ライフサイエンスコーパス: Anopheles

1 e Plasmodium infection in the malaria vector Anopheles.

2 mum spatial resolution of the malaria vector Anopheles.

3 linkage between reproduction and immunity in Anopheles.

4 Of 1494 female Anopheles (89.8% Anopheles gambiae sensu lato), 88.3% we

5 d with sexually-transferred steroids or with Anopheles ability to transmit human malaria.

6 ment in female Anophelinae mosquitoes, using Anopheles albimanus as a model.

7 As previously reported for the Anopheles albimanus orthologue anophelin, cE5 binds both

8 m the salivary glands of the malaria vector, Anopheles albimanus The inhibitor was purified from sali

9 Haiti from May to September, 2012, where the Anopheles albimanus vector bites primarily outdoors and

10 nes (Anopheles gambiae, Anopheles dirus, and Anopheles albimanus) with Pfs47-Africa (GB4) and Pfs47-S

11 VU041, a submicromolar-affinity inhibitor of Anopheles (An.) gambiae and Aedes (Ae.) aegypti Kir1 cha

12 Antibody-based biomarkers of exposure to Anopheles and Aedes mosquito bites could be helpful tool

13 ing antibody-based biomarkers of exposure to Anopheles and Aedes mosquito bites.

14 were evaluated against Drosophila as well as Anopheles and Aedes mosquitoes, the former identified as

15 ists were previously shown to strongly repel Anopheles and Culex mosquitoes, we examined the bioactiv

16 oglobulin G (IgG) responses to both gSG6-P1 (Anopheles) and Nterm-34-kDa (Aedes) salivary peptides as

17 cate that mosquitoes of three genera, Aedes, Anopheles, and Culex are able to locate and land on surf

18 ales of the important malaria vector species Anopheles arabiensis and An. gambiae produce and release

19 to differ in their innate host preferences: Anopheles arabiensis and An. gambiae sensu stricto.

20 We show that gravid Anopheles arabiensis are attracted and oviposit in respo

21 increased prevalence of the malaria vector, Anopheles arabiensis, in sub-Saharan Africa.

22 introgression to the sibling vector species Anopheles arabiensis.

23 nsity of the partly zoophilic malaria vector Anopheles arabiensis.

24 dably and accurately measure the activity of Anopheles as well as Aedes over several days.

25 seeking and heat-stimulated blood feeding in Anopheles At a cellular level, Ir21a is essential for th

26 se change and high proportions of the vector Anopheles balabacensis.

27 ich may contribute to the impact of wAnga on Anopheles biology and vectorial capacity.

28 ices (P = .027), whereas that of exposure to Anopheles bites varied according to insecticide-treated

29 measure the risk among humans of exposure to Anopheles bites.

30 effects on female reproductive traits across Anopheles by using a set of 16 mosquito species (five An

31 onging to only three out of eight subgenera: Anopheles, Cellia and Nyssorhynchus.

32 ter IRS, An. arabiensis accounted for 86% of Anopheles collected by PSC and 66% by CDC light trap in

33 ce of Wolbachia in females from a laboratory Anopheles coluzzii (A. gambiae, M form) colony experimen

34 es and consequences of a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissa

35 sent in malaria-transmitting mosquitoes like Anopheles coluzzii and Anopheles sinensis.

36 43) is required for parasite evasion of the Anopheles coluzzii complement-like response.

37 d molecular and metabolic mechanisms in wild Anopheles coluzzii from South-East Cote d'Ivoire in 2019

38 major malaria vectors Anopheles gambiae and Anopheles coluzzii in Burkina Faso.

39 Aedes aegypti and Anopheles coluzzii mosquitoes exhibit diurnal and noctur

40 e chemical composition and attractiveness to Anopheles coluzzii mosquitoes of skin odours from partic

41 ised transmission and resistance profiles of Anopheles coluzzii populations from three sites in north

42 Behavioural attraction of the malaria vector Anopheles coluzzii to human host odour increases during

43 that the common insect repellent DEET repels Anopheles coluzzii upon contact with their legs.

44 ain insecticide (97.5%) used by farmers, and Anopheles coluzzii was the main mosquito identified.

45 ed Anopheles gambiae s.l. males (all M form (Anopheles coluzzii)) were collected from mating swarms i

46 frican malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aedes aegypti, the cosmop

47 quito populations of a major malaria vector, Anopheles coluzzii, in Burkina Faso.

48 In contrast, night-biting mosquitoes, Anopheles coluzzii, specifically avoid ultraviolet (UV)

49 In the African malaria vector, Anopheles coluzzii, we identified candidate enhancers in

50 pecies, including the primary malaria vector Anopheles coluzzii, were identified among 235 anopheline

51 Anopheles VKR in the Gambiae complex member, Anopheles coluzzii.

52 , Aedes albopictus, Anopheles stephensi, and Anopheles coluzzii.

53 content and permissiveness to Plasmodium of Anopheles coluzzii.

54 tory neurons of the African malaria mosquito Anopheles coluzzii.

55 human malaria in this biome is the mosquito Anopheles cruzii.

56 smission potential of novel, uncharacterized Anopheles cryptic species in western Kenya.

57 y, for the first time, reports extensive new Anopheles cryptic species involved in the malaria transm

58 ansion of multiple species within the Aedes, Anopheles, Culex, and Psorophora genera.

59 es of the New World species A. albimanus and Anopheles darlingi) also played some functionally releva

60 ria vector, Nyssorhynchus darlingi (formerly Anopheles darlingi), ranges from 0 degrees S-23 degrees

61 Moreover, human landing catches of sylvan Anopheles demonstrated the propensity of these three vec

62 119 falciparum-infected Cambodian adults to Anopheles dirus mosquitoes by membrane feeding.

63 Laboratory-reared Anopheles dirus were fed on blood from 119 Pf-infected C

64 from distant anophelines (Anopheles gambiae, Anopheles dirus, and Anopheles albimanus) with Pfs47-Afr

65 nd Plasmodium vivax to Anopheles gambiae and Anopheles dirus, respectively.

66 by using a set of 16 mosquito species (five Anopheles, eight Cellia, and three Nyssorhynchus), inclu

67 ne (Culex, Aedes) and anopheline mosquitoes (Anopheles), embryo polarity rests on a previously unname

68 own in bioassays with pyrethroid susceptible Anopheles farauti mosquitoes.

69 The blood-feeding behavior of Anopheles females delivers essential nutrients for egg d

70 r) to pyrethroids/DDT resistance observed in Anopheles funestus across Africa.

71 Anopheles funestus and An. arabiensis were fully suscept

72 died anopheline radiations, the Afrotropical Anopheles funestus complex (AFC), to investigate the rol

73 control of major malaria vectors, including Anopheles funestus Effective management of resistance re

74 lative rate [RR] 0.23; 95% CI 0.16-0.33) and Anopheles funestus group (RR 0.08; 0.04-0.16), which wer

75 Up to 15% of morphologically identified Anopheles funestus mosquitoes in insecticide resistance

76 The resistance levels of an Anopheles funestus population from Palmeira, southern Mo

77 Plasmodium infection in field population of Anopheles funestus s.s.

78 treatment to determine Anopheles gambiae and Anopheles funestus survival and infection rates.

79 eive IRS based on predicted probabilities of Anopheles funestus, and (3) prioritizing communities to

80 In the major malaria vector Anopheles funestus, little is currently known about the

81 oid resistance in the African malaria vector Anopheles funestus.

82 istance) in the major African malaria vector Anopheles funestus.

83 tor coreceptor of the African malaria vector Anopheles gambiae (AgamORco) in a small collection of na

84 50s associated with pyrethroid resistance in Anopheles gambiae (CYPs 6M2, 6P3, 6P4, 6P5, 9J5, 9K1, 6Z

85 ing pockets of odorant binding proteins from Anopheles gambiae (OBP1 and OBP47) were analysed using i

86 diator of heat seeking in the malaria vector Anopheles gambiae Although Ir21a mediates heat avoidance

87 Anopheles gambiae and A. stephensi larvae were bred unde

88 Anopheles gambiae and Aedes aegypti are perhaps the best

89 file the transcriptomes of 8506 hemocytes of Anopheles gambiae and Aedes aegypti mosquito vectors.

90 essential AChE1 enzymes from the mosquitoes Anopheles gambiae and Aedes aegypti.

91 Anopheles gambiae and An. arabiensis are major malaria v

92 b-Saharan Africa, two major malaria vectors, Anopheles gambiae and An. coluzzii, breed in distinct la

93 of key vector species from Africa and Asia (Anopheles gambiae and An. stephensi) to transmit the hum

94 cts populations of the major malaria vectors Anopheles gambiae and Anopheles coluzzii in Burkina Faso

95 They include the African malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aed

96 f Plasmodium berghei and Plasmodium vivax to Anopheles gambiae and Anopheles dirus, respectively.

97 s after initiation of treatment to determine Anopheles gambiae and Anopheles funestus survival and in

98 -kDa) protein related to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant al

99 andidate genes were successfully cloned from Anopheles gambiae and expressed in insect cells.

100 of mosquitoes (all p<0.0001), especially the Anopheles gambiae complex (relative rate [RR] 0.23; 95%

101 resistance phenotypes in mosquitoes from the Anopheles gambiae complex across Africa.

102 ga-Mali) was identified in mosquitoes of the Anopheles gambiae complex collected in the Malian villag

103 n male mating behavior, recent data from the Anopheles gambiae complex suggests that, apart from the

104 Mosquitoes of the Anopheles gambiae complex were identified to species usi

105 However, in the Anopheles gambiae complex, the primary African vectors o

106 cur in all intercrosses among members of the Anopheles gambiae complex.

107 nces restricted to the member species of the Anopheles gambiae complex.

108 ulations of the major African malaria vector Anopheles gambiae Contact with LLINs reduced the immedia

109 e monitored miRNA expression in the mosquito Anopheles gambiae during the 72-h period immediately aft

110 ed heme peroxidase is required for long-term Anopheles gambiae fertility.

111 Aedes aegypti and Anopheles gambiae harbor the causative agents of disease

112 PHs) are positive and negative regulators of Anopheles gambiae immune responses mediated by the compl

113 a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissau, where high hybridisa

114 ne expression to develop transgenic lines of Anopheles gambiae in which olfactory receptor neurons ex

115 the principal role of hormonal signaling in Anopheles gambiae initiated shortly after blood-feeding,

116 revealed that the single hodor orthologue in Anopheles gambiae is an essential gene.

117 9-based homing system for the suppression of Anopheles gambiae is encouraging; however, with current

118 ere, we show that the primary malaria vector Anopheles gambiae is targeted and killed by small insect

119 parated four distinct cell lineages from the Anopheles gambiae male gonads: premeiotic, meiotic (prim

120 hough the presence of Pe. chrysogenum in the Anopheles gambiae midgut does not affect mosquito surviv

121 termine the antiviral immune pathways of the Anopheles gambiae midgut, the initial site of viral infe

122 nvestigate the function of P. berghei P47 in Anopheles gambiae mosquito infections.

123 The reproductive fitness of the Anopheles gambiae mosquito represents a promising target

124 Anopheles gambiae mosquitoes are the most important vect

125 A study of Anopheles gambiae mosquitoes shows that a molecule invol

126 e rapidly and completely blocked when female Anopheles gambiae mosquitoes take up low concentrations

127 ingested blood enhance the susceptibility of Anopheles gambiae mosquitoes to malaria infection by dis

128 Exposure of Anopheles gambiae mosquitoes to Plasmodium infection enh

129 nd Plasmodium berghei expressing PfCelTOS in Anopheles gambiae mosquitoes.

130 asite to evade the mosquito immune system of Anopheles gambiae mosquitoes.

131 h Plasmodium falciparum infection in natural Anopheles gambiae populations at malaria endemic areas i

132 resistance and its underlying mechanisms in Anopheles gambiae populations from a subset of trial vil

133 In the African malaria vector, Anopheles gambiae quantitative and qualitative variance

134 ot competitively linked to processes shaping Anopheles gambiae reproduction.

135 Tracking multiple free-flying Anopheles gambiae responding to human-occupied bed nets

136 een carbamate and pyrethroid resistance with Anopheles gambiae s.l.

137 he probability that pyrethroid resistance in Anopheles gambiae s.l. exceeds the World Health Organiza

138 ll this knowledge gap, coupled and uncoupled Anopheles gambiae s.l. males (all M form (Anopheles colu

139 Anopheles gambiae s.s. mosquitoes are efficient vectors

140 d to assess the safety and immunogenicity of Anopheles gambiae saliva vaccine (AGS-v), a peptide-base

141 ssed by passive case detection and number of Anopheles gambiae sensu lato mosquitoes collected per li

142 Of 1494 female Anopheles (89.8% Anopheles gambiae sensu lato), 88.3% were fed, 51.9% had

143 he prevailing wind direction included 81,000 Anopheles gambiae sensu stricto, 6 million A. coluzzii a

144 ssays were conducted using laboratory-reared Anopheles gambiae sensu stricto.

145 nversion is a widespread polymorphism in the Anopheles gambiae species complex, the major African mos

146 rotein from the major African malaria vector Anopheles gambiae that specifically, tightly, and quickl

147 nding site of an OR from the malaria vector, Anopheles gambiae The closely related odorant-specificit

148 or molecules to the malaria vector mosquito, Anopheles gambiae The drive system targets the cardinal

149 actometer, we investigated the attraction of Anopheles gambiae to 50 Kenyan children (aged 5-12 years

150 anges in species composition from endophilic Anopheles gambiae to exophilic An. arabiensis.

151 Resistance in Anopheles gambiae to members of all 4 major classes (pyr

152 ive contribution of AgTRIO to the ability of Anopheles gambiae to transmit Plasmodium berghei to mice

153 ngs of heterologously expressed and purified Anopheles gambiae TRPA1 (AgTRPA1), with and without the

154 icide resistance in the major malaria vector Anopheles gambiae was assessed.

155 Anopheles gambiae was genetically partitioned into inlan

156 ome of the primary African malaria mosquito, Anopheles gambiae We find that the An. gambiae Y consist

157 repeats, located in a single cluster on the Anopheles gambiae X chromosome.

158 ob, for the M factor in the malaria mosquito Anopheles gambiae Yob, activated at the beginning of zyg

159 In Anopheles gambiae, a likely candidate for sexual selecti

160 f a survey of 1280 genomes of the mosquitoes Anopheles gambiae, An. coluzzii, and Aedes aegypti in wh

161 ated with Plasmodium falciparum infection in Anopheles gambiae, and FREP1 is important for Plasmodium

162 main, ZLD orthologs from Drosophila virilis, Anopheles gambiae, and Nasonia vitripennis activate tran

163 cificity of P47Rec from distant anophelines (Anopheles gambiae, Anopheles dirus, and Anopheles albima

164 nthetically in the main human malaria vector Anopheles gambiae, by selectively destroying the X-chrom

165 ective pressures on the major malaria vector Anopheles gambiae, caused by the widespread use of insec

166 ocidal to important vector species including Anopheles gambiae, Culex quinquefasciatus, and Aedes aeg

167 atment to deplete phagocytic immune cells in Anopheles gambiae, demonstrating the role of phagocytes

168 ory system in Plasmodium falciparum infected Anopheles gambiae, Plasmodium berghei infected Anopheles

169 s application using genome variation data of Anopheles gambiae, Plasmodium falciparum and Plasmodium

170 Afrotropical human malaria vector mosquito, Anopheles gambiae, remains a significant threat to globa

171 ructs that function as gene drive systems in Anopheles gambiae, the main vector for malaria.

172 ss of selection for pyrethroid resistance in Anopheles gambiae, the most important malaria vector in

173 After 24 h, the mortality of Anopheles gambiae, the principal African malaria vector,

174 sistance markers in the major malaria vector Anopheles gambiae, which we used to screen mosquitoes fr

175 Ago1 is the predominant carrier of miRNAs in Anopheles gambiae.

176 larvae of the mosquitoes, Aedes aegypti and Anopheles gambiae.

177 uestions regarding SGs of the malaria vector Anopheles gambiae.

178 IIS6, in pyrethroid-resistant populations of Anopheles gambiae.

179 ene drive (SDGD) in the human malaria vector Anopheles gambiae.

180 n the legs, confers pyrethroid resistance to Anopheles gambiae.

181 cturally diverse compounds against Orco from Anopheles gambiae.

182 ly understood in many species, including the Anopheles genus of mosquitoes-an emerging model system f

183 nsmitted between humans by mosquitoes of the Anopheles genus.

184 ra is required for full viral infectivity to Anopheles, in contrast to malaria infection, where the p

185 icate that the evolution of blood feeding in Anopheles involves repurposing an ancestral thermorecept

186 of male-biased genes on the X Chromosomes in Anopheles is a conserved feature in this genus and can b

187 Although Anopheles is known to be principally nocturnal and Aedes

188 resistance is a major obstacle to control of Anopheles malaria mosquitoes in sub-Saharan Africa and r

189 ly repetitive content of the Y chromosome in Anopheles malaria mosquitoes.

190 nsecticide resistance in wild populations of Anopheles malaria vectors emphasises the need for novel

191 nsects, but VKR function is required in both Anopheles males and females for development of larval pr

192 : Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role in transmission was conf

193 est wAnga may influence or interact with the Anopheles microbiota, which may contribute to the impact

194 ongly activated small numbers of ORNs in the Anopheles mosquito antennae at low concentrations.

195 ction of distance from the hotspot boundary, Anopheles mosquito density, mosquito breeding site produ

196 Malaria is transmitted when an infected Anopheles mosquito deposits Plasmodium sporozoites in th

197 After being ingested by a female Anopheles mosquito during a bloodmeal on an infected hos

198 on of malaria parasites occurs when a female Anopheles mosquito feeds on an infected host to acquire

199 th injection of Plasmodium sporozoites by an Anopheles mosquito into the skin of the mammalian host.

200 due to widespread insecticide resistance in Anopheles mosquito populations, a major public health ch

201 n vectorial capacity for human malaria among Anopheles mosquito species is determined by many factors

202 Most Anopheles mosquito species that are of major importance

203 sis based on ITS2 and COX1 genes revealed 21 Anopheles mosquito species, including two previously unr

204 Among Anopheles mosquito species, these phenotypic differences

205 present in the genomes of Aedes, Culex, and Anopheles mosquito species.

206 Six pyrethroid-resistant Anopheles mosquito strains from across Africa were expos

207 the year have suggested that some species of Anopheles mosquito use long-distance migration(3).

208 arum malaria parasites occurs when nocturnal Anopheles mosquito vectors feed on human blood.

209 a elimination is the effective management of Anopheles mosquito vectors.

210 the transmission of malaria parasites by the Anopheles mosquito.

211 modium species, and is transmitted by female Anopheles mosquitoes and presents with generic febrile s

212 Anopheles mosquitoes are vectors of the human malaria pa

213 t of participants (11/16) were infectious to Anopheles mosquitoes at peak gametocytemia.

214 ealth problem, is transmitted by a subset of Anopheles mosquitoes belonging to only three out of eigh

215 f agrochemicals to insecticide resistance in Anopheles mosquitoes breeding on vegetable farms in sout

216 " trap caught nearly ten times the number of Anopheles mosquitoes caught by a human collector.

217 IRS, An. funestus accounted for over 80% of Anopheles mosquitoes collected by light trap and PSC in

218 ate (HBR), expressed as the number of female Anopheles mosquitoes collected per house-night of collec

219 with the ingestion of gametocytes by female Anopheles mosquitoes during a blood meal.

220 nhibit parasite development when ingested by Anopheles mosquitoes during blood meals.

221 Anopheles mosquitoes employ complex behavioral and physi

222 ve pathway (AP) of complement by saliva from Anopheles mosquitoes facilitates feeding by blocking pro

223 of 300 and 600 mcg/kg/day were shown to kill Anopheles mosquitoes for at least 28 days post-treatment

224 Overall, our findings highlight that Anopheles mosquitoes have evolved different reproductive

225 tal temperature to influence the capacity of Anopheles mosquitoes to transmit the human malaria paras

226 Bites of Anopheles mosquitoes transmit Plasmodium falciparum para

227 Blood fed Anopheles mosquitoes were collected from a malaria endem

228 Anopheles mosquitoes were sampled in human settlements,

229 asmodium parasites and insecticide-resistant Anopheles mosquitoes, and first generation vaccines offe

230 ome sequence data from Plasmodium parasites, Anopheles mosquitoes, and global human populations, this

231 After transmission by Anopheles mosquitoes, Plasmodium sporozoites travel to t

232 hia on the biology and vectorial capacity of Anopheles mosquitoes, the vectors of malaria parasites.

233 nhibit parasites during their development in Anopheles mosquitoes, thus breaking the cycle of transmi

234 he genetic architecture of osmoregulation in Anopheles mosquitoes, vectors of human malaria.

235 es are frequently overexpressed in resistant Anopheles mosquitoes, were analyzed.

236 r blocking Plasmodium transmission by female Anopheles mosquitoes, which has promising implications f

237 d CO(2) is an important host-seeking cue for Anopheles mosquitoes, which is detected by a highly cons

238 skin surface, rendering humans invisible to Anopheles mosquitoes.

239 d reduced oocyst infection and prevalence in Anopheles mosquitoes.

240 ed pyrethroid, by more than 10 times against Anopheles mosquitoes.

241 nsecticide susceptibility phenotype of adult Anopheles mosquitoes.

242 nthesis and malaria parasite transmission to Anopheles mosquitoes.

243 chrysogenum, from the midgut of field-caught Anopheles mosquitoes.

244 um berghei during its life cycle in mice and Anopheles mosquitoes.

245 olism of Plasmodium-infected and -uninfected Anopheles mosquitoes.

246 l and potential biological control agent for Anopheles mosquitoes.

247 ronments and enhancing vectorial capacity in Anopheles mosquitoes.

248 nsmitted through the bite of infected female Anopheles mosquitos, is one of the main causes of mortal

249 cted with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role

250 toluamide (DEET) and IR3535 did not activate Anopheles odorant receptor co-receptor (Orco)-expressing

251 erratia bacterium strain (AS1) isolated from Anopheles ovaries that stably colonizes the mosquito mid

252 tissues of four vector species spanning the Anopheles phylogeny to explore the genomic and evolution

253 nce to insecticides has become widespread in Anopheles populations(2-4), which has led to the threat

254 alating resistance to pyrethroids in African Anopheles populations, threatening to reverse the gains

255 p of closely related mosquitoes known as the Anopheles punctulatus group.

256 To test this in the field, we quantified Anopheles responses to olfactory, visual and thermal sti

257 we address the relevance of using a specific Anopheles salivary biomarker to measure the risk among h

258 Here, we use defined panels of Anopheles samples from West Africa to test two experimen

259 tified by PCR in five species of mosquitoes (Anopheles sinensis, Armigeres subalbatus, Aedes albopict

260 tting mosquitoes like Anopheles coluzzii and Anopheles sinensis.

261 ively associated with the abundance of total Anopheles species and primary malaria vectors and the EI

262 Anopheles species assignments based on ribosomal DNA ITS

263 elationships between specific Plasmodium and Anopheles species combinations.

264 1 is highly conserved (>90% identical) among Anopheles species from different continents, suggesting

265 ings underscore the importance of non-common Anopheles species in malaria transmission and the need t

266 The likelihood of capturing Anopheles species increased with altitude (the height of

267 st every other insect species, males of some Anopheles species produce steroid hormones which are tra

268 rphological identifications recognized eight Anopheles species while 18 distinct sequence groups or s

269 or example, malaria parasites transmitted by Anopheles species, and viruses such as dengue, Zika and

270 n of multiple Plasmodium species to multiple Anopheles species.

271 gs of which are found thus far only in other Anopheles species.

272 ain about its key protagonists, a handful of Anopheles species.

273 tricto, 6 million A. coluzzii and 44 million Anopheles squamosus.

274 vides insights into the molecular anatomy of Anopheles stephensi and the distribution and localizatio

275 omics analysis to compare SGs mRNA levels in Anopheles stephensi fed on non-infected and P. berghei-i

276 an overwhelming insecticidal effect against Anopheles stephensi mosquitoes in laboratory conditions

277 e NF54 laboratory strain of P. falciparum in Anopheles stephensi mosquitoes using the standard membra

278 cid supplementations were more infectious to Anopheles stephensi mosquitoes, essentially doubling pre

279 ulses to kill or disable anesthetized female Anopheles stephensi mosquitoes, which were chosen as a r

280 lasers and pulse conditions on anesthetized Anopheles stephensi mosquitoes.

281 ssion of mature P. falciparum gametocytes to Anopheles stephensi mosquitoes.

282 tely blocked mouse-to-mouse transmission via Anopheles stephensi mosquitoes.

283 natomical structure of rodent malaria vector Anopheles stephensi mosquitoes.

284 to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant albicin was produced in

285 d the presence of an Asian mosquito species; Anopheles stephensi, a species known to thrive in urban

286 ene-drive system in the Asian malaria vector Anopheles stephensi, adapted from the mutagenic chain re

287 dynamics of Aedes aegypti, Aedes albopictus, Anopheles stephensi, and Anopheles coluzzii.

288 opheles gambiae, Plasmodium berghei infected Anopheles stephensi, and P. berghei infected An. gambiae

289 osome gene of a major urban malaria mosquito Anopheles stephensi, confers 100% female lethality when

290 pulation modification of the malaria vector, Anopheles stephensi, that relieves the load in females c

291 h for analysis of the genome and proteome of Anopheles stephensi, which is one of the most important

292 creation of conditional male-only transgenic Anopheles strains for malaria control programs.

293 lus high contrast visual stimuli caught more Anopheles than traps with odour alone, showing that desp

294 losing efficacy against pyrethroid-resistant Anopheles vectors(3,4), methods that restore performance

295 les were found infected with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles mar

296 Results showed that Anopheles VKR can be activated by L-amino acids, with L-

297 n for reproduction and immunity suggest that Anopheles VKR could be a potentially druggable target fo

298 Anopheles VKR function is also required for protection a

299 In this study we functionally characterized Anopheles VKR in the Gambiae complex member, Anopheles c

300 Among them, only three species of Anopheles were found infected with ape Plasmodium: Anoph