ライフサイエンスコーパス: BHA

1 BHA administration also inhibited estradiol- or estrone-

2 BHA decreased yeast resistance to oxidative treatments a

3 BHA inhibits de novo interferon regulator factor (IRF)-1

4 BHA presented cell damaging effects even in the absence

5 BHA showed the highest OS, with p-anisidine, PV and inhi

6 BHA shows extraordinary reactivity toward the triester d

7 BHA treatment significantly attenuated RSV-induced lung

8 e decreased by 30-60% in animals fed a 0.75% BHA diet for 18 days prior to the injection of estrogen.

9 In conclusion, feeding a 0.75% BHA-supplemented diet to female CD-1 mice for 2-3 weeks

10 Feeding a 0.75% BHA-supplemented diet to ovariectomized CD-1 mice for 18

11 al, environment and the benzhydroxamic acid (BHA) substrate binding pocket.

12 complexes with INH and benzohydroxamic acid (BHA) are reported.

13 hols catalyzed by Hf(IV)-bishydroxamic acid (BHA) complexes is described.

14 ession of Broadband High-frequency Activity (BHA; 80-180 Hz), both locked to fixation onset supportin

15 lase inhibitor, aminotriazole, also affected BHA- and tBHQ-stimulated ERK2 activity but not JNK1, ind

16 ave been designed as alternatives to BHT and BHA antioxidants.

17 imple LC-MS/MS method for estimating BHT and BHA levels in Salmo salar, butter, and milk.

18 The presence of lycopene and BHA increases the total phenolic compounds in the enrich

19 c compounds in the enriched egg powders, and BHA exhibits the most antioxidant activity, as quantifie

20 layed good detection limits of both TBHQ and BHA (0.73 and 5.6 nM for TBHQ and BHA, respectively).

21 he nanosensor successfully detected TBHQ and BHA in food samples proved its potential for the develop

22 vily on synthetic antioxidants like TBHQ and BHA to prevent spoilage.

23 h TBHQ and BHA (0.73 and 5.6 nM for TBHQ and BHA, respectively).

24 widely used, the butylated hydroxyl anisole (BHA).

25 se kinases was abrogated by the antioxidants BHA and vitamin C.

26 cerola extracts with synthetic antioxidants (BHA and BHT).

27 phy (HPLC) to detect synthetic antioxidants; BHA, BHT, and TBHQ in the deep-UV region below 300 nm.

28 justifying the Briggs-Haldane approximation (BHA).

29 EXT was not as effective as BHA or DHM in soybean oil.

30 tion from digestion was extremely stable, as BHA-inhibitor complexes could be incubated for 24 h in l

31 igh-shattering weed groups, Blackhull Awned (BHA) and Spanish Weedy Rice (SWR), respectively.

32 , these genes may hint at a mechanism behind BHA's evident, but minimally detrimental effect on the g

33 mple molecular backbone of benzohydroxamate (BHA) and on the more complex structure of the widely use

34 d to 7 synthetic antioxidants including BHT, BHA, TBHQ and PG with regard to their ability to protect

35 2, is responsible for the ERK2 activation by BHA and tBHQ, whereas the JNK1 activation may require a

36 E attenuated ERK2 but not JNK1 activation by BHA and tBHQ.

37 Activation of ERK2 by BHA was rapid and transient, whereas the JNK1 activation

38 ng a kinetics different from that induced by BHA.

39 ss and body weight loss were also reduced by BHA treatment, which inhibited neutrophil recruitment to

40 tment of c-Jun to this proximal AP-1 site by BHA was confirmed by chromatin immunoprecipitation analy

41 rmally when BHA was supplemented in trans by BHA-expressing cells.

42 In contrast, BHA administration had little or no effect on the liver

43 liver or Hepa1c1c7 cells treated with either BHA or acrolein.

44 e study showed that at POC/MWCNTs electrodes BHA oxidation occurred at 0.27 V.

45 s alphamin values of 1.8 x 10(-5) cm(-1) for BHA, 2.8 x 10(-5) cm(-1) for BHT, and 1.9 x 10(-5) cm(-1

46 Furthermore, BHA and tBHQ activation of ERK2 was strongly inhibited b

47 Furthermore, BHA-induced apoptosis appeared to be independent of form

48 nts decreased in the order ETX>TR>PG>AA>TBHQ>BHA.

49 , purified HA trimers (bromelain-cleaved HA [BHA]) are used to examine the properties and binding cha

50 Influenza B virus hemagglutinin (BHA) contains a predicted cytoplasmic tail of 10 amino a

51 ith the antioxidant butylated hydroxyanisol (BHA), as well a panel of chemically unrelated antioxidan

52 n of 0.75% 2(3)-tert-butyl-4-hydroxyanisole (BHA) in AIN-76A diet to female CD-1 mice for 3 weeks inc

53 similar to that of butylated hydroxyanisole (BHA) and higher than that of other synthetic antioxidant

54 ytoluene (BHT) and butylated hydroxyanisole (BHA) are two commonly used antioxidants with potential h

55 e determination of butylated hydroxyanisole (BHA) by electropolymerization of O-cresolphthalein compl

56 Butylated hydroxyanisole (BHA) continues to raise consumer concerns.

57 O-induced ROS with butylated hydroxyanisole (BHA) does not affect Nrf2 activation or cell death.

58 enolic antioxidant butylated hydroxyanisole (BHA) is a commonly used food preservative with broad bio

59 quinone (TBHQ) and butylated hydroxyanisole (BHA) was designed.

60 Butylated hydroxyanisole (BHA), a commonly used food preservative, is reported to

61 rally administered butylated hydroxyanisole (BHA), an antioxidant.

62 ed the antioxidant butylated hydroxyanisole (BHA), and others were given daily intraperitoneal inject

63 olic antioxidants, butylated hydroxyanisole (BHA), butylated hydroxytoluene (BHT) and tert-butyl hydr

64 Butylated hydroxyanisole (BHA), butylated hydroxytoluene (BHT), and tert-butylhydr

65 ives [acetic acid, butylated hydroxyanisole (BHA), ethylenediaminetetraacetic acid (EDTA), and sodium

66 rbamate (PDTC) and butylated hydroxyanisole (BHA), indicating that the generation of free oxygen radi

67 th the antioxidant butylated hydroxyanisole (BHA).

68 hin, lycopene, and butylated hydroxyanisole (BHA)] for conjugated dienes (during a 90-day period) and

69 nolic antioxidant (butylated hydroxyanisole: BHA), a common ingredient in food additives and preserva

70 acrolein p.o.) or butylated hydroxylanisole (BHA) (0.45% in the diet) and of cytosolic NAD(+)-depende

71 aken together, our results indicate that (i) BHA and its metabolite tBHQ differentially regulate MAPK

72 for 24 h in low pH with almost no change in BHA structure.

73 rial permeability transition pore, inhibited BHA-induced loss of Deltapsi(m), cytochrome c release, c

74 Instead, BHA and tBHQ substantially reduced the amount of intrace

75 ion but acquired additional mutations in its BHA, neuraminidase (NA), and M1 proteins.

76 onstrating the flexible nature of the Hf(IV)-BHA system.

77 A dose of 0.35 g/L BHA elicited no statistically significant changes in the

78 duced particles containing dramatically less BHA.

79 erivatives from 2-tert-butyl-4-methylphenol, BHA, creosol, isoeugenol and di-o-propenyl p-cresol, few

80 6-well format after O-benzylhydroxylamine (O-BHA) derivatization under aqueous conditions.

81 is effect is mediated through the ability of BHA to inhibit RSV-induced interferon regulatory factor

82 derlying these diverse biological actions of BHA is thus of great importance.

83 is known about the intracellular activity of BHA, these genes may hint at a mechanism behind BHA's ev

84 Analysis of BHA after photoaffinity analog binding and UV cross-link

85 ytoplasmic tail abrogated the association of BHA with Triton X-100-insoluble lipid rafts.

86 se results indicate that the cytotoxicity of BHA is due to the induction of apoptosis that is mediate

87 as shown a linear range for the detection of BHA from 0.33 uM to 110 uM with the detection limit of 0

88 confirm the accurate, reliable detection of BHA in food products in the future.

89 Amperometric determination of BHA was also done using chronoamperometric techniques an

90 indicate that the cytoplasmic tail domain of BHA is important for efficient incorporation of BHA into

91 vivo model was used to assess the effect of BHA on microbial communities from 24 donors, aged infant

92 is important for efficient incorporation of BHA into virions and tight lipid raft association.

93 Indeed, direct incubation of BHA with isolated mitochondria triggered cytochrome c re

94 e LSV response (anodic peak current, Ipa) of BHA and BHT by 2- and 20-times, respectively.

95 Although the levels of BHA cell surface expression were indistinguishable betwe

96 r the multiplex detection and measurement of BHA, BHT, and TBHQ levels in complex food samples using

97 significance to understand the mechanism of BHA-induced toxicity.

98 A major metabolite of BHA, tert-butylhydroquinone (tBHQ), also activated ERK2

99 estradiol and estrone by liver microsomes of BHA-treated animals as determined by substrate disappear

100 electrochemical performance in oxidation of BHA and the study showed that at POC/MWCNTs electrodes B

101 kinetic analysis showed that replication of BHA cytoplasmic tailless viruses could be improved by co

102 However, the use of BHA as a chemopreventive agent against cancer in human h

103 ing revealed predictive pY sites for HFD- or BHA-induced metabolite changes.

104 Addition of either INH or BHA to KatG induces only minor changes in the resonance

105 Ps at 121 degrees C exhibited higher AA than BHA.

106 DHM was more potent than BHA in preventing soybean oil oxidation.

107 MRPs showed higher PRS and RP than BHA.

108 logy during the de-domestication of SWR than BHA, fewer genes exhibited a comparable AZ-region exclus

109 Here we demonstrate that BHA is capable of activating distinct mitogen-activated

110 has been challenged by the observation that BHA may exert toxic effect in some tissues of animals.

111 Here, we report that BHA induces apoptosis in freshly isolated rat hepatocyte

112 BHA compared with controls, suggesting that BHA-induced expression is independent of nuclear factor

113 and substrate concentrations where both the BHA and the QEA are invalid and allows us to define prec

114 ectomy, whereas Nemo(Deltahepa) mice fed the BHA diet were protected from carcinogenesis.

115 a recombinant influenza B virus lacking the BHA cytoplasmic tail domain.

116 ly, long-term culture of a virus lacking the BHA cytoplasmic tail in Madin-Darby canine kidney (MDCK)

117 in that overlaps the validity domains of the BHA and the QEA and only slightly extends them.

118 SA) overlaps and extends the validity of the BHA and the QEA, and that it is at least roughly valid f

119 assignment strategy is then extended to the BHA:HRPC-CN complex.

120 nding pocket and suggest a model whereby the BHA is able to undergo a partial, reversible structural

121 of enzyme-to-substrate ratios for which the BHA is invalid, including the extreme of enzyme excess.

122 Cross-linking of the photoaffinity analog to BHA under neutral (native) pH conditions identified a st

123 eneration of H2O2 after exposure of cells to BHA or tBHQ using a H2O2-sensitive fluorescent probe, 2'

124 re shown to be in primary dipolar contact to BHA.

125 by different pulse voltammetry (DPV) towards BHA with a low limit of detection (LOD) and sensitivity

126 real samples (corn oil and noodles) towards BHA.

127 The addition of Trolox, BHA, and propyl gallate had no significant effect on fur

128 tinguishable between truncated and wild-type BHA, the BHATail(-) virus produced particles containing

129 at of wild-type virus but grew normally when BHA was supplemented in trans by BHA-expressing cells.

130 a mass-to-charge ratio (m/z) of 219.4, while BHA was detected using multiple-reaction monitoring, wit

131 icetin (DHM) were analysed and compared with BHA in two model systems, soybean oil and cooked ground

132 Treatment of hepatocytes with BHA also induced loss of mitochondrial transmembrane pot

133 rf2(+)/(+) and Nrf2(-)/(-) mice treated with BHA compared with controls, suggesting that BHA-induced

134 ost significantly affected by treatment with BHA across age groups are those involved in lipopolysacc