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1 quisition in the parasitic filarial nematode Brugia malayi.
2  ability to combat infection by the nematode Brugia malayi.
3  of a metalloprotease from the filarial worm Brugia malayi.
4 ss in the important human parasitic nematode Brugia malayi.
5 ainst the Wolbachia-dependent model filaria, Brugia malayi.
6 e tropical parasites Onchocerca volvulus and Brugia malayi.
7 r chronic exposure to the nematode parasite, Brugia malayi.
8 en developed for the human filarial parasite Brugia malayi.
9 a nematode model with the filarial parasite, Brugia malayi.
10 parasitic nematodes Wuchereria bancrofti and Brugia malayi.
11 gy to identify candidate vaccine antigens of Brugia malayi.
12  C. elegans and the human-parasitic nematode Brugia malayi.
13 rial nematodes, including Brugia pahangi and Brugia malayi.
14  nematode parasites Wuchereria bancrofti and Brugia malayi.
15 interleukin-4 (IL-4) in host defense against Brugia malayi.
16  nematode parasites Wuchereria bancrofti and Brugia malayi.
17 in two nematodes; Caenorhabditis elegans and Brugia malayi.
18 ge larvae (L3) or live microfilariae (Mf) of Brugia malayi, a causative agent of human lymphatic fila
19 has been isolated from the filarial nematode Brugia malayi, a causative agent of human lymphatic fila
20 ertaken the first study of heme transport in Brugia malayi, a causative agent of lymphatic filariasis
21  of almost all the major mammalian stages of Brugia malayi, a causative agent of lymphatic filariasis
22 e cloned an AVR-14B GluCl subunit from adult Brugia malayi, a causative agent of lymphatic filariasis
23  for sequencing and assembling the genome of Brugia malayi, a human parasite widely used in filariasi
24 ges in serum N-glycans during infection with Brugia malayi, a parasitic nematode of humans responsibl
25 nts generated from MF patients, filarial Ag (Brugia malayi adult Ag (BmA))-stimulated supernatants fr
26  basophils released histamine in response to Brugia malayi Ag (BmAg).
27 this strategy also blocks transmission-stage Brugia malayi, an agent of human lymphatic filariasis.
28                                           In Brugia malayi, an etiological agent of LF, chemoreceptor
29  Brugian filariasis (caused by the nematodes Brugia malayi and B. timori) is an important cause of di
30 During our investigations into the course of Brugia malayi and Brugia pahangi infections in immunodef
31                                  Extracts of Brugia malayi and Onchocerca volvulus, which contain Wol
32 e (Mb) genome of the human filarial parasite Brugia malayi and predict approximately 11,500 protein c
33 river blindness, or Wuchereria bancrofti and Brugia malayi and related parasitic nematodes resulting
34 ical to the MIFs from the parasitic nematode Brugia malayi, and 22-35% identical to mammalian MIFs.
35  cells were exposed to microfilariae (mf) of Brugia malayi, and their phenotypic and functional chara
36 enomes to that of another filarial parasite, Brugia malayi, and to those of several other nematodes,
37 duals (n = 11) were stimulated in vitro with Brugia malayi antigen (BMA) or mycobacterial purified pr
38  association between ratios of polyclonal to Brugia malayi antigen (BmAg)-specific IgE (range, 14:1 t
39 (-/-) mice were first immunized with soluble Brugia malayi antigens and then inoculated intravenously
40 d secondary murine and human infections with Brugia malayi are characterized by substantial increases
41                                              Brugia malayi are thread-like parasitic worms and one of
42 s brenneri, using Pristionchus pacificus and Brugia malayi as outgroups, and identified numerous oper
43 chocerca volvulus, Wuchereria bancrofti, and Brugia malayi available, new drug targets have been iden
44 on passive transfer, led to the clearance of Brugia malayi (Bm) microfilariae (mf) from infected jird
45 inth parasites Wuchereria bancrofti (Wb) and Brugia malayi (Bm).
46 nce tag, from the distantly related parasite Brugia malayi (Bm-her-1).
47               Characterization of viruses of Brugia malayi (BMRV1) and Onchocerca volvulus (OVRV1) sh
48  response to live, infective-stage larvae of Brugia malayi but not to microfilariae of this parasite.
49 rgillus nidulans, Schizosaccharomyces pombe, Brugia malayi, Caenorhabditis elegans, Trypanosoma cruzi
50 uble extracts of the human filarial parasite Brugia malayi can induce potent inflammatory responses,
51        Infection with the parasitic helminth Brugia malayi can result in development of a severe asth
52 sponding genes in Caenorhabditis elegans and Brugia malayi contained a cysteine codon in place of TGA
53 la melanogaster, Caenorhabditis elegans, and Brugia malayi EST sequences.
54  behaviour towards host cues when exposed to Brugia malayi filarial parasites.
55 rbamazine acts on the muscle of adult female Brugia malayi, generating temporary spastic paralysis ma
56         The filarial nematode gene Bmgmf for Brugia malayi glia maturation factor contains six predic
57                                              Brugia malayi GMF (BmGMF) is also related to a large fam
58                  The human filarial parasite Brugia malayi harbors an endosymbiotic bacterium Wolbach
59 e heme; however, the human filarial parasite Brugia malayi has acquired a bacterial gene encoding fer
60 sponse to the infective-stage larvae (L3) of Brugia malayi has not been well-characterized in vivo (b
61  while others, such as the filarial parasite Brugia malayi, have an XY mechanism.
62 he genomic environment of a newly identified Brugia malayi Hox6-8 ortholog (Bm-ant-1) revealed that i
63 N-glycans and GSLs of the parasitic nematode Brugia malayi in a glycoanalytical workflow.
64 ilarial Ag (MfAg) from the filarial parasite Brugia malayi in the presence of APCs.
65     We have shown that live microfilariae of Brugia malayi induce caspase-dependent apoptosis in huma
66    We demonstrate that the filarial nematode Brugia malayi induced lymphatic remodeling and impaired
67  immune responses were assayed in a group of Brugia malayi-infected rhesus monkeys.
68 luated the contributions of T and B cells in Brugia malayi infection by utilizing knockout mice on a
69  This study aimed to evaluate the impacts of Brugia malayi infection on Aedes aegypti flight paramete
70 ablated, and susceptibility to chronic adult Brugia malayi infection was established, promoting a fun
71  after treatment with diethylcarbamazine for Brugia malayi infection, and recorded the severity of an
72 uired for host protection in mouse models of Brugia malayi infection.
73                 A cDNA clone isolated from a Brugia malayi infective-stage larva expression library e
74         Together, these results suggest that Brugia malayi mf employ mechanisms of metabolic modulati
75                                              Brugia malayi microfilariae and adults were exposed in v
76                                  Blood-borne Brugia malayi microfilariae survived for significantly l
77 tics and anti-Wolbachia efficacy in a murine Brugia malayi model of minocycline versus doxycycline.
78                 We found three phases of the Brugia malayi motility responses as they adapted to leva
79 ivates TRP-2, GON-2 & CED-11 TRP channels in Brugia malayi muscle cells producing spastic paralysis.
80             We have isolated and sequenced a Brugia malayi nematode 3.4-kb genomic DNA fragment and i
81 del of alternative activation in which adult Brugia malayi nematodes are implanted surgically in the
82 bitor of NO synthase abrogated resistance to Brugia malayi, one of the causative agents of human lymp
83  interactions between the infective stage of Brugia malayi--one of the extracellular parasites respon
84 termine, via preclinical infection models of Brugia malayi or Onchocerca ochengi that elevated exposu
85 nsoni ova, larvae from the filarial helminth Brugia malayi, or CFA.
86 IL4Ralpha(-/-) mice elicited by the nematode Brugia malayi, or via intraperitoneal thioglycollate inj
87 ells (PEC) from mice transplanted with adult Brugia malayi parasites suppress the proliferation of ly
88 a donovani, Toxoplasma gondii) and helminth (Brugia malayi) parasites were examined, each of which pr
89                     Examples of glycosylated Brugia malayi proteins have been previously identified b
90                        The filarial nematode Brugia malayi represents a leading cause of disability i
91 ted IgE mAbs were then tested for binding to Brugia malayi somatic extracts using ImmunoCAP, immunobl
92 ocheilonema viteae, Onchocerca volvulus, and Brugia malayi, strongly supporting the concept that the
93 ympahtic filariasis is the nematode parasite Brugia malayi that requires a competent mosquito vector
94 cation of binding protein(s) of the helminth Brugia malayi to CaGC and the ability of binding complex
95       Here we have studied the adaptation of Brugia malayi to exposure to the anthelmintic, levamisol
96 single-cell approaches in the human parasite Brugia malayi to generate an annotated cell expression a
97         The resistance of the human parasite Brugia malayi to the antiparasitic activity of cyclospor
98 pod Drosophila melanogaster and the nematode Brugia malayi, together with the partial genome sequenci
99 ylcarbamazine's action is dependent upon the Brugia malayi TRP-2 channel and may also activate endoge
100         These studies have demonstrated that Brugia malayi TRP-2 is activated by diethylcarbamazine.
101  this study, we heterologously expressed the Brugia malayi TRP-2b channel in the Human Embryonic Kidn
102 e, C. remanei, C. brenneri, C. japonica, and Brugia malayi using some of the best-performing gene-fin
103 our Wolbachia genomes: the filarial nematode Brugia malayi, wBm, (21-fold enrichment), Drosophila mau
104 e infective-stage larvae or microfilariae of Brugia malayi, we found significant impairment of both T
105  our research on the human nematode parasite Brugia malayi, which causes elephantiasis.
106 issue and developing embryos of adult female Brugia malayi worms.
107 t the filarial nematodes responsible for LF (Brugia malayi, Wuchereria bancrofti) or onchocerciasis (

 
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