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1 d after 95 days by a microbiota dominated by Actinobacillus.
6 Porphyromonas gingivalis (P. gingivalis) and Actinobacillus actinomycetemcomitans (A. actinomycetemco
7 counter known periodontal pathogens, such as Actinobacillus actinomycetemcomitans (A.a.) and, therefo
8 tle vector that is capable of replicating in Actinobacillus actinomycetemcomitans (Aa) and Escherichi
9 nes from a lambda gt11 expression library of Actinobacillus actinomycetemcomitans (Aa) genomic DNA th
11 ing protein lactoferrin (LF) may either kill Actinobacillus actinomycetemcomitans (Aa) or interfere w
12 s of four randomized treatment modalities on Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
13 study evaluated the reinfection incidence by Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
14 dentify the following target microorganisms: Actinobacillus actinomycetemcomitans (Aa), Tannerella fo
15 on dioxide (CO(2)) is required for growth of Actinobacillus actinomycetemcomitans (Aa), the reputed c
16 tion has tested the hypothesis that HGF from Actinobacillus actinomycetemcomitans (Aa)-infected patie
17 lla nigrescens (OR=1.7; 95% CI, 1.1 to 2.6), Actinobacillus actinomycetemcomitans (OR=1.7; 95% CI, 1.
19 5 (PG-2, PG-3, and PG-5) were tested against Actinobacillus actinomycetemcomitans (three strains) and
21 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans [Aa]), and divided
22 [IgG Bf], Porphyromonas gingivalis [IgG Pg], Actinobacillus actinomycetemcomitans [IgG Aa]), and asse
24 acteria, including the periodontal pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
25 G2 dominates responses to carbohydrates from Actinobacillus actinomycetemcomitans and Porphyromonas g
27 ntigens associated with host immunity (using Actinobacillus actinomycetemcomitans and Porphyromonas g
28 antigens associated with host immunity (with Actinobacillus actinomycetemcomitans and Porphyromonas g
29 es to the periodontitis-associated pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
30 lms produced by the human periodontopathogen Actinobacillus actinomycetemcomitans and the porcine res
31 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans are naturally compe
35 uced by the Gram-negative periodontopathogen Actinobacillus actinomycetemcomitans as well as by the G
36 entrations and the IgG2 antibody response to Actinobacillus actinomycetemcomitans can be influenced b
38 enomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycetemcomitans could be identified
40 that treatment of human lymphocytes with the Actinobacillus actinomycetemcomitans cytolethal distendi
43 ells of the gram-negative periodontopathogen Actinobacillus actinomycetemcomitans express a surface-e
44 ere, we report that the periodontal pathogen Actinobacillus actinomycetemcomitans expresses a homolog
45 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans form tenaciously ad
47 eficient mutants of the periodontal pathogen Actinobacillus actinomycetemcomitans from a library of r
49 risk factor for periodontitis in adults, and Actinobacillus actinomycetemcomitans has been implicated
51 utilized to screen for exported proteins of Actinobacillus actinomycetemcomitans in Escherichia coli
52 ducer 2 (AI-2) produced by the oral pathogen Actinobacillus actinomycetemcomitans influences growth o
57 The cytolethal distending toxin (CDT) of Actinobacillus actinomycetemcomitans is a typical member
58 The cytolethal distending toxin (Cdt) of Actinobacillus actinomycetemcomitans is an atypical A-B-
59 The oral commensal Gram-negative bacterium Actinobacillus actinomycetemcomitans is believed to be t
63 own to confer a hyperleukotoxic phenotype in Actinobacillus actinomycetemcomitans IS1, but the mechan
68 lasts (HGF) were incubated for 24 hours with Actinobacillus actinomycetemcomitans lipopolysaccharide
69 linical isolates of the periodontal pathogen Actinobacillus actinomycetemcomitans live as autoaggrega
70 IL-1beta, Porphyromonas gingivalis LPS, and Actinobacillus actinomycetemcomitans LPS, MIP-1alpha mRN
78 at the oral bacterium and periodontopathogen Actinobacillus actinomycetemcomitans secretes an immunos
79 ip exists between antibody reactive with the Actinobacillus actinomycetemcomitans serotype b lipopoly
80 le periodontitis (LJP) patients colonized by Actinobacillus actinomycetemcomitans serotype b often co
81 r gene probe hybridized to Southern blots of Actinobacillus actinomycetemcomitans strain JP2 (serotyp
86 ded by the tad (for tight adhesion) locus in Actinobacillus actinomycetemcomitans that is involved in
87 ere assayed for Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans using a 16S rRNA po
88 a membrane protein of the periodontopathogen Actinobacillus actinomycetemcomitans was achieved by con
92 pe b-specific carbohydrate antigen (SbAg) of Actinobacillus actinomycetemcomitans Y4 is reported to b
93 annerella forsythensis, Treponema denticola, Actinobacillus actinomycetemcomitans) and dental caries
94 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
95 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
96 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
97 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
98 monas gingivalis, Bacteroides forsythus, and Actinobacillus actinomycetemcomitans) and serum antibody
99 coccus mutans, Porphyromonas gingivalis, and Actinobacillus actinomycetemcomitans) selected based upo
100 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), a capnophilic fac
101 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Campylobacter
102 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Porphyromonas
103 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Streptococcus
104 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Campylobacter rec
105 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
106 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
107 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
108 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Prevotella interm
109 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans)-biofilm colonizing
116 in a polymorphic region of the chromosome of Actinobacillus actinomycetemcomitans, a predominant oral
117 red gene required for tenacious adherence of Actinobacillus actinomycetemcomitans, also has significa
118 microorganisms, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, also invade the pe
119 usobacterium nucleatum, Eikenella corrodens, Actinobacillus actinomycetemcomitans, and Campylobacter
120 sed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomitans, and Fusobacterium
121 4 subjects were analyzed for the presence of Actinobacillus actinomycetemcomitans, Bacteroides forsyt
122 eled synthetic oligonucleotides specific for Actinobacillus actinomycetemcomitans, Bacteroides forsyt
124 tivation by microbial or protein Ags (namely Actinobacillus actinomycetemcomitans, bovine insulin, an
125 CEK (species other than Haemophilus species, Actinobacillus actinomycetemcomitans, Cardiobacterium ho
126 , Capnocytophaga, Propionibacterium, yeasts, Actinobacillus actinomycetemcomitans, coagulase-negative
127 Gram-negative bacterial species, such as Actinobacillus actinomycetemcomitans, contain lipopolysa
128 pathogens (such as Porphyromonas gingivalis, Actinobacillus actinomycetemcomitans, etc.) into the lun
129 levels 8 to 20 h after infection with either Actinobacillus actinomycetemcomitans, F. nucleatum, or P
130 BD-3 against a collection of oral organisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucl
131 ed to distinguish among clinical isolates of Actinobacillus actinomycetemcomitans, Haemophilus aphrop
134 ntal pathogens, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, may contribute to
136 ned risk indicators for oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
138 d a 16S rRNA PCR detection method identified Actinobacillus actinomycetemcomitans, Porphyromonas ging
139 the following 3 major periodontal pathogens: Actinobacillus actinomycetemcomitans, Porphyromonas ging
140 imultaneous detection and differentiation of Actinobacillus actinomycetemcomitans, Porphyromonas ging
142 obability for detecting oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
143 Previously, we reported that intracellular Actinobacillus actinomycetemcomitans, Porphyromonas ging
144 croscopy to detect the periodontal pathogens Actinobacillus actinomycetemcomitans, Porphyromonas ging
145 nce of known periodontal pathogens including Actinobacillus actinomycetemcomitans, Prevotella interme
146 t bacteriostatic effect, particularly on the Actinobacillus actinomycetemcomitans, Prevotella oris, S
147 oral pathogens Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, skin commensal Sta
150 ridement is recommended for the treatment of Actinobacillus actinomycetemcomitans-associated periodon
151 The purpose of this study was to evaluate Actinobacillus actinomycetemcomitans-responsive B lympho
152 amma is clearly associated with (i) enhanced Actinobacillus actinomycetemcomitans-specific RANKL-expr
166 gatibacter actinomycetemcomitans [previously Actinobacillus actinomycetemcomitans], Prevotella interm
168 The periodontal pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans has an irregular o
169 required for the growth of Aggregatibacter (Actinobacillus) actinomycetemcomitans in culture under c
173 oral and systemic pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans produces a leukoto
176 ng decreases in the genera Streptococcus and Actinobacillus as well as increases in Oribacterium and
177 s that localized juvenile and other forms of Actinobacillus-associated periodontitis are primarily as
178 d average time to detection for Haemophilus, Actinobacillus, Cardiobacterium, Eikenella, and Kingella
180 containing a periplasmic binding protein in Actinobacillus for its potential role in nutrient acquis
182 as the only Actinomyces spp. coisolated with Actinobacillus (Haemophilus) actinomycetemcomitans; Acti
185 ix host-associated microbial clades spanning Actinobacillus, Pasteurella, Streptococcus, and Erysipel
186 y be applied to other members of Haemophilus-Actinobacillus-Pasteurella family, where genetic manipul
188 previous studies demonstrated that Adh from Actinobacillus pleuropneumoniae (A. pleuropneumoniae) is
189 a coli (Ec) was constructed by modifying the Actinobacillus pleuropneumoniae (Ap) plasmid pYG53.
192 of its ability to confer NAD independence on Actinobacillus pleuropneumoniae and H. influenzae, has b
193 ting of two fastidious veterinary pathogens, Actinobacillus pleuropneumoniae and Haemophilus somnus,
194 nimals intranasally co-infected (n = 7) with Actinobacillus pleuropneumoniae and Pasteurella multocid
199 Here we report the crystal structure of Actinobacillus pleuropneumoniae HMW1C (ApHMW1C), a funct
201 system, we introduced the P5PAB operon from Actinobacillus pleuropneumoniae into an Escherichia coli
209 s, which differentiates serovars 3, 6, and 8 Actinobacillus pleuropneumoniae isolates, is described.
211 sequence optimized for glycosylation by the Actinobacillus pleuropneumoniae N-glycosyltransferase (N
212 directly quantify glycopeptide formation by Actinobacillus pleuropneumoniae NGT and determine its su
214 t a vaccine prepared from outer membranes of Actinobacillus pleuropneumoniae serotype 5 can elicit pr
216 ee extracts isolated from colony biofilms of Actinobacillus pleuropneumoniae serotype 5 were found to
218 tiplex PCR assays were developed to identify Actinobacillus pleuropneumoniae serotypes 1, 2, and 8.
221 -ray crystal structure of the Cu,Zn SOD from Actinobacillus pleuropneumoniae, a major porcine pathoge
222 steurellaceae indicates that M. haemolytica, Actinobacillus pleuropneumoniae, and Haemophilus ducreyi
223 genic Neisseriaceae, Haemophilus influenzae, Actinobacillus pleuropneumoniae, and M. catarrhalis.
225 Haemophilus parasuis, Pasteurella multocida, Actinobacillus pleuropneumoniae, Bordetella bronchisepti
231 apsule type 3), Streptococcus zooepidemicus, Actinobacillus spp., and Mycoplasma equirhinis and equin
232 biosuccinic acid using the bacterial strain Actinobacillus succinogenes 130 Z, and simultaneously pr
233 pathway and the central carbon metabolism of Actinobacillus succinogenes for the production of succin
234 ethod of sampling is applied to the organism Actinobacillus succinogenes for the production of succin
236 ling either cells or membranes purified from Actinobacillus succinogenes to drive electron transfer a
238 19 mm and resistance at < or =11 mm, and for Actinobacillus suis, Erysipelothrix rhusiopathiae, and S