ライフサイエンスコーパス: AVNRT

1 AVNRT is an uncommon AF trigger seen more frequently in

2 Six of 22 AVNRT recurrences (27%) occurred >/=5 years after ablati

3 Analyses comprised 2384 AVNRT cases and 106 489 referents, and 2811 AVAP/AVRT ca

4 about the long-term results >/=3 years after AVNRT ablation in pediatric patients.

5 he test accurately predicted AVRT (n=29) and AVNRT (n=44) in all cases.

6 d predicted cardiac expression of NKX2-5 and AVNRT.

7 sought to characterize patients with AF and AVNRT and assess clinical outcomes after ablation.

8 The responses to PHCs distinguished AVRT and AVNRT with 100% specificity and sensitivity.

9 development and the sarcomere (AVAP/AVRT and AVNRT) as important potential effectors of supraventricu

10 nt from that required for atrial flutter and AVNRT.

11 Cs during tachycardia can distinguish JT and AVNRT with 100% specificity in adult patients.

12 al activation between ventricular pacing and AVNRT in only 21 of 46 patients.

13 Atypical AVNRT with eccentric retrograde left-sided activation wa

14 ricular node reentry (AVNRT; n=82), atypical AVNRT (n=13), permanent junctional reciprocating tachyca

15 uction intervals during typical and atypical AVNRT that occurred in the same patient.

16 tients with AVNRT, both typical and atypical AVNRT were induced at electrophysiology study by pacing

17 nd 65% of patients with typical and atypical AVNRT, respectively.

18 s usually possible to differentiate atypical AVNRT from ORT using a septal accessory pathway, a defin

19 Thirteen patients (65%) displayed atypical AVNRT with fast-slow characteristics.

20 PI-TCL are useful in distinguishing atypical AVNRT from ORT using a septal accessory pathway.

21 n age 48.5+/-18.1 years, 68 female, atypical AVNRT or coexistent atypical and typical AVNRT without o

22 n, as applied for typical cases, in atypical AVNRT.

23 with typical AVNRT (0%) P<0.001, or atypical AVNRT (0%) but similar to those with AT (11%) and PJRT (

24 In 30 patients with atypical AVNRT and 44 patients with ORT using a septal accessory

25 All 30 patients with atypical AVNRT and none of the 44 patients with ORT using a septa

26 r variables predicted ablation success (AVJ, AVNRT, or left free wall AP ablation and an experienced

27 To determine the prevalence of concomitant AVNRT and AF, 629 consecutive patients referred for cath

28 During AVNRT, only 2 patients had a single early site with foca

29 Retrograde atrial activation during AVNRT (337 +/- 43 ms) and ventricular pacing at a simila

30 ventricular pacing and in 43 patients during AVNRT.

31 ventricular pacing and in 26 patients during AVNRT.

32 hazard ratio of at least 3.6 for exhibiting AVNRT compared with the general population.

33 Familial AVNRT prevalence is higher than previously believed sugg

34 This indicates a familial AVNRT prevalence of 127 cases per 10 000 (95% confidence

35 e prevalence and characteristics of familial AVNRT among patients who underwent radiofrequency ablati

36 nts at ablation were younger in the familial AVNRT group when compared with the sporadic AVNRT group

37 lation reports of all patients with familial AVNRT (at least 2 first-degree family members) who under

38 After inclusion of 4 families with familial AVNRT who underwent ablation at another hospital our pop

39 ade fast pathway conduction during slow-fast AVNRT and anterograde fast pathway conduction during fas

40 ing slow-pathway radiofrequency ablation for AVNRT were assigned to autonomic blockade (0.2 mg/kg pro

41 Cryo-therapy may be more effective for AVNRT than septal AVRT.

42 ciation studies to identify genetic loci for AVNRT (4 studies) and AVAP/AVRT (7 studies).

43 We identified 2 significant loci for AVNRT, which implicate NKX2-5 and TTN as disease suscept

44 The success rate for AVNRT was higher than for AVRT (95.5% vs. 62.5%, p < 0.0

45 pothesized that JT can be distinguished from AVNRT based on specific responses to premature atrial co

46 tial AVNRT ablation, calculated freedom from AVNRT was 96% at 1 year, 94% at 3 years, 93% at 5 years,

47 f AVNRT ablation did not impact freedom from AVNRT.

48 Further, AVNRT could not be perturbed by a late PHC (<=20 ms ahea

49 Twenty-two had AVNRT, 8 AVRT, and 1 VT.

50 ation of any non-PV trigger of AF, including AVNRT.

51 ndeterminate rhythm, the technique indicated AVNRT in 1 patient and JT in 7 patients, and the test wa

52 re were 27 patients (4.3%) who had inducible AVNRT at the time of AF ablation.

53 t initiated AF and the presence of inducible AVNRT.

54 After the initial AVNRT ablation, calculated freedom from AVNRT was 96% at

55 re divided into 3 groups: clinically obvious AVNRT, clinically obvious JT, and clinically indetermina

56 In the 26 cases of clinically obvious AVNRT, the sensitivity and specificity of the test were

57 Cumulatively, catheter ablation of AVNRT continued to be effective in >90% of our pediatric

58 Ablation of AVNRT in patients with AF was associated with improved o

59 n for patients who had undergone ablation of AVNRT.

60 tion is involved and confirms a diagnosis of AVNRT.

61 Successful elimination of AVNRT was achieved in all patients.

62 The slow-fast form of AVNRT was also inducible in 17 of the 20 patients.

63 ither the fast-slow or the slow-slow form of AVNRT.

64 tion energy, and the procedural end point of AVNRT ablation did not impact freedom from AVNRT.

65 Procedural end point of AVNRT ablation had been either SP ablation (no residual

66 thway conduction times during the 2 types of AVNRT were calculated.

67 Although there are abundant data on AVNRT ablation in adult patients, little is known about

68 with 44 tachycardias suggesting either JT or AVNRT based on a short ventriculo-atrial interval and ap

69 n prematurity of the PHC required to perturb AVNRT was 48 ms (range, 28-70 ms) and the advancement le

70 =59), typical atrioventricular node reentry (AVNRT; n=82), atypical AVNRT (n=13), permanent junctiona

71 ion intervals during slow-fast and fast-slow AVNRT in the same patient, fast pathway conduction times

72 hat typical slow-fast and atypical fast-slow AVNRT use different anatomic pathways for fast conductio

73 ade fast pathway conduction during fast-slow AVNRT was 41.8+/-39.7 ms and was significantly different

74 AVNRT group when compared with the sporadic AVNRT group (44.2+/-19 versus 54.8+/-18 years old, P=0.0

75 ged </=18 years who had undergone successful AVNRT ablation were analyzed.

76 way is the therapy of choice for symptomatic AVNRT, regardless of whether the typical or atypical for

77 rioventricular nodal re-entrant tachycardia (AVNRT) do not use the same limb for fast conduction, but

78 trioventricular node re-entrant tachycardia (AVNRT) from orthodromic reciprocating tachycardia (ORT)

79 atrioventricular nodal re-entry tachycardia (AVNRT), atrioventricular reciprocating tachycardia (AVRT

80 atrioventricular node re-entry tachycardia (AVNRT).

81 oventricular node reciprocating tachycardia (AVNRT) can be associated with eccentric retrograde left-

82 trioventricular nodal reentrant tachycardia (AVNRT) and atrioventricular accessory pathways or atriov

83 trioventricular nodal reentrant tachycardia (AVNRT) are scarce, and the optimal ablation method has n

84 trioventricular nodal reentrant tachycardia (AVNRT) can also cause or coexist with AF.

85 trioventricular nodal reentrant tachycardia (AVNRT) is considered a sporadic disease occurring in app

86 ical form of AV nodal reentrant tachycardia (AVNRT) underwent selective radiofrequency ablation of th

87 trioventricular nodal reentrant tachycardia (AVNRT) who underwent fluoroscopically guided procedures

88 substrate for AV node reentrant tachycardia (AVNRT), ablation of the slow pathway potentially may be

89 trioventricular nodal reentrant tachycardia (AVNRT), an accessory pathway (AP), or the atrioventricul

90 trioventricular nodal reentrant tachycardia (AVNRT).

91 atients with AV nodal reentrant tachycardia (AVNRT).

92 t AP who had AV nodal reentrant tachycardia (AVNRT).

93 f atrioventricular node reentry tachycardia (AVNRT) and atrioventricular reentry tachycardia (AVRT) l

94 ing in cure of AV nodal reentry tachycardia (AVNRT) has led to the concept that these pathways are di

95 Earlier PHCs can advance the AVNRT circuit but only by a quantity less than the prema

96 cal AVNRT or coexistent atypical and typical AVNRT without other concomitant arrhythmia was diagnosed

97 logy study and catheter ablation for typical AVNRT.

98 those with AVRT (15%) compared with typical AVNRT (0%) P<0.001, or atypical AVNRT (0%) but similar t

99 l (VA) intervals (13%) compared with typical AVNRT (0%), P<0.005.

100 nd sex-matched control patients with typical AVNRT.

101 Of these, 13 underwent AVNRT ablation without PV isolation.

102 mination at the AV node in AVRT (85%) versus AVNRT (86%) after adenosine, but patients with AVRT show

103 However, it is not known whether AVNRT can occur with eccentric retrograde left-sided act

104 d with the rest of the cohort, patients with AVNRT and AF were younger at the time of symptom onset (

105 was demonstrated in 6% of all patients with AVNRT masquerading as tachycardia using a left-sided acc

106 First-degree relatives of patients with AVNRT presented a hazard ratio of at least 3.6 for exhib

107 We studied 2079 patients with AVNRT subjected to slow pathway ablation.

108 p 21.4+/-9.4 months); however, patients with AVNRT targeted for ablation were more likely to be AF fr

109 We studied 356 patients with AVNRT who underwent catheter ablation.

110 In 20 of 1299 patients with AVNRT, both typical and atypical AVNRT were induced at e

111 cluded AP conduction in all 53 patients with AVNRT.

112 ed a total of 24 families (50 patients) with AVNRT.

113 whom 20 had >/=1 first-degree relatives with AVNRT.