the anterior-temporal (AT) memory network appears to drive higher tau depositio

223 In contrast, tau in anterior-temporal (AT) regions is most predictive of memory decline in Abet

224 ed from patients with Achilles tendinopathy (AT) or Achilles rupture (AR) under baseline and IL-1beta

225 ENSE system in guiding ablation to terminate AT with the first lesion set and with reduced entrainmen

226 equences are observed to be more stable than AT-rich ones, with the specific response dependent on th

227 In vitro studies demonstrate that AT-rich downstream DNA enhances pausing by Pol I and inh

228 unwrapping, supporting the possibility that AT-rich segments may signal the start of transcription b

229 We propose that AT-rich centromeres drive karyotype diversity in the Mal

230 ected even in safe contexts, suggesting that AT has trait-like neural signatures within the brain.

231 Furthermore, we find that (AT)12 forms stable periodic structures with non-canonica

232 The AT genotype frequencies for CFB was non significantly lo

233 The AT-tract alters DNA conformation and enhances contacts b

234 In addition, the AT was a predictor of measures of upper airway collapsib

235 tingly, weight loss after obesity alters the AT immune compartment, which may account for the decreas

236 ts multiple, productive conformations at the AT binding interface, allowing the complex to sustain hi

237 cesses behind the sound pressure gain in the AT by numerically modeling the tracheal acoustic behavio

238 so has single-strand binding activity in the AT-rich region of its origin.

239 on the change in sound pressure level in the AT.

240 , the best conserved promoter feature is the AT-rich -10 element; a sequence essential for DNA unwind

241 A key feature of the AT is its capacity to reduce the velocity of sound propa

242 nt in humans and can block in particular the AT(1) R axis.

243 Since the AT environment rapidly changes based upon systemic energ

244 In Arabidopsis thaliana, the AT-hook motif nuclear-localized (AHL) transcription fact

245 ruitment with those for Gq signaling via the AT(1) receptor.

246 As conceived by Karlman Wasserman, the AT coalesced the increase of blood lactate concentration

247 on is likely through suramin binding to the "AT-hook" DNA-binding motifs and therefore preventing HMG

248 ical roles in the binding of suramin to the "AT-hook" DNA-binding motifs.

249 omized studies comparing antibiotic therapy (AT) and surgical therapy-appendectomy (ST) for uncomplic

250 relation to biofilm for active thermography (AT).

251 Here, we show that three AT-rich introns were retained in the TNP2-like transposa

252 The anaerobic threshold (AT) remains a widely recognized, and contentious, concep

253 d M1-like phenotype in obese adipose tissue (AT) and may contribute to AT inflammation and insulin re

254 the resident immune cells in adipose tissue (AT) and regulate both tissue homeostasis in the lean sta

255 and adipocytes are among the adipose tissue (AT) APCs that differentiate and activate naive CD4(+) T

256 ade inflammation of visceral adipose tissue (AT) characterized by an increasing number of AT macropha

257 SPO3 may limit gluteofemoral adipose tissue (AT) expansion by suppressing adipogenesis and increasing

258 tissue, and (3) human frozen adipose tissue (AT) from six individuals.

259 P4) is elevated in serum and adipose tissue (AT) in obesity-induced insulin resistance and correlates

260 Adipose tissue (AT) inflammation contributes to systemic insulin resista

261 tivity due to dysfunction of adipose tissue (AT) is one of the earliest pathogenic events in type 2 d

262 ic clamping with concomitant adipose tissue (AT) microdialysis and an in-depth analysis of subcutaneo

263 ls have shown that decreased adipose tissue (AT) oxygenation is involved in the pathogenesis of obesi

264 Adipose tissue (AT) plays a central role in both metabolic health and pa

265 Adipose tissue (AT) regulatory T cells (T regs) control inflammation and

266 but not the total undigested adipose tissue (ATs), from obese patients has decreased LAL expression c

267 inked the central nucleus of the amygdala to AT and its underlying neural circuit.

268 Agonists or antagonists to AT(2)R are potential therapeutics in cardiovascular dise

269 sometimes antagonistic, of their binding to AT(1)R.

270 proteins that outcompete H-NS for binding to AT-rich foreign DNA.

271 sors in Pseudomonas aeruginosa by binding to AT-rich regions of the chromosome.

272 se adipose tissue (AT) and may contribute to AT inflammation and insulin resistance.

273 dala to determine its causal contribution to AT.

274 ot observed by cryo-ET, potentially owing to AT-2 inactivation.

275 rapy who received ampicillin and tobramycin (AT), ampicillin and cefotaxime (AC), or ampicillin, tobr

276 ree dimensions, we applied Array Tomography (AT) and Stimulated Emission Depletion microscopy (STED),

277 e respiratory trachea, the acoustic trachea (AT), which transfers sound from the mesothoracic acousti

278 Aerobic exercise training (AT) improves endothelial function.

279 The trans-AT family of modular PKSs, whose biosynthetic frameworks

280 eta-methyl branch installation in this trans-AT PKS.

281 stinctive enzymatic features common to trans-AT PKSs is their ability to introduce methyl groups at p

282 ve chemotherapy and focal radiation to treat AT/RT.

283 Incubation of IL-1beta treated AT and AR tendon-derived stromal cells in 15-epi-LXA(4)

284 Atypical teratoid/rhabdoid tumor (AT/RT) is an aggressive, early-childhood brain tumor wit

285 Under AT and HT, the lipidome was dominated by phosphatidylcho

286 ses hyperinsulinemia and diminishes visceral AT (VAT) T reg number and function, but whether these tw

287 While AT-TA exhibits non-distinguishable behaviors from random

288 IL1beta is elevated in perigonadal white AT (PGWAT) of chow-fed RBP4-overexpressing mice and in s

289 ty of immune cells within the visceral white AT and their contributions to homeostasis and pathology.

290 The expansion of visceral white AT promotes dysregulation of its immune cell composition

291 Patients from birth to 22 years of age with AT/RT were eligible.

292 AT pO2 was negatively associated with AT gene expression of markers of inflammation and fibros

293 ostaglandin E(2) release in AR compared with AT cells.

294 MUO group and was negatively correlated with AT pO2, whereas the plasma concentrations of other cytok

295 Furthermore, the treatment of mice with AT-RvD1 during a period of smoke-cessation further enhan

296 Moreover, treatment of mice with AT-RvDs (e.g., AT-RvD1 and AT-RvD3) or AT-LXA(4) inhibit

297 were optimized and the images obtained with AT were compared with those from scanning electron micro

298 ten time to ALT&100 U/L in participants with AT-DILI, but significantly reduced length of hospital st

299 Patients with AT were randomized to either RM or LAT mapping using the

300 with historical therapies for patients with AT/RT.

301 the work to date has been accomplished with ATs that are either naturally promiscuous and/or located