ライフサイエンスコーパス: B

1 B cell antigen receptor (BCR) signals induce Syk activat

2 B lymphocytes are important players of the adaptive immu

3 B, T, and myeloid cells were analyzed before anti-CD20 a

4 B-1 B cells derive from a developmental program distinct

5 B. licheniformis PGA elicited more TLR4 signal than B. a

6 B. mojavensis EPS (BM-EPS) was recovered, fractionated b

7 B., & Plowright, R.

8 B-1 B cells derive from a developmental program distinct fro

9 k plasma sintered Fe(48)Cr(15)Mo(14)Y(2)C(15)B(6) metallic glass is established by analyzing the crys

10 , we make dysprosiacarboranes, namely [(C(2) B(9) H(11) )(2) Ln(THF)(2) ][Na(THF)(5) ] (Ln=Dy, 1Dy) a

11 y{eta(2) :eta(5) -C(6) H(4) (CH(2) )(2) C(2) B(9) H(9) }(2) Li] 3Dy and show that dicarbollide ligand

12 Li](2) [{eta(5) -C(6) H(4) (CH(2) )(2) C(2) B(9) H(9) }Dy{eta(2) :eta(5) -C(6) H(4) (CH(2) )(2) C(2)

13 The thioglycosylated trans-A(2)B(2) porphyrins showed decent in vitro singlet oxygen ge

14 A borinic acid functionalized 1,4-B,N-anthracene 1 was found to display unprecedented reac

15 d naive IL-4Ralpha(-/lox) or IL-4Ralpha(-/-) B cells into muMT(-/-) mice a day before sensitization o

16 lassified in BCLC stage 0 (9.4%), A (59.5%), B (16.2%), and C (14.9%).

17 Li(5) B(4) /Li electrodes are coupled with a garnet-type ceram

18 Cr(0.4) Mo(0.6) B(2) has excellent long-term stability and durability sh

19 In chronological order, I have a B.A. in Scandinavian languages and literature from UCLA,

20 ates are formed by the direct oxidation of a B-B bond between a boron cluster cage and an exopolyhedr

21 the Fe(III) and Fe(IV) =O redox states of a B-type DyP.

22 Patients were categorized into 4 groups (A, B, C, and D) corresponding to the manufacturer from whic

23 kaloids and their analogues with modified A, B, and C rings, as well as hybrid compounds derived from

24 itor of tropomyosin receptor kinase (TRK) A, B, and C, which has been shown to have anti-tumour activ

25 compound led to tumor stasis in an activated B-cell-like (ABC) diffuse large B-cell lymphoma (DLBCL)

26 cquisition of founder mutations in activated B cells favors the development of aberrant MBs prone to

27 ctor kappa-light-chain-enhancer of activated B cells)-dependent inflammation, caspase-dependent apopt

28 ity to effectively recruit T cell help after B cell activation.

29 ll enforced, and there was selection against B cells with high affinity for self-antigen.

30 nt or as step-down therapy from amphotericin B.

31 along elevated viaducts of Metro lines 4 and B, as well as differential displacements in Pantitlan st

32 Human herpesvirus-6 (HHV-6) A and B are ubiquitous betaherpesviruses, infecting the majori

33 pproved in 2018 for treating influenza A and B virus infections.

34 ders, including carriers of hemophilia A and B, or with von Willebrand disease, have an increased ris

35 Serum toxin A- and B-specific neutralizing antibodies were measured.

36 pha activation lowered surface BCR, CD19 and B cell-activating factor receptor and increased expressi

37 gens, cluster of differentiation 3 (CD3) and B-cell maturation antigen (BCMA).

38 r reference samples (breast cancer cells and B cells), captured either separately or in mixtures, we

39 metathesis mediated (prevalent with B-H and B-R electrophiles).

40 ferences favored the MN assay for A/H1N1 and B strains in study 1, whereas the titers determined by H

41 ins: A/WSN/33 (H1N1), A/Udorn/72 (H3N2), and B/Lee/40.

42 cterized clinical parameters, mortality, and B cell phenotypes in blood and cerebrospinal fluid (CSF)

43 assays for the detection of B. pertussis and B. parapertussis.

44 memory Th cells as well as regulatory T and B cells were increased.

45 rograde motor IFT dynein, and the IFT-A and -B complexes.

46 ID1A) (13%), albumin (11.4%), apolipoprotein B (APOB) (9.4%), and AXIN1 (9.2%).

47 andard deviation) values for apolipoproteins B and A-I were 1.03 (0.24) g/L and 1.54 (0.27) g/L, resp

48 uced dosing/treatment discontinuation in arm B vs arm C.

49 : A subpopulation of B cells (age-associated B cells [ABCs]) is increased in mice and humans with inf

50 Thymus-associated B cell clones were detected in the circulation by both m

51 Many circulating thymus-associated B cell clones were inferred to have originated and initi

52 ments with liposomes revealed that the Aster-B GRAM domain binds to membranes in a cholesterol concen

53 Aurora B kinase has a critical role in regulating attachments b

54 Furthermore, we discovered that the BARLEY B RECOMBINANT/BASIC PENTACYSTEINE transcription factors

55 tage of Barcelona Clinic Liver Cancer (BCLC) B and C, and the presence of cirrhosis, respectively.

56 Because B vitamins are essential for survival, null mutations of

57 paper reports the oxidation of Remazol black B dye by employing iron ions catalyst based gas diffusio

58 ipid interactions and create a modified BoNT/B with improved therapeutic efficacy.

59 erminal receptor-binding domain (HC) of BoNT/B (HC/B) has been proposed to also contribute to toxin b

60 of human transcription factor TFIIH, in both B lymphocytes and epithelial cells, we hypothesized that

61 s of the marine natural product (+)-bretonin B.

62 ilms revealed that C-H functionalization by [B(12)X(11)](-) occurred in the presence of other more re

63 ctivity with the cysteine protease cathepsin B.

64 r these ADCs involve cleavage with cathepsin B or papain to release and measure the antibody-conjugat

65 lly infected biopsies indicated that CD20(+) B lymphocytes, CD8(+) T lymphocytes, and CD11c(+) cells

66 n of CD4(+) T cells while suppressing CD5(+) B-1a cell development.

67 as well as increased populations of CD73(+) B lymphocytes.

68 for the brain data and granulocytes/T cells/B cells/monocytes for the blood data.

69 Mice developed expanded germinal center B lymphocyte populations as in other models of AID defic

70 CL, NOS was predominantly of germinal center B-cell (GCB) subtype and carried gene mutations similar

71 ing a M(B2) permutational mechanism (Channel B).

72 Ps) and complexes of SEDS proteins and class B PBPs (bPBPs).

73 s, these provide coverage of the major class B GPCR subfamilies.

74 are a thirty-three-member subfamily of Class B GPCRs that control a wide array of physiological proce

75 he events that drive the activation of class B receptors.

76 -A1-CD15-LDE225 via scavenger receptor class B type 1 (SR-B1) and CD15 on brain SHH MB cells in vitro

77 ducts BB0406 and BB0405, members of a common B. burgdorferi paralogous gene family, share 59% similar

78 imulation and survival in vivo Consequently, B. fragilis acquires essential heme from host tissues du

79 l program distinct from that of conventional B cells, through B cell receptor (BCR)-dependent positiv

80 lity using legacy models (e.g. Brooks-Corey (B-C), van Genuchten, etc.) that were developed for conve

81 he product of an obligate gene for FAO (CPT2(B-/-)).

82 Dermotype B was characterized by reduced microbial richness, deple

83 been shown that patients with HFpEF develop B-lines upon submaximal exercise stress echocardiography

84 tatus of signaling proteins across different B cell subpopulations present in a single sample.

85 Our approach was to utilize differential B-cell depletion with anti-CD20 to retain B cells whose

86 We find aberrant activation of early B cell factor 1 (EBF1) to promote transformation of LMP1

87 lozoyensis than to those of the echinocandin B gene cluster from A. pachycristatus.

88 irus(RV-C)microbiome(mixed)T2(low); endotype B, virus(RV-A)microbiome(Haemophilus)T2(low); endotype C

89 a number of exceptionally potent epothilone B analogues, demonstrating the potency enhancing effects

90 patients, we hypothesize that ET(A)R and ET(B)R play dichotomous roles in oral carcinogenesis and pa

91 such that ET(A)R activation and silenced ET(B)R expression result in increased carcinogenesis and pa

92 to promote transformation of LMP1-expressing B cells by inhibiting their differentiation to plasma ce

93 o-access, near-UV-emitting linearly extended B,N-doped heptacene with high thermal stability is desig

94 ncharacterized population of extrafollicular B helper T cells, which produced IL-10 and could play a

95 e of a 1.5 kb platelet derived growth factor B (PDGFB) promoter.

96 Recent structures of family B GPCR-G(s) protein complexes reveal a disruption in the

97 al organization of T(FH) cells to form T(FH):B cell conjugates in germinal centers.

98 a ferromagnetic liquid with a magnetic field B(S) = 10(12+/-1) T.

99 We report here that DksA is essential for B. burgdorferi to infect a mammalian host.

100 stablished a pan-transcriptome reference for B. juncea, to which we mapped transcriptome data from a

101 This study supports an important role for B cells in indirect T cell priming and further emphasize

102 Recent work has uncovered a role for B cells in the induction and activation of Tregs during

103 00 monoclonal antibodies (mAbs) derived from B cells induced by numerous influenza virus vaccines and

104 ere caused by species that are distinct from B. dermatitidis Increasing clinical awareness and access

105 DFD regimen favors persistence of functional B cells after the third dose.

106 Children with ABL-class fusion B-cell acute lymphocytic leukaemia have poor outcomes wh

107 Baclofen is a GABA(B) agonist prescribed as a treatment for spasticity in s

108 ated whether a single 10 mg dose of the GABA(B) agonist baclofen impaired motor sequence learning and

109 Within lymph nodes, we observed augmented GC B cell responses and the promotion of T(h)1 gene express

110 ignancy, namely follicular lymphoma (FL), GC B cell-diffuse large B cell lymphoma (GCB-DLBCL), and Bu

111 olled a unique transcriptional program in GC B cells that promoted optimal GC polarization and choles

112 However, how apoptotic caspases regulate GC B cell fate has not been fully characterized.

113 VZV glycoprotein B (gB) peptides assembled into fibrils and catalyzed amy

114 Older age (OR-1.07), grade B/C pancreatic fistula (OR-3.84), and epidural use (OR-3

115 classified as Grade A, 193 (66.1%) as Grade B, and 65 (22.2%) as Grade C.

116 ts in the death of tumor cells in a granzyme B (GrB)-dependent manner.

117 1 oncogene Tax, driven by the human granzyme B promoter (Tax(+)), develop osteolytic tumors.

118 x 107 HSV DNA copies, and surges in granzyme B and IFN-gamma occurred within the early hours after re

119 ve on Galalpha1-3Gal but not the blood group B antigen.

120 ortality was higher in group A than in group B (17.8% and 8.1%, respectively; P = .001).

121 l specimens was significantly lower in Group B than in Group A (128.68cm3 vs. 189.37cm3; p<0.05).

122 s licensed to protect against invasive group B meningococcal disease.

123 The meningococcal group B vaccine 4CMenB is a new, recombinant protein-based vac

124 s degradation of xeroderma pigmentosum group B-complementing protein (XPB), a component of human tran

125 ectively induce ex vivo expansion of GZMB(+) B cells.

126 zygotic contributions, we show that the H2A.B status of both the father and mother, but not of the z

127 l receptor-binding domain (HC) of BoNT/B (HC/B) has been proposed to also contribute to toxin binding

128 Hepatitis B and C virus-associated HCC became less common, and mor

129 Hepatitis B virus (HBV) is an important but difficult to study hum

130 e intravenous group (sepsis [n=2], hepatitis B reactivation [n=1], and Pneumocystis jirovecii pneumon

131 r, smoking status, hepatitis C and hepatitis B virus coinfection, group of exposure, nadir CD4 count,

132 ve mixed tailing in transcripts of hepatitis B virus (HBV) and human cytomegalovirus (HCMV), generate

133 iate analysis were the etiology of hepatitis B, the stage of Barcelona Clinic Liver Cancer (BCLC) B a

134 exam, including tuberculosis (TB), hepatitis B, hepatitis C, malaria, strongyloidiasis, schistosomias

135 larly in sub-Saharan Africa, where hepatitis B virus (HBV) is an important risk factor.

136 HLA-B*5701-negative adults without hepatitis B virus co-infection to receive coformulated bictegravir

137 models is that bnAb expression often hinders B cell development.

138 Study 1 randomly assigned (1:1) HLA-B*5701-negative adults without hepatitis B virus co-infe

139 one of the most prevalent HLA-B alleles, HLA-B*52:01, present in 22.5% of infected individuals.

140 s a potential effect on T-lymphocytes at HLA-B(*)55:01.

141 nificantly increased the presentation of HLA-B, -C, and -E on lung epithelial cells.

142 rt in Delhi on one of the most prevalent HLA-B alleles, HLA-B*52:01, present in 22.5% of infected ind

143 The HLA-B*57:01-restricted, HIV epitope-specific CD8 T-cell resp

144 was US $58.64 (range: US $37.25 at Hospital B, US $73.27 at Hospital A).

145 Previously identified B. burgdorferi proteins, lipid immunogens, and live muta

146 sitivity and 100% specificity in identifying B. microti antibody containing sera in an ELISA.

147 hat at low doses these SCFAs directly impact B cell intrinsic functions to moderately enhance class-s

148 lues of the transposase genes were higher in B. oleracea than in B. rapa.

149 t1 to achieve near-saturation mutagenesis in B. distachyon, which will aid in functional genomics stu

150 ase genes were higher in B. oleracea than in B. rapa.

151 es, decreased T cell infiltration, increased B cell numbers, and decreased macrophage recruitment.

152 use today depend on their ability to induce B cell memory, we have not yet succeeded in developing v

153 ct, a large proportion of islet-infiltrating B lymphocytes in the NOD mouse model of T1D produce Abs

154 ence materials, and contextual information, (B) factors that relate to the specific person doing the

155 evels had significantly higher serum Inhibin B and total sperm count than men with the lowest serum K

156 iversity microbiota, Community State Type IV-B, was the most prevalent in both HIV-negative (38% at b

157 re in K, S is the Seebeck coefficient, and k(B) /e = 86.3 uV K(-1) .

158 ral boron-based substituent (e.g., -BF(3)K, -B(OH)(2)).

159 es as well as NFkappaB (nuclear factor kappa B).

160 n receptor activator of unclear factor kappa-B ligand, periostin, and peroxidasin gene expressions in

161 se B pathway by injecting the protein kinase B activator SC79 in Lgr4(-/-) mice can effectively rever

162 ntly, the reactivation of the protein kinase B pathway by injecting the protein kinase B activator SC

163 ) in a TGF-beta receptor/PI3K/protein kinase B-dependent manner, to regulate hepatic acetyl-CoA and c

164 mouse line with Tropomyosin receptor kinase B (TrkB) receptor deletion from D1-expressing cells (D1-

165 ese catalysts have low linear-to-branched (L:B) regioselectivity for simple linear alkenes.

166 cision medicine approaches for diffuse large B cell lymphoma (DLBCL) is confounded by its pronounced

167 cular lymphoma (FL), GC B cell-diffuse large B cell lymphoma (GCB-DLBCL), and Burkitt lymphoma (BL).

168 ainst recurrent and refractory diffuse large B cell lymphomas (DLBCL).

169 an activated B-cell-like (ABC) diffuse large B-cell lymphoma (DLBCL) xenograft model, but this compou

170 dent deformability increase upon latrunculin B-induced actin disassembly was detected only with cDC a

171 included 28 P/LP variants, 96 VUS, and 34 LB/B variants, mostly in cancer (40%) and cardiac (27%) ris

172 and adults with Philadelphia chromosome-like B cell acute lymphoblastic leukemia (Ph-like B-ALL) expe

173 B cell acute lymphoblastic leukemia (Ph-like B-ALL) experience high relapse rates despite best-availa

174 ucture of pri-let-7, which also binds Lin28A/B.

175 tert-butanol as a solvent, 20 g/L of lipase B from Candida Antarctica, and vinyl cinnamate as acyl d

176 proportion of Hispanic residents have lower B-CPR rates and lower survival.

177 nthesizing compounds with electron-precise M=B triple bonds analogous to classical carbyne systems.

178 he immune environments associated with major B-cell lymphomas with an emphasis on the immune escape p

179 Memory B cells (MBCs) expressing the transcription factor T-bet

180 lls (CD3(+)CD8(+)CD161(+)PD1(+)), and memory B cells (CD3(-)CD19(+)CD20(+)CD24(+)CD27(+)) were found

181 ration of long-lived plasma cells and memory B cells and highlight the challenges for successful vacc

182 ence that primary FCRL4-bearing human memory B cells are constitutively bound to IgA.

183 ~2,200 IgG-secreting activated human memory B cells, activated ex vivo, demonstrating its versatilit

184 th B cells led to greater tissue like memory B cell frequencies.

185 reaction, limiting the generation of memory B cell and long-lived plasma cell responses.

186 Alternatively, subsets of memory B cells were lower in abundance in cirrhotic relative to

187 ed upon antigen re-encounter, whereas memory B cells expressed receptors capable of neutralizing viru

188 stable transfection of the Babesia microti (B. microti) in the in vivo conditions.

189 in Mongolia from the late second millennium B.C.E. through the Mongol Empire are traditionally assum

190 49 gliomas (86%; 95% CI: 76%, 96%) for model B.

191 ss is abolished in C18orf8-, Ccz1- and Mon1A/B-deficient cells and restored by a constitutively activ

192 selective formation of bromo-substituted B,N,B-benzo[4]helicenes 2 a/2 b via intramolecular borylatio

193 Memory and Naive B cells are good potential predictors of LC and HCCin di

194 tors determining whether an individual naive B cell will proliferate following Ag encounter remains u

195 ype possessing the ability to suppress naive B cells.

196 tic interaction of MYC and MNT in neoplastic B-cell development, but the underlying mechanism remaine

197 cause of parenterally transmitted non-A, non-B hepatitis.

198 parallel, MCL cells as compared with normal B cells expressed elevated levels of WNT16, a NF-kappaB

199 All vaccines rely on the ability of B cells to remember pathogen infections and respond more

200 ation as is the case with the Co-C5' bond of B(12).

201 ead to germinal centers that are composed of B-cells that come from a single strain-specific clone, a

202 ed the potential functional contributions of B cells via bulk and single-cell RNA sequencing, which d

203 other molecular assays for the detection of B. pertussis and B. parapertussis.

204 nations for the puzzling fact that growth of B. subtilis does not result in the significant accumulat

205 We demonstrate that lytic MHV68 infection of B cells, macrophages, and fibroblasts leads to robust ac

206 ne and M3 significantly increased killing of B. cenocepacia and P. aeruginosa in CF MDMs in a dose-de

207 d transcriptome data from a diverse panel of B. juncea accessions.

208 We have identified a novel promoter of B. microti.

209 its substrate Notch, a critical regulator of B cell and T cell development.

210 Our aim was to analyze the role of B cells in a house dust mite (HDM)-based murine asthma m

211 In particular, the central role of B cells in the disease has been demonstrated by both the

212 individuals, indicating strong selection of B cell antigen receptors even in the absence of microbio

213 al expansion and unique functional states of B cells in responders.

214 Rationale: A subpopulation of B cells (age-associated B cells [ABCs]) is increased in

215 wever, it is unclear which subpopulations of B cells express GZMB under normal conditions and which p

216 04 patients comprising different subtypes of B cell neoplasms, we demonstrate that IgCaller identifie

217 cts of M.tb infection and BCG vaccination on B cell responses to heterologous pathogen recall antigen

218 r mixed models, including biomarker [log10(P/B ratio) and/or AMY1 CN] diet-group interactions.

219 calculations, show that (sigma, B p)->(pai, B p) transitions accelerate the ISC process.

220 eous opacities within 5 foveal or parafoveal B-scans (vitreous opacity ratio).

221 owed by a 40-week open-label extension (part B), during which time all participants received clazakiz

222 Patients in part B were given the recommended phase 2 dose.

223 PDE4 family comprises four subtypes, PDE4A, B, C, and D.

224 sing mAbs (n = 14) generated from peripheral B cells of two patients.

225 12 vg per animal, n = 10 per group) with PHP.B vectors.

226 pressive symptoms and care partner physical (B=0.05, P<0.001) and psychological (B=0.04, P<0.001) QOL

227 ls during the production phase, or in plasma B cells.

228 %] of 7 patients) and PDGFRB fusion-positive B-cell acute lymphocytic leukaemia (43 [88%] of 49 patie

229 omerular filtration rate 1-y posttransplant (B, 0.58; 95% CI, -2.07 to 3.22; P = 0.67), and long-term

230 Pre-B cell leukemia factor 1 (PBX1) is an essential developm

231 optosis and markedly decreasing premalignant B lymphoid cell populations.

232 initiated the transformation of premalignant B cells.

233 agreement with structural features present: B. subtilis gyrase is a minimal enzyme, and its subunits

234 Holland (now Janssen Vaccines and Prevention B.V.), European Union's Horizon 2020 research and innova

235 ally under negative selection during primary B cell development.

236 elevated cardiac biomarkers (N-terminal pro-B-type natriuretic peptide [>40 pg/mL] or troponin T [>0

237 red with placebo, the single-species product B animalis subsp lactis or L reuteri significantly reduc

238 t NLRC5 signaling in macrophages can promote B-cell lymphomagenesis during chronic Helicobacter infec

239 ation of multiple self-antigens that promote B cell expansion.

240 glycoprotein non-metastatic melanoma protein B (GPNMB) signaling in human PD SN.

241 Follicular T helper (TFH) cells provide B-cell help and are crucial for generating long-term hum

242 hysical (B=0.05, P<0.001) and psychological (B=0.04, P<0.001) QOL.

243 this method, the natural product pyrolaside B was synthesized for the first time.

244 mptomatology and survivor psychological QOL (B=0.03, P<0.05) and moderated the association between ca

245 w that high-affinity vaccines targeting rare B cells capable of broadly protective antibody responses

246 were few effects of light or flow in the RAS+B system, potentially highlighting the importance of het

247 ight had a more pronounced effect in the RAS-B system, while flow affected certain coral response var

248 MN), we studied the cholecystokinin receptor B-expressing (CCKBR-expressing) VMN targets of glucose-e

249 In normal resting B cells, IFITM3 was minimally expressed and mainly local

250 al B-cell depletion with anti-CD20 to retain B cells whose presence were required to achieve EAE reco

251 rded as possibly carcinogenic, and rhodamine B was reported to cause local sarcomas.

252 vine fetuin, asialofetuin, IgG, ribonuclease B, and alpha-1 acid glycoprotein (AGP) by PGC-LC-MS.

253 main products rishirilide A and rishirilide B.

254 althy had significantly higher RSV-A and RSV-B titers compared to infants that subsequently contracte

255 via intramolecular borylation and sequential B-Mes bond cleavage in the presence of BBr(3) .

256 simple mathematical model, increasing sigma(B) expression shifts the peak of sporulation to the midd

257 ith DFT/MRCI calculations, show that (sigma, B p)->(pai, B p) transitions accelerate the ISC process.

258 e evolutionary constraints of antigenic site B have changed over time.

259 a virus Ankara vector to induce HBV-specific B- and T-cell responses.

260 for selective formation of bromo-substituted B,N,B-benzo[4]helicenes 2 a/2 b via intramolecular boryl

261 Treatment with Tat-A and Tat-B caused membrane disruption and pore formation in HeLa

262 generating a CoA ester of tiglic acid (Taxol B side chain), TmAAE3 and TmAAE13 as suitable for the ac

263 ench-stable Bi(III) precursor via telescoped B-to-Bi transmetallation and oxidation.

264 l (pseudo)halides using tetrahydroxydiboron (B(2)(OH)(4)).

265 ) and CD8(+) T cells to a larger extent than B. burgdorferi.

266 eniformis PGA elicited more TLR4 signal than B. anthracis PGA, whereas B. subtilis PGA elicited none.

267 Finally, we showed that B. miyamotoi-stimulated DCs induced proliferation of nai

268 The B cell response showed converging IGHV3-driven BCR clust

269 e delineation of the CD22 interactome in the B cell line DT40.

270 latency and lytic programmes to navigate the B-cell compartment and evade immune responses.

271 However, engagement of the B cell receptor (BCR) induced both expression of IFITM3

272 sociated with increased plasma levels of the B-cell-attracting chemokine CXCL13.

273 nable other researchers in understanding the B. microti biology, host modulation and diverse parasite

274 t from that of conventional B cells, through B cell receptor (BCR)-dependent positive selection of fe

275 r the detection of IgM and IgG antibodies to B. burgdorferi The BioPlex 2200 Lyme Total assay exhibit

276 Compared to B-type (non-variant) cells of P. polymyxa strain E681, i

277 robust clinical responsiveness of IgG4-RD to B cell depletion and by the identification of multiple s

278 The authors revealed a distinct tolerant B cell phenotype possessing the ability to suppress naiv

279 ding measurements performed on cholera toxin B subunit homopentamer (CTB(5)) and nanodiscs containing

280 ne, and nanodomains induced by cholera toxin B.

281 We developed transgenic B. distachyon plants expressing Tnt1 (R0) and in the sub

282 ated with specific expansion of transitional B cells, extrafollicular IgG2c-producing plasma cells, a

283 ice intranasally challenged with a wild-type B anthracis strain or with an isogenic mutant deficient

284 activation of proteins belonging to the type-B Response Regulator family of cytokinin response activa

285 works of the five most relevant HIV-1 types (B and circulating recombinant forms [CRFs] CRF01_AE, CRF

286 Ultraviolet B (UVB) radiation is a natural nonchemical stressor posi

287 B versus placebo and narrowband-ultraviolet B in patients with nonsegmental vitiligo.

288 rase 4 inhibitor, and narrowband-ultraviolet B versus placebo and narrowband-ultraviolet B in patient

289 ults are not only valuable for understanding B cell biology, but also have important implications for

290 We present a model for UVR8 action in UV-B-acclimated plants growing in photoperiodic conditions

291 Deficient compared with higher vitamin B-12 was significantly associated with lower cognitive s

292 able medium to provide water soluble vitamin B(12) and fat soluble vitamin D(3) in single product.

293 e TLR4 signal than B. anthracis PGA, whereas B. subtilis PGA elicited none.

294 er in germ-free than in SPF mice, and winner B cells in germ-free germinal centres are enriched in 'p

295 sidents in a neighborhood is associated with B-CPR delivery and survival from out-of-hospital cardiac

296 ylation of a wide range of chloroarenes with B(2)pin(2) at room temperature in excellent yields and w

297 rect NV grading was 87.8% using SS-OCTA with B-scans and 86.2% using FA (P = .92).

298 gma-bond metathesis mediated (prevalent with B-H and B-R electrophiles).

299 philic aromatic substitution (prevalent with B-X electrophiles); (ii) sigma-bond metathesis mediated

300 Culture of MAIT cell supernatants with B cells led to greater tissue like memory B cell frequen