ライフサイエンスコーパス: AV conduction

1 short PR intervals indicative of accelerated AV conduction.

2 AC(VI) gene transfer in treating diseases of AV conduction.

3 changing atrial rates and premature beats to AV conduction.

4 important role of Rho GTPases in regulating AV conduction.

5 o NA-VL cause bradycardia without decreasing AV conduction.

6 gation of the AH interval with continued 1:1 AV conduction.

7 r of the PR interval is consistent with dual AV conduction.

8 he central vagal control of cardiac rate and AV conduction.

9 2 microinjections into rNA-VL did not affect AV conduction.

10 atrioventricular (AV) node are essential for AV conduction.

11 th bradycardia and a decrease in the rate of AV conduction.

12 ailure, left bundle branch block, and intact AV conduction.

13 AV conduction abnormalities are far less common and gene

14 effects, including hypotension, chest pain, AV conduction abnormalities, and bronchospasm.

15 and were associated with varying degrees of AV conduction abnormalities, including complete AV block

16 urvived for 4 weeks and significantly slowed AV conduction and ventricular rate in acutely induced AF

17 agonist into the rNA-VL on atrioventricular (AV) conduction: and (3) find ultrastructural evidence fo

18 s in the AV nodal area and marked effects on AV conduction, AV block did not occur.

19 AV conduction axis pathology caused by environmental NPs

20 The atrioventricular (AV) conduction axis is key for the passage of electrical

21 Complete AV conduction block is typically treated by implanting a

22 nical model of severe bradyarrhythmia due to AV conduction block.

23 have intrinsic AV conduction or intermittent AV conduction block.

24 ptal defects and result in atrioventricular (AV) conduction block during postnatal life.

25 A mutations that result in atrioventricular (AV) conduction block.

26 dycardia (25 cases), ictal atrioventricular (AV)-conduction block (11 cases), postictal AV-conduction

27 (AV)-conduction block (11 cases), postictal AV-conduction block (2 cases), (post)ictal atrial flutte

28 Ictal asystole, bradycardia and AV-conduction block were self-limiting in all but one of

29 During seizures the occurrence of AV conduction blocks increased, predisposing Kv1.1-defic

30 To determine whether the interictal AV conduction blocks were of cardiac or neural origin, w

31 stered separately, only atropine ameliorated AV conduction blocks, indicating that excessive parasymp

32 ing a fivefold increase in atrioventricular (AV) conduction blocks, as well as bradycardia and premat

33 Radiofrequency catheter modification of AV conduction can be used to control the ventricular rat

34 ectrum consisting of sinus node dysfunction, AV conduction defects, and hypertrophic cardiomyopathy.

35 n patients with congenital atrioventricular (AV) conduction defects.

36 because of the potential for bradycardia or AV conduction delay, which may be induced by BAY 2413555

37 (Cx30.2) and a resulting loss of physiologic AV conduction delay.

38 egree or complete block of atrioventricular (AV) conduction developed in the absence of CAR in the ad

39 Atrioventricular (AV) conduction disturbances requiring permanent pacemake

40 We studied AV conduction during this transition and its sensitivity

41 Modification of atrioventricular (AV) conduction during atrial fibrillation (AF) may be ac

42 ions between substrate (atrial fibrillation, AV conduction, heart failure, MI, EF) and pacing promote

43 l sino-atrial (SA) rate or atrioventricular (AV) conduction; i.e., the SA and AV ganglia, respectivel

44 obtained with radiofrequency modification of AV conduction in patients with atrial fibrillation.

45 vented an evaluation of potential effects on AV conduction in the iNA-VL.

46 In this study, we examined postnatal AV conduction in the knockin mice.

47 the peptide tertiapin-Q could improve HR and AV conduction in these mouse strains.

48 The least common manifestation of dual AV conduction is an abrupt PR interval change in the set

49 ry mediating the vagal baroreflex control of AV conduction may be composed of as few as four neurons

50 injections, and transient nocturnal Mobitz I AV conduction occurred early postinjection in both group

51 e adult mouse heart and that prolongation of AV conduction occurred in the embryonic heart of the glo

52 h dual-chamber pacemakers who have intrinsic AV conduction or intermittent AV conduction block.

53 ble to induce bradycardia without decreasing AV conduction or ventricular contractility.

54 ructs in rat hearts to create an alternative AV conduction pathway.

55 ted arrhythmias, or when the location of the AV conduction pathways remains uncertain.

56 determination of sinus node recovery times, AV conduction properties, and atrial, AV, and ventricula

57 the AV ring, although no longer required for AV conduction, remains transiently able to conduct, prov

58 nglion which selectively mediates changes in AV conduction, retrogradely labeled neurons were histoch

59 During development, AV conduction switches from base-to-apex to apex-to-base

60 cient to direct expression to the developing AV conduction system (AVCS).

61 emporal patterning of Hcn4 expression in the AV conduction system required cis-regulatory elements wi

62 actility without influencing cardiac rate or AV conduction, this ganglion contains a functionally sel

63 l analyses, we provide evidence of permanent AV conduction through the implant in one-third of recipi

64 1 CA, 7 CMs) with immediate return of normal AV conduction upon cessation of application.

65 The AV conduction was characterized using a pacing electrode

66 cular function, and intact atrioventricular (AV) conduction were studied with serial-gated blood pool

67 of Glu into the rNA-VL caused a decrease in AV conduction without an effect on cardiac rate.