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1 s to DEA/NO, atrial natriuretic peptide, and C-type natriuretic peptide.
2 the B-type natriuretic peptide (BNP) and the C-type natriuretic peptide.
3 t of recent advances with a special focus on C-type natriuretic peptide.
4 of the Npr2 abolished protective effects of C-type natriuretic peptide, a ligand for Npr2, against d
7 tic peptide acts primarily on the artery and C-type natriuretic peptide acts predominantly on the vei
8 rophy and pathologic calcification, and with C-type natriuretic peptide, an essential factor in endoc
9 om these vessels, the pattern of response to C-type natriuretic peptide and atrial natriuretic peptid
10 donors was mimicked by 2 cGMP analogues and C-type natriuretic peptide and blocked by a specific inh
11 racrine signaling by extracellular peptides (C-type natriuretic peptide and EGF receptor ligands) mai
12 r affinity for [125I]-DNP binding sites than C-type natriuretic peptide and the natriuretic peptide r
14 pulmonary vasculature to atrial, B-type, and C-type natriuretic peptides (ANP, BNP, and CNP) during a
16 Here, we focus on the cardioprotective CNP (C-type natriuretic peptide) as a novel antiarrhythmic pr
21 ions of atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) are elaborated by membr
25 ently, CD-NP, a chimeric peptide composed of C-type natriuretic peptide (CNP) fused to the C-terminal
28 te important roles for nitric oxide (NO) and C-type natriuretic peptide (CNP) in the regulation of ca
29 Both atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP) increased the accumulat
36 minal pro-BNP (NT-proBNP), proBNP(1-108), or C-type natriuretic peptide (CNP) is not well understood.
38 he binding of atrial natriuretic peptide and C-type natriuretic peptide (CNP) to the guanylyl cyclase
39 be coimmunoprecipitated with Galpha(i), and C-type natriuretic peptide (CNP) treatment induced trans
40 domain of the mammalian receptor (GC-B) for C-type natriuretic peptide (CNP) was fused to the cataly
41 uced by atrial natriuretic peptide (ANP) and C-type natriuretic peptide (CNP), activators of particul
42 ial-specific deletion of Nppc, which encodes C-type natriuretic peptide (CNP), and determined that th
44 Activating mutations in the receptor for C-type natriuretic peptide (CNP), guanylyl cyclase B (GC
46 GMP signaling pathway composed of the ligand C-type natriuretic peptide (CNP), its receptor, the guan
47 r S-nitroso-N-acetylpenicillamine (SNAP) and C-type natriuretic peptide (CNP), reduced TNF-alpha-indu
48 t marked elevations of cyclic GMP induced by C-type natriuretic peptide (CNP), the ligand of GC-B, bl
49 istically, Musclin enhances the abundance of C-type natriuretic peptide (CNP), thereby promoting card
50 ngest candidate therapy employs an analog of C-type natriuretic peptide (CNP), which antagonizes the
63 ic peptides, atrial natriuretic peptide, and C-type natriuretic peptide evoked fluorescence resonance
64 inin-potentiating peptides originated from a C-type natriuretic peptide gene backbone; and 4) VEGF-F
65 markedly increased by natriuretic peptides (C-type natriuretic peptide >> atrial natriuretic peptide
66 muscle cells cultured from these vessels to C-type natriuretic peptide in comparison with atrial nat
69 ocytochemically localized atrial, brain, and C-type natriuretic peptide-like immunoreactivity (ANP-LI
70 L658F, Y708C, R776W, and G959A) bound (125)I-C-type natriuretic peptide on the surface of cells but f
71 clase B is the receptor for a small peptide (C-type natriuretic peptide) produced locally in many dif
73 l value) was more effective than 1 microM of c-type natriuretic peptide (three-fold increase in cycli
74 se with the discovery that administration of C-type natriuretic peptide to achondroplastic mice ameli
75 by daily injection of a recombinant form of C-type natriuretic peptide to post-natal pups for 18 day
76 ase in cyclic GMP in response to 1 microM of C-type natriuretic peptide vs. six-fold increase in cycl
78 the order ANP > brain natriuretic peptide >> C-type natriuretic peptide) was also similar to that of