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1 and viability of the Gram-negative bacterium Burkholderia cenocepacia.
2 ved during experimental biofilm evolution of Burkholderia cenocepacia.
3 utely conserved enzyme in this pathway, from Burkholderia cenocepacia.
4 e, we have investigated the BCP homologue of Burkholderia cenocepacia.
5 d via the type VI secretion system (T6SS) of Burkholderia cenocepacia.
6 the N-terminal domain of BC2L-C lectin from Burkholderia cenocepacia, a pathogen causing severe resp
7 ction by Burkholderia species, in particular Burkholderia cenocepacia, accelerates tissue damage and
10 eins present in the cultured supernatants of Burkholderia cenocepacia and Burkholderia multivorans we
11 ents examined the growth of two Bcc species, Burkholderia cenocepacia and Burkholderia multivorans, i
12 of Escherichia coli, Pseudomonas aeruginosa, Burkholderia cenocepacia, and Staphylococcus aureus.
13 nosa but also the corresponding orthologs in Burkholderia cenocepacia, another notorious opportunisti
17 on stress stimulate the transcription of the Burkholderia cenocepacia bcnA gene that encodes a secret
18 s organelles expressed by certain strains of Burkholderia cenocepacia, believed to facilitate coloniz
19 that lectins from Pseudomonas aeruginosa and Burkholderia cenocepacia bind and stimulate FL cells.
20 tides were chosen to distinguish B. cepacia, Burkholderia cenocepacia, Burkholderia multivorans, and
23 ly decreased the amount of C8-HSL present in Burkholderia cenocepacia cultures and inhibited a quorum
24 vitamin D level, and a higher prevalence of Burkholderia cenocepacia ET12 infection were associated
25 e whose host range includes human pathogenic Burkholderia cenocepacia (formally B. cepacia genomovar
26 im, and ciprofloxacin has been identified in Burkholderia cenocepacia (genomovar III), an important c
27 ed with infections in CF patients, including Burkholderia cenocepacia, Haemophilus influenzae, and St
29 nts with the opportunist betaproteobacterium Burkholderia cenocepacia in a biofilm model, mutations w
30 osis (CF) pathogen and endemic soil dweller, Burkholderia cenocepacia, in conditions mimicking these
44 henotypic analyses of 215 serially collected Burkholderia cenocepacia isolates from 16 cystic fibrosi
45 is of genomic DNA from pathogenic strains of Burkholderia cenocepacia J2315 and Escherichia coli O104
46 s of human and mouse genomes and between the Burkholderia cenocepacia J2315 genome and the Ralstonia
47 es enhanced their resistance to infection by Burkholderia cenocepacia J2315, Mycobacterium tuberculos
49 Here, we used the alpha-mannose-specific Burkholderia cenocepacia lectin A (BC2L-A) and show that
52 ections caused by Pseudomonas aeruginosa and Burkholderia cenocepacia pose a severe threat to immunoc
54 riophages (phages) in a mouse model of acute Burkholderia cenocepacia pulmonary infection was assesse
55 are outcompeted by the epidemic Bcc isolate Burkholderia cenocepacia strain AU1054 in a T6SS-depende
59 is a membrane-bound hybrid sensor kinase of Burkholderia cenocepacia that negatively regulates quoru
61 in structural homologs include a lectin from Burkholderia cenocepacia, the C1q component of complemen
63 A previous study identified mutations in the Burkholderia cenocepacia tilS gene affecting locations d
65 g on a pathogen of the cystic fibrosis lung, Burkholderia cenocepacia, we sequenced clones and metage