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1                                              Albright hereditary osteodystrophy (AHO), a disorder cha
2                                              Albright hereditary osteodystrophy (AHO), an autosomal d
3                                              Albright's Hereditary Osteodystrophy (AHO) was the first
4 den Dystonia Rating Scale-Motor Scale, Barry Albright Dystonia Scale), disability (Burke Fahn Marsden
5 otal score of at least 3 points on the Barry-Albright Dystonia (BAD) scale, and no evidence of iron d
6 kinson's Disease Rating Scale (UPDRS), Barry-Albright Dystonia (BAD) scale, Schwab and England Activi
7  skeletal abnormalities in a syndrome called Albright's hereditary osteodystrophy.
8 d hyperphosphatemia but without evidence for Albright hereditary osteodystrophy who has paternal unip
9 n the 1940s with the demonstration by Fuller Albright that treatment with oestrogen could reverse the
10 ings potentially have implications for human Albright hereditary osteodystrophy, a condition caused b
11                                           In Albright Hereditary Osteodystrophy (AHO) heterozygous lo
12 ha-inactivating mutations lead to obesity in Albright hereditary osteodystrophy (AHO) patients, but o
13  reports of mild heterotopic ossification in Albright's hereditary osteodystrophy (AHO) and a recent
14 e resistance that is limited to PTH and lack Albright hereditary osteodystrophy.
15                                       McCune-Albright syndrome (MAS) is a mosaic disorder arising fro
16                                       McCune-Albright syndrome (MAS), a mosaic condition associated w
17 and from induced hPSCs derived from a McCune-Albright patient.
18 ently, IPMNs have been described as a McCune-Albright syndrome-associated tumor, present in about 15%
19  of polyostotic fibrous dysplasia and McCune-Albright syndrome among patients operated on for presump
20 ts with fibrous dysplasia of bone and McCune-Albright syndrome generated more basal cAMP accumulation
21 use of fibrous dysplasia of bone (FD)/McCune-Albright syndrome (MAS).
22 utations) and hormone hypersecretion (McCune-Albright syndrome caused by gain-of-function mutations).
23 ary mucinous neoplasms (IPMNs) and in McCune-Albright syndrome, characterized by fibrous dysplasia, p
24 trimeric G-protein disorders, joining McCune-Albright and Sturge-Weber syndromes.
25 predisposing to pituitary neoplasias: McCune-Albright syndrome, multiple endocrine neoplasia type 1,
26 e of a syndromic IPMN as a feature of McCune-Albright syndrome, this observation is further evidence
27 on clinical examination suggestive of McCune-Albright syndrome.
28    Because of its similarities to the McCune-Albright syndrome and other features, such as paradoxica
29 tic fibrous dysplasia, and 26 had the McCune-Albright syndrome.
30 sia of bone is a major finding in the McCune-Albright syndrome.
31 nd bone age advancement in girls with McCune-Albright syndrome (MAS), despite ovarian enlargement.
32 raphy was performed on a patient with McCune-Albright syndrome and acromegaly.
33 have been identified in patients with McCune-Albright syndrome, but the mechanism leading to the spec
34  tumors are observed in patients with McCune-Albright syndrome.
35 H) resistance, but lack physical features of Albright hereditary osteodystrophy.
36 denylyl cyclase plus somatic features termed Albright hereditary osteodystrophy.
37 PHP), a disease which shares features of the Albright hereditary osteodystrophy (AHO) phenotype.
38 ygous Gsalpha inactivating mutations lead to Albright hereditary osteodystrophy.
39 ors of this Special Feature, CSTL alumni Tom Albright and Jennifer Mnookin have recruited articles at
40 is mutation was identified in a patient with Albright hereditary osteodystrophy.
41                     Studies in patients with Albright hereditary osteodystrophy suggest a similar G(s
42 an homologue GNAS1, mutated in patients with Albright hereditary osteodystrophy, is also imprinted.