コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 stringent response to nutrient starvation in Borrelia burgdorferi.
2 has an essential role in the eradication of Borrelia burgdorferi.
3 reliosis model of infection of C3H mice with Borrelia burgdorferi.
4 ly or indirectly modulate gene expression of Borrelia burgdorferi.
5 is serologic detection of antibodies against Borrelia burgdorferi.
6 one of the major outer membrane proteins of Borrelia burgdorferi.
7 curring triacylated lipoprotein agonist from Borrelia burgdorferi.
8 olonization for the Lyme disease spirochaete Borrelia burgdorferi.
9 us life cycle of the Lyme disease spirochete Borrelia burgdorferi.
10 in, thyroglobulin, LPS, influenza virus, and Borrelia burgdorferi.
11 uding the Lyme disease spirochete bacterium, Borrelia burgdorferi.
12 ritical components to the immune response to Borrelia burgdorferi.
13 mented genome of the Lyme disease spirochete Borrelia burgdorferi.
14 ops in C3H mice infected with the spirochete Borrelia burgdorferi.
15 P-producing protein (diguanylate cyclase) of Borrelia burgdorferi.
16 ) was studied in the Lyme disease spirochete Borrelia burgdorferi.
17 were infected with the causative bacterium, Borrelia burgdorferi.
18 ribed, including Torque teno virus (TTV) and Borrelia burgdorferi.
19 d infectivity in the Lyme disease spirochete Borrelia burgdorferi.
20 ease caused by infection with the spirochete Borrelia burgdorferi.
21 gellar motor in the Lyme disease spirochete, Borrelia burgdorferi.
22 ment assembly in the Lyme disease spirochete Borrelia burgdorferi.
23 three phases of disease after infection with Borrelia burgdorferi.
24 bacterial enhancer binding protein (bEBP) in Borrelia burgdorferi.
25 acterized the hypothetical protein BB0794 in Borrelia burgdorferi.
26 ultisystem disorder caused by the spirochete Borrelia burgdorferi A common late-stage complication of
30 e neuroborreliosis, caused by the spirochete Borrelia burgdorferi, affects both peripheral and centra
31 The persistence of dormant, noncultivable Borrelia burgdorferi after ceftriaxone treatment was exa
33 During the natural enzootic life cycle of Borrelia burgdorferi (also known as Borreliella burgdorf
35 m falciparum and the Lyme disease spirochete Borrelia burgdorferi, among others, have been shown to s
38 5 cells/ml for B. afzelii and 8 cells/ml for Borrelia burgdorferi and Borrelia garinii Clinical sampl
39 sal agents of Lyme disease in North America, Borrelia burgdorferi and Borrelia mayonii, are transmitt
40 orrelia burgdorferi bound to AMP, GluRS from Borrelia burgdorferi and Burkholderia thailandensis boun
43 isease is caused by the bacterial spirochete Borrelia burgdorferi and other related Borrelia species.
44 colonization by the Lyme disease spirochete Borrelia burgdorferi and the rickettsial agent Anaplasma
45 acteria, that is the Lyme disease spirochete Borrelia burgdorferi and the rickettsial agents Anaplasm
48 ion liposomes, His-tagged antigen rOspA from Borrelia burgdorferi, and lipophilic analogue norAbuMDP-
49 model of Lyme borreliosis, we observed that Borrelia burgdorferi antigens, but not infectious spiroc
50 determination of the serologic responses to Borrelia burgdorferi antigens, with the exception of the
52 s studies showed that the immune response to Borrelia burgdorferi appears to lack robust T-dependent
53 pression quantitative trait locus underlying Borrelia burgdorferi arthritis-associated locus 1 (Bbaa1
55 lying flagellar rotational switching, we use Borrelia burgdorferi as a model system to determine high
56 ification (LAMP), the CHB probe could detect Borrelia burgdorferi (B. burgdorferi) recA gene with a s
64 ce in the host of the Lyme disease bacterium Borrelia burgdorferi (Bb), but to date, there are no sol
66 f the RpoS regulon in mammalian host-adapted Borrelia burgdorferi, bb0728 (cdr) was found to be duall
68 se is caused by the three spirochete species Borrelia burgdorferi, Borrelia afzelii, and Borrelia gar
69 structures were determined for a CysRS from Borrelia burgdorferi bound to AMP, GluRS from Borrelia b
71 domains, interacted with Leptospira spp. and Borrelia burgdorferi, but not with Escherichia coli or S
73 To further characterize the function of the Borrelia burgdorferi C-terminal protease CtpA, we used s
81 The genome of the Lyme disease spirochete Borrelia burgdorferi contains multiple homologues of che
83 We recently showed that a homolog of CsrA in Borrelia burgdorferi (CsrA(Bb), BB0184) was upregulated
85 tick-borne infection caused by the bacteria Borrelia burgdorferi Current diagnosis of early Lyme dis
86 ecific DNA-binding protein was purified from Borrelia burgdorferi cytoplasmic extracts, and determine
87 ouse model of Lyme disease has revealed that Borrelia burgdorferi differentially expresses numerous o
88 s, we found that the Lyme disease spirochete Borrelia burgdorferi displays a complex pattern of growt
90 jor lipoproteins expressed on the surface of Borrelia burgdorferi during tick feeding and the early p
105 egy adopted by the Lyme disease spirochaete, Borrelia burgdorferi, for adaptation and survival in the
106 hemoreceptors of the Lyme disease spirochete Borrelia burgdorferi form long, thin arrays near both ce
110 merging for differential pathogenicity among Borrelia burgdorferi genotypes in the United States.
113 ligate pathogen, the Lyme disease spirochete Borrelia burgdorferi has a streamlined genome that encod
114 cytosis of the Lyme disease-causing pathogen Borrelia burgdorferi has been shown to be important for
117 interactions of the Lyme disease spirochete Borrelia burgdorferi Here, we investigated how B. burgdo
118 rray of human and animal pathogens including Borrelia burgdorferi; however, how tick immune component
120 014, in whom routine serologic screening for Borrelia burgdorferi (IgG as determined by enzyme-linked
122 od culture based on microscopic detection of Borrelia burgdorferi in Barbour-Stoenner-Kelly medium af
126 spC genotypes of the Lyme disease spirochete Borrelia burgdorferi in the tick vector by in vitro cult
127 was previously implicated in development of Borrelia burgdorferi-induced arthritis in C3H mice, and
130 ion between neuroborreliosis (nervous system Borrelia burgdorferi infection) and Lyme encephalopathy
137 major vector of the Lyme disease spirochete Borrelia burgdorferi, influence spirochete colonization
145 ary, we have found substantial evidence that Borrelia burgdorferi is capable of forming biofilm in vi
156 The aetiological agent of Lyme disease, Borrelia burgdorferi, is transmitted via infected Ixodes
158 proposed that round morphologic variants of Borrelia burgdorferi, known variably as "cyst forms" and
161 ected with the Lyme disease-causing bacteria Borrelia burgdorferi, leading to induction of antimicrob
165 ious studies, we showed that localization of Borrelia burgdorferi monomeric surface lipoprotein OspA
166 Lyme arthritis-susceptible C3H/HeJ mice with Borrelia burgdorferi, mRNA expression of 5-LO and 5-LO-a
168 nlike the genomes of most organisms, that of Borrelia burgdorferi notably encodes a single HtrA gene
173 brane (OM), we generated constructs in which Borrelia burgdorferi outer surface lipoprotein A (OspA)
181 (n = 241 samples), to select the best of 12 Borrelia burgdorferi proteins to improve our microfluidi
183 This study describes the selection of two Borrelia burgdorferi recombinant proteins and evaluation
185 , and serologic evaluation for antibodies to Borrelia burgdorferi remains the recommended modality fo
187 C3H 5-lipoxygenase (LO)-deficient mice with Borrelia burgdorferi results in prolonged nonresolving L
189 d samples were analysed by real-time PCR for Borrelia burgdorferi s.l., Borrelia miyamotoi, Anaplasma
191 o 34 degrees C, leading to a hypothesis that Borrelia burgdorferi senses environmental temperature to
202 me borreliosis, spirochetes belonging to the Borrelia burgdorferi sensu lato group, have developed se
205 caused by the tick-borne spirochetes of the Borrelia burgdorferi sensu lato species complex has been
206 caused by the tick-borne spirochetes of the Borrelia burgdorferi sensu lato species complex, has bee
210 to resist the shear force of the blood flow.Borrelia burgdorferi sensu lato spirochetes, the causati
211 plasmataceae, six genospecies in the complex Borrelia burgdorferi sensu lato, 11 species of Babesia,
212 infected tick, the Lyme disease spirochete, Borrelia burgdorferi sensu lato, colonizes the mammalian
218 strains representing 3 Borrelia genospecies, Borrelia burgdorferi sensu stricto, Borrelia garinii, an
223 nts with Lyme arthritis, and supernatants of Borrelia burgdorferi-stimulated peripheral blood mononuc
228 al serious diseases, including Lyme disease (Borrelia burgdorferi), syphilis (Treponema pallidum) and
229 borne illness in North America, is caused by Borrelia burgdorferi The long-term survival of B. burgdo
230 ck Ixodes scapularis limits proliferation of Borrelia burgdorferi, the aetiologic agent of Lyme disea
245 ing protein expressed on the cell surface of Borrelia burgdorferi, the causative agent of Lyme diseas
252 an important lipoprotein from the bacterium Borrelia burgdorferi, the causative agent of Lyme diseas
253 onses modulate disease during infection with Borrelia burgdorferi, the causative agent of Lyme diseas
257 esis for many infectious bacteria, including Borrelia burgdorferi, the causative agent of Lyme diseas
258 HGA), shares the same enzootic life cycle as Borrelia burgdorferi, the causative agent of Lyme diseas
261 ) respond to CD1d-presented glycolipids from Borrelia burgdorferi, the causative agent of Lyme diseas
265 and ordered (raft) membrane domains exist in Borrelia burgdorferi, the causative agent of Lyme diseas
278 Treponema pallidum and Lyme disease pathogen Borrelia burgdorferi, the pertinent glycan structures an
279 orrelate with protection from infection with Borrelia burgdorferi, the primary cause of Lyme disease
281 of gene regulation in the enzootic cycle of Borrelia burgdorferi, the spirochete that causes Lyme di
282 Persistent infection of a mammalian host by Borrelia burgdorferi, the spirochete that causes Lyme di
285 trast, Dae2 has no intrinsic ability to kill Borrelia burgdorferi, the tick-borne Lyme disease bacter
286 nfections with tick-transmitted Borreliella (Borrelia) burgdorferi, the cause of Lyme disease, repres
287 logy utilizing peptide antigens derived from Borrelia burgdorferi to stimulate interferon-gamma (IFN-
288 mammals forces the Lyme disease spirochete, Borrelia burgdorferi, to adapt to different host milieus
289 gands or the Lyme disease causing bacterium, Borrelia burgdorferi, to LAMP-1 lysosomal compartments.
290 y a positive IgG or IgM immunoblot assay for Borrelia burgdorferi--to receive a 12-week oral course o
293 derstanding of the molecular mechanisms that Borrelia burgdorferi uses to survive during mammalian in
294 HP) with 36 residues, a surface protein from Borrelia burgdorferi (VlsE) with 341 residues, and two p
296 In this report, the Lyme disease spirochete Borrelia burgdorferi was used as a genetic model to inve
297 , the only PilZ domain-containing protein in Borrelia burgdorferi, was reported to bind c-di-GMP, nei
298 ns with a positive skin or blood culture for Borrelia burgdorferi were enrolled in a prospective stud
300 c ligand for TLR2/1 heterodimers, as well as Borrelia burgdorferi, which is a strong activator of TLR