ライフサイエンスコーパス: BAL fluid

1 or lung tissue, r = 0.887 and P < 0.0001 for BAL fluid).

2 pecimens (sputum and bronchoalveolar lavage [BAL] fluid).

3 ; three patients cultured Aspergillus sp. in BAL fluid.

4 frequency of dual-positive TH2/TH17 cells in BAL fluid.

5 in lung homogenates, and ELISA of Muc5ac in BAL fluid.

6 sure increased numbers of macrophages in the BAL fluid.

7 Macrophages were isolated from BAL fluid.

8 uring both acute and latent infection in the BAL fluid.

9 flammatory cytokines to higher levels in the BAL fluid.

10 of albumin and lactate dehydrogenase in the BAL fluid.

11 L-33 induced IL-13-producing nuocytes in the BAL fluid.

12 constituting up to 50% of the total cells in BAL fluid.

13 f Pneumocystis colonization by nested PCR of BAL fluid.

14 trophil chemoattractant capacity seen in BOS BAL fluid.

15 oniae and HRV loads in cell supernatants and BAL fluid.

16 manual method was more sensitive for GHE in BAL fluid.

17 correlated with the concentration of IL-5 in BAL fluid.

18 Aspergillus galactomannan antigen or DNA in BAL fluid.

19 ly with the number of neutrophils present in BAL fluid.

20 related against neutrophil concentrations in BAL fluid.

21 the host defense protein lactotransferrin in BAL fluid.

22 hanced secretory IgA and reduced tryptase in BAL fluid.

23 al growth factor-A, and IL-1RA production in BAL fluid.

24 atory effects reducing cells and proteins in BAL fluid.

25 lymphoid cells and basophils were scarce in BAL fluid.

26 I(+) myeloid dendritic cells were present in BAL fluid.

27 luated biomarkers in bronchoalveolar lavage (BAL) fluid.

28 vated in BOS patient bronchoalveolar lavage (BAL) fluid.

29 IP-10, and IL-33 in bronchoalveolar lavage (BAL) fluid.

30 levels of chemokines in the bronchoalveolar (BAL) fluid.

31 nts were measured in bronchoalveolar lavage (BAL) fluid.

32 6, and leukocytes in bronchoalveolar lavage (BAL) fluids.

33 hich showed greater sensitivity than qCx for BAL fluid (100% versus 67%; P = 0.04; n = 15).

34 increased levels of IL-4, IL-5, and IL-13 in BAL fluid after 11 challenges whereas IL-10 and TGF-beta

35 polyunsaturated acyl groups predominated in BAL fluid after bleomycin injury, with 22:5 and 22:6 spe

36 numbers of leukocytes and cytokine levels in BAL fluid after OVA sensitization and 7 airway challenge

37 D metabolites were increased in concentrated BAL fluids after allergen compared to saline challenge.

38 Cytokine (IL-5) levels in BAL fluid also increased markedly following 100 mug down

39 evidenced by neutrophilia or eosinophilia on BAL fluid analysis before CYC therapy and on repeat anal

40 Persistently abnormal results on BAL fluid analysis following CYC treatment is a common f

41 f PFTs and had abnormal findings on followup BAL fluid analysis.

42 e in sialylated glycans from eosinophils and BAL fluid analyzed by mass spectrometry.

43 ease in HIV that was detectable in acellular BAL fluid and a slower decrease in the HIV RNA and DNA l

44 ildren with STRA had significantly increased BAL fluid and biopsy specimen eosinophil counts compared

45 Airway inflammation, remodeling, and BAL fluid and biopsy specimen T(H)2 cytokines were quant

46 As a control group, BAL fluid and biopsy specimens from 22 patients with SCI

47 rofiles of antigen-specific CD4 T cells from BAL fluid and blood before and after the initiation of h

48 ge and correlated with eosinophil numbers in BAL fluid and blood.

49 the soluble form of IL-13Ralpha2 in serum or BAL fluid and did not affect IL-13-dependent signal tran

50 mined the effects of SIV infection on AMs in BAL fluid and IMs in lung tissue of rhesus macaques.

51 In cells collected from BAL fluid and in PBMCs, the concentrations of interferon

52 s well as increased levels of adiponectin in BAL fluid and increased adiponectin expression by airway

53 Mononuclear cells from BAL fluid and peripheral blood mononuclear cells (PBMCs)

54 dioidal antigen preparation, cells from both BAL fluid and peripheral blood obtained from patients wi

55 398 and 368 dysregulated metabolites in the BAL fluid and plasma of sensitized and challenged mice w

56 that is observed at higher concentrations in BAL fluid and plasma of subjects with chronic obstructiv

57 We analyzed BAL fluid and serum for a total of 244 biomolecules usin

58 lecules studied, 52 and 14 biomolecules from BAL fluid and serum, respectively, were significantly di

59 y Gram-positive cocci (GPC), but cultures of BAL fluid and subcarinal lymph node biopsy tissue were n

60 , T cell responses were detected only in the BAL fluid and were lower and more restricted than those

61 These BAL fluids and exogenous polyamines were able to induce

62 Bronchoalveolar lavage (BAL) fluid and blood were collected before initiation of

63 s were determined in bronchoalveolar lavage (BAL) fluid and blood.

64 rvival in both human bronchoalveolar lavage (BAL) fluid and fetal bovine serum (FBS), (ii) survival i

65 Bronchoalveolar lavage (BAL) fluid and lung biopsy specimens were stained with h

66 g sporangiospores in bronchoalveolar lavage (BAL) fluid and lung tissue homogenates from rabbits was

67 r viral loads in the bronchoalveolar lavage (BAL) fluid and nasal and ocular swabs in aged animals, s

68 and viral genomes in bronchoalveolar-lavage (BAL) fluid and nasal swab specimens were assessed by pol

69 d cellularity in the bronchoalveolar lavage (BAL) fluid and notable lung eosinophilia not seen in con

70 g acute infection in bronchoalveolar lavage (BAL) fluid and peripheral blood.

71 rgic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were collected from three groups o

72 as been validated on bronchoalveolar lavage (BAL) fluid and serum specimens, but recent evidence sugg

73 mmatory cytokines in bronchoalveolar lavage (BAL) fluid and the lungs.

74 and CD4(+) cells in bronchoalveolar lavage (BAL) fluid and was associated with significantly increas

75 et correlated with the number of MSCs in the BAL fluid, and myofibroblasts present in the fibrotic le

76 (flow cytometry) immune parameters in blood, BAL fluid, and sputum, before and after mepolizumab and

77 6, IL-23, C3a, and serum amyloid A levels in BAL fluid, and these correlated with IL-1beta and C3a le

78 less protein in the bronchoalveolar lavage (BAL) fluid, and a significant reduction in the number of

79 both in vivo, in the bronchoalveolar lavage (BAL) fluid, and in vitro, following Ag stimulation of pe

80 on, and lung tissue, bronchoalveolar lavage (BAL) fluid, and plasma were investigated.

81 med transport media, bronchoalveolar lavage (BAL) fluid, and sputum.

82 ng, IL1RN protein in bronchoalveolar lavage (BAL) fluid, and trafficking of lymphocytes and neutrophi

83 sed by the inhibition of extracellular NE by BAL fluid antiproteases and its binding to leukocytes.

84 Higher values of CXCL10 (IP-10) in BAL fluid are associated with ACR in LTRs, suggesting a

85 ice restored levels of IL-10 and TGF-beta in BAL fluid as well as suppressed AHR and inflammation.

86 HIT1) bioactivity in bronchoalveolar lavage (BAL) fluid, as well as CCL18, YKL-40 and CHIT1 mRNA leve

87 iferation of plasmacytoid dendritic cells in BAL fluid at 3 days postinfection in RMs infected with S

88 Free neutrophil elastase activity in BAL fluid at 3 months of age was associated with persist

89 vels were reduced in bronchoalveolar lavage (BAL) fluid, BAL cells, and primary alveolar macrophages

90 At the end of each phase, BAL fluid (BALF) and plasma samples were obtained.Measur

91 esized that proteins and protein pathways in BAL fluid (BALF) would distinguish children with neuroen

92 active TGF-beta1 in bronchoalveolar lavage (BAL) fluid (BALF).

93 ed that MV, found in bronchoalveolar lavage (BAL) fluids (BALF) of LTR at CLAD diagnosis, are elevate

94 lated from blood and bronchoalveolar lavage (BAL) fluid before and after segmental allergen challenge

95 aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

96 T cell proliferation was higher in the BAL fluid but delayed and reduced in peripheral blood in

97 e blood, spleen, and bronchoalveolar lavage (BAL) fluid, but not in the lung interstitium.

98 ion of IL-8 reduced migration of PMNs toward BAL fluid by approximately 50%, indicating that IL-8 is

99 Viral replication was not detectable in BAL fluid by day 2 after challenge in seven of eight ani

100 Lung nitrates were measured in BAL fluid by use of the Greiss reaction, and cytokines w

101 -glucan detection in bronchoalveolar lavage (BAL) fluid by Fungitell assay aims to further improve up

102 subsets constituted secondary TH types, and BAL fluid CD8(+) T cells were almost exclusively IFN-gam

103 However, it is unknown whether the BAL fluid cell count obtained after CYC treatment of alv

104 ificantly differ from that in patients whose BAL fluid cell counts had normalized (P = 0.70 and P = 0

105 Of the 25 study patients, in only 6 did the BAL fluid cell counts normalize after CYC therapy.

106 1), fraction of exhaled nitric oxide values, BAL fluid cell counts, and corticosteroid use.

107 Expression profiling of BAL fluid cells collected before and after challenge sho

108 Recognition was significantly absent from BAL fluid cells of patients with other lung diseases, in

109 /=2% eosinophilia in bronchoalveolar lavage (BAL) fluid, chronic lung allograft dysfunction (CLAD)-fr

110 anscriptional profiling of AMs purified from BAL fluid collected from 35 subjects with ARDS.

111 creased IL-5 and IL-13 protein expression in BAL fluid compared to vehicle-treated mice.

112 ith IPF had double the burden of bacteria in BAL fluid compared with 44 control subjects.

113 ammation were expressed at greater levels in BAL fluid compared with blood ILC2s, whereas blood ILC2s

114 llenge, and reduced IL-13 cytokine levels in BAL fluid compared with values seen in vehicle-treated a

115 ke morphology in the bronchoalveolar lavage (BAL) fluid, constituting up to 50% of the total cells in

116 secutive patients with ADA deficiency, 7 had BAL fluid containing periodic acid-Schiff-positive surfa

117 Prostaglandin D(2) and CXCL12 levels in BAL fluid correlated with decreased ILC2 numbers in bloo

118 culture-based diagnostic methods applied to BAL fluid could facilitate accurate diagnosis and more-t

119 us antigen or DNA in bronchoalveolar lavage (BAL) fluid could facilitate earlier diagnosis, thereby g

120 and identified some specific limitations of BAL fluid culture in the diagnosis of pneumonia.

121 report the first correlation of quantitative BAL fluid culture results with culture-independent evide

122 d positive histology or microscopy, positive BAL fluid culture, galactomannan optical index of 1 or m

123 on one of the seminal papers on quantitative BAL fluid culture, to explain why she believes that quan

124 o explain why she believes that quantitative BAL fluid cultures are the optimal strategy for VAP diag

125 -tocotrienol abated HDM-induced elevation of BAL fluid cytokine and chemokine levels, total reactive

126 exposing healthy alveolar macrophages to CF BAL fluid decreased bacterial killing, and this was reve

127 HBD2 is present in bronchoalveolar lavage (BAL) fluid during conditions associated with airway infl

128 F) levels in hamster bronchoalveolar lavage (BAL) fluid early after intranasal challenge.

129 imals showed reduced bronchoalveolar lavage (BAL) fluid eosinophil counts, reduced airway resistance

130 n favor of obtaining bronchoalveolar lavage (BAL) fluid for lymphocyte cellular analysis, and suggest

131 BAL fluid from 40% of patients with RA was positive for

132 studies, we characterized lung microbiota in BAL fluid from 68 patients with IPF.

133 Overall greater expression of cytokines in BAL fluid from African American patients as compared wit

134 cells were present at a higher frequency in BAL fluid from asthmatic patients compared with numbers

135 BAL fluid from asthmatic patients had increased TSLP but

136 BAL fluid from BMT mice at day 21 after infection contai

137 At day 5 after infection, the BAL fluid from C-EX (but not A-EX) mice had less IL-6, i

138 rominent neutrophil chemoattractant found in BAL fluid from individuals at the time of diagnosis of B

139 heightened inflammatory response detected in BAL fluid from infected pregnant mice was characterized

140 In vitro, BAL fluid from KGF-treated subjects inhibited pulmonary

141 d quantitative cultures in experiments using BAL fluid from neutropenic rabbits with experimentally i

142 These cells were not seen in the BAL fluid from normal animals or from Pneumocystis-infec

143 BAL fluid from patients living in a coccidioidomycosis-e

144 The sFasL in BAL fluid from patients with ALI was bioactive and prese

145 The concentration of L-ficolin in BAL fluid from patients with fungal infection was signif

146 No other priority toxicants were found in BAL fluid from the case patients or the comparator group

147 -1-infected subjects, compared with those in BAL fluid from the HIV-1-uninfected control subjects (me

148 of the beta -chemokine RANTES were found in BAL fluid from the human immunodeficiency virus (HIV)-1-

149 ages during infection, after incubation with BAL fluids from Pneumocystis-infected animals, or after

150 rformed on acellular bronchoalveolar lavage (BAL) fluid from 30 subjects infected with HIV with advan

151 We obtained bronchoalveolar lavage (BAL) fluid from 45 Malawian adults, to measure the conce

152 ve TH2/TH17 cells in bronchoalveolar lavage (BAL) fluid from asthmatic patients, examine their respon

153 2), secretory IgA in bronchoalveolar lavage (BAL) fluid from immunized mice, and Yersinia-specific CD

154 olin was measured in bronchoalveolar lavage (BAL) fluid from lung transplant recipients by enzyme-lin

155 vity is increased in bronchoalveolar lavage (BAL) fluid from patients with cystic fibrosis (CF).

156 tion of toxicants in bronchoalveolar-lavage (BAL) fluid from patients with EVALI can provide direct i

157 We screened bronchoalveolar lavage (BAL) fluids from COPD-E (chronic obstructive pulmonary d

158 presence of virus in bronchoalveolar lavage (BAL) fluids from patients during the first year after su

159 Bronchoalveolar lavage (BAL) fluids from Pneumocystis-infected animals were foun

160 cific PCRs were comparable in sensitivity to BAL fluid galactomannan (GM) assay.

161 er severity scores, mortality, and serum and BAL fluid galactomannan and 1,3-beta-d-glucan concentrat

162 erum 1,3-beta-d-glucan and galactomannan and BAL fluid galactomannan concentrations were observed in

163 nd SLPI (R2=0.29 and P=.001) correlated with BAL fluid HIV-1 load but not with plasma HIV-1 load.

164 asthma had neutrophilic asthma and increased BAL fluid IL-1alpha, IL-6, IL-8, granulocyte colony-stim

165 There was no increase in BAL fluid IL-4, IL-5, or IL-13 levels in patients with S

166 BAL fluid ILC2s from asthmatic patients with increased T

167 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic patients were resistant

168 Platelia EIA index cut-off > or =1.0 in the BAL fluid in a lung transplant recipient with a compatib

169 Neutrophil elastase activity in BAL fluid in early life was associated with early bronch

170 than culture in detection of A. fumigatus in BAL fluid in experimentally induced IPA (P+/-0.04).

171 l characteristics of bronchoalveolar lavage (BAL) fluid in acute pulmonary coccidioidomycosis have no

172 to culture-negative bronchoalveolar lavage (BAL) fluid in order to identify septate hyphae noted by

173 nnan (GM) testing of bronchoalveolar lavage (BAL) fluid in the diagnosis of invasive pulmonary asperg

174 IL-7 concentrations in BAL fluid increased significantly 48 h after segmental a

175 Field Effect genes were higher in cell-free BAL fluid, indicating they may be secreted by the TAMs.

176 summary, we found that bacterial culture of BAL fluid is largely effective in discriminating acute i

177 cells as measured in bronchoalveolar lavage (BAL) fluid is more moderate and similar to the depletion

178 Concomitantly, we phenotyped blood and BAL fluid leukocytes by flow and image cytometry, and me

179 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines, mediators, and epithel

180 injury increases the bronchoalveolar lavage (BAL) fluid levels of ATX protein 17-fold.

181 Following extraction from BAL fluids, levels of 25-hydroxy-vitamin D (25(OH)D) and

182 F-beta derived from parenchymal cells in the BAL fluid, lower nitrite levels, and reduced apoptosis,

183 Bronchoalveolar lavage (BAL) fluid, lung tissues, and whole blood were collected

184 sponses in blood and bronchoalveolar lavage (BAL) fluid lymphocytes compared with all other adjuvants

185 nged elevation of these chemokines in serial BAL fluid measurements predicted the development of CLAD

186 L fluid neutrophils strongly correlated with BAL fluid myeloperoxidase, IL-8, IL-1alpha, IL-6, granul

187 had positive galactomannan in serum (n = 4), BAL fluid (n = 16), or both (n = 4); three patients cult

188 r CYC therapy for active lung disease alters BAL fluid neutrophil and eosinophil counts and whether t

189 , bronchial basement membrane thickness, and BAL fluid neutrophil and lymphocyte counts but not eosin

190 BAL fluid neutrophil counts negatively and positively co

191 irteen of these 52 molecules correlated with BAL fluid neutrophil counts.

192 BAL fluid neutrophils strongly correlated with BAL fluid

193 CF but not disease control children harbored BAL fluid neutrophils with high exocytosis of primary gr

194 RVs were found in 61% (41% in BAL fluid/NPAs and 20% in NPAs only).

195 Vitamin E acetate was identified in BAL fluid obtained from 48 of 51 case patients (94%) in

196 T cells derived from bronchoalveolar lavage (BAL) fluid obtained during diagnostic bronchoscopy.

197 advocate the use of bronchoalveolar lavage (BAL) fluid obtained either bronchoscopically or by the u

198 0.008), free neutrophil elastase activity in BAL fluid (odds ratio, 3.02; 95% CI, 1.70 to 5.35; P<0.0

199 cognition of ESAT-6 and KatG in T cells from BAL fluid of 32/44 sarcoidosis subjects, compared to 1/2

200 was measured by tandem mass spectrometry in BAL fluid of CS- or elastase-exposed mice, and GCS was d

201 and release of IL-5, IL-9, and GM-CSF in the BAL fluid of mice, indicating an involvement of pro-eosi

202 observed in the peripheral blood, MLNs, and BAL fluid of SIV-infected RMs.

203 When polyamines were depleted from the BAL fluids of infected animals, the ability of these BAL

204 , these markers remained elevated within the BAL fluids of treated animals, which was consistent with

205 ) were determined in bronchoalveolar lavage (BAL) fluid of 10 asthmatics and 15 normal controls taken

206 e sputum, serum, and bronchoalveolar lavage (BAL) fluid of asthma patients and are known to correlate

207 (MLNs), jejunum, and bronchoalveolar lavage (BAL) fluid of healthy and SIV-infected rhesus macaques (

208 be isolated from the bronchoalveolar lavage (BAL) fluid of human lung transplant recipients.

209 The bronchoalveolar lavage (BAL) fluid of infected BHS also contained higher levels

210 is detectable in the bronchoalveolar lavage (BAL) fluid of patients with ALI, but the mechanisms cont

211 ting its presence in bronchoalveolar lavage (BAL) fluid of subjects with atopic asthma before and aft

212 s collected from the bronchoalveolar lavage (BAL) fluid of the animals.

213 f neutrophils in the bronchoalveolar lavage (BAL) fluid of the lungs.

214 galactomannan optical index of 1 or more in BAL fluid or 0.5 or more in serum.Measurements and Main

215 ahydrocannabinol (THC) or its metabolites in BAL fluid or had reported vaping THC products in the 90

216 creased TNF-alpha in bronchoalveolar lavage (BAL) fluid (p < .001).

217 mpared with those found in control subjects (BAL fluid, P < .001; biopsy specimen, P < .01); within t

218 BAL fluid pan-Aspergillus and species-specific PCRs were

219 Confirming previous results, BAL fluid PGD(2) levels were highest in patients with se

220 yte phenotyping was performed, and acellular BAL fluid, plasma HIV RNA load, and BAL cell and periphe

221 eases in HGF concentrations were detected in BAL fluid, plasma, and fibroblast culture medium from Ca

222 testing of serum and bronchoalveolar lavage (BAL) fluid pools from positive and negative patient spec

223 generated from neutrophil myeloperoxidase in BAL fluid promoted aggregation of sFasL in vitro and in

224 Bronchoalveolar lavage (BAL) fluid prostaglandin D(2)(PGD(2)) levels are increas

225 These results show that pre-HCT BAL fluid RV positivity was a predictor for allo-LSs.

226 redicted asthma status with 75% accuracy for BAL fluid samples and 80% accuracy for EB samples.

227 Treatment of both the crude BAL fluid samples and P. carinii lysate and the 200 mM m

228 Alveolar macrophages treated with these BAL fluid samples became defective in phagocytosis but r

229 We tested archived BAL fluid samples from 132 HCT recipients with CMV pneum

230 he galactomannan (GM) test were used with 32 BAL fluid samples from 32 patients at risk of IPA.

231 A total of 78 BAL fluid samples from 78 patients at risk for IPA (74 s

232 BAL fluid samples from consecutive lung transplant recip

233 test, and an Aspergillus PCR assay by using BAL fluid samples from immunocompromised patients.

234 32/33) of the serum and 78.8% (26/33) of the BAL fluid samples from infected animals were in agreemen

235 ytosis but remained normal when treated with BAL fluid samples from noninfected or Toxoplasma gondii-

236 The suppressive activity of the BAL fluid samples from P. carinii-infected rats on phago

237 , CTSS activity was detectable in 100% of CF BAL fluid samples from patients without Pseudomonas aeru

238 Also, the CMV viral load in BAL fluid samples increased along with immunoreactivity

239 In BAL fluid samples the dominant genera were Cladosporium,

240 re size of 0.45 microm but was lost when the BAL fluid samples were digested with proteases such as t

241 Calcofluor staining and culture of all BAL fluid samples were negative for fungal infection.

242 d rats on phagocytosis was retained when the BAL fluid samples were passed through a filter with a po

243 Among 23 BAL fluid samples, all were positive by qPCR, while 22 w

244 methods for diagnosing the disease by using BAL fluid samples.

245 gene expression using microarray analysis of BAL fluid samples.

246 tests are useful for the diagnosis of IPA in BAL fluid samples.

247 omannan detection in bronchoalveolar lavage (BAL) fluid samples (GM test) is currently considered the

248 tection of Pc DNA in bronchoalveolar lavage (BAL) fluid samples by use of PCR.

249 nalysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observational prospective cohor

250 Bronchoalveolar lavage (BAL) fluid samples were analyzed using cytokine array te

251 Serial plasma and bronchoalveolar lavage (BAL) fluid samples were examined for changes in antibody

252 lung homogenates and bronchoalveolar lavage (BAL) fluid showed that LPS-induced lung inflammation and

253 each batch were prepared by spiking a pooled BAL fluid specimen with a known dilution of Coccidioides

254 se, 72 paired NP and bronchoalveolar lavage (BAL) fluid specimen sets, mostly from transplant recipie

255 Bacteria were identified in 44 of 46 (95.7%) BAL fluid specimens by culture-independent sequencing, s

256 In 19 sets (26.4%), the NP and BAL fluid specimens were both positive for an RV; in 3 s

257 In a retrospective case series, 202 BAL fluid specimens were collected at the Johns Hopkins

258 .2%), the NP specimens were positive but the BAL fluid specimens were negative; and in 3 other sets,

259 sets, the NP specimens were negative but the BAL fluid specimens were positive.

260 In BAL fluid specimens with no culture growth, the amount o

261 iratory pathogens in bronchoalveolar lavage (BAL) fluid specimens from subjects with suspected respir

262 clinically obtained bronchoalveolar lavage (BAL) fluid specimens from symptomatic and asymptomatic l

263 d a blinded study of bronchoalveolar lavage (BAL) fluid specimens from well-characterized models of I

264 sites, including bronchial alveolar lavage (BAL) fluid; sputum and lung tissue samples; and pleural

265 An analysis of the BAL fluid suggested that Hsp70 is critically required fo

266 = 15) of children with STRA with detectable BAL fluid T(H)2 cytokines had significantly lower lung f

267 r lung function than those with undetectable BAL fluid T(H)2 cytokines.

268 On the basis of the dominance of BAL fluid TH2 or TH2/TH17 cells, we identified 3 subgrou

269 ds of infected animals, the ability of these BAL fluids to induce apoptosis was lost.

270 ralizing activity in vitro and inhibition of BAL fluid total, eosinophil, and neutrophil counts in HD

271 BAL fluid TSLP levels correlated (r = 0.74) with steroid

272 nses to mycobacterial antigens in cells from BAL fluid used for sarcoidosis diagnosis suggests a stro

273 Memory CCR5(+) TH1 cells were enriched in BAL fluid versus blood, and pathogenic respiratory virus

274 ifungal therapy to the time of collection of BAL fluid was 6 days.

275 RV positivity in BAL fluid was a predictor for allo-LSs (hazard ratio, 3.

276 earch for bacteria, mycobacteia and fungi in BAL fluid was negative.

277 Fiberoptic bronchoscopy with collection of BAL fluid was performed on seven patients with GC-sensit

278 inducing activity of the polyamine-depleted BAL fluids was restored when polyamines were added back.

279 Bronchoalveolar lavage (BAL) fluid was assessed for total and differential cell

280 A Gram stain of bronchoalveolar lavage (BAL) fluid was notable for many Gram-positive cocci (GPC

281 Using the BAL fluid, we performed isotope dilution mass spectromet

282 Stored serum and BAL fluid were available from 194 nonneutropenic patient

283 ARP results, and all microbiologic data from BAL fluid were collected.

284 /antiproteinase, and endotoxin levels in the BAL fluid were compared.

285 Epithelial cells and BAL fluid were evaluated for HPGDS (quantitative real-ti

286 cells was elevated and inflammatory cells in BAL fluid were increased.

287 -Aspergillus and species-specific qPCRs with BAL fluid were more sensitive than culture for detection

288 The fungal burdens in BAL fluid were more variable and not indicative of a pro

289 rplasia, and IL-4, IL-5, and IL-13 levels in BAL fluid were significantly reduced in fB-/- mice compa

290 t survival rates in yeast media, saline, and BAL fluid were similar to those of the wild type; howeve

291 BAL fluids were collected from 51 patients with EVALI in

292 hough the absolute numbers of eosinophils in BAL fluids were similar between the strains, very few CD

293 nflammatory cells in bronchoalveolar lavage (BAL) fluid were assessed by flow cytometry.

294 culation), blood and bronchoalveolar lavage (BAL) fluid were collected from infected and uninfected a

295 kocytes in blood and bronchoalveolar lavage (BAL) fluid were compared for expression of FoxP3 by flow

296 factor alpha in the bronchoalveolar lavage (BAL) fluid were lower in C-EX and A-EX than in NON-EX mi

297 pearance of proinflammatory cytokines in the BAL fluid, whereas the inflammatory and cytokine respons

298 evels of transforming growth factor beta1 in BAL fluid, while simultaneously reducing the number of i

299 and R. oryzae GHE in bronchoalveolar lavage (BAL) fluid with either extraction method was 1 GHE/ml.

300 A. niger, culture and PCR amplification from BAL fluid yielded similar sensitivities for untreated an