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1  regulated in response to coaggregation with Actinomyces naeslundii.
2 n of KB cells by other oral streptococci and Actinomyces naeslundii.
3 ion with only group A and group E strains of Actinomyces naeslundii.
4 us gordonii but was required for adhesion of Actinomyces naeslundii.
5 sitive coaggregations of these bacteria with Actinomyces naeslundii.
6 uman erythrocytes, and to the oral bacterium Actinomyces naeslundii.
7 eponema denticola, Tannerella forsythia, and Actinomyces naeslundii.
8 monas gingivalis, Peptostreptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococ
9             The presence of higher levels of Actinomyces Naeslundii (An) enhanced the effect of being
10                                  Only the Sg/Actinomyces naeslundii (An)/Fn multispecies biofilms eli
11                                              Actinomyces naeslundii, an early colonizer of the oral c
12                            The oral bacteria Actinomyces naeslundii and Actinomyces viscosus are know
13 he Orange-Blue cluster score (which included Actinomyces naeslundii and Eubacterium nodatum) was inve
14 al pathogens including Streptococcus mutans, Actinomyces naeslundii and Prevotella intermedia.
15                                              Actinomyces naeslundii and Streptococcus gordonii, oral
16   Biofilms of S. mutans, alone or mixed with Actinomyces naeslundii and Streptococcus oralis, were in
17 terium spp., and Tannerella forsythia, while Actinomyces naeslundii and Streptococcus sanguinis were
18 ngle-species biofilms: Streptococcus mutans, Actinomyces naeslundii, and Enterococcus faecalis.
19 terococcus faecalis, Streptococcus gordonii, Actinomyces naeslundii, and Lactobacillus acidophilus),
20 hogen Streptococcus mutans UA159, as well as Actinomyces naeslundii ATCC 12104 and Streptococcus oral
21 y in vitro on streptococcal biofilms than on Actinomyces naeslundii biofilms.
22 increased the ability of enterococci to bind Actinomyces naeslundii cells.
23 sequence of the chromosomal DNA flanking the Actinomyces naeslundii (formerly A. viscosus) T14V type
24 eptococcus sanguis, Haemophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A.
25 tobacillus acidophilus, Lactobacillus casei, Actinomyces naeslundii genospecies (gsp) 1 and 2, total
26 immune response in saliva to colonization by Actinomyces naeslundii genospecies 1 and 2 was studied i
27                       These species included Actinomyces naeslundii II, Actinomyces israelii, Actinom
28 d of species found in healthy oral biofilms (Actinomyces naeslundii, Lactobacillus casei, Streptococc
29 eponema denticola, Streptococcus oralis, and Actinomyces naeslundii levels.
30  hydroxyapatite and did not coaggregate with Actinomyces naeslundii PK606.
31 which was composed of Streptococcus sanguis, Actinomyces naeslundii, Porphyromonas gingivalis, and Fu
32                                              Actinomyces naeslundii, Prevotella spp., and Porphyromon
33  in the presence of Streptococcus oralis and Actinomyces naeslundii steadily formed exopolysaccharide
34 brial gene clusters present in the genome of Actinomyces naeslundii strain MG-1.
35 he oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggreg
36 ive with antibody against type 2 fimbriae of Actinomyces naeslundii T14V (anti-type-2) were much less
37 saliva of two human oral commensal bacteria, Actinomyces naeslundii T14V and Streptococcus oralis 34,
38                       The type 1 fimbriae of Actinomyces naeslundii T14V mediate adhesion of this gra
39               The nucleotide sequence of the Actinomyces naeslundii T14V type 2 fimbrial structural s
40 s gordonii DL1, Streptococcus oralis 34, and Actinomyces naeslundii T14V).
41 romonas gingivalis, Fusobacterium nucleatum, Actinomyces naeslundii, Tannerella forsythia, and Strept
42                               The ability of Actinomyces naeslundii to convert sucrose to extracellul
43 organisms such as Streptococcus gordonii and Actinomyces naeslundii to the saliva-coated tooth surfac
44            When the main fimbrial subunit of Actinomyces naeslundii type I fimbriae, FimA, is express
45 ide to support fimbriae-mediated adhesion of Actinomyces naeslundii was explained by the position of
46 f antibodies against Eubacterium nodatum and Actinomyces naeslundii) was inversely associated with al
47  Streptococcus sanguis and type 2 fimbriated Actinomyces naeslundii, which bound terminal sialic acid
48 and characterized the urease gene cluster of Actinomyces naeslundii, which is one of the pioneer orga
49        A gene encoding FTF was isolated from Actinomyces naeslundii WVU45; the deduced amino acid seq