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1 BCV Drep RNA that lacked a poly(A) tail did not replicat
2 BCV is a prototype for the coronaviruses that express an
12 chi Calossi Versaci front index (BCV(f)) and BCV back index (BCV(b)), root mean square of front and b
14 ia monocytogenes, a Gram-positive bacterium, BCV rupture by Gram-negative pathogens such as Shigella
16 g superior vena cava (SVC), brachiocephalic (BCV), subclavian (SCV) and internal jugular vein (IJV).
17 s used to study the effect of brincidofovir (BCV) in normal and immune-deficient (nu/nu) mice infecte
18 stchallenge administration of brincidofovir (BCV, CMX001) was studied in normal and immune-deficient
19 rucella replication in the ER is followed by BCV conversion into a compartment with autophagic featur
21 igenic relationship with bovine coronavirus (BCV) and porcine hemagglutinating encephalomyelitis viru
24 spike protein subunit of bovine coronavirus (BCV) reacted with the virus in formalin-fixed intestines
26 eplaced by the 3' UTR of bovine coronavirus (BCV), which diverges overall by 31% in nucleotide sequen
28 ine (BCNU), cyclophosphamide, and etoposide (BCV) preparative regimen (27 patients) and an ABMT at 16
30 a published nucleoprotein gene sequence for BCV (Mebus isolate), we arbitrarily designed two primers
32 aci front index (BCV(f)) and BCV back index (BCV(b)), root mean square of front and back corneal surf
33 rface, Baiocchi Calossi Versaci front index (BCV(f)) and BCV back index (BCV(b)), root mean square of
36 Postchallenge administration of 20 mg/kg BCV rescued normal and immune-deficient mice partially r
37 oVEMPs: to BCV with the B81 on the mastoid, BCV with the B81 on the forehead, and BCV with the Mini-
41 n IpaH7.8 promotes Rab13 retention on moving BCV membrane remnants, thereby facilitating membrane unc
43 an abundant lipid of the luminal leaflet of BCV membranes, and normally absent from the cytosol, bec
45 sequence of NC99 and published sequences of BCV (Mebus and F15 strains) and human coronavirus (strai
46 of both ECV isolates were similar to that of BCV as determined by sodium dodecyl sulfate-polyacrylami
48 ectron microscopy (CLEM), we discovered that BCV rupture proceeds through two distinct successive sta
51 host-pathogen interactions that occur at the BCV membrane is limited by the difficulty in purifying m
52 CUAAAC element which occurs only once in the BCV 5' untranslated region was either deleted or complet
53 substitution of a particular portion of the BCV 3' UTR for the corresponding region of the MHV 3' UT
54 acuoles (BCVs) and subsequent rupture of the BCV membrane, thereby exposing to the cytosol intralumin
55 anslocated into the cytosolic leaflet of the BCV, invariably followed by cytosolic exposure of glycan
58 Specific host protein interactions with the BCV 3' UTR [287 nt plus poly(A) tail] were identified us
59 ely related genetically and antigenically to BCV and will be a new member of antigenic group 2 of the
61 he B81 transducer on the mastoid; oVEMPs: to BCV with the B81 on the mastoid, BCV with the B81 on the
63 by forming the Brucella-containing vacuole (BCV), which traffics from the endocytic compartment to t
66 with 10(4) PFU of IHD-J-Luc and treated with BCV postchallenge survived the infection, cleared the vi