ライフサイエンスコーパス: BPA

1 BPA and BPS altered the expression of an identical set o

2 BPA and BPS did not degrade (1 h; 100 degrees C) in wate

3 BPA and GEN exposed females had increased number of meta

4 BPA and phthalate metabolites were not associated with e

5 BPA and TBBPA both interfere with skeletal muscle functi

6 BPA bioaccessibility was evaluated in six positive sampl

7 BPA disrupts endocrine pathways in fish, but the long-te

8 BPA exposure down-regulated WNT2 expression, and elevate

9 BPA exposure impeded the interaction between the human c

10 BPA exposure increased Nrf2 binding to a putative antiox

11 BPA has been associated with numerous adverse health out

12 BPA increased Na(v)-mediated ramp currents elicited with

13 BPA may have toxic effects on the female reproductive sy

14 BPA or vehicle-treated rats from an FDA facility were us

15 BPA showed the lowest concentrations (8.3 pg/g f.w.), fa

16 BPA was detected in 17 out of 40 samples (42.5%); in som

17 BPA was not associated with most BASC-2 scales; however,

18 BPA was not associated with WPPSI-III scores; child sex

19 BPA was positively associated with the relationship prob

20 bited statistically significant (p < 0.0001) BPA removal (40% and 46%, respectively) over approximate

21 Significantly higher (p < 0.005) BPA accumulation in roots than shoots and nonsignificant

22 Five of the 6 BPA activities significantly (likelihood ratio rest, P <

23 Conclusions Delivering a BPA through the electronic health record recommending to

24 In the study group, a BPA alerted providers of the diagnostic results suggesti

25 In the study group, a BPA alerted providers of the diagnostic results suggesti

26 were randomized to receive or not receive a BPA recommending consideration for ICD implantation.

27 The thermal degradation of bisphenol A (BPA) and bisphenol S (BPS) was investigated in water and

28 vide a definitive evaluation of bisphenol A (BPA) and explain disparities between traditional regulat

29 f serially assessed exposure to bisphenol A (BPA) and phthalates on measures of kidney function, tubu

30 Phthalates and bisphenol A (BPA) are endocrine disruptors, and previous research has

31 Phthalates and bisphenol A (BPA) are used in some personal care products (PCPs) and

32 l sensor to detect and quantify Bisphenol A (BPA) contamination.

33 nol S (BPS) as a substitute for bisphenol A (BPA) could actually increase the risk of endocrine disru

34 ealed that prenatal exposure to bisphenol A (BPA) disrupted the transcriptome profiles of genes in th

35 A positive association between Bisphenol A (BPA) exposure and coronary heart disease has been shown,

36 Early-life exposure to bisphenol A (BPA) has been implicated to play a role in the developme

37 Bisphenol A (BPA) has been linked with pediatric asthma development a

38 vironmental disrupting chemical bisphenol A (BPA) has estrogenic activity, but its implications in pa

39 Pressures to ban bisphenol A (BPA) has led to the use of alternate chemicals such as B

40 Although prenatal exposure to bisphenol A (BPA) has recently been associated with the ASD risk, whe

41 on an innovative heterogeneous bisphenol A (BPA) immunoassay based on an electrolyte-gated organic f

42 Bisphenol A (BPA) is an endocrine disruptor that affects fetal growth

43 Bisphenol A (BPA) is an environmental endocrine disruptor and has bee

44 Bisphenol A (BPA) is known to be biologically active in experimental

45 The plastic monomer bisphenol A (BPA) is one of the highest production volume chemicals i

46 vironmental endocrine disruptor bisphenol A (BPA) is ubiquitous and associated with the increased ris

47 Bisphenol A (BPA) is widely recognized being an endocrine disrupter a

48 Laboratory studies show that bisphenol A (BPA) leaches from bisphenol A-glycidyl methacrylate (bis

49 The lowest concentration of bisphenol A (BPA) that induces a response was ~0.1 mg/L (detected in

50 In this study, bisphenol A (BPA) was selected as a model compound.

51 e endocrine disrupting chemical bisphenol A (BPA) while exerting estrogenic effects of comparable pot

52 Bisphenol A (BPA), a chemical incorporated into plastics and resins,

53 gests that prenatal exposure to bisphenol A (BPA), a ubiquitous endocrine-disrupting chemical, is ass

54 increasingly used to substitute bisphenol A (BPA), a widespread environmental endocrine disruptor and

55 nol F diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF) and bisphenol

56 ignin-derivable alternatives to bisphenol A (BPA), but limited bioassay data are available on their e

57 in industrial chemicals, e.g., bisphenol A (BPA), present in plastics and other products that are pr

58 gers inhibited the oxidation of bisphenol A (BPA), suggesting the possible involvement of ROSs; howev

59 tudies to assess the effects of bisphenol A (BPA).

60 early-life exposure to low-dose bisphenol A (BPA).

61 e-disrupting chemicals, such as bisphenol A (BPA).

62 In contrast, Bisphenol-A (BPA) elicited a non-monotonic response.

63 Bisphenol-A (BPA), a potent endocrine disrupting compound, is a synth

64 er of this family of compounds, bisphenol-A (BPA), when spiked (at 1 mug.g(-1) concentration) in the

65 sequencing in d-200 dorsal prostates across BPA doses.

66 Additionally, BPA and TNF-alpha levels in cord blood were inversely as

67 irect electron transfer between the adsorbed BPA and complexed PMS as the mechanism.

68 ealth record-based "best practice advisory" (BPA) that prompted primary care providers to perform HCV

69 ectronic medical record best practice alert (BPA) based on procalcitonin and respiratory polymerase c

70 tigated the impact of a best practice alert (BPA) through the electronic health records on the rates

71 lectronic health record best practice alert [BPA], and patient solicitation), evaluated hepatitis C v

72 Although BPA and phthalate metabolites were not associated with c

73 10 genes shared only between the BPA(25) and BPA(250) groups, with 115 of them predicted to be regula

74 or phosphoric acid (37% PA) to yield BCA and BPA etchants respectively.

75 significant differences between control and BPA dose groups at PND21, whereas at PND90 there were si

76 d among 80 pregnant women and 55 infants and BPA and phthalate concentrations in multiple maternal an

77 lysis, we reported that DEHP metabolites and BPA were significantly associated with an approximate 20

78 We aimed to investigate urinary NP and BPA levels in relation to biomarkers of oxidative/nitrat

79 tions of four phthalates, seven parabens and BPA were determined in forty-two milk products.

80 sodilators or balloon pulmonary angioplasty (BPA).

81 causes almost identical placental effects as BPA.

82 ed to the use of alternate chemicals such as BPA analogues bisphenol S (BPS) and bisphenol AF (BPAF)

83 ing a bead-based opsonic phagocytosis assay (BPA).

84 used PA gel yielded higher SBS than the BCA/BPA etchants, it exhibited greater adhesive remnants wit

85 al variations of the benzylphenoxyacetamide (BPA) molecular skeleton were explored as a viable starti

86 The association between BPA and BRIEF-P scores was modified by child sex (BPAxse

87 expressed, although the association between BPA-responsive genes and AD-related genes has not been t

88 ed significant positive associations between BPA and fat weight [SMD=0.67 (95% CI: 0.53, 0.81)], trig

89 ved consistent positive associations between BPA exposure and measures of asthma morbidity.

90 The observed associations between BPA, MnBP, and behavior in boys are consistent with prev

91 sure and 8-OHdG and 8-NO2Gua levels, between BPA and 8-isoPF2alpha levels, and between maternal CRP l

92 lorophyll (p > 0.19), and peroxidase between BPA-treated and untreated plants indicates substantial B

93 sing statistical models, 25 mug/kg BW/d BPA [BPA(25)], or 250 mug/kg BW/d BPA [BPA(250)] exerted effe

94 BW/d BPA [BPA(25)], or 250 mug/kg BW/d BPA [BPA(250)] exerted effects similar to that of EE2 at 0.5

95 f BPA from thermal paper receipts occurs but BPA pharmacokinetics following dermal exposure is not un

96 ves were primary sites for ERE activation by BPA and MBP, and transcriptomic analysis of microdissect

97 urrents of Na(v) channels, were unchanged by BPA.

98 etween ERE activation in the heart valves by BPA's reactive metabolite MBP and the development of val

99 Upon PMS addition into one cell, BPA was quickly oxidized in the other cell, confirming t

100 For some cells, BPA caused rapid Ca(2+) transient loss without marked ch

101 Regulatory Insights on Bisphenol-A (CLARITY-BPA) is a rare collaboration of guideline-compliant (cor

102 ulatory Insights on Toxicity of BPA (CLARITY-BPA; henceforth CLARITY), was launched by three US agenc

103 day 6 up to 1 y old according to the CLARITY-BPA consortium protocol.

104 tors, and urinary creatinine concentrations, BPA, but not BPF or BPS, was significantly associated wi

105 In conclusion, BPA and pulmonary vasodilators both improve 6MWD and hem

106 on, using statistical models, 25 mug/kg BW/d BPA [BPA(25)], or 250 mug/kg BW/d BPA [BPA(250)] exerted

107 ug/kg BW/d BPA [BPA(25)], or 250 mug/kg BW/d BPA [BPA(250)] exerted effects similar to that of EE2 at

108 CD-1 mice exposed to 4 to 40,000 mug/kg/day BPA or 0.02 to 2 mug/kg/day EE from conception until 12-

109 The GOP system successfully degraded BPA in both surface water and hypersaline shale gas-prod

110 s, relative to BPA and one petroleum-derived BPA analogue (bisphenol F, BPF), and the incorporation o

111 e the pharmacokinetics of dermal and dietary BPA exposure, six male participants handled simulated re

112 Compared to dietary BPA exposure, dermal absorption of BPA leads to prolonge

113 The bis-difluoromethylated BPA was conveniently detected on the organic layers from

114 n limit (MDL) for the bis-difluoromethylated BPA was determined to be 0.01 mug.mL(-1).

115 ic chemicals was further examined to disrupt BPA at various human-relevant levels.

116 Lifelong exposure of rats to low-dose BPA at 25 and 250 mug/kg BW/d altered the estrous cycle

117 ta provide compelling evidence that low-dose BPA exposure induces marked adverse effects.

118 d dental materials and that chronic low-dose BPA exposure may adversely affect child health, strategi

119 to carcinoma in rats given neonatal low-dose BPA with adult T+E but not in rats given adult T+E alone

120 he exposure also disrupted a unique low-dose BPA-gene signature with predictive value for survival ou

121 tions of 6 chemical exposure measures (i.e., BPA, PA, and 4 phthalate metabolite groups) and clinical

122 rved evidence of sexually dimorphic effects; BPA concentrations were associated with increased odds o

123 To satisfy the increasing need to ensure "BPA-free" articles, a liquid chromatography-tandem mass

124 fter the year 2011, which was when the first BPA restrictions became effective.

125 f DEHP metabolites and 1.22 (1.01, 1.47) for BPA] at median 10 wk and 26 wk, respectively.

126 underscoring switchgrass's effectiveness for BPA removal.

127 orst-case (10x ) maximum fetal exposures for BPA, or equivalent concentrations of its metabolite MBP,

128 ntrations of 1.0, 1.0, 0.10, and 50 mg/L for BPA, BPF, BPAF, and BPS, respectively.

129 To investigate its potential for BPA removal, two United States native switchgrass variet

130 or has an excellent wide detection range for BPA from 0.1 to 150 ppm with LODs of 0.02 ppm.

131 nol S (BPS), which have been substituted for BPA in some consumer products, have also shown endocrine

132 s on average about 250 times higher than for BPA for an equal oral molar dose of the two compounds.

133 ve lower estimated pKa values than those for BPA.

134 oning were measured at several pH values for BPA, BPAF, and BPS.

135 lateral prostates despite undetectable free BPA 1 hr postexposure.

136 The rank order for toxicity was BPAF > BPA > BPF > BPS.

137 The rank order for estrogenicity was BPAF > BPA = BPF > BPS.

138 High BPA responses of nMSP3K1, GLURP-R2, MSP23D7, MSP119k, an

139 Higher BPA, BPF, and BPS concentrations were observed in obese

140 that leads to protein degradation at higher BPA concentrations.

141 t migration pattern groups were highlighted: BPA and Zn (and Pb to a lesser extent) showed similar mi

142 To understand how BPA and the reputedly more inert bisphenol S (BPS) affec

143 [95% CI =1.28-3.51]) per 10-fold increase in BPA concentration.

144 C dopamine immunoreactivity was increased in BPA- and BPS-exposed placentas.

145 Children experience short-term increases in BPA from dental treatment.

146 inants in different food matrices, including BPA, BPS, bis(2-ethylhexyl) adipate, dibutyl adipate, he

147 ning relevant levels of BPA (isotope-labeled BPA-d16) for 5 min, followed by hand-washing 2 h later.

148 d BPAP (1.6 ng L(-1)), while bottles leached BPA (<222 ng L(-1)) and BPF, BPE, BPB and BPZ (1.1-4.6 n

149 Cans leached BPA (<5865 ng L(-1)), three BPF isomers (8.2-1286 ng L(-

150 Nevertheless, Mn oxides may limit BPA migration in near-surface environments and have pote

151 t differences between control and the lowest BPA dose and between control and the lowest EE2 dose in

152 The lowest BPA dose initiated maximal hormonal carcinogenesis in la

153 dence in the stop-dose animals at the lowest BPA dose tested.

154 ndings from this study suggest that maternal BPA exposure may increase AD risk in offspring by dysreg

155 in the hippocampus and suggest that maternal BPA exposure may increase ASD susceptibility by dysregul

156 We measured BPA concentration in urine samples collected at approxim

157 In pregnant mice, BPA-exposed mice exhibited preeclampsia-like features in

158 ffect child health, strategies that minimize BPA exposure could potentially improve child health.

159 sal root ganglia (DRG) nociceptors with 1 nM BPA increases the frequency of action potential firing.

160 Patients belonging to the BPA and no BPA groups of providers were followed to the end points

161 (n=997 patients) and 102 providers in the no BPA (n=909 patients) groups.

162 ronic PND21 specimens displayed nonmonotonic BPA effects, with a breaking point between the 25 and 25

163 ss are a function of prenatal exposure to NP/BPA and inflammation in 241 mother-fetus pairs.

164 Dermal absorption of BPA from thermal paper receipts occurs but BPA pharmacok

165 o dietary BPA exposure, dermal absorption of BPA leads to prolonged exposure and may lead to higher p

166 curonide (BPSG) and gavage administration of BPA and BPS.

167 nd urine after intravenous administration of BPA, BPS, and BPS glucuronide (BPSG) and gavage administ

168 ereas there were significant associations of BPA exposure with general and abdominal obesity, BPF or

169 The TK behavior of BPA and BPS was investigated by administering the two co

170 ting for the low systemic bioavailability of BPA (0.50%).

171 ion between baseline urine concentrations of BPA and of ClxBPA and incident MI was determined.

172 th percentiles) of urinary concentrations of BPA, BPF, and BPS were 1.40 (0.19-3.85), 0.65 (0.34-1.39

173 ic associations of urinary concentrations of BPA, BPF, and BPS with size at birth.

174 We quantified concentrations of BPA, BPS, and BPF in 660 urine samples from 148 predomin

175 Urinary concentrations of BPA, PA, and phthalate metabolites were positively assoc

176 ndard deviation [SD] 1.6), concentrations of BPA, phthalic acid (PA), and phthalate metabolites were

177 In some studies, the daughters of BPA-exposed dams have shorter AGD than controls.

178 determined, which will inform the design of BPA removal phytotechnologies for a variety of soil cond

179 walled carbon nanotubes for the detection of BPA in water.

180 The development of BPA allowed profiling of functional antibodies in an LCS

181 based TK model describing the disposition of BPA and BPS and their glucuronides was built from these

182 times lower than the current 'safe' dose of BPA for humans.

183 In contrast, only 77% of the oral dose of BPA was absorbed and underwent an extensive first-pass g

184 rague-Dawley rats were exposed to 5 doses of BPA [0, 25, 250, 2,500, or 25,000 mug/kg body weight (BW

185 ther lifelong exposure to different doses of BPA induced uterine abnormalities and molecular changes

186 Both doses of BPA significantly impaired insulin secretion in male but

187 We also observed dose-specific effects of BPA on islets in males.

188 ive methods revealed nonmonotonic effects of BPA.

189 ucted in piglets to evaluate the kinetics of BPA, BPS, and their glucuronoconjugated metabolites in p

190 sed to two relevant human exposure levels of BPA (10mug/kg/d-LowerB and 10mg/kg/d-UpperB).

191 lated receipts containing relevant levels of BPA (isotope-labeled BPA-d16) for 5 min, followed by han

192 Results shown significantly higher levels of BPA in recycled PET.

193 Levels of BPA migrating from cans did not exceed the specific migr

194 itment to the Srebp-1c promoter in livers of BPA-exposed mice was observed.

195 r rapid, sensitive and on-site monitoring of BPA in food samples.

196 quality monitoring system for monitoring of BPA in water.

197 We compared efficacy and safety outcomes of BPA with or without pulmonary vasodilators to pulmonary

198 Kinetic parameters of BPA removal and translocation factors were also determin

199 The degradation percentage of BPA was 33.0 +/- 1.5% and 35.4 +/- 1.2% in incurred and

200 ts show minimal hydrolysis and photolysis of BPA over 55 days, confirming its persistence.

201 ephthalate (PET) food grade, the presence of BPA in recycled PET should not be neglected.

202 r was also applied for the quantification of BPA present drinking water stored in the plastic bottles

203 omparing the highest with lowest quartile of BPA, 1.02 (0.70-1.47) for BPF, and 1.22 (0.81-1.83) for

204 A wide range of BPA activity was observed across the different antigens.

205 line was mainly caused by the replacement of BPA by BPS in thermal paper and the lower dermal uptake

206 altered cellular excitability as a result of BPA-induced Vm hyperpolarization.

207 s facilitated the electrochemical sensing of BPA.

208 Second, a major target of BPA/BPS is either spongiotrophoblast or GCs within the j

209 temic persistency, were higher than those of BPA.

210 demic and Regulatory Insights on Toxicity of BPA (CLARITY-BPA; henceforth CLARITY), was launched by t

211 The LD but not UD of BPA exposure resulted in individuals spending more time

212 ighting health concerns regarding the use of BPA alternatives.

213 concluded that currently authorized uses of BPA continue to be safe.

214 Migration of bisphenol compounds (only BPA and BADGE.2H(2)O detected) was mainly affected by st

215 ernal diet to GEN (250 mg/kg feed weight) or BPA (5 mg/kg feed weight, low dose- LD or 50 mg/kg, uppe

216 We recently demonstrated that perinatal BPA exposure is associated with higher body fat, impaire

217 ths prior to BPA to 72% over the 1 year post-BPA.

218 e estimated the association between prenatal BPA exposure and child neurobehavior at 3 y of age in a

219 Sex-specific effects of prenatal BPA exposure on the susceptibility of AD deserve further

220 dings from this study indicate that prenatal BPA exposure alters the expression of ASD-linked genes i

221 iling data from several previously published BPA studies consistently showed that BPA-responsive gene

222 ome profiling data from previously published BPA studies consistently showed that BPA-responsive gene

223 hly sensitive methods are needed to quantify BPA in various matrices including water, serum, and food

224 Here, we quantify BPA oxidation rates and the formation of its predominant

225 n of 476 human orthologous genes in this rat BPA signature robustly predicted the overall survival (p

226 d humans, our results suggest that replacing BPA with BPS will likely lead to increased internal expo

227 tion process (GOP) was developed to separate BPA and PMS into two half cells.

228 er but with different kind of sauces, showed BPA levels ranging from <1microgkg(-1) (limit of quantif

229 ld successfully recover the levels of spiked BPA.

230 The automated antimicrobial stewardship BPA effectively reduced antibiotic use and discharge pre

231 The automated antimicrobial stewardship BPA effectively reduced antibiotic use and discharge pre

232 d and untreated plants indicates substantial BPA tolerance in both varieties.

233 The impact of relatively high, short-term BPA exposure on child health is unknown.

234 This biosensor was applied to test BPA in canned food samples and could successfully recove

235 ), that have higher estrogenic activity than BPA.

236 mportantly, with significantly lower EA than BPA over an environmentally relevant range of 10(-10)-10

237 cts to BPA and that BPAF is more potent than BPA, further highlighting health concerns regarding the

238 zed at a rate that is 12.6 times slower than BPA and accumulates in solution.

239 Taken together, these data demonstrate that BPA exposure alters trophoblast cell invasion and causes

240 The study conclusively showed that BPA facilitated in vitro enamel adhesion without detrime

241 blished BPA studies consistently showed that BPA-responsive genes were significantly associated with

242 blished BPA studies consistently showed that BPA-responsive genes were significantly associated with

243 Our data suggest that BPA at environmentally relevant doses affects the abilit

244 We found evidence to suggest that BPA exposure in a predominantly low-income, minority ped

245 ly shorter AGD in daughters, suggesting that BPA may alter the hormonal environment of the female fet

246 The BPA also reduced the percentage of patients prescribed a

247 The BPA also reduced the percentage of patients prescribed a

248 The BPA reduced inpatient antibiotic days of therapy by 2.2

249 The BPA reduced inpatient antibiotic-days of therapy by a me

250 The BPA response was different from the EE2 effect for many

251 d a set of 710 genes shared only between the BPA(25) and BPA(250) groups, with 115 of them predicted

252 he pseudo-second-order rate constant for the BPA pollutant is 182.3 g mg(-1) min(-1), which is the hi

253 Furthermore, the BPA etchant caused lower enamel decalcification with ext

254 fraction were managed by 93 providers in the BPA (n=997 patients) and 102 providers in the no BPA (n=

255 More complications occurred in the BPA arm.

256 Patients in the BPA group were more likely to be referred to electrophys

257 In the BPA trial, data from 14,475 patients (BC, n = 8928; cont

258 xhibit significant inhibitory effects on the BPA metabolism, especially the sulfate conjugation.

259 t demonstrates significant inhibition on the BPA metabolism, indicating the possible natural existenc

260 Patients belonging to the BPA and no BPA groups of providers were followed to the

261 We thus show that these BPA alternatives induce similar toxic and estrogenic eff

262 DRG from ERbeta(-/-) mice indicate that this BPA effect involves ERalpha and phosphoinositide 3-kinas

263 wed as promising sustainable alternatives to BPA and BPF.

264 around exposures to substitutes compared to BPA.

265 d the lower dermal uptake of BPS compared to BPA.

266 duce similar toxic and estrogenic effects to BPA and that BPAF is more potent than BPA, further highl

267 the extent to which children are exposed to BPA from dental treatment with bisGMA materials, by amou

268 Males exposed to BPA or GEN showed alterations in lysine degradation and

269 ted from neonatal pups prenatally exposed to BPA was conducted and revealed a list of differentially

270 Sprague-Dawley rats were exposed to BPA, vehicle, or positive control [ethinyl estradiol (EE

271 ernal and/or ancestral embryonic exposure to BPA affects liver metabolism leading to development-dist

272 ric asthma burden and widespread exposure to BPA in the United States.

273 Low-level exposure to BPA is ubiquitous in human populations due to its widesp

274 c review suggest that early-life exposure to BPA may increase adiposity and circulating lipid levels

275 e for concern that developmental exposure to BPA or GEN might affect the microbiome-gut-brain axis.

276 Whether exposure to BPA or its structural analogs bisphenol S (BPS) and bisp

277 Exposure to BPA was 31% in the ESTHER cohort and 18% in the SURDIAGE

278 Despite ubiquitous human exposure to BPA, and the wide-spread clinical use of EE as oral cont

279 valuate the relationship between exposure to BPA, ClxBPA and the occurrence of myocardial infarction

280 care provider visit in the 6 months prior to BPA to 72% over the 1 year post-BPA.

281 nt reduction in EA in all cases, relative to BPA and one petroleum-derived BPA analogue (bisphenol F,

282 ed and showed sex differences in response to BPA exposure.

283 and estradiol, also occurring in response to BPA/BPS exposure, likely affect the placental-brain axis

284 Total BPA metabolite concentrations were lower in exclusively

285 After dietary exposure, urine total BPA-d16 peaked within 5 h and quickly cleared within 24

286 Higher first-trimester BPA exposure was associated with significantly shorter A

287 y lead to higher proportions of unconjugated BPA in systemic circulation.

288 Urinary BPA concentrations in different trimesters were inversel

289 We hypothesized that posttreatment urinary BPA (uBPA) concentrations would be higher among patients

290 f MI was significantly associated with urine BPA detection: adjusted OR = 1.97 (1.05-3.70), p = 0.04.

291 Here, we demonstrated that in utero BPA exposure also disrupted the transcriptome profiles o

292 J mouse dams were fed 200 mug/kg body weight BPA or BPS daily for 2 wk and then bred.

293 f soil conditions, including landfills where BPA accumulation is greatest.

294 We aimed to a) determine whether BPA showed effects on the developing rat mammary gland u

295 We investigated whether BPA exposure causes preeclampsia-like features in pregna

296 y been associated with the ASD risk, whether BPA dysregulates ASD-related genes in the developing bra

297 While BPA may offer greater functional and hemodynamic improve

298 emic availability of BPS, in comparison with BPA, and its lower plasma clearance (3.5 times lower), t

299 (71.0 meters, 95% CI: 47.4-94.5 meters with BPA versus 47.8 meters, 95% CI: 34.5-61.2 meters with pu

300 ere we show that treatment of male mice with BPA (50 ug/kg/day) during 8 days, decreases the latency

301 the PMS decomposition tests with and without BPA and the comparison with a (1)O(2)-generation system