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1 uated two real-time PCR assays for detecting Brugia DNA in human blood and compared the results of th
2 4 in the clearance of a primary infection of Brugia from the murine host.
3 gulation of the Th2 response associated with Brugia infection.
4  immunologically intact permissive models of Brugia infection.
5 ype that reacts strongly with the surface of Brugia L3.
6 on passive transfer, led to the clearance of Brugia malayi (Bm) microfilariae (mf) from infected jird
7 inth parasites Wuchereria bancrofti (Wb) and Brugia malayi (Bm).
8 nce tag, from the distantly related parasite Brugia malayi (Bm-her-1).
9               Characterization of viruses of Brugia malayi (BMRV1) and Onchocerca volvulus (OVRV1) sh
10 nts generated from MF patients, filarial Ag (Brugia malayi adult Ag (BmA))-stimulated supernatants fr
11  basophils released histamine in response to Brugia malayi Ag (BmAg).
12  Brugian filariasis (caused by the nematodes Brugia malayi and B. timori) is an important cause of di
13 During our investigations into the course of Brugia malayi and Brugia pahangi infections in immunodef
14                                  Extracts of Brugia malayi and Onchocerca volvulus, which contain Wol
15 e (Mb) genome of the human filarial parasite Brugia malayi and predict approximately 11,500 protein c
16 river blindness, or Wuchereria bancrofti and Brugia malayi and related parasitic nematodes resulting
17 duals (n = 11) were stimulated in vitro with Brugia malayi antigen (BMA) or mycobacterial purified pr
18  association between ratios of polyclonal to Brugia malayi antigen (BmAg)-specific IgE (range, 14:1 t
19 (-/-) mice were first immunized with soluble Brugia malayi antigens and then inoculated intravenously
20 d secondary murine and human infections with Brugia malayi are characterized by substantial increases
21                                              Brugia malayi are thread-like parasitic worms and one of
22 s brenneri, using Pristionchus pacificus and Brugia malayi as outgroups, and identified numerous oper
23 chocerca volvulus, Wuchereria bancrofti, and Brugia malayi available, new drug targets have been iden
24  response to live, infective-stage larvae of Brugia malayi but not to microfilariae of this parasite.
25 uble extracts of the human filarial parasite Brugia malayi can induce potent inflammatory responses,
26        Infection with the parasitic helminth Brugia malayi can result in development of a severe asth
27 sponding genes in Caenorhabditis elegans and Brugia malayi contained a cysteine codon in place of TGA
28 la melanogaster, Caenorhabditis elegans, and Brugia malayi EST sequences.
29  behaviour towards host cues when exposed to Brugia malayi filarial parasites.
30         The filarial nematode gene Bmgmf for Brugia malayi glia maturation factor contains six predic
31                                              Brugia malayi GMF (BmGMF) is also related to a large fam
32                  The human filarial parasite Brugia malayi harbors an endosymbiotic bacterium Wolbach
33 e heme; however, the human filarial parasite Brugia malayi has acquired a bacterial gene encoding fer
34 sponse to the infective-stage larvae (L3) of Brugia malayi has not been well-characterized in vivo (b
35 he genomic environment of a newly identified Brugia malayi Hox6-8 ortholog (Bm-ant-1) revealed that i
36 N-glycans and GSLs of the parasitic nematode Brugia malayi in a glycoanalytical workflow.
37 ilarial Ag (MfAg) from the filarial parasite Brugia malayi in the presence of APCs.
38     We have shown that live microfilariae of Brugia malayi induce caspase-dependent apoptosis in huma
39    We demonstrate that the filarial nematode Brugia malayi induced lymphatic remodeling and impaired
40 luated the contributions of T and B cells in Brugia malayi infection by utilizing knockout mice on a
41  This study aimed to evaluate the impacts of Brugia malayi infection on Aedes aegypti flight paramete
42 ablated, and susceptibility to chronic adult Brugia malayi infection was established, promoting a fun
43  after treatment with diethylcarbamazine for Brugia malayi infection, and recorded the severity of an
44 uired for host protection in mouse models of Brugia malayi infection.
45                 A cDNA clone isolated from a Brugia malayi infective-stage larva expression library e
46         Together, these results suggest that Brugia malayi mf employ mechanisms of metabolic modulati
47                                              Brugia malayi microfilariae and adults were exposed in v
48                                  Blood-borne Brugia malayi microfilariae survived for significantly l
49 tics and anti-Wolbachia efficacy in a murine Brugia malayi model of minocycline versus doxycycline.
50                 We found three phases of the Brugia malayi motility responses as they adapted to leva
51 ivates TRP-2, GON-2 & CED-11 TRP channels in Brugia malayi muscle cells producing spastic paralysis.
52             We have isolated and sequenced a Brugia malayi nematode 3.4-kb genomic DNA fragment and i
53 del of alternative activation in which adult Brugia malayi nematodes are implanted surgically in the
54 termine, via preclinical infection models of Brugia malayi or Onchocerca ochengi that elevated exposu
55 ells (PEC) from mice transplanted with adult Brugia malayi parasites suppress the proliferation of ly
56                     Examples of glycosylated Brugia malayi proteins have been previously identified b
57                        The filarial nematode Brugia malayi represents a leading cause of disability i
58 ted IgE mAbs were then tested for binding to Brugia malayi somatic extracts using ImmunoCAP, immunobl
59 ympahtic filariasis is the nematode parasite Brugia malayi that requires a competent mosquito vector
60 cation of binding protein(s) of the helminth Brugia malayi to CaGC and the ability of binding complex
61       Here we have studied the adaptation of Brugia malayi to exposure to the anthelmintic, levamisol
62 single-cell approaches in the human parasite Brugia malayi to generate an annotated cell expression a
63         The resistance of the human parasite Brugia malayi to the antiparasitic activity of cyclospor
64 ylcarbamazine's action is dependent upon the Brugia malayi TRP-2 channel and may also activate endoge
65         These studies have demonstrated that Brugia malayi TRP-2 is activated by diethylcarbamazine.
66  this study, we heterologously expressed the Brugia malayi TRP-2b channel in the Human Embryonic Kidn
67 e, C. remanei, C. brenneri, C. japonica, and Brugia malayi using some of the best-performing gene-fin
68 issue and developing embryos of adult female Brugia malayi worms.
69 a donovani, Toxoplasma gondii) and helminth (Brugia malayi) parasites were examined, each of which pr
70 ge larvae (L3) or live microfilariae (Mf) of Brugia malayi, a causative agent of human lymphatic fila
71 has been isolated from the filarial nematode Brugia malayi, a causative agent of human lymphatic fila
72 e cloned an AVR-14B GluCl subunit from adult Brugia malayi, a causative agent of lymphatic filariasis
73 ertaken the first study of heme transport in Brugia malayi, a causative agent of lymphatic filariasis
74  of almost all the major mammalian stages of Brugia malayi, a causative agent of lymphatic filariasis
75  for sequencing and assembling the genome of Brugia malayi, a human parasite widely used in filariasi
76 ges in serum N-glycans during infection with Brugia malayi, a parasitic nematode of humans responsibl
77 this strategy also blocks transmission-stage Brugia malayi, an agent of human lymphatic filariasis.
78                                           In Brugia malayi, an etiological agent of LF, chemoreceptor
79 ical to the MIFs from the parasitic nematode Brugia malayi, and 22-35% identical to mammalian MIFs.
80  cells were exposed to microfilariae (mf) of Brugia malayi, and their phenotypic and functional chara
81 enomes to that of another filarial parasite, Brugia malayi, and to those of several other nematodes,
82 rgillus nidulans, Schizosaccharomyces pombe, Brugia malayi, Caenorhabditis elegans, Trypanosoma cruzi
83 rbamazine acts on the muscle of adult female Brugia malayi, generating temporary spastic paralysis ma
84  while others, such as the filarial parasite Brugia malayi, have an XY mechanism.
85 bitor of NO synthase abrogated resistance to Brugia malayi, one of the causative agents of human lymp
86 nsoni ova, larvae from the filarial helminth Brugia malayi, or CFA.
87 IL4Ralpha(-/-) mice elicited by the nematode Brugia malayi, or via intraperitoneal thioglycollate inj
88 ocheilonema viteae, Onchocerca volvulus, and Brugia malayi, strongly supporting the concept that the
89 pod Drosophila melanogaster and the nematode Brugia malayi, together with the partial genome sequenci
90 our Wolbachia genomes: the filarial nematode Brugia malayi, wBm, (21-fold enrichment), Drosophila mau
91 e infective-stage larvae or microfilariae of Brugia malayi, we found significant impairment of both T
92  our research on the human nematode parasite Brugia malayi, which causes elephantiasis.
93 t the filarial nematodes responsible for LF (Brugia malayi, Wuchereria bancrofti) or onchocerciasis (
94  interactions between the infective stage of Brugia malayi--one of the extracellular parasites respon
95  immune responses were assayed in a group of Brugia malayi-infected rhesus monkeys.
96  nematode parasites Wuchereria bancrofti and Brugia malayi.
97 in two nematodes; Caenorhabditis elegans and Brugia malayi.
98 quisition in the parasitic filarial nematode Brugia malayi.
99  ability to combat infection by the nematode Brugia malayi.
100  of a metalloprotease from the filarial worm Brugia malayi.
101 ss in the important human parasitic nematode Brugia malayi.
102 ainst the Wolbachia-dependent model filaria, Brugia malayi.
103 e tropical parasites Onchocerca volvulus and Brugia malayi.
104 r chronic exposure to the nematode parasite, Brugia malayi.
105 en developed for the human filarial parasite Brugia malayi.
106 a nematode model with the filarial parasite, Brugia malayi.
107 parasitic nematodes Wuchereria bancrofti and Brugia malayi.
108 gy to identify candidate vaccine antigens of Brugia malayi.
109  C. elegans and the human-parasitic nematode Brugia malayi.
110 rial nematodes, including Brugia pahangi and Brugia malayi.
111  nematode parasites Wuchereria bancrofti and Brugia malayi.
112 interleukin-4 (IL-4) in host defense against Brugia malayi.
113 ed with intracorneal binding complex or live Brugia microfilariae.
114       Reverse genetic studies implicate both Brugia osm-9 and the cyclic nucleotide-gated (CNG) chann
115 L3s, suggesting a polymodal sensory role for Brugia osm-9.
116  occur in many filarial nematodes, including Brugia pahangi and Brugia malayi.
117            Apoptosis of human monocytes with Brugia pahangi antigen (BpA) was demonstrated by scoring
118 ctivity against Acanthocheilonema viteae and Brugia pahangi in jirds.
119       Intraperitoneal (i.p.) infections with Brugia pahangi in Mongolian gerbils, or jirds (Meriones
120 gations into the course of Brugia malayi and Brugia pahangi infections in immunodeficient mouse model
121 limination following primary intraperitoneal Brugia pahangi infections in mice.
122  lymphocytes in antifilarial immunity, using Brugia pahangi infections in the murine peritoneal cavit
123 cleared a challenge infection with infective Brugia pahangi L3 in an accelerated manner, whereas coho
124 es aimed to define early tissue migration of Brugia pahangi L3 in the gerbil (Meriones unguiculatus)
125  four veterinary parasitic nematode species: Brugia pahangi, Teladorsagia circumcincta and Heligmosom
126 solated from the filarial parasitic nematode Brugia pahangi.
127 ifferent cell types following infection with Brugia pahangi.
128  hsp83 was cloned from the filarial nematode Brugia pahangi.
129  PCR assay is a sensitive means of detecting Brugia parasite DNA in human blood.
130 ms, arthropod and mammalian infectious stage Brugia parasites were incubated in nicotinamide, an agon
131 a abortus-killed S19 was inoculated into the Brugia-permissive gerbil host to induce gamma interferon
132  of transmission of Wuchereria bancrofti and Brugia spp. through the application of annual mass drug
133 s and potent against parasitic worms such as Brugia, which causes lymphatic filariasis and Trichuris,
134 aused by the insect-borne filarial nematodes Brugia, Wuchereria and Loa.
135  malaria (Plasmodium), lymphatic filariasis (Brugia,Wuchereria bancrofti), giardiasis (Giardia), toxo
136 22 million years ago (Myr ago) involving the Brugia/Wuchereria lineage and >20-17 Myr ago involving t

 
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