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1                                              Adams Cowley Shock Trauma Center between November 2015 a
2                                              Adams Cowley Shock Trauma Center of the University of Ma
3                                              Adams et al. in this issue of Immunity provide evidence
4                                              Adams, "Introduction of Indus River Basin: Water securit
5                                              Adams, et al. describe a multisite study evaluation of p
6                                              Adams-Oliver syndrome (AOS) is a rare developmental diso
7                                              Adams-Oliver syndrome (AOS) is a rare disorder character
8 pply evolved magmas to Mounts St. Helens and Adams, and possibly Rainier, and could contain approxima
9  eastern Nebraska (Sarpy, Dakota, Wayne, and Adams) have a higher risk of flooding consequences due t
10 ade before her language deficits arose, Anne Adams (AA), a lifelong scientist, developed an intense d
11                  In an accompanying article, Adams et al. used a subsample of the National Institutes
12                      In this issue of Blood, Adams et al provide evidence for an important novel func
13                              Therefore, both Adams and the miR-29 family represent therapeutic target
14 BPJ) in two independent families affected by Adams-Oliver syndrome (AOS), a rare multiple-malformatio
15                       See related article by Adams et al., Cancer Res 1999;59:2615-22Visit the Cancer
16 sumer expenditures for alcohol, published by Adams Business Research.
17 function variants in DLL4 are known to cause Adams-Oliver syndrome, this is the first report of a hyp
18 tigated a recognized developmental disorder, Adams-Oliver syndrome (AOS), characterized by the combin
19 but mesotrione-sensitive population (ACR for Adams County mesotrione-sensitive but atrazine-resistant
20 ghly cited 1999 Cancer Research article from Adams and colleagues was published during the period whe
21                                      Helens, Adams, and Rainier these pathways connect subduction-ind
22              Impaired RBPJ-DNA binding as in Adams-Oliver-Syndrome affect both target site associatio
23  (i) gain-of-function CdGAP mutants found in Adams-Oliver Syndrome patients strongly destabilize cell
24  model renal fibrosis, we observed increased Adams gene expression that was blocked by oral administr
25                                         Jean Adams and colleagues argue that population interventions
26                                         Jean Adams discusses the evidence around food marketing restr
27                                       Julian Adams and scientists at ProScript (later Millennium Phar
28                      The Carpentier-McCarthy-Adams (CMA) IMR ETlogix annuloplasty ring is the first r
29 ace1), A disintegrin and metalloproteinases (Adams), and presenilins (Psen).
30 e melt source region lies east towards Mount Adams.
31        Two patients demonstrated features of Adams-Oliver syndrome, with genetic testing identifying
32 ls results in a significant up-regulation of Adams 10, 17, 12, and 19.
33                                  C., Li, R., Adams, N., Winuthayanon, W., Hamilton, K.
34 ic Reactions and the initiation of the Roger Adams Award are discussed.
35                        This study shows that Adams are involved in renal fibrosis and are regulated b
36 al solute concentration that incorporate the Adams-Fujita assumption for composition-dependence of ac
37  numerical simulation of our model using the Adams-Bashforth 3-step method to verify the theoretical
38 d breakthrough curves were best fit with the Adams-Bohart and the Clark models, as they provided the
39 ent with theoretical predictions coined the "Adams instability." When scaled properly, slip pulses ex
40               A dataset is generated through Adams method for the proposed NNs-BLMS in case of variou
41 Pases that regulate Rac1 are associated with Adams-Oliver syndrome (AOS), a syndrome characterized by
42 OTCH1 variants in unrelated individuals with Adams-Oliver syndrome (AOS), a rare disease with major f
43 posed by a father and two affected sibs with Adams Oliver syndrome (AOS) (OMIM 100300).