ライフサイエンスコーパス: ATP

1 ATP and Gly induced common, but also specific, alternati

2 ATP binding and hydrolysis by StXPB could lead to a spir

3 ATP sulfurylase, an enzyme which catalyzes the conversio

4 ATP synthase inhibition in myotubes triggers the ISR via

5 ATP triggers the recruitment of microglial protrusions a

6 ATP-binding cassette (ABC) transporters constitute one o

7 ATP-binding cassette (ABC) transporters play several cri

8 ATP-dependent chromatin remodelling enzymes facilitate d

9 ATP-dependent chromatin-remodeling enzymes control acces

10 In silico analyses revealed that 1 ATP synthase is [Formula: see text]-dependent and the ot

11 hat each global unfolding cycle consumes ~10 ATPs.

12 nd the energetic requirement for at least 12 ATP molecules to be hydrolyzed per revolution of the mot

13 erium Pseudobutyrivibrio ruminis possesses 2 ATP synthases and 2 distinct respiratory enzymes, the fe

14 ic mitochondrial Ca(2+) uptake to accelerate ATP production.

15 ne protease family and an iteratively acting ATP-grasp protein.

16 urbs mitochondrial Ca2+ homeostasis, affects ATP production, and attenuates DNA repair.

17 al genome replication, RNA-binding affinity, ATP hydrolysis activity, and helicase-mediated unwinding

18 and identification of the biomolecules AMP, ATP, and CoA, which are fundamental for numerous biochem

19 a stable complex with Pol II and acts as an ATP-dependent processivity factor that helps Pol II acro

20 is proceeds through an ester generated by an ATP-dependent adenylating enzyme.

21 Eukaryotic initiation factor 4A (eIF4A), an ATP-dependent DEAD-box RNA helicase, is a critical compo

22 deletion in the ABCC4/MRP4 gene encoding an ATP-binding cassette (ABC) transporter in PEL-negative i

23 SpoIVFB has an ATP-binding domain exposed to the MC cytoplasm, but the

24 the fly ortholog of H2A.Z) genome-wide in an ATP-dependent manner, like the yeast SWR1 complex.

25 ks DNA plectonemes and can compact DNA in an ATP-dependent manner.

26 d unwinds ~2 turns of the double helix in an ATP-independent fashion.

27 ABCB4 is an ATP-binding cassette transporter that extrudes phosphati

28 ference/activation screen integrated with an ATP biosensor.

29 ncrease in both k(f)(CK) (+86%, P<0.001) and ATP delivery (+80%, P<0.001).

30 ator infusions (DeltaFVC: ACh: -31 +/- 3 and ATP: -30 +/- 4%).

31 Extracellular purines, adenosine and ATP, protected against ALI induced by purified LPS.

32 Time-lapse imaging of autophagosomes and ATP/ADP levels in migrating cells in the rostral migrato

33 ardiac cells in response to Blebbistatin and ATP drug exposure in real tme.

34 as well as mitochondrial quality control and ATP production.

35 hrome C oxidase copper chaperone (COX17) and ATP Synthase, H(+) transporting, Mitochondrial Fo Comple

36 hile phosphate moieties present in crRNA and ATP molecules enhance IRMPD, an increase in the IR cross

37 y-3-methylglutaryl-CoA reductase (HMGCR) and ATP-citrate lyase (ACLY) in a TGF-beta receptor/PI3K/pro

38 Consistently, ATP levels and ATP synthesis are lower in mitosis than in G2 in synchro

39 same signals used to regulate metabolism and ATP production, such as calcium and reactive oxygen spec

40 a manner involving fatty acid oxidation and ATP-citrate lyase activity.

41 ription, co-chaperone "helper" proteins, and ATP binding and hydrolysis.

42 (SphK1 or SphK2) from sphingosine (Sph) and ATP.

43 ilament with single-stranded DNA (ssDNA) and ATP.

44 show that the DSB motion is subdiffusive and ATP-dependent and exhibits unique dynamical signatures,

45 are temporally linked and how substrate and ATP alter protein dynamics to achieve active transport.

46 SGs assembled by stem-loop RNA triggers are ATP-sensitive, regulated by helicase/chaperone activity,

47 ate the release of sensory mediators such as ATP, which in turn modulates afferent nerve activity in

48 which suggests that Rv1819c has a bacterial ATP-binding cassette (ABC)-exporter fold(1).

49 A model of structure-based ATP production predicts profound energy stress in older

50 Unlike the bacterial flagellar motor (BFM), ATP (adenosine-5'-triphosphate) hydrolysis probably driv

51 avenger beta-methylcyclodextrin also blocked ATP release and IL-33 secretion by decreasing the level

52 in) or statin compound (simvastatin) blocked ATP and IL-33 release by lowering the expression of VDAC

53 te polypeptides upon hydrolysis of its bound ATP.

54 midine-4-amine (PP) compounds that are bulky ATP mimetics.

55 be gradually imported into the cytoplasm by ATP-powered transport, however, the proton motive force

56 by dynamic nucleosome unwrapping governed by ATP-dependent chromatin remodelers.

57 partially lysis independent and inhibited by ATP.

58 containing mycobacterial GyrB are limited by ATP hydrolysis.

59 -cellular, and the signal can be mediated by ATP and/or gap junctions, and is species dependent.

60 eveal that a single conformational switch by ATP binding drives unidirectional substrate translocatio

61 ehyde-3-phosphate dehydrogenase, calmodulin, ATP synthase, sperm equatorial segment protein 1, peroxi

62 of P-loop NTPase fold enzymes that catalyze ATP-hydrolysis and utilize its free energy for a stagger

63 nal changes in the F(1) motor that catalyzes ATP production.

64 lism of ATP by two ecto-nucleotidases, CD39 (ATP -> AMP) and CD73 (AMP -> ADO).

65 Cellular ATP levels did not change.

66 ), mtND1 expression (p < 0.001) and cellular ATP content (p < 0.001) in sepsis patients.

67 bution and autocrine stimulation by cellular ATP release and was perturbed by indiscriminate global s

68 P production, but a net decrease in cellular ATP levels.

69 Increased cellular ATP underpins increased myeloid cell superoxide producti

70 of genes and pathways that regulate cellular ATP-the ATPome-we conducted a genome-wide CRISPR interfe

71 As results, cellular ATP and reactive oxygen species (ROS) levels are elevate

72 on of proteolytic fragments of chloroplastic ATP synthase, termed inceptins.

73 th-dependent activation requires the cognate ATP/GTP substrate pair, while negative-cooperativity sup

74 m mitochondrial disease mutations compromise ATP synthesis and render cells vulnerable to nutrient an

75 ify the mechanism responsible for conductive ATP release.

76 Consistently, ATP levels and ATP synthesis are lower in mitosis than i

77 The cosubstrate, ATP, enforces a four-coordinate cob(II)alamin geometry,

78 at allows us to follow the steps that couple ATP turnover to mechanical work.

79 y function as molecular machines by coupling ATP binding, hydrolysis, and phosphate release to transl

80 L1) to prevent incorporation of N6-methyl-(d)ATP into DNA and RNA.

81 Decreased ATP production and NADH recycling, associated with mitoc

82 Our results show how decreasing ATP concentrations lead to both increasing dwell times o

83 , but not FX synapses, by stimulus-dependent ATP synthase beta subunit translation; this increases th

84 tilize the localized proton density to drive ATP synthesis.

85 butes to the proton motive force that drives ATP synthesis in many mammalian tissues.

86 tivity suppresses Mn2+ utilization by either ATP or GTP alone.

87 Many viruses employ ATP-powered motors during assembly to translocate DNA in

88 synthase enzyme to its c-subunit, enhancing ATP production efficiency and synaptic growth.

89 thases, peroxisomal acyl-activating enzymes, ATP binding cassette (ABC) transporters, and central car

90 are the porcine mitochondrial ND2 and equine ATP 6-8 genes.

91 The objective of this study was to examine ATP level and PG production of porcine IVD cells under p

92 nstrate that both condensin I and II exhibit ATP-dependent motor activity and promote extensive and r

93 Extracellular ATP (eATP) is a signaling molecule that variably affects

94 Extracellular ATP and subsequent P2Y(2) receptor function stimulate DN

95 Extracellular ATP mitigates pneumolysin-induced neutrophil activation.

96 mage was indeed exacerbated by extracellular ATP, subsequent P2Y(2)R activation, and downstream intra

97 Elevated levels of extracellular ATP induced by SARS-CoV-2 infection may trigger hyperact

98 -/-) tumor cells, depletion of extracellular ATP, or inactivation of the ATP-gated P2X7R channel also

99 very of a novel class of anti-tuberculosis F-ATP synthase inhibitors.

100 Here, we inhibited F1Fo ATP synthase function in primary cultured hippocampal ne

101 ss in diseases, at least, in those with F1Fo ATP synthase defects.

102 vel agonist, the synthetic analogue 2-fluoro-ATP, and to confirm its agonist activity on rat P2X2 rec

103 Selectivity remains a challenge for ATP-mimetic kinase inhibitors, an issue that may be over

104 nd for NAD(+) is in excess of the demand for ATP.

105 nels; (3) antagonist of P2Y(1) receptors for ATP; and (4) inhibitors of phospholipase C or IP3 recept

106 We observed high nucleotide selectivity for ATP driving motor rotation, negative cooperativity in AT

107 tial mechanoenzyme that uses the energy from ATP hydrolysis to physically reshape and remodel, and th

108 hich synthesizes cyclic oligoadenylates from ATP)(3-5).

109 ipocytes uncouples fatty acid oxidation from ATP generation in mitochondria and promotes energy dissi

110 Uncoupling respiration from ATP synthesis or increasing ATP hydrolysis restores NAD(

111 has the impact of the channels on MC and FS ATP levels.

112 and 6 in the D2 ring, work together to fuel ATP-dependent degradation is not understood.

113 ilizes oxygen during respiration to generate ATP.

114 Glutamate, ATP, Arabidopsis PLANT ELICITOR PEPTIDE, and glutathione

115 ar NADH:NAD(+) ratio, upregulated glycolytic ATP production and restored cellular proliferation.

116 atine/ATP), but potentially limiting greater ATP delivery during increased workload.

117 Vacuolar H+-ATP complex (V-ATPase) is a multisubunit protein complex

118 These results show the importance of high ATP in DnaA oligomerization and its dependence on the Hi

119 rophic growth fully supported by this highly ATP-efficient CO(2) fixation pathway.

120 hm was developed to track and categorise how ATP and NAD(P)H pools are affected in the presence of a

121 However, how ATP powers substrate remodelling and whether a shared me

122 icle in the presence of the non-hydrolysable ATP analogue AMP-PNP at an overall resolution of 3.1 ang

123 monomeric, AMP-PNP-DnaA (a non-hydrolysable ATP-analog bound-DnaA) was oligomeric, primarily dimeric

124 h or without adenosine or a non-hydrolyzable ATP analog, adenosine 5'-(gamma-thio)-triphosphate (ATPg

125 uolar-type H(+)-ATPases (V-ATPase) hydrolyze ATP to pump protons across the plasma or intracellular m

126 t is incapable of binding DNA or hydrolyzing ATP.

127 Pol II nucleosome bypass without hydrolyzing ATP.

128 metabolites accumulated in higher amounts in ATP sulfurylase transgenic seeds.

129 ng motor rotation, negative cooperativity in ATP hydrolysis, and the energetic requirement for at lea

130 y stream of mouse revealed that decreases in ATP levels force cells into the stationary phase and ind

131 In conclusion, increases in ATP and H(2)O(2) constitute an essential signal that swi

132 Mitochondria fulfill essential roles in ATP production, metabolic regulation, calcium signaling,

133 d in numerous biological functions including ATP production, cofactor biosyntheses, apoptosis, lipid

134 s mediated by the ABCG1 transporter increase ATP release by volume-regulated anion channels under hyp

135 secretion was not a consequence of increased ATP-binding cassette subfamily G member 5/8 activity giv

136 UA significantly increased ATP and NAD(+) levels in mice skeletal muscle.

137 kload, although the nonobese heart increases ATP delivery through CK, the obese heart does not; this

138 respiration from ATP synthesis or increasing ATP hydrolysis restores NAD(+)/NADH homeostasis and prol

139 creen for modulators of hypotonicity-induced ATP release using HEK-293 cells and murine cerebellar gr

140 TRPV4 contributed to 50% stretch-induced ATP release.

141 6) modulates sAC by increasing intracellular ATP levels, with accompanying cAMP accumulation lost in

142 s is uniformly affected by the intracellular ATP level of the living muscle cells, further demonstrat

143 nels that are regulated by the intracellular ATP/ADP ratio.

144 system uses a consistent mechanism involving ATP release from ISCs and activation of P2RY1 autorecept

145 p, we found that uptake by the 14A mutant is ATP hydrolysis-, substrate concentration-, and time-depe

146 radecamer in its apo-form or loaded with its ATP cofactor, to a 1 MDa capsid-like homo-hexacontamer,

147 K(ATP) channel subunits Kir6.1 and SUR2B were expressed in

148 those of WT vessels, suggesting that basal K(ATP) channel activity in LSM is not an essential compone

149 We examined the contribution of central K(ATP) channels to glucose effectiveness.

150 Ca(2+) oscillations, and the patch-clamped K(ATP) channel opened more frequently when glucose was hig

151 the ability of bound nucleotide to inhibit K(ATP).

152 es, inhibit pancreatic ATP-sensitive K(+) (K(ATP) ) channels to increase insulin release.

153 tention to the role of ATP-sensitive K(+) (K(ATP)) channels and the exact site of action of the hormo

154 NaHS plus glibenclamide, an antagonist of K(ATP) opening (NaHS Glib), and Glib alone (Glib).

155 are equipped with ATP-regulated potassium (K(ATP) ) channels that are regulated by the intracellular

156 For patients taking sulfonylureas, K(ATP) channel inhibition may exacerbate exercise intolera

157 Our findings demonstrate that systemic K(ATP) channel inhibition reduces V O(2) max and critical

158 running in rats, before and after systemic K(ATP) channel inhibition via glibenclamide.

159 Vascular K(ATP) channel function (topical glibenclamide superfused

160 The alpha-cells are also equipped with K(ATP) channels but they are under strong tonic inhibition

161 Using pMRI, we found that in pig kidney, ATP was rapidly generated in presence of oxygen (100 kPa

162 Known ATP-competitive small-molecule IRE1 kinase inhibitors ei

163 scently labeled boron-dipyrromethene-labeled ATP molecules in relaxed skeletal muscle sarcomeres from

164 ded increased mitochondrial matrix-localized ATP via substrate-level phosphorylation.

165 e for active amino acid transport, and lower ATP availability contribute to reduced amino acid uptake

166 of the electron transport chain by lowering ATP and increasing ROS productions.

167 FTS mutations deregulate metabolism lowering ATP levels, as a result of increased purine catabolism a

168 Furthermore, a rise in the MC ATP level was not necessary for Pro-sigma(K) cleavage by

169 Measured ATP concentrations exhibited high variance between repli

170 otrusions and is converted by the microglial ATP/ADP hydrolysing ectoenzyme CD39 into AMP; AMP is the

171 expectedly partially activates mitochondrial ATP-generating activity.

172 Glycolytic and mitochondrial ATP production were increased in ABA-treated human myotu

173 of mRNAs for functions such as mitochondrial ATP synthesis, the chloroplast thylakoids, vesicle traff

174 l pathways, we noted increased mitochondrial ATP production, but a net decrease in cellular ATP level

175 l of mitochondria, we quantify mitochondrial ATP synthesis rates in mitosis from the single-cell time

176 We find that mitochondrial ATP synthesis decreases by approximately 50% during earl

177 Addition of 2 mm ATP to the lysate or homogenate did not decrease the sta

178 axial cores, whose formation requires MukBEF ATP hydrolysis.

179 e therapeutic range lowered cell G6P but not ATP and decreased G6pc mRNA at high glucose.

180 alpha exhibited a sufficient availability of ATP resulted from higher activities of succinate dehydro

181 turn the enzyme on or off by the binding of ATP or dATP, respectively.

182 ation-loop threonine residues and binding of ATP-Mg(2+).

183 ADO depends on the sequential catabolism of ATP by two ecto-nucleotidases, CD39 (ATP -> AMP) and CD7

184 ally ignored the biophysical consequences of ATP-fueled active processes acting on chromatin.

185 phorylation abolishes the K(+)-dependence of ATP hydrolysis and blocks the catalytic cycle after form

186 Depletion of ATP by adding azide also results in IFT slowdown and IFT

187 3 relieves the negative regulatory effect of ATP through a 2.5- angstrom-resolution crystal structure

188 ylation attenuated the allosteric effects of ATP on NAT1.

189 al DNA configurations and uses the energy of ATP hydrolysis to promote their compaction.

190 Q-bodies, is triggered by the exhaustion of ATP.

191 dwell times and processivity as functions of ATP concentration, providing experimental evidence for t

192 (~30 aa) steps, each coupled to hundreds of ATP hydrolysis events.

193 l optogenetic inhibition and interruption of ATP-P2X4 signaling reduced reflexive behavioral response

194 Loss of ATP sensing in macrophages may reduce their secretory ca

195 respiration, allowing for the maintenance of ATP levels under conditions of impaired mitochondrial re

196 vide insight into the catalytic mechanism of ATP-grasp enzymes.

197 These results challenge sequential models of ATP hydrolysis and coupled mechanical work by ClpAP and

198 dition, enabling detection of 10(-14) mol of ATP within 10 min.

199 X7L are predicted to have reduced numbers of ATP-binding sites, which potentially alters receptor fun

200 resolution structure reveals the presence of ATP and GMP at the canonical sites of the Bateman domain

201 lits into two TrkA dimers in the presence of ATP and releases the constraints on TrkH, resulting in a

202 to increase during mitosis, but the rates of ATP synthesis and consumption during mitosis have not be

203 nit translation; this increases the ratio of ATP synthase enzyme to its c-subunit, enhancing ATP prod

204 eveal novel mechanisms for the regulation of ATP release and volume-regulated anion channel activity

205 We aimed to investigate the role of ATP in neutrophil response to pneumococcal infections.

206 exposed to the MC cytoplasm, but the role of ATP in regulating Pro-sigma(K) cleavage has been unclear

207 on, with particular attention to the role of ATP-sensitive K(+) (K(ATP)) channels and the exact site

208 mination or elongation rescue, which rely on ATP-dependent Mfd translocation on DNA.

209 TrkH-TrkA complex in the presence of ADP or ATP.

210 educing mitochondrial redox potential and/or ATP synthesis impaired proline biosynthesis.

211 that transgenic soybean seeds overexpressing ATP sulfurylase accumulated very low levels of the beta-

212 prescribed for diabetes, inhibit pancreatic ATP-sensitive K(+) (K(ATP) ) channels to increase insuli

213 We also demonstrate that mutants perturbing ATP hydrolysis or DNA cleavage in vitro impair P2 OLD-me

214 for depleted energy stores (phosphocreatine/ATP), but potentially limiting greater ATP delivery duri

215 he loss of a functional signal (e.g. plasma [ATP]) as opposed to an intrinsic endothelial dysfunction

216 tional change and is activated to polymerize ATP into 2'-5'-oligoadenylate chains.

217 Significantly, kinking of TM6 in the post-ATP hydrolysis state stabilized by MgADPVO(4) eliminates

218 suggested that 6-OH-BDE-47 is a promiscuous ATP-competitive kinase inhibitor; 2) in vitro in dissoci

219 the terminal subunit, which likely promotes ATP hydrolysis and rapid phosphate release.

220 y for gene expression, but they also provide ATP, the primary fuel driving gene expression.

221 er patient, both have gain-of-function Rad50 ATP hydrolysis activity that results not from faster ass

222 s led to membrane depolarization and reduced ATP production.

223 l morphology and membrane potential, reduced ATP production, and increased superoxide ion levels); fu

224 Mitochondria regulate ATP production, metabolism, and cell death.

225 role for the host Golgi compartment-resident ATP-powered calcium pump (secretory pathway calcium ATPa

226 emical ion gradient is harnessed by a rotary ATP synthase to phosphorylate adenosine diphosphate to A

227 ulates ATPase activity, and mutating Dnmt5's ATP-binding pocket disproportionately reduces ATPase sti

228 While several ATP analogues gave responses of similar magnitude to ATP

229 Here, we show ATP citrate lyase (Acly) to be activated in inflammatory

230 e that requires sensing of the damage signal ATP.

231 ns in the microtubule-bound state by slowing ATP-binding, resulting in high-force production at both

232 arm priming (recovery stroke) while slowing ATP hydrolysis, demonstrating that it uncouples these tw

233 ped a user-friendly, ready-to-use and stable ATP sensing paper biosensor that can be combined with sm

234 esin-5 tails decrease microtubule-stimulated ATP-hydrolysis by specifically engaging motor domains in

235 cassettes strongly influence RNA-stimulated ATP hydrolysis by the N-terminal cassette.

236 Our results show that substrate stimulates ATP hydrolysis by accelerating the IF-to-OF transition.

237 bstrates, instead of its canonical substrate ATP.

238 We hypothesized that ATP transfer rate through creatine kinase (CK) (k(f)(CKr

239 Previously, we showed that ATP-DnaA, not ADP-DnaA, undergoes a conformational chang

240 tathione transport activity, suggesting that ATP hydrolysis and substrate transport by Atm1 may invol

241 The analysis also suggests that ATP and NAD(P)H balancing cannot be assessed in isolatio

242 The ATP citrate lyase (ACLY) enzyme cleaves cytosolic citrat

243 The ATP sensing paper includes a lyophilized "nano-lantern"

244 The ATP-triggered half-site release rate of KIF1A was simila

245 ibition of adenosine receptors abrogated the ATP effect on CXCL8 secretion.

246 mechanism of energy transduction between the ATP and RNA binding pockets using molecular dynamics sim

247 t Motif V controls communication between the ATP-binding pocket and the helical gate.

248 bactin is imported into the cytoplasm by the ATP binding cassette (ABC) transporter IrtAB(4), which f

249 lity of the human genome is modulated by the ATP-driven SWI/SNF chromatin remodeling multiprotein com

250 s a membrane-bound enzyme that catalyzes the ATP-dependent phosphorylation of diacylglycerol to form

251 of active site constituents that engage the ATP phosphates and the metal cofactors.

252 Antagonists for the ATP-gated ion channel receptor P2X1 have potential as an

253 ing an irreversible bond with Cys 215 in the ATP-binding pocket, a residue that is not present in hum

254 dicted to delete critical amino acids in the ATP-binding site.

255 he active site entailing dislocations of the ATP phosphates, altered contacts to ATP, and variations

256 d in Fmr1(-/y) mouse neurons, closure of the ATP synthase leak channel by mild depletion of its c-sub

257 t results not from faster association of the ATP-bound form but faster dissociation leading to less s

258 stress resilience through high levels of the ATP-dependent chaperonin TRiC/CCT.

259 of extracellular ATP, or inactivation of the ATP-gated P2X7R channel also compromised the effects of

260 P2X1 receptor by measuring inhibition of the ATP-induced calcium influx.

261 g site on FlhG, their binding depends on the ATP-dependent dimerization state of FlhG.

262 In this system, the ATP-binding cassette (ABC) transporter LptB(2) FGC extra

263 rse cholesterol transportation targeting the ATP binding cassette protein.

264 predicts that indole docks perfectly to the ATP binding site of the GyrB subunit.

265 d proton density to the crista tip where the ATP synthase can readily utilize the localized proton de

266 Our measurements with the ATP-depleted cells reveal that the diffusion dynamics of

267 It maintains indirect interactions with the ATP-pocket and mediates a critical salt bridge with a gl

268 ular docking of plant metabolites within the ATP and D-cycloserine binding pockets of Ddl.

269 een human and preclinical species within the ATP binding site highlights a single amino acid (I960 ->

270 acellular levels of anticancer drugs through ATP-binding cassette (ABC) pumps.

271 We demonstrate that, through ATP-dependent RNA binding, the DEAD-box protein eIF4A re

272 he P2K2 extracellular lectin domain binds to ATP with higher affinity than P2K1 (dissociation constan

273 However, when AAA4 is bound to ATP, the gating of AAA1 by AAA3 prevails and dynein moti

274 when bound to ADP and opens it when bound to ATP.

275 sence of complementary (positive) charges to ATP enhance reaction rates, though the impact of these c

276 s of the ATP phosphates, altered contacts to ATP, and variations in the numbers and positions of the

277 se to phosphorylate adenosine diphosphate to ATP.

278 Brief exposure to ATP induces the opening of a nonselective cation channel

279 ogues gave responses of similar magnitude to ATP, including the previously identified agonists ATPgam

280 ting the allosteric transition that triggers ATP hydrolysis.

281 ae and Haemophilus influenzae use tripartite ATP-independent periplasmic transporters (TRAPs) to scav

282 re fully bound to adenosine 5'-triphosphate (ATP) analogs, which is expected to induce opening of the

283 and the number of adenosine 5'-triphosphate (ATP) molecules hydrolyzed per step is demonstrated to be

284 " pockets within the adenosine triphosphate (ATP) binding site of PI3Kgamma.

285 EAH helicases couple adenosine triphosphate (ATP) hydrolysis to conformational changes of their catal

286 o myofibrils because adenosine triphosphate (ATP) is needed to fuel sarcomere shortening.

287 The adenosine triphosphate (ATP) synthase in human mitochondria is a membrane bound

288 ed cells can release adenosine triphosphate (ATP) to the extracellular medium, which can be hydrolyze

289 s the Rnl5 family of adenosine triphosphate (ATP)-dependent polynucleotide ligases that seal 3'-OH RN

290 enzymes, purine ribonucleoside triphosphate, ATP and propanoate, which are considered as the basal fu

291 its the tendency of UPF1 to release RNA upon ATP binding and hydrolysis.

292 mplexes, cohesin, condensin, and Smc5/6, use ATP hydrolysis to power a plethora of functions requirin

293 nstructed the torque-speed curve at various [ATP]s and discuss rotary models in which the archaellum

294 of substrates for production of heat versus ATP.

295 ve or inactive form of kinases, compete with ATP, stabilize inactive kinase conformations, or act thr

296 fective NgrRnl-Ala mutants in complexes with ATP/Mn2+.

297 Both cells are equipped with ATP-regulated potassium (K(ATP) ) channels that are regu

298 cludes the TOMM34 interaction interface with ATP-bound HSP70 dimers, which leaves them intact and the

299 of C. thermosuccinogenes that operates with ATP and GTP exhibits unusual kinetics toward F6P, as it

300 phosphorylated but is again stabilized with ATP-Mg(2+) bound.