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1 hilum, supporting its placement in the genus Anaplasma.
2                                        Thus, Anaplasma actively induces autophagy by secreting Ats-1
3         The JAK-STAT pathway exerts its anti-Anaplasma activity presumably through STAT-regulated eff
4          Rickettsial pathogens in the genera Anaplasma and Ehrlichia cause acute infection in immunol
5            Bacterial pathogens in the genera Anaplasma and Ehrlichia encode a protein superfamily, pf
6 obligatory intracellular bacterial pathogens Anaplasma and Ehrlichia infect leukocytes by hijacking h
7         APH_1235 homologs are found in other Anaplasma and Ehrlichia species but not in other bacteri
8 porates melt curve analysis to differentiate Anaplasma and Ehrlichia species with blood smear and ser
9  24 amino acids and is conserved among other Anaplasma and Ehrlichia species.
10 th intracellular survival and replication of Anaplasma and Ehrlichia spp. in granulocytes or monocyte
11 ription of the three virB genes in these two Anaplasma and Ehrlichia spp. is regulated by factors tha
12 lection for divergence in Ank function among Anaplasma and Ehrlichia spp. is supported by both locus
13    Although the basic developmental cycle of Anaplasma and Ehrlichia spp. within the tick has been de
14  widely applicable to bacteria in the genera Anaplasma and Ehrlichia, removing a major technical impe
15 n fragmentation are common in the genomes of Anaplasma and other obligate intracellular bacteria.
16                                              Anaplasma and related Ehrlichia spp. are important tick-
17 ich is conserved among bacteria in the genus Anaplasma and the closely related genus Ehrlichia.
18              The assay was tested on various Anaplasma, Borrelia, Erhlichia, and Rickettsia species,
19  positive amplicons revealed the presence of Anaplasma bovis, an agent not known to be present in Nor
20                We provisionally nominate it "Anaplasma capra".
21 lasma marginale subsp. centrale (synonym for Anaplasma centrale) induces protection against severe di
22 atrix and gut barrier-critical obstacles for Anaplasma colonization.
23  The broad-range assay detected Ehrlichia or Anaplasma DNA in 20 (26%) of the symptomatic dogs and 2
24                                              Anaplasma (Ehrlichia) phagocytophila and Ehrlichia chaff
25                                              Anaplasma (Ehrlichia) phagocytophila's major immunodomin
26 roEL and gltA genes revealed the presence of Anaplasma, Ehrlichia, Candidatus Neoehrlichia, and Ricke
27                       Organisms in the genus Anaplasma express an immunodominant major surface protei
28                                              Anaplasma (formerly Ehrlichia) phagocytophilum and Ehrli
29 hermal in situ technique to detect localized Anaplasma gene sequences by using rolling-circle amplifi
30             Bacterial pathogens in the genus Anaplasma generate surface coat variants by gene convers
31                                 Furthermore, Anaplasma growth arrest by the class III PI3K inhibitor
32                                Prevalence of Anaplasma in the Minnesota ticks was 63.9%; 23 of 36 tic
33 utophagy proteins were also localized to the Anaplasma inclusion.
34     Ectopically expressed Ats-1 targeted the Anaplasma inclusions and enhanced infection, whereas hos
35       Cytospin preparations of uninfected or Anaplasma-infected cell cultures were examined using thi
36 nd preliminary immunofluorescent staining of Anaplasma-infected tissues suggest that endothelial cell
37                  Population distributions of Anaplasma infection were assessed by targeting the p44 g
38 eterozygous-deficient mice were resistant to Anaplasma infection.
39                Following neutrophil binding, Anaplasma invasion requires at least 240 min.
40 incubated at 34 degrees C, and 8 days later, Anaplasma-like inclusions were demonstrated in Giemsa-st
41 coplasma spp. (79 to 83% similarity) than to Anaplasma marginale (72 to 75% similarity).
42                                              Anaplasma marginale (order Rickettsiales, family Anaplas
43 , major surface protein 2 (MSP2) and MSP3 of Anaplasma marginale and Anaplasma ovis, Anaplasma phagoc
44  an immunodominant outer membrane protein of Anaplasma marginale and Anaplasma phagocytophilum pathog
45 er membrane protein expression sites in both Anaplasma marginale and Anaplasma phagocytophilum.
46 to the model obligate intracellular pathogen Anaplasma marginale and the human pathogen Anaplasma pha
47 red unique to the intraerythrocytic agent of Anaplasma marginale and the intragranulocytic agent of A
48 of the persistent bovine ehrlichial pathogen Anaplasma marginale are immunodominant proteins that und
49                    Major surface proteins of Anaplasma marginale are vaccine candidates.
50 he system, we used Dermacentor andersoni and Anaplasma marginale as a tick-pathogen interaction model
51                     The rickettsial pathogen Anaplasma marginale assembles an actin filament bundle d
52                                              Anaplasma marginale causes bovine anaplasmosis, a debili
53                                              Anaplasma marginale colonies differed in their developme
54 SP1a and MSP1b) of the erythrocytic stage of Anaplasma marginale conferred protection against homolog
55         Strains of the tick-borne rickettsia Anaplasma marginale differ markedly in transmission effi
56                     The rickettsial pathogen Anaplasma marginale establishes lifelong persistent infe
57                                              Anaplasma marginale establishes persistent infection cha
58                     The rickettsial pathogen Anaplasma marginale expresses a variable immunodominant
59 the hypothesis that challenge of cattle with Anaplasma marginale expressing MSP2 variants to which th
60               Immunization of cattle with an Anaplasma marginale fraction enriched in outer membranes
61                                 In addition, Anaplasma marginale generates variants by recombination
62      Like several other bacterial pathogens, Anaplasma marginale has an outer membrane that induces c
63 ive immunity against the ehrlichial pathogen Anaplasma marginale has been hypothesized to require ind
64                                              Anaplasma marginale illustrates this transition: in the
65 he highly transmissible St. Maries strain of Anaplasma marginale in Dermacentor andersoni as a positi
66  protein 1 (MSP1) of the ehrlichial pathogen Anaplasma marginale induces protective immunity in calve
67 r membranes induces protection against acute Anaplasma marginale infection and disease, and a proteom
68 -cell response is central for the control of Anaplasma marginale infection in cattle.
69                                   Throughout Anaplasma marginale infection, recombination results in
70 acterize tick genes regulated in response to Anaplasma marginale infection.
71 ve major surface protein 1 (MSP1) complex of Anaplasma marginale is a heteromer of MSP1a and MSP1b, e
72                     The St. Maries strain of Anaplasma marginale is a high-transmission-efficiency st
73                                              Anaplasma marginale is a prototypical highly antigenical
74                                              Anaplasma marginale is a tick-borne pathogen, one of sev
75                                              Anaplasma marginale is a tick-transmitted pathogen of ca
76                                              Anaplasma marginale is an ehrlichial pathogen of cattle
77                                              Anaplasma marginale is an ehrlichial pathogen of cattle,
78                                              Anaplasma marginale is an intraerythrocytic rickettsial
79 y the intraerythrocytic rickettsial pathogen Anaplasma marginale is endemic in South Africa.
80 ly variant major surface protein 2 (MSP2) of Anaplasma marginale is expressed from a 3.5-kb operon th
81                               The rickettsia Anaplasma marginale is the most prevalent tick-borne liv
82 nalyzing the CD4(+) T lymphocyte response to Anaplasma marginale major surface protein 1a (MSP1a).
83                                     Specific Anaplasma marginale major surface protein 2 (MSP2) varia
84 erogeneity in this species, the homologue of Anaplasma marginale major surface protein 4 gene (msp4)
85 gene family, with sequence similarity to the Anaplasma marginale msp-2 genes.
86 termini of the Cowdria ruminantium MAP-1 and Anaplasma marginale MSP-4 proteins.
87 ly unique strains of the tick-borne pathogen Anaplasma marginale occur and are transmitted within reg
88         In this study we demonstrate that in Anaplasma marginale outer membrane-vaccinated cattle, Vi
89 1b1 occur as naturally complexed OMPs in the Anaplasma marginale outer membrane.
90                   Immunization with purified Anaplasma marginale outer membranes induces complete pro
91 n superinfections of the tick-borne pathogen Anaplasma marginale predominate.
92             Comparison between A. platys and Anaplasma marginale proteins showed sequence identities
93 sed an unbiased proteomic screen to identify Anaplasma marginale proteins specifically upregulated in
94                                           In Anaplasma marginale pseudogenes for two antigenically va
95                        Live vaccination with Anaplasma marginale subsp. centrale (synonym for Anaplas
96                                              Anaplasma marginale subsp. centrale is a naturally atten
97 s were fed on animals superinfected with the Anaplasma marginale subsp. centrale vaccine strain (low
98                                              Anaplasma marginale subsp. centrale was the first vaccin
99 ody isotype induced by live vaccination with Anaplasma marginale subsp. centrale.
100                                              Anaplasma marginale subspecies centrale also infects cat
101                                              Anaplasma marginale superinfection occurs when the secon
102  distinct strains of the tick-borne pathogen Anaplasma marginale that encode distinctly different sur
103                                              Anaplasma marginale utilizes gene conversion of a repert
104  This question was addressed by tracking the Anaplasma marginale variant population and corresponding
105      We examined allelic usage in generating Anaplasma marginale variants during in vivo infection in
106 ofluorescence, Anaplasma phagocytophilum and Anaplasma marginale were successfully localized in situ
107 e protein 2 (MSP2) variants are expressed by Anaplasma marginale within the tick salivary gland and,
108 t study, we examined the strain structure of Anaplasma marginale, a genogroup II ehrlichial pathogen,
109 , broad population immunity develops against Anaplasma marginale, a highly antigenically variant rick
110 immunodominant surface proteins, we examined Anaplasma marginale, a rickettsia with two highly immuno
111                                              Anaplasma marginale, a rickettsial pathogen, evades clea
112 We investigated this by using infection with Anaplasma marginale, a ruminant pathogen that replicates
113                                              Anaplasma marginale, a tick-borne rickettsial pathogen o
114       In this study, the surface proteome of Anaplasma marginale, a tick-transmitted bacterial pathog
115                                              Anaplasma marginale, an intraerythrocytic ehrlichial pat
116 nulocytic ehrlichiosis, msp-2 and msp-4 from Anaplasma marginale, and map-1 from Cowdria ruminantium.
117 s on three unrelated vector-borne pathogens, Anaplasma marginale, Borrelia hermsii and Trypanosoma br
118         The tick-borne rickettsial organism, Anaplasma marginale, causes a disease in cattle of world
119     Native major surface protein 1 (MSP1) of Anaplasma marginale, composed of covalently associated M
120 chia phagocytophila, and the bovine pathogen Anaplasma marginale, express a markedly immunodominant o
121 protein 2 (Msp2) of the tick-borne pathogen, Anaplasma marginale, is thought to be involved in antige
122                      In the related organism Anaplasma marginale, persistence is associated with anti
123                                              Anaplasma marginale, the causative agent of bovine anapl
124 f template DNA from closely related species (Anaplasma marginale, the white-tailed deer agent, and ad
125               We examined this balance using Anaplasma marginale, which generates antigenic variants
126  a hallmark of the tick transmitted pathogen Anaplasma marginale.
127 tein in the outer membrane of the rickettsia Anaplasma marginale.
128 perate-region strains of the cattle pathogen Anaplasma marginale.
129 ydiae, Orientia tsutsugamushi, Wolbachia and Anaplasma marginale.
130  of world-wide economic importance caused by Anaplasma marginale.
131  protein homologous to the MSP-2 proteins of Anaplasma marginale.
132  Ehrlichia species but distinct from that of Anaplasma marginale.
133 d antigenically polymorphic among strains of Anaplasma marginale.
134 ces of selected amplicons from the assay for anaplasma matched sequences of the white-tailed deer age
135 s technique to detect, localize, and analyze Anaplasma nucleotide sequences in the tissues of infecte
136 heart base, and skin of coinfected mice, but Anaplasma numbers remained constant.
137                Anaplasma phagocytophilum and Anaplasma ovis cause human infections.
138        Goats which have recovered from acute Anaplasma ovis infection remain seropositive, although i
139 2 (MSP2) and MSP3 of Anaplasma marginale and Anaplasma ovis, Anaplasma phagocytophilum MSP2 (p44), Eh
140 netically distinct A. marginale strains, and Anaplasma ovis.
141                                              Anaplasma PCR results from a 3-year period, with associa
142 ewardship algorithm would reduce unnecessary Anaplasma PCR testing, without missing active cases.
143 sms in the genera Rickettsia, Ehrlichia, and Anaplasma, persists in ticks and mammalian hosts; howeve
144 burgdorferi (33.6%), Babesia microti (8.4%), Anaplasma phagocytophila (1.9%), and Bartonella spp. (34
145 a low prevalence of antibodies reactive with Anaplasma phagocytophila (2 of 112; 2%).
146 nant 44 kDa major outer membrane proteins of Anaplasma phagocytophila (human granulocytic ehrlichiosi
147 ck-vectored pathogen transmission, including Anaplasma phagocytophila an etiologic agent of granulocy
148 ng tick-borne zoonosis caused by a strain of Anaplasma phagocytophila called the HGE agent, an obliga
149                                              Anaplasma phagocytophila is an obligatory intragranulocy
150                                              Anaplasma phagocytophila persists within neutrophils and
151                                              Anaplasma phagocytophila, an obligately intracellular ba
152   The human granulocytic ehrlichiosis agent, Anaplasma phagocytophila, resides and multiplies exclusi
153  determined the frequencies of antibodies to Anaplasma phagocytophila, the agent of human granulocyti
154                                              Anaplasma phagocytophila, the etiologic agent of human g
155                                              Anaplasma phagocytophilum (Ap) is an obligate intracellu
156                                              Anaplasma phagocytophilum (Ap), the agent of the tick-bo
157                                              Anaplasma phagocytophilum (Ap), the etiologic agent of t
158 ing of the region directly downstream of the Anaplasma phagocytophilum (strain MRK) 16S rRNA gene ide
159                   Francisella tularensis and Anaplasma phagocytophilum alter host autophagy, Shigella
160 relia burgdorferi and the rickettsial agents Anaplasma phagocytophilum and A. marginale.
161 nd von Willebrand factor immunofluorescence, Anaplasma phagocytophilum and Anaplasma marginale were s
162                                              Anaplasma phagocytophilum and Anaplasma ovis cause human
163  Pennsylvania and tested for the presence of Anaplasma phagocytophilum and Borrelia burgdorferi by PC
164  developed for the simultaneous detection of Anaplasma phagocytophilum and Borrelia burgdorferi.
165 amine TH 1 and TH 17 immunity infection with Anaplasma phagocytophilum and Citrobacter rodentium resp
166 , O. tsutsugamushi, and infections caused by Anaplasma phagocytophilum and Ehrlichia chaffeensis with
167                    Ehrlichia chaffeensis and Anaplasma phagocytophilum are agents of human monocytic
168 nt outer membrane proteins (P44 proteins) of Anaplasma phagocytophilum are encoded by the p44 polymor
169   The mechanisms used by the tick to control Anaplasma phagocytophilum are not known.
170 ngton State suspected of being infected with Anaplasma phagocytophilum because of the finding of moru
171                                              Anaplasma phagocytophilum causes granulocytic anaplasmos
172                   The intracellular organism Anaplasma phagocytophilum causes human granulocytic ehrl
173                                              Anaplasma phagocytophilum causes human granulocytic ehrl
174 Colonization of neutrophils by the bacterium Anaplasma phagocytophilum causes the disease human granu
175 rrelia burgdorferi and the rickettsial agent Anaplasma phagocytophilum Collectively, we highlight the
176          The tick-borne rickettsial pathogen Anaplasma phagocytophilum develops within membrane-bound
177                                              Anaplasma phagocytophilum DNA was detected in one dog, a
178      A polymorphic multigene family (p44) of Anaplasma phagocytophilum encodes the immunodominant 44-
179 from 16S rRNA gene-based genetic variants of Anaplasma phagocytophilum from dogs in the western Unite
180  to determine the presence and prevalence of Anaplasma phagocytophilum human agent (AP-ha) and a gene
181                                              Anaplasma phagocytophilum immunodominant polymorphic maj
182                               Infection with Anaplasma phagocytophilum in white-footed mice results i
183                                              Anaplasma phagocytophilum infection induces functional n
184          A total of 60 sheep were exposed to Anaplasma phagocytophilum infection on an enclosed area
185 tick vector Ixodes scapularis in response to Anaplasma phagocytophilum infection, the causative agent
186 auses meningoencephalitis, this is rare with Anaplasma phagocytophilum infection.
187                                              Anaplasma phagocytophilum is a bacterium that is transmi
188                                              Anaplasma phagocytophilum is a gram-negative obligate in
189                                              Anaplasma phagocytophilum is a tick-borne rickettsial pa
190                                              Anaplasma phagocytophilum is an intracellular pathogen t
191                                              Anaplasma phagocytophilum is an intragranulocytic bacter
192                                              Anaplasma phagocytophilum is an obligate intracellular b
193                                              Anaplasma phagocytophilum is an obligate intracellular b
194                                              Anaplasma phagocytophilum is an obligate intracellular b
195                                              Anaplasma phagocytophilum is an obligate intracellular b
196                                              Anaplasma phagocytophilum is an obligate intracellular p
197                                              Anaplasma phagocytophilum is an obligate intracellular t
198                                              Anaplasma phagocytophilum is an obligate vacuolar bacter
199                                              Anaplasma phagocytophilum is an obligatory intracellular
200                                              Anaplasma phagocytophilum is an obligatory intracellular
201  heterogeneity of anthropozoonosis caused by Anaplasma phagocytophilum is poorly understood.
202                                              Anaplasma phagocytophilum is the agent of human anaplasm
203                                              Anaplasma phagocytophilum is the causative agent of an e
204                                              Anaplasma phagocytophilum is the etiologic agent of huma
205                                              Anaplasma phagocytophilum is the etiologic agent of huma
206                                              Anaplasma phagocytophilum is the tick-transmitted obliga
207  during infection with the rickettsial agent Anaplasma phagocytophilum Macrophages deficient in annex
208 3 of Anaplasma marginale and Anaplasma ovis, Anaplasma phagocytophilum MSP2 (p44), Ehrlichia chaffeen
209  membrane protein of Anaplasma marginale and Anaplasma phagocytophilum pathogens that cause bovine an
210        The human intragranulocytic bacterium Anaplasma phagocytophilum promotes variation of P44s, wh
211                                              Anaplasma phagocytophilum propagates within neutrophils
212    Borellia burgdorferi, Babesia microti and Anaplasma phagocytophilum rely almost exclusively on a s
213  et al. demonstrate that ticks infected with Anaplasma phagocytophilum show enhanced fitness against
214  first tissue culture isolates of the unique Anaplasma phagocytophilum strain, Ap-Variant 1, were obt
215 ligate intracellular Gram-negative bacterium Anaplasma phagocytophilum The disease often presents wit
216 owledge regarding the strategies employed by Anaplasma phagocytophilum to evade or subvert neutrophil
217 ce-exposed major membrane proteins, P44s, of Anaplasma phagocytophilum were hypothesized to be garner
218 he agent of human granulocytic ehrlichiosis (Anaplasma phagocytophilum) is intensely enzootic in rabb
219                               Infection with Anaplasma phagocytophilum, a gram-negative, lipopolysacc
220                                              Anaplasma phagocytophilum, a member of the family Anapla
221                                              Anaplasma phagocytophilum, a recently reclassified bacte
222 he agent of human granulocytic anaplasmosis, Anaplasma phagocytophilum, among other pathogens.
223                    The natural life cycle of Anaplasma phagocytophilum, an obligatory intracellular b
224                                              Anaplasma phagocytophilum, an obligatory intracellular b
225                                              Anaplasma phagocytophilum, an unusual obligate intracell
226 y pathogens, including Borrelia burgdorferi, Anaplasma phagocytophilum, and Babesia microti.
227                          Borrelia miyamotoi, Anaplasma phagocytophilum, and Babesia spp. were not det
228 gen, seventy-four (40%) were seroreactive to Anaplasma phagocytophilum, and five (2.7%) were seroposi
229 ate substrates, an ankyrin repeat protein of Anaplasma phagocytophilum, AnkA, is delivered into the h
230 intracellular bacteria such as Ehrlichia and Anaplasma phagocytophilum, as well as obligate intracell
231 ted real-time PCR assays for Ehrlichia spp., Anaplasma phagocytophilum, Babesia spp., or Lyme Borreli
232 d 100% sensitivity for the identification of Anaplasma phagocytophilum, Borrelia miyamotoi, Borrelia
233 egative for all tick-borne pathogens tested (Anaplasma phagocytophilum, Ehrlichia canis, and Ricketts
234 hogens in the order Rickettsiales, including Anaplasma phagocytophilum, Ehrlichia canis, E. chaffeens
235 ighly conserved with orthologous proteins in Anaplasma phagocytophilum, Ehrlichia chaffeensis, and Eh
236 ptional regulator, tr1, and a homolog of the Anaplasma phagocytophilum, identified here as A. platys
237 n Anaplasma marginale and the human pathogen Anaplasma phagocytophilum, in order to examine the abili
238 marginale and the intragranulocytic agent of Anaplasma phagocytophilum, respectively.
239 rrelia burgdorferi s.l., Borrelia miyamotoi, Anaplasma phagocytophilum, Rickettsia spp., Candidatus N
240 ated first to Anaplasma platys and second to Anaplasma phagocytophilum, supporting its placement in t
241  recent studies of the granulocyte pathogen, Anaplasma phagocytophilum, the agent of granulocytic ehr
242  burgdorferi, the agent of Lyme disease, and Anaplasma phagocytophilum, the agent of human anaplasmos
243                                              Anaplasma phagocytophilum, the agent of human anaplasmos
244                                              Anaplasma phagocytophilum, the agent of human granulocyt
245                                              Anaplasma phagocytophilum, the agent of human granulocyt
246 s harbor numerous human pathogens, including Anaplasma phagocytophilum, the agent of human granulocyt
247 y proteomics and transcriptome sequencing to Anaplasma phagocytophilum, the agent of human granulocyt
248                                              Anaplasma phagocytophilum, the causative agent of human
249                                              Anaplasma phagocytophilum, the causative agent of human
250      Infection of neutrophil precursors with Anaplasma phagocytophilum, the causative agent of human
251 increases tick colonization by the bacterium Anaplasma phagocytophilum, the causative agent of human
252                                              Anaplasma phagocytophilum, the causative agent of human
253 ertain Ixodes ticks, which can also transmit Anaplasma phagocytophilum, the cause of human granulocyt
254 des scapularis tick, which can also transmit Anaplasma phagocytophilum, the cause of human granulocyt
255                                              Anaplasma phagocytophilum, the etiologic agent of human
256                                              Anaplasma phagocytophilum, the etiologic agent of human
257                                              Anaplasma phagocytophilum, the etiologic agent of human
258                                              Anaplasma phagocytophilum, the etiologic agent of human
259                                     However, Anaplasma phagocytophilum, the obligatory intracellular
260 ic ehrlichiosis was recently reclassified as Anaplasma phagocytophilum, unifying previously described
261 his report, we show that the AnkA protein of Anaplasma phagocytophilum, which is translocated into th
262 using DNA extracted from serial dilutions of Anaplasma phagocytophilum-infected HL-60 cells.
263                                              Anaplasma phagocytophilum-infected neutrophil degranulat
264 athogens including human anaplasmosis agent, Anaplasma phagocytophilum.
265 used by the obligate intracellular bacterium Anaplasma phagocytophilum.
266  a critical role in the early eradication of Anaplasma phagocytophilum.
267 a focus on the model intracellular bacterium Anaplasma phagocytophilum.
268 sease, or a variant strain, Ap-Variant 1, of Anaplasma phagocytophilum.
269 a spp., Rickettsia spp., Ehrlichia spp., and Anaplasma phagocytophilum.
270 ession sites in both Anaplasma marginale and Anaplasma phagocytophilum.
271 used by the obligate intracellular bacterium Anaplasma phagocytophilum.
272 aris transmits the human anaplasmosis agent, Anaplasma phagocytophilum.
273  host stimulation with the rickettsial agent Anaplasma phagocytophilum.
274                  All positive specimens were Anaplasma phagocytophilum; no Ehrlichia species were ide
275 he isolate was most closely related first to Anaplasma platys and second to Anaplasma phagocytophilum
276                                              Anaplasma platys infects peripheral blood platelets and
277                                              Anaplasma platys is an obligate intracellular rickettsia
278 closely-related pathogenic bacteria, such as Anaplasma platys, Ehrlichia chaffeensis, Orientia tsutsu
279   Targeted pathogens were Borrelia, Babesia, Anaplasma, Rickettsia, Ehrlichia, Bartonella, Francisell
280 cells and other potentially cryptic sites of Anaplasma sp. infection in mammalian tissues, a sensitiv
281     Transmission electron microscopy of this Anaplasma sp. organism in tick cell cultures revealed la
282                 Tick cells infected with the Anaplasma sp. organism were used to successfully infect
283 3%), Ehrlichia ewingii (44 of 217; 20%), and Anaplasma species (214 of 217; 99%).
284 tients assessed were infected with the novel Anaplasma species according to PCR and sequencing.
285  antibody (MAb) ANAF16C1, is conserved among Anaplasma species and is expressed in the salivary gland
286 May 1, to June 10, 2014, to detect the novel Anaplasma species by PCR.
287 ota and represent the first isolations of an Anaplasma species directly from ticks.
288 l for cross-reactivity among closely related Anaplasma species has made the accurate determination of
289 or human pathogenicity of a newly discovered Anaplasma species infecting goats in China.
290 rrhagic fever virus, Babesia, Theileria, and Anaplasma species, identifies arboviruses with the poten
291 ajor outer membrane protein gene families in Anaplasma species.
292 hat the pathogen was distinct from all known Anaplasma species.
293 ans that is closely related to Ehrlichia and Anaplasma species.
294 ealed A. pullorum is most closely related to Anaplasma spp.
295                               Infectivity of Anaplasma spp. develops when infected ticks feed on a ma
296 r membrane suggests that the porin feeds the Anaplasma TCA cycle and that the relatively large pore s
297        In humans and ruminants infected with Anaplasma, the major surface protein 2 (MSP2) is immunod
298 ts known pathogenic species of Ehrlichia and ANAPLASMA: The species was determined by using species-s
299                          Here, we found that Anaplasma translocated substrate 1 (Ats-1), a type IV se
300 that the relatively large pore size provides Anaplasma with the necessary metabolic intermediates fro

 
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