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1 cterium tumefaciens, and the animal pathogen Brucella abortus.
2 such as trinitrophenyl (TNP)-Ficoll and TNP-Brucella abortus.
3 mefaciens, and the bovine and human pathogen Brucella abortus.
4 Lon protease homologue has been cloned from Brucella abortus.
5 nd we show here that it is also essential in Brucella abortus.
6 li and the glucose/galactose:H+ symporter of Brucella abortus.
7 lar survival of the global zoonotic pathogen Brucella abortus.
8 ion (AI) induced by injection of heat-killed Brucella abortus.
9 tiple life processes, including virulence in Brucella abortus.
10 n zinc transport has been found to attenuate Brucella abortus.
11 proteobacteria Agrobacterium tumefaciens and Brucella abortus.
12 important factor for growth and virulence of Brucella abortus.
13 th the Gram-negative intracellular bacterium Brucella abortus.
14 y skin disease, 7.46% (6.71 to 8.81) against Brucella abortus, 11.57% (10.29 to 13.36) against anthra
15 s (Brucella cell surface proteins [BCSP]) of Brucella abortus 19 and later challenge exposed with B.
16 e responses and resistance to infection with Brucella abortus 2308 (S2308) were measured in mice foll
17 s, the intracellular trafficking of virulent Brucella abortus 2308 and attenuated hfq and bacA mutant
18 lectrophoretic analysis of cell lysates from Brucella abortus 2308 and the isogenic hfq mutant Hfq3 r
19 ent (IRF-1(-/-)) mice infected with virulent Brucella abortus 2308 at 5 x 10(5) CFU developed acute h
20 -responsive regulation of mntH expression in Brucella abortus 2308 but also identify the cis-acting e
21 coding gene, galE or exoB, was isolated from Brucella abortus 2308 by complementation of an exoB muta
24 nsporter BhuA exhibits maximum expression in Brucella abortus 2308 during growth under iron-deprived
25 nt (designated CAM220) derived from virulent Brucella abortus 2308 exhibited increased sensitivity to
26 The genes designated BAB2_0837-0840 in the Brucella abortus 2308 genome sequence are predicted to e
27 annotated as BAB2_0350 and BAB2_0351 in the Brucella abortus 2308 genome sequence are predicted to e
30 H is required for the wild-type virulence of Brucella abortus 2308 in mice and indicated that the mnt
31 evaluation of isogenic mutants derived from Brucella abortus 2308 indicates that the AlcR homolog Dh
32 redox-responsive two-component regulator in Brucella abortus 2308 that responds to decreasing levels
33 this protein plays a role in the ability of Brucella abortus 2308 to use heme as an iron source, an
35 -mediated immune response, a gene library of Brucella abortus 2308 was screened for the expression of
36 mutant (DeltaentC) constructed from virulent Brucella abortus 2308, however, we found that production
41 n) with the HtrA stress-response proteins of Brucella abortus (59%), Ec (37%) and Salmonella typhimur
46 inorhizobium meliloti, a legume symbiont and Brucella abortus, a phylogenetically related mammalian p
47 norhizobium meliloti, a legume symbiont, and Brucella abortus, a phylogenetically related mammalian p
49 hese bacteria, including Rhizobium meliloti, Brucella abortus, Agrobacterium tumefaciens, and Rhodoba
50 RIV and MRIV), USDA standard tube tests with Brucella abortus and Brucella melitensis antigens (SATA
51 ntified an OPS epitope equally common to all Brucella abortus and Brucella melitensis strains but uni
53 d Fz phages that are predominantly lytic for Brucella abortus and Brucella neotomae; Group II include
54 nemia induced by an injection of heat-killed Brucella abortus and examined the contribution of hepcid
55 on the functional architecture of MucR from Brucella abortus and its homolog Ml5 from Mesorhizobium
57 iales order, and that sequence homologs from Brucella abortus and Liberobacter solanacearum complemen
58 role of O antigen in the interaction between Brucella abortus and macrophages, we have monitored the
65 was identical to the homologous sequences of Brucella abortus, B. melitensis, B. canis, and B. suis.
66 olates of human and animal origin, including Brucella abortus, B. melitensis, B. ovis, B. neotomae, m
67 onary relationships of five Brucella species-Brucella abortus, B. melitensis, B. suis, B. canis, and
71 athogens that form chronic infections, e.g., Brucella abortus, Bartonella henselae, and Legionella pn
73 lipid A of the medically relevant pathogens Brucella abortus, Brucella melitensis, Bartonella hensel
74 react with several other bacteria including Brucella abortus, Brucella melitensis, Yersinia enteroco
75 Clostridium botulinum, Brucella melitensis, Brucella abortus, Brucella suis, and Brucella canis were
76 s pathogenic in humans (Brucella melitensis, Brucella abortus, Brucella suis, and Brucella canis) is
77 adiations, producing four clades as follows: Brucella abortus-Brucella melitensis, Brucella suis-Bruc
78 1 and the control of in vitro replication of Brucella abortus but not Salmonella enterica serovar Dub
79 the heat-inactivated gram-negative bacterium Brucella abortus can induce IFN-gamma secretion by T cel
82 Several intracellular pathogens, such as Brucella abortus, display a biphasic infection process s
84 l reporter systems to show that the pathogen Brucella abortus does encounter alkylating stress during
86 e report the x-ray crystal structures of the Brucella abortus DRL enzyme in its apo form and in compl
87 resent Brucella-secreted protein L (BspL), a Brucella abortus effector that interacts with Herp, a ce
89 ed that the prokaryotes Brucella melitensis, Brucella abortus, Erythrobacter litoralis, and Pseudomon
90 The intracellular, gram-negative pathogen Brucella abortus establishes chronic infections in host
91 Here we use a genomic approach to examine Brucella abortus evolution, cross-species transmission a
93 We report the complete genome sequence of Brucella abortus field isolate 9-941 and compare it to t
100 and BAB2_0612 are highly over-produced in a Brucella abortus hfq mutant compared with the parental s
101 se sigma factor RpoS in many bacteria, and a Brucella abortus hfq mutant displays a phenotype in vitr
104 bacterial DNA in the context of heat-killed Brucella abortus (HKBA) engages TLR9 in dendritic cells
105 rototypic Th1-inducing adjuvant, heat-killed Brucella abortus (HKBA) to assess stimulation of DC subs
106 use model of AI by administering heat-killed Brucella abortus (HKBA) to germ line TNFalpha knockout (
109 The genetic basis for chronic persistence of Brucella abortus in lymphoid organs of mice, cows, and h
111 the description of cell-cycle progression of Brucella abortus, including unipolar growth and the orde
112 t in vitro and in vivo evidence showing that Brucella abortus-induced inflammatory response leads to
113 oteins or peptides conjugated to inactivated Brucella abortus induces the secretion of virus-neutrali
114 R2(+) Tregs contribute to the persistence of Brucella abortus infection and that inactivation of Treg
118 cells (HBMEC) in response to infection with Brucella abortus Infection of HBMEC with B. abortus indu
119 for the diagnosis of Mycobacterium bovis and Brucella abortus infection simultaneously in bovine milk
123 at immunoglobulin M and complement-opsonized Brucella abortus infects and survives inside primary mur
124 to determine its accuracy in differentiating Brucella abortus into three categories: field strains, v
135 a abortus double-mutant (DeltaznuA DeltanorD Brucella abortus-lacZ [znBAZ]) was assessed for its prot
139 is revealed that transcription of the native Brucella abortus lon increases in response to heat shock
143 ce to elicit systemic infections with either Brucella abortus or B. melitensis at various doses.
144 h DNP-keyhole limpet hemocyanin, heat-killed Brucella abortus, or infection with Nippostrongylus bras
145 We report the cloning and sequencing of the Brucella abortus oxyR homolog and provide evidence that
152 ntation by MHC II and allows host control of Brucella abortus replication in IL-4-treated DCs and in
157 analysis of sera from cattle vaccinated with Brucella abortus S19 exhibit an elevated serologic respo
165 system, PhyR and sigma(E1), are required for Brucella abortus stress survival in vitro and maintenanc
166 Here we show that the intracellular pathogen Brucella abortus survives and replicates preferentially
167 II TA cassette in the intracellular pathogen Brucella abortus that consists of the toxin gene, brnT,
168 Yersinia pestis, Francisella tularensis, and Brucella abortus), the last four of which are biothreat
171 yllumazine synthase (lumazine synthase) from Brucella abortus, the infectious organism of the disease
172 s about the molecular mechanisms employed by Brucella abortus to combat host defenses or to persist a
177 same AT-rich site is recognized by MucR from Brucella abortus using a similar mechanism involving con
178 ge when compared with conventional livestock Brucella abortus vaccines, the smooth S19 (smooth B. abo
185 -alum (OVA/alum) immunization by heat-killed Brucella abortus was not reversed by anti-IL-12 antibody
186 IL-6 production triggered by infection with Brucella abortus, which induces ER stress by injecting t
187 natural hosts of the intracellular pathogen Brucella abortus, which inflicts a significant burden on
188 -term survival of Sinorhizobium meliloti and Brucella abortus within acidic compartments in plant and
189 as Salmonella enterica serovar Typhimurium, Brucella abortus, Yersinia enterocolitica, and Pseudomon