ライフサイエンスコーパス: AV block

1 diomyopathy, one of whom had a pacemaker for AV block.

2 AVN) region of adult rats to create complete AV block.

3 ar basis for adenosine-induced second-degree AV block.

4 n upfront pacing technique for patients with AV block.

5 ntricular response during AF without causing AV block.

6 ndent hypoglycemia, myocardial ischemia, and AV block.

7 cy stimulation identified GP sites producing AV block.

8 hythm, except for 1 with undetected complete AV block.

9 congenital and childhood nonimmune isolated AV block.

10 ose with abnormalities that increase risk of AV block.

11 epair, 48 (1.1%) underwent PPM placement for AV block.

12 ary SND associated with different degrees of AV block.

13 for those with subsequent PPM placement for AV block.

14 holine-induced first-degree and third-degree AV block.

15 injected fibroblasts without the creation of AV block.

16 Acetylcholine induced dose-dependent AV block.

17 tients with atrial fibrillation and advanced AV block.

18 ime, significant sinus bradycardia preceding AV block.

19 icular (AV) block, and 8 congenital complete AV block.

20 ide the homeodomain, and not associated with AV block.

21 used as a candidate gene in 2 children with AV block.

22 ant have been recognized in association with AV block.

23 tricular tachycardia and a high incidence of AV block.

24 ortion of tetralogy of Fallot and idiopathic AV block.

25 d by atrial tachycardia or atrioventricular (AV) block.

26 c2 in zebrafish results in atrioventricular (AV) block.

27 characterized by complete atrioventricular (AV) block.

28 , and one had first-degree atrioventricular (AV) block.

29 her incidence of both new-onset third-degree AV block (6.5% versus 2.5%; P=0.001) and new-onset LBBB

30 d the highest risk of new-onset third-degree AV block (adjusted odds ratio [aOR], 8.33 [95% CI, 3.31-

31 AV block after adenosine was only observed in AT patient

32 ement for atrioventricular conduction block (AV block) after operative repair of perimembranous ventr

33 Subjects with congenital AV block, all presenting with bradycardia, showed large

34 g conditions of this case, recapitulated 2:1 AV block and arrhythmia.

35 Only 3 days of complete AV block and autonomic denervation did not affect the pr

36 different myocardial layers in conditions of AV block and His-bundle pacing.

37 12 mongrel dogs, after creation of complete AV block and implantation of a ventricular inhibited pac

38 The overall incidence of third-degree AV block and new-onset LBBB after SAVR was 4.5% and 7.8%

39 Operative AV block and PPM placement occurred in 1.1% of patients

40 s evoked complex brady-tachycardia with both AV block and premature ventricular contractions (PVCs).

41 m) of canine left ventricle in conditions of AV block and right ventricular pacing.

42 reentrant tachycardia, the incidence of 2:1 AV block and the response to atropine and a single ventr

43 I AV block frequently progressed to complete AV block and was associated with seizures, death, and pa

44 ar basis for second-degree atrioventricular [AV] block), and the recovery of excitability in rabbit i

45 2 congenital second-degree atrioventricular (AV) block, and 8 congenital complete AV block.

46 eeks of age rapidly progressed into complete AV block as early as 4 weeks of age.

47 which further progressed into second-degree AV block at 4 weeks of age before the development of car

48 lure, left ventricular systolic dysfunction, AV block, atrial or ventricular arrhythmias, and sudden

49 tellate ganglion in dogs with chronic MI and AV block augments sympathetic nerve sprouting and create

50 tological findings in goat models for AF and AV block (AVB) using JavaCyte.

51 e presence of first-degree atrioventricular (AV) block, bifascicular block, left bundle branch block,

52 Another 6 dogs with MI and complete AV block but without NGF infusion served as controls (n=

53 Atropine abolished the bradycardia and AV block, but the atropine-insensitive tachycardia and P

54 rlier reports do not report the incidence of AV block by VSD type.

55 etus with cardiac failure caused by complete AV block (CAVB) may allow delivery of a full-term, stabl

56 athways may account for sporadic examples of AV block, complicating posteroseptal ablation in patient

57 Complete AV block created by segmental atrial isolation was achie

58 and ventricular rhythms in 22 (6%)-although AV block decreased (3 hearts, 1%).

59 , from 1980-2009) included 141 children with AV block diagnosed in utero, at birth, or before 15 year

60 al area and marked effects on AV conduction, AV block did not occur.

61 The first-degree AV block dose prolonged AV and AH intervals by 26% and 2

62 At the first-degree AV block dose, AVN effective refractory period increased

63 The incidence of induced 2:1 AV block during AV node reentrant tachycardia is approxi

64 In patients with 2:1 AV block during AV node reentrant tachycardia, the absen

65 APD90 and ERP were measured before and after AV block during pacing at 120 beats/min.

66 arify the mechanism of 2:1 atrioventricular (AV) block during AV node reentrant tachycardia induced i

67 conduction disturbances, and intraprocedural AV block emerged as predictors of PPM implantation after

68 interval in separate groups of dogs that had AV block for 1 week or 3 days with and without rapid pac

69 In dogs that had AV block for 1 week, 1 hour of rapid pacing prolonged V-

70 Type II AV block frequently progressed to complete AV block and

71 Patients with 2:1 AV block had a shorter tachycardia cycle length than did

72 ses, including atrial fibrillation, complete AV block, heart failure and supraventricular tachycardia

73 In AV-block hearts, CRTopt exclusively depended on interven

74 In left bundle-branch block and AV-block hearts, optimal hemodynamic effect of CRT depen

75 2.30; 95% CI, 1.70-3.11), atrioventricular (AV) block (HR, 1.48; 95% CI, 1.11-1.97), and myocardial

76 ands with cardiac anomalies and first-degree AV block, idiopathic AV block, or tetralogy of Fallot.

77 s were uncommon under basal conditions, with AV block in 14 (4%) and junctional rhythms in 4 (1%).

78 used transient PR prolongation and transient AV block in 30% and 50%, respectively.

79 achieved without the induction of pathologic AV block in 50 (81%) of 62 patients.

80 d in transient PR prolongation and transient AV block in 59% and 6%, while PF(REV-HIGH) caused transi

81 n RV PICM was evident 4 weeks after complete AV block in both groups; subsequent intervention led to

82 ering the major consequences associated with AV block in patients with complex congenital heart disea

83 onduction, whereas adenosine infusion caused AV block in TG(WT) mice but not TG(N488I) mice with pre-

84 n humans consistently caused bradycardia and AV block in the zebrafish.

85 The data imply that AV block in utero is accompanied by hypertrophy, which i

86 nsitive bradycardia with atrial-ventricular (AV) block in conscious WKY rats, thus indicating a paras

87 n have CHB induce complete atrioventricular (AV) block in the human fetal heart perfused by the Lange

88 dies revealed first-degree atrioventricular (AV) block in the transgenic heart at 1 week of age, whic

89 conduction abnormalities, including complete AV block, in the pups.

90 ow and fast pathway, and a Wenckebach type I AV block; in the third ECG, findings compatible with sim

91 Complications other than AV block included polymorphic ventricular tachycardia 10

92 High-grade AV block is a potential risk of lesions placed in the sy

93 ndle-branch block (n=8) or atrioventricular (AV) block (n=6) through atrial (A), right ventricular (R

94 tinuation, and no symptomatic bradycardia or AV blocks observed.

95 n the cycle length (CL) at which anterograde AV block occurred between group I (305 +/- 63 ms) and gr

96 Inadvertent high degree AV block occurred in 10 (16%) of 62 patients, with the A

97 Persistent 2:1 AV block occurred in 13 of 139 patients with AV node ree

98 Transient atrioventricular (AV) block occurred during eight cryo-applications (1 CA,

99 ccurred in 10 (16%) of 62 patients, with the AV block occurring at the time of the procedure in 6 pat

100 equire a permanent pacemaker for inadvertent AV block or because of AV nodal ablation after a failed

101 Features favoring AT were the presence of AV block or marked shortening of atrial cycle length bef

102 mes of interests were new-onset third-degree AV block or new-onset LBBB during the index hospitalizat

103 uded number of participants with high degree AV block or symptomatic pauses/ bradycardia and changes

104 sinus node disease (SND), atrioventricular (AV) block or pacemaker implantation, and intraventricula

105 09, 95% CI 1.05-1.14), the composite of SND, AV-block or pacemaker implantation (HR 1.06, 95% CI 0.94

106 17, 95% CI 1.16-1.18), the composite of SND, AV-block or pacemaker implantation (HR 1.40, 95% CI 1.37

107 h block (OR 39; P = 0.002), and first-degree AV block (OR 14; P = 0.001) on the baseline ECG are inde

108 malies and first-degree AV block, idiopathic AV block, or tetralogy of Fallot.

109 ffinity for calcium (P < 0.015), and reduced AV block (P = 0.04).

110 The highly penetrant and progressive AV block phenotype seen in human heterozygous missense m

111 Patients with atrioventricular (AV) block receiving right ventricular (RV) pacing are at

112 ion (AF) without producing atrioventricular (AV) block remains a clinical challenge.

113 ildhood nonimmune isolated atrioventricular (AV) block remains unknown.

114 but the risk of complications, in particular AV block, remains non-negligible.

115 cts, including progressive atrioventricular (AV) block requiring pacemaker implantation.

116 nd 28+/-8 bpm (first-degree and third-degree AV block, respectively; P<0.05).

117 n parents of children affected by idiopathic AV block revealed a high prevalence of conduction abnorm

118 3; p < 0.01); for patients with first-degree AV block (RR: 1.52; p < 0.01), left anterior hemiblock (

119 eline; and for patients with intraprocedural AV block (RR: 3.49; p < 0.01).

120 s accompanied by ECG abnormalities including AV block, sinus bradycardia, and ventricular dysfunction

121 p developed either second-degree or complete AV block spontaneously.

122 during AT (at doses sufficient to result in AV block) terminated or transiently suppressed focal AT

123 One group had uninterrupted AV block; the other group underwent a period of rapid le

124 In pigs with complete atrioventricular (AV) block, transcription factor T-box 18 (TBX18) was inj

125 Complete AV block was achieved in each of the 10 dogs with 6.5+/-

126 at 10 microg/min were infused until complete AV block was achieved.

127 Advanced AV block was also occasionally demonstrated in the mutan

128 The most significant risk factor for AV block was Down syndrome (odds ratio 3.62, 95% confide

129 Type I AV block was most often due to digitalis and was reversi

130 0]; P=0.011), whereas new-onset third-degree AV block was not associated with worse prognosis.

131 No AV block was seen in any cohort at 4 weeks.

132 PR prolongation (first degree AV block) was present at 4 weeks, 7 months, and 17 month

133 rest and during exertion, without pathologic AV block, was achieved long term in 45 (73%) of 62 patie

134 dogs with MI and complete atrioventricular (AV) block, we induced cardiac sympathetic nerve sproutin

135 ction, 14% to 30%), atrial fibrillation, and AV block were studied by pressure-volume analysis.

136 al (V-A) conduction, two suffered high-grade AV block when ablation of the systemic venous portion of

137 sent a rodent model of a complete and stable AV block, which enhances the rigor and reproducibility o

138 Associated phenotypes included AV block, which was the primary manifestation of cardiac

139 Tjp1 (Tjp1(fl/fl); Hcn4(CreERt2)) developed AV block while tamoxifen-induced conduction system delet

140 onal ablation because it can create complete AV block, while in effect permitting the equivalent of H

141 postoperative third-degree atrioventricular (AV) block with subsequent permanent pacemaker requiremen

142 sinus node dysfunction or atrioventricular (AV) block, with need for pacing, is common after the Fon

143 a feasible energy source to create complete AV block within the beating heart without damaging the o

144 secutive patients with pacing indication for AV block without CRT indication, with a 1:1 randomizatio

145 nduced loss of ZO-1 led to atrioventricular (AV) block without changes in heart rate, as measured by