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1 erobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii).
2 and the full-length acetyltransferase WeeI (Acinetobacter baumannii).
3 istant strains of Pseudomonas aeruginosa and Acinetobacter baumannii.
4 ctivity do not address MDR pathogens such as Acinetobacter baumannii.
5 (ESBL)-producing Enterobacteriaceae and MDR Acinetobacter baumannii.
6 c, rifabutin (RBT), against highly resistant Acinetobacter baumannii.
7 t defense against the opportunistic pathogen Acinetobacter baumannii.
8 acteriaceae and the class D (OXA) enzymes of Acinetobacter baumannii.
9 ncy against the opportunistic human pathogen Acinetobacter baumannii.
10 designed an assay for the emerging pathogen Acinetobacter baumannii.
11 ella pneumoniae, Pseudomonas aeruginosa, and Acinetobacter baumannii.
12 ns caused by Gram-negative pathogens such as Acinetobacter baumannii.
13 consensus method for the active screening of Acinetobacter baumannii.
14 ing the pathogens Pseudomonas aeruginosa and Acinetobacter baumannii.
15 ed protein (Bap) in a bloodstream isolate of Acinetobacter baumannii.
16 Escherichia coli, Klebsiella pneumoniae and Acinetobacter baumannii.
17 n antibiotic with promising activity against Acinetobacter baumannii.
18 d drug resistance of the nosocomial pathogen Acinetobacter baumannii.
19 ylococcus aureus, Klebsiella pneumoniae, and Acinetobacter baumannii.
20 monas maltophilia, Burkholderia cepacia, and Acinetobacter baumannii.
22 lecular Microbiology, structural analysis in Acinetobacter baumannii 17978 revealed that a pentasacch
26 deaths were hospital-acquired, mainly due to Acinetobacter baumannii (47.1%) and K. pneumoniae (35.3%
27 acquired, mainly due to multidrug-resistant Acinetobacter baumannii (52.2%), Klebsiella pneumoniae (
28 siella pneumoniae [10], carbapenem-resistant Acinetobacter baumannii [8], carbapenem- and quinolone-r
34 trate, carnitine by non-heme iron containing Acinetobacter baumannii (Ab) oxygenase CntA/reductase Cn
36 sted against the class C cephalosporinase of Acinetobacter baumannii [Acinetobacter-derived cephalosp
38 lmonella enterica, Klebsiella pneumoniae and Acinetobacter baumannii against 28 different antibiotics
39 options exist for extensively drug resistant Acinetobacter baumannii, an emerging threat in ICUs worl
40 l monomicrobial necrotizing fasciitis due to Acinetobacter baumannii, an unusual finding that may be
41 Pathogenic Acinetobacter species, including Acinetobacter baumannii and Acinetobacter nosocomialis,
45 s and virulence with phage susceptibility in Acinetobacter baumannii and investigated draft genomes o
50 gues when used alone lacked activity against Acinetobacter baumannii and Klebsiella pneumoniae; howev
51 n associated with extensively drug-resistant Acinetobacter baumannii and multidrug-resistant Klebsiel
52 ilar protocols have been proposed to include Acinetobacter baumannii and Pseudomonas aeruginosa (SNAP
53 ymyxin resistant recent clinical isolates of Acinetobacter baumannii and Pseudomonas aeruginosa .
54 ority bacteria included carbapenem-resistant Acinetobacter baumannii and Pseudomonas aeruginosa, and
56 e-scaffolds would be more effective against Acinetobacter baumannii and Staphylococcus aureus biofil
60 tive (Staphylococcus aureus), gram-negative (Acinetobacter baumannii), and fungal (Candida albicans)
61 tive (Staphylococcus aureus), Gram-negative (Acinetobacter baumannii), and fungal (Candida albicans)
62 Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methicillin-resistant Staph
63 assifies carbapenemases in Enterobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa duri
64 d 100 well-characterized Enterobacteriaceae, Acinetobacter baumannii, and Pseudomonas aeruginosa isol
66 he majority of isolates of Enterobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa test
67 ococcus spp., members of the orange complex, Acinetobacter baumannii, and Pseudomonas aeruginosa were
69 cally troublesome pathogens, the prokaryote, Acinetobacter baumannii, and the eukaryote, Candida albi
70 of VIM-2 and SPM-1 from Escherichia coli and Acinetobacter baumannii, and their confinement to Pseudo
71 Infections caused by the bacterial pathogen Acinetobacter baumannii are a mounting concern for healt
72 essential genes in Acinetobacter lwoffii and Acinetobacter baumannii are active in vitro and in vivo.
73 esistant forms of the Gram-negative pathogen Acinetobacter baumannii are an emerging threat to human
76 -negative bacilli Pseudomonas aeruginosa and Acinetobacter baumannii are increasingly acquiring carba
78 yed for the treatment of multidrug-resistant Acinetobacter baumannii, as it can rapidly develop resis
79 ein 33 (Omp33) is an outer membrane porin of Acinetobacter baumannii associated with carbapenem resis
80 plified samples were Prevotella tannerae and Acinetobacter baumannii at frequencies between 89 and 10
81 activity against the Gram-negative pathogen Acinetobacter baumannii ATCC 17961 (MIC = 0.0078 muM).
82 The efficacy of antimicrobial therapy for Acinetobacter baumannii bacteremia has been difficult to
84 al targets in 51% of all wound samples, with Acinetobacter baumannii being the most frequently detect
85 ut in vitro screen to identify inhibitors of Acinetobacter baumannii biofilms using a library of natu
86 thogens that express a T2S system(s) include Acinetobacter baumannii, Burkholderia pseudomallei, Chla
87 nonflagellated, and pathogenic coccobacillus Acinetobacter baumannii, but there is considerable varia
88 smic domains from Pseudomonas aeruginosa and Acinetobacter baumannii by BOCILLIN FL, aztreonam, merop
90 the susceptibilities of 107 isolates of the Acinetobacter baumannii-calcoaceticus complex to amikaci
92 infected or not with Staphylococcus aureus, Acinetobacter baumannii, Candida albicans, or Aspergillu
93 aphylococcus aureus, Pseudomonas aeruginosa, Acinetobacter baumannii, carbapenem-resistant Enterobact
97 cteriaceae spp., Pseudomonas aeruginosa, and Acinetobacter baumannii chosen to provide extreme diagno
98 cient killing by GA was also demonstrated in Acinetobacter baumannii clinical isolates and approximat
102 In this study, we prospectively collected Acinetobacter baumannii complex clinical isolates from a
103 nterobacterales, Pseudomonas aeruginosa, and Acinetobacter baumannii complex for patient care until t
104 119 Pseudomonas aeruginosa isolates, and 83 Acinetobacter baumannii complex isolates) were tested.
105 s, from whom the Acinetobacter calcoaceticus-Acinetobacter baumannii complex was isolated over a 14-m
107 ydroxyphenylacetate (HPA) 3-hydroxylase from Acinetobacter baumannii consists of a reductase componen
110 ruginosa (CP-PA) and carbapenemase-producing Acinetobacter baumannii (CP-AB) and perform a multicente
111 anisms of resistance of carbapenem-resistant Acinetobacter baumannii (CRAB) were determined in hospit
112 tam antibiotics against carbapenem-resistant Acinetobacter baumannii (CRAB), a WHO "critical priority
113 rbapenem-resistant Enterobacteriaceae (CRE), Acinetobacter baumannii (CRAB), and Pseudomonas aerugino
116 tasets, as well as Klebsiella pneumoniae and Acinetobacter baumannii datasets, with whole genome sequ
117 ecium, and silver/copper combination against Acinetobacter baumannii demonstrated antimicrobial syner
118 with MenC or another Gram-negative bacteria, Acinetobacter baumannii, did not inhibit augmented PS-sp
119 jor protein species in the outer membrane of Acinetobacter baumannii does not belong to the high-perm
122 es its properties to those published for the Acinetobacter baumannii enzyme, an example of the altern
123 igm of clinical infectious disease research, Acinetobacter baumannii, Escherichia coli, Klebsiella pn
126 s with molecular methods used to distinguish Acinetobacter baumannii from other members of the A. cal
127 We performed WGS on longitudinal isolates of Acinetobacter baumannii from patients undergoing colisti
128 ideal sampling method for identification of Acinetobacter baumannii from the health care environment
129 comparative analysis on currently completed Acinetobacter baumannii genomes revealed extensive and d
130 the in-hospital mortality was higher in the Acinetobacter baumannii group (16% vs. 13%; odds ratio,
131 cute liver failure were more frequent in the Acinetobacter baumannii group compared to the control gr
138 t emergence of multidrug resistance (MDR) in Acinetobacter baumannii has raised concern in health car
141 rk done over the past 5 years has shown that Acinetobacter baumannii has the remarkable capability to
142 multidrug-resistant, opportunistic pathogen, Acinetobacter baumannii, has spread swiftly through hosp
144 and incidence rates of carbapenem-resistant Acinetobacter baumannii (Hungary), extended-spectrum bet
145 gative pathogens, such as colistin-resistant Acinetobacter baumannii in a mouse peritonitis-sepsis mo
146 of both drug-susceptible and drug-resistant Acinetobacter baumannii in a soft-tissue infection model
147 fect of the presence of carbapenem-resistant Acinetobacter baumannii in accordance with surveillance
148 domonas aeruginosa, and carbapenem-resistant Acinetobacter baumannii in the pediatric population.
149 nfectivity of bacterial pathogens, including Acinetobacter baumannii, in animal models, but the mecha
150 re unit length of stay, and complications of Acinetobacter baumannii including multidrug-resistant st
151 observed with a second respiratory pathogen, Acinetobacter baumannii, indicating that the phenomenon
154 e or clinical indifference, the impact of an Acinetobacter baumannii infection on mortality is inconc
156 intensive care unit patients with confirmed Acinetobacter baumannii infection were defined as cases.
158 -deficient mice were protected from systemic Acinetobacter baumannii infection, suggesting that facto
159 between mortality and colistin resistance in Acinetobacter baumannii infections and the interaction w
160 s for treatment of multidrug-resistant (MDR) Acinetobacter baumannii infections are extremely limited
162 d Clostridioides difficile and pan-resistant Acinetobacter baumannii infections in murine models.
163 ulfonate (CMS) to treat carbapenem-resistant Acinetobacter baumannii infections, colistin resistance
165 s Diseases (NSRB) against BasE, an AAAE from Acinetobacter baumannii involved in production of the si
192 enem-associated outer membrane protein) from Acinetobacter baumannii is a small OM protein that has b
218 n antibiotic with promising activity against Acinetobacter baumannii, is mediated by an intrinsically
219 CPNFs), including Pseudomonas aeruginosa and Acinetobacter baumannii, is necessary to prevent their d
220 contains this coumarate catabolism pathway, Acinetobacter baumannii, is resistant to inhibition by t
221 se collection of epidemiologically unrelated Acinetobacter baumannii isolates to compare the robustne
223 rbapenems (imipenem and meropenem); 90.8% of Acinetobacter baumannii isolates were susceptible to min
225 or 99 isolates of Pseudomonas aeruginosa, 26 Acinetobacter baumannii isolates, and 11 Stenotrophomona
227 Moraxella catarrhalis, and most recently in Acinetobacter baumannii, it has become increasingly appa
228 lethal infections with clinical isolates of Acinetobacter baumannii, Klebsiella pneumoniae and E. co
229 en overlooked for priority pathogens such as Acinetobacter baumannii, Klebsiella pneumoniae and Pseud
230 ogenic carbapenem-resistant strains, such as Acinetobacter baumannii, Klebsiella pneumoniae, and Esch
231 stant clinical isolates of Escherichia coli, Acinetobacter baumannii, Klebsiella pneumoniae, Pseudomo
232 important Gram-negative pathogens including Acinetobacter baumannii, Klebsiella pneumoniae, Pseudomo
233 am-negative bacteria, but colistin-resistant Acinetobacter baumannii lacking lipid A were isolated af
236 ers for identifying carbapenem resistance in Acinetobacter baumannii, methicillin resistance in Staph
237 To successfully establish an infection, Acinetobacter baumannii must overcome the iron starvatio
240 mophilus influenzae, Pseudomonas aeruginosa, Acinetobacter baumannii, Neisseria meningitidis, Bactero
250 6, Pseudomonas aeruginosa, pandrug-resistant Acinetobacter baumannii (PDRAB), Staphylococcus aureus,
253 activation of MDR Pseudomonas aeruginosa and Acinetobacter baumannii (planktonic cells and biofilms)
259 taphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ent
260 taphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ent
261 taphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ent
262 taphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ent
263 in Escherichia coli, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ste
264 onfermenting gram-negative bacteria, such as Acinetobacter baumannii, Pseudomonas aeruginosa, and Ste
265 panresistant Gram-negative bacilli, such as Acinetobacter baumannii, requires consideration of nonan
266 Klebsiella spp., Pseudomonas aeruginosa, and Acinetobacter baumannii) resistant to cephalosporins or
268 n of NAD metabolism in the emerging pathogen Acinetobacter baumannii revealed unique features suggest
269 Investigating the opportunistic pathogen Acinetobacter baumannii's response to Zn starvation, we
270 ork, we show that the opportunistic pathogen Acinetobacter baumannii senses and responds to blue ligh
272 ly, that this activity is maintained against Acinetobacter baumannii, Stenotrophomonas maltophilia, S
273 Pse-containing exopolysaccharide (EPS) from Acinetobacter baumannii strain 54149 (Ab-54149) to form
275 e and rapid spreading of multidrug-resistant Acinetobacter baumannii strains has become a major healt
277 (3) CFU mL(-1) of Pseudomonas aeruginosa and Acinetobacter baumannii strains that were undetectable u
280 t strains of some bacterial species, such as Acinetobacter baumannii strains, cannot be reliably iden
281 lls and antimicrobial activity against seven Acinetobacter baumannii strains, resistant to polymyxin
283 ed genes in a multidrug-resistant isolate of Acinetobacter baumannii that are required for growth and
285 o MDR isolates of Pseudomonas aeruginosa and Acinetobacter baumannii through in vitro disk diffusion,
287 a representative nosocomial human pathogen, Acinetobacter baumannii, to chlorhexidine to identify th
290 comial pathogens, Pseudomonas aeruginosa and Acinetobacter baumannii, was also detected using this me
294 l intensive care unit stay was prolonged for Acinetobacter baumannii when compared to controls (media
296 d carbacephalosporinase producing strains of Acinetobacter baumannii, which have been listed by the W
297 ella pneumoniae, Pseudomonas aeruginosa, and Acinetobacter baumannii with an even higher affinity (K(
298 erobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii with various beta-lactamase susc
299 la tularensis, Burkholderia pseudomallei and Acinetobacter baumannii, with a view to exploiting RNase