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1 nas gingivalis, Fusobacterium nucleatum, and Aggregatibacter actinomycetemcomitans).
2 t organisms, namely Streptococcus mutans and Aggregatibacter actinomycetemcomitans.
3 biofilm formation by the periodontopathogen Aggregatibacter actinomycetemcomitans.
4 h levels of biofilm Campylobacter rectus and Aggregatibacter actinomycetemcomitans.
5 the OFD group had a significant reduction in Aggregatibacter actinomycetemcomitans.
6 se caused by Cdt-producing organisms such as Aggregatibacter actinomycetemcomitans.
7 omyces naeslundii, Streptococcus mutans, and Aggregatibacter actinomycetemcomitans.
8 scherichia coli and leukotoxin A (LtxA) from Aggregatibacter actinomycetemcomitans.
9 4 to 5 mm and >/= 6 mm, BOP, and ABL, except Aggregatibacter actinomycetemcomitans.
10 files, especially those testing positive for Aggregatibacter actinomycetemcomitans.
11 .884) had a decreased colonization risk with Aggregatibacter actinomycetemcomitans.
12 in that is secreted from the oral bacterium, Aggregatibacter actinomycetemcomitans.
13 the remaining five implants, per group, with Aggregatibacter actinomycetemcomitans.
14 odontopathogens Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans.
15 is a protein secreted by the oral bacterium Aggregatibacter actinomycetemcomitans A. actinomycetemco
16 usobacterium nucleatum (a middle colonizer), Aggregatibacter actinomycetemcomitans (a late colonizer)
20 high orange" bacterial complexes, and "high" Aggregatibacter actinomycetemcomitans (Aa) colonization.
21 duce the proinflammatory response induced by Aggregatibacter actinomycetemcomitans (Aa) in human coro
22 ent species in a bacterial biofilm of Sg and Aggregatibacter actinomycetemcomitans (Aa) in terms of h
23 of periodontal disease (PD) associated with Aggregatibacter actinomycetemcomitans (Aa) infection.
29 rsythia (Tf), Porphyromonas gingivalis (Pg), Aggregatibacter actinomycetemcomitans (Aa), Fusobacteriu
30 nd quantifying Fusobacterium nucleatum (Fn), Aggregatibacter actinomycetemcomitans (Aa), Porphyromona
31 tal amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomycetemcomitans (Aa), Tannerella f
32 n (Ig) G response, and alveolar bone loss in Aggregatibacter actinomycetemcomitans (Aa)-inoculated Fa
33 ylneuraminate substrate-binding protein from Aggregatibacter actinomycetemcomitans (AaSiaP) in both t
35 gnificant differences in serum IgG levels to Aggregatibacter actinomycetemcomitans among the four dia
37 xamined in vivo and in vitro colonization by Aggregatibacter actinomycetemcomitans, an organism highl
38 lus systems from several bacteria, including Aggregatibacter actinomycetemcomitans and Caulobacter cr
39 mice were infected on days 0, 2, and 4 with Aggregatibacter actinomycetemcomitans and divided into g
41 oad, whereas PCT3 and PCT5 were dominated by Aggregatibacter actinomycetemcomitans and Porphyromonas
43 was observed against the periodontopathogens Aggregatibacter actinomycetemcomitans and Porphyromonas
44 by a 3-log reduction in periodontopathogenic Aggregatibacter actinomycetemcomitans and Porphyromonas
45 In the present study, the aim is to measure Aggregatibacter actinomycetemcomitans and Porphyromonas
47 ofilm consisting of the periodontal pathogen Aggregatibacter actinomycetemcomitans, and a commensal S
48 ingivalis, Prevotella intermedia/nigrescens, Aggregatibacter actinomycetemcomitans, and Fusobacterium
49 iodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, and Fusobacterium
50 susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, and Fusobacterium
51 d substantial evidence of the association of Aggregatibacter actinomycetemcomitans, and its highly le
52 Campylobacter rectus, Tannerella forsythia, Aggregatibacter actinomycetemcomitans, and Peptostreptoc
53 a denticola (Td), Tannerella forsythia (Tf), Aggregatibacter actinomycetemcomitans, and Prevotella in
54 phyromonas gingivalis, Tannerella forsythia, Aggregatibacter actinomycetemcomitans, and total bacteri
55 formation and virulence of the oral pathogen Aggregatibacter actinomycetemcomitans, and we previously
57 by Staphylococcus aureus, S. epidermidis and Aggregatibacter actinomycetemcomitans, but not by A. ple
58 cy strains were clustered into eight groups: Aggregatibacter actinomycetemcomitans, Campylobacter spp
60 parainfluenzae, Aggregatibacter aphrophilus, Aggregatibacter actinomycetemcomitans, Cardiobacterium h
61 ng toxin (Cdt) from the periodontal pathogen Aggregatibacter actinomycetemcomitans causes extensive d
62 the essential functions of the oral pathogen Aggregatibacter actinomycetemcomitans change during pair
63 the phagocytic ability of mast cells against Aggregatibacter actinomycetemcomitans compared with macr
64 arrest in lymphocytes after exposure to the Aggregatibacter actinomycetemcomitans cytolethal distend
65 est in lymphocytes following exposure to the Aggregatibacter actinomycetemcomitans cytolethal distend
66 s physiologic support, and pathogens such as Aggregatibacter actinomycetemcomitans display resource p
67 growth with streptococci, the oral pathogen Aggregatibacter actinomycetemcomitans displays enhanced
72 onstrate that CDTs from Haemophilus ducreyi, Aggregatibacter actinomycetemcomitans, Escherichia coli,
75 s protease-deficient mutant KDP128, and live Aggregatibacter actinomycetemcomitans In contrast, infec
76 carrier rate of JP2 and non-JP2 genotypes of Aggregatibacter actinomycetemcomitans in the Ghanaian ad
77 eponema denticola, Tannerella forsythia, and Aggregatibacter actinomycetemcomitans in the subgingival
78 ssion could be attenuated by the presence of Aggregatibacter actinomycetemcomitans in tri-species bio
80 injections of lipopolysaccharide (LPS) from Aggregatibacter actinomycetemcomitans in wild-type (WT)
81 es of the Gram-negative periodontal pathogen Aggregatibacter actinomycetemcomitans include serotype a
83 d with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycetemcomitans, increased GCF-IL-
86 (Cdt), expressed by the periodontal pathogen Aggregatibacter actinomycetemcomitans, inhibits the prol
88 esin A (EmaA) of the Gram-negative bacterium Aggregatibacter actinomycetemcomitans is a fibrillar col
99 iofilm formation by the periodontal pathogen Aggregatibacter actinomycetemcomitans is dependent upon
103 gram-negative fastidious human oropharyngeal Aggregatibacter actinomycetemcomitans is implicated in t
106 s were found to inhibit biofilm formation by Aggregatibacter actinomycetemcomitans, Klebsiella pneumo
107 obacterium nucleatum (middle colonizer), and Aggregatibacter actinomycetemcomitans (late colonizer).
109 eponema denticola, Tannerella forsythia, and Aggregatibacter actinomycetemcomitans levels in subgingi
110 PISA index, and Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans levels, when compa
111 MKP-1 induction and preclinical models using Aggregatibacter actinomycetemcomitans lipopolysaccharide
112 ontrol lean rats by periodontal injection of Aggregatibacter actinomycetemcomitans lipopolysaccharide
117 oral pathogens Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans perturbed the tran
118 me PCR was used for quantitative analysis of Aggregatibacter actinomycetemcomitans, Porphyromonas gin
121 ations and higher prevalence of the bacteria Aggregatibacter actinomycetemcomitans, Porphyromonas gin
122 thia dentocariosa), and cultivable bacteria (Aggregatibacter actinomycetemcomitans, Porphyromonas gin
123 e chain reaction determined total bacterial, Aggregatibacter actinomycetemcomitans, Porphyromonas gin
124 species were assessed at baseline, including Aggregatibacter actinomycetemcomitans, Porphyromonas gin
125 d total and quantitative bacterial counts of Aggregatibacter actinomycetemcomitans, Porphyromonas gin
128 gation is to quantify periodontal pathogens (Aggregatibacter actinomycetemcomitans, Porphyromonas gin
131 tive real-time polymerase chain reaction for Aggregatibacter actinomycetemcomitans, Porphyromonas gin
132 smoking by pack-years, periodontal bacteria (Aggregatibacter actinomycetemcomitans, Porphyromonas gin
133 Subgingival plaque samples were analyzed for Aggregatibacter actinomycetemcomitans, Porphyromonas gin
134 l animal model to study the host response to Aggregatibacter actinomycetemcomitans (previously Actino
135 ove the minimal inhibitory concentration for Aggregatibacter actinomycetemcomitans (previously Actino
136 ide PMNs could enhance their ability to kill Aggregatibacter actinomycetemcomitans (previously Actino
138 w cytometry in response to Escherichia coli, Aggregatibacter actinomycetemcomitans (previously Actino
139 (tooth and site) factors on the detection of Aggregatibacter actinomycetemcomitans (previously Actino
140 lowed to incubate in a bacterial solution of Aggregatibacter actinomycetemcomitans (previously Actino
142 for serum immunoglobulin G (IgG) antibody to Aggregatibacter actinomycetemcomitans (previously Actino
143 rase chain reaction determined the number of Aggregatibacter actinomycetemcomitans (previously Actino
144 iodontitis are associated with infections by Aggregatibacter actinomycetemcomitans (previously Actino
146 sly T. forsythensis), Prevotella intermedia, Aggregatibacter actinomycetemcomitans (previously Actino
147 serum immunoglobulin G antibody response to Aggregatibacter actinomycetemcomitans (previously Actino
148 esent study evaluated seven target bacteria, Aggregatibacter actinomycetemcomitans (previously Actino
149 igher frequency of Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans (previously Actino
150 odontal pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans [previously Actino
154 primary gingival fibroblast cells (GFs) with Aggregatibacter actinomycetemcomitans protein extract (A
156 similarity to the Escherichia coli LsrB and Aggregatibacter actinomycetemcomitans RbsB proteins that
158 generally increase protection, against live Aggregatibacter actinomycetemcomitans, significant incre
160 Filifactor alocis, Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, Streptococcus par
163 pecific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, Tannerella forsyt
165 ted with aggressive periodontitis-associated Aggregatibacter actinomycetemcomitans triggered a type I
166 lla intermedia, Fusobacterium nucleatum, and Aggregatibacter actinomycetemcomitans was examined using
167 votella intermedia, Treponema denticola, and Aggregatibacter actinomycetemcomitans was identified by
169 g toxin (Cdt), from the periodontal pathogen Aggregatibacter actinomycetemcomitans, was conjugated to
170 ium saphenum in saliva and serum IgG against Aggregatibacter actinomycetemcomitans were negatively co
173 a/nigrescens, Streptococcus constellatus, or Aggregatibacter actinomycetemcomitans, were resistant in
174 gens, including Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, were similarly re
175 al activity against the periodontal pathogen Aggregatibacter actinomycetemcomitans when the bacteria
176 ein that is secreted from the oral bacterium Aggregatibacter actinomycetemcomitans, which targets and